ライフサイエンスコーパス: phosphatidylethanolamine

1 f the membrane caused by the presence of PE (phosphatidylethanolamine).

2 was correlated with phosphatidylcholine and phosphatidylethanolamine.

3 chidonate into liver phosphatidylcholine and phosphatidylethanolamine.

4 (CL), mono-lyso CL, phosphatidylcholine, and phosphatidylethanolamine.

5 s involved in the synthesis of mitochondrial phosphatidylethanolamine.

6 otoisomerization of N-ret-PE (N-retinylidene-phosphatidylethanolamine.

7 ctive, causing increased inward transport of phosphatidylethanolamine.

8 te through cellular membranes via binding to phosphatidylethanolamine.

9 e biosynthetic pathways for the synthesis of phosphatidylethanolamine.

10 interaction prevents PfAtg8 lipidation with phosphatidylethanolamine.

11 by PtdSer decarboxylase 2 (Psd2p) to produce phosphatidylethanolamine.

12 omes containing only phosphatidylcholine and phosphatidylethanolamine.

13 utral phospholipids, phosphatidylcholine, or phosphatidylethanolamine.

14 specially dilinoleoyl phosphatidylcholine or phosphatidylethanolamine.

15 osphatidylcholine, phosphatidylinositol, and phosphatidylethanolamine.

16 hat synthesizes the major amount of cellular phosphatidylethanolamine.

17 hosphatidylcholine, phosphatidylinositol and phosphatidylethanolamine.

18 rease was associated with a reduction in DHA-phosphatidylethanolamine.

19 ygenase-derived hydroxyeicosatetraenoic acid-phosphatidylethanolamines.

20 s NAPA spectra were dominated by signal from phosphatidylethanolamines.

21 r both treatment groups (q<0.00005), whereas phosphatidylethanolamine (36:3e) contributed most to dis

22 boxylation of phosphatidylserine to generate phosphatidylethanolamine, a critical step in phospholipi

23 bisretinoids A2E, A2-GPE, A2-dihydropyridine-phosphatidylethanolamine (A2-DHP-PE), and all-trans-reti

24 P4-ATPase that flips phosphatidylserine and phosphatidylethanolamine across cell membranes.

25 phosphatidylserine and, to a lesser extent, phosphatidylethanolamine across the lipid bilayers of in

26 epare blank oil (O), glucose added oil (OG), phosphatidylethanolamine added oil (OP), and both phosph

27 ial for synthesizing the ATG12-ATG5 and ATG8-phosphatidylethanolamine adducts that are central to aut

28 ine (A2-DHP-PE), and all-trans-retinal dimer-phosphatidylethanolamine (all-trans-retinal dimer-PE) al

29 With dioleoyl phosphatidylethanolamine alone, the proteins had no effe

30 , and Arg69, ensures stable insertion of the phosphatidylethanolamine anchor into membranes.

31 hosphate-limited plants, phosphatidylcholine/phosphatidylethanolamine and associated transcript level

32 n not only locally curved membranes but also phosphatidylethanolamine and cardiolipin, lipids with hi

33 at high levels of Mdm33 affect the levels of phosphatidylethanolamine and cardiolipin, the two key in

34 Conical lipids, phosphatidylethanolamine and diacylglycerol, enhanced tr

35 hatidylethanolamine added oil (OP), and both phosphatidylethanolamine and glucose added oil (OPG).

36 Phosphatidylethanolamine and glucose were added to the d

37 tion of synthesis involves the conversion of phosphatidylethanolamine and glycerol into PG and is cat

38 her saturated lyso-PLs (e.g., palmitoyl lyso-phosphatidylethanolamine and lyso-sphingomyelin) also fa

39 By targeting phosphatidylethanolamine and lysylphosphatidylglycerol,

40 lycerophospholipid in both seed oils whereas phosphatidylethanolamine and phosphatidic acid were less

41 The major phospholipids, phosphatidylethanolamine and phosphatidylcholine (PtdCho

42 treatment and not by total fat content; milk phosphatidylethanolamine and phosphatidylcholine increas

43 headgroups found in mitochondrial membranes, phosphatidylethanolamine and phosphatidylcholine.

44 Phosphatidylethanolamine and phosphatidylserine are foun

45 maintain membrane asymmetry by translocating phosphatidylethanolamine and phosphatidylserine from the

46 tion required to induce PFO binding, whereas phosphatidylethanolamine and phosphatidylserine in the i

47 Cellular debris lipids such as phosphatidylethanolamine and phosphatidylserine were onl

48 s of host-encoded cofactor molecules such as phosphatidylethanolamine and RNA molecules are required

49 in aqueous extracts and phosphatidylcholine, phosphatidylethanolamine and sphingomyelin in lipid extr

50 s were observed by reduced ratios between PC:phosphatidylethanolamine and sphingomyelin:cholesterol,

51 ment of other host factors and enrichment of phosphatidylethanolamine and sterol lipids within the su

52 host yeast has revealed essential roles for phosphatidylethanolamine and sterols in viral replicatio

53 sly associated with cardiometabolic disease (phosphatidylethanolamine and triglycerides).

54 of lipid A-core molecules, cardiolipins, and phosphatidylethanolamines and decreased levels of specif

55 SMALP-extracted ZipA showed an enrichment in phosphatidylethanolamines and depletion in cardiolipins

56 osphocoenzyme A, spermidine, putrescine, and phosphatidylethanolamines and elevated alanine, leucine,

57 NAPA favored the ionization of phosphatidylethanolamines and glycosylated ceramides, wh

58 Correlations between phosphatidylethanolamines and sphingomyelin, and glycine

59 re made for neutral (phosphatidylcholine and phosphatidylethanolamine) and anionic (phosphatidylserin

60 ted by zwitterionic (phosphatidylcholine and phosphatidylethanolamine) and cationic (sphinganine) lip

61 authentic standards (phosphatidylcholine and phosphatidylethanolamine) and two purified PL from marin

62 erification of arachidonic/adrenic acid into phosphatidylethanolamine), and depletion of glutathione

63 phosphatidic acid, a phosphatidylcholine, a phosphatidylethanolamine, and a phosphatidylglycerol wer

64 sphatidic acid together with sphingomyelins, phosphatidylethanolamine, and cholesterol) gives rise to

65 ly found in F1, with higher triglyceride and phosphatidylethanolamine, and lower sphingomyelin concen

66 Defective cardiolipin (CL), phosphatidylethanolamine, and phosphatidylglycerol remod

67 ain-containing lipase 1) that hydrolyzes CL, phosphatidylethanolamine, and phosphatidylglycerol.

68 cids, diacylglycerides, phosphatidylcholine, phosphatidylethanolamine, and phosphatidylinositol in th

69 r phosphatidylcholine, phosphatidylinositol, phosphatidylethanolamine, and sphingomyelin between phos

70 SG phosphatidylserine, phosphatidylinositol, phosphatidylethanolamine, and sphingomyelin, fatty acids

71 tabolite classes including triacylglycerols, phosphatidylethanolamines, and phosphatidylcholines; dec

72 es, triacylglycerides, phosphatidylcholines, phosphatidylethanolamines, and sphingomyelins.

73 for the synthesis of phosphatidylcholine and phosphatidylethanolamine are attractive drug targets to

74 ilayer-forming phospholipids cardiolipin and phosphatidylethanolamine are required for the activity o

75 Phosphatidylcholine and phosphatidylethanolamine are two major phospholipid clas

76 ovide new molecular insight into the role of phosphatidylethanolamine as a membrane component that ca

77 nvolves transfer of LC3/GABARAP from ATG3 to phosphatidylethanolamine at the target membrane.

78 al lipid groups were altered, with increased phosphatidylethanolamines being the most affected lipid

79 To study the evolution of phosphatidylethanolamine-binding protein (PEBP) gene fam

80 ether 15-lipoxygenase-1 (15LO1) binding with phosphatidylethanolamine-binding protein 1 (PEBP1) contr

81 nases, and selected gene family members with phosphatidylethanolamine-binding protein domains.

82 nd assisted by two proteins with homology to phosphatidylethanolamine-binding proteins.

83 We previously demonstrated increased phosphatidylethanolamine biosynthesis subsequent to elev

84 trol blood samples as an indication of liver phosphatidylethanolamine biosynthesis.

85 thanolamine branch of the Kennedy pathway of phosphatidylethanolamine biosynthesis.

86 wo specific lipid (cholesterol/oxysterol and phosphatidylethanolamine) biosynthetic cascades as being

87 egulatory enzyme for de novo biosynthesis of phosphatidylethanolamine by the CDP-ethanolamine pathway

88 TG), diglycerides (DG), phosphatidylcholine, phosphatidylethanolamine, ceramide and sphingomyelin lip

89 etworks 'triacylglycerols' and 'cardiolipins-phosphatidylethanolamines (CL-PE)' characterized by lowe

90 evealed the abundance of arachidonic/adrenic-phosphatidylethanolamine compared with other arachidonic

91 From day 100 postpartum on, phosphatidylethanolamine concentration was constant in t

92 Atg7 and other autophagy factors and undergo phosphatidylethanolamine conjugation to preautophagic me

93 mine, derivatives of phosphatidylcholine and phosphatidylethanolamine containing a 7-nitro-2-1,3-benz

94 id oxidation products, specifically oxidized phosphatidylethanolamine containing arachidonic or adren

95 id bilayer triggered by a change in membrane phosphatidylethanolamine content, both in vivo and in vi

96 Unsaturated triglycerides and phosphatidylethanolamines decreased in abundance before

97 ation of arachidonic/adrenic acid-containing-phosphatidylethanolamine, decreased the number of termin

98 We now show that carboxyalkylpyrrole-phosphatidylethanolamine derivatives (CAP-PEs) are prese

99 o phosphatidylserine, phosphatidylcholine or phosphatidylethanolamine destabilizes the Na,K-ATPase.

100 ps that tend to form non-bilayer structures (phosphatidylethanolamine, diacylglycerol, and ergosterol

101 The polar lipid profile consisted of phosphatidylethanolamine, diphosphatidylglycerol, phosph

102 dylglycerol (DOPG), and zwitterionic dioleyl phosphatidylethanolamine (DOPE), with the addition of mo

103 d between the two cell types, including four phosphatidylethanolamines elevated in astrocytes and nin

104 Synthetic mouse prions formed with phosphatidylethanolamine exhibit levels of specific infe

105 is revealed alteration to levels of specific phosphatidylethanolamine fatty acyl species in patients,

106 or the final step in Kennedy pathway forming phosphatidylethanolamine from CDP-ethanolamine.

107 te that AtMic60 contributes to the export of phosphatidylethanolamine from mitochondria and the impor

108 urated phosphatidylcholines, sphingomyelins, phosphatidylethanolamines, glucosylceramides, and phosph

109 the abundances of endogenous cardiolipin and phosphatidylethanolamine halve during elongation of the

110 y-arachidonoyl (C20:4)- or adrenoyl (C22:4)- phosphatidylethanolamine (Hp-PE).

111 rate proferroptotic hydroperoxy-arachidonoyl-phosphatidylethanolamines (HpETE-PEs) as proximate death

112 ine in TBSV replication and a novel role for phosphatidylethanolamine in asymmetrical (+)RNA synthesi

113 Duramycin, a peptide that binds phosphatidylethanolamine in enveloped virions and preclu

114 and offer insight into the critical role of phosphatidylethanolamine in fusion.

115 the presence of the negatively curved lipid phosphatidylethanolamine in membranes.

116 SDs) play a central role in the synthesis of phosphatidylethanolamine in numerous species of prokaryo

117 3/GABARAP are membrane phospholipids such as phosphatidylethanolamine in the autophagosome membrane.

118 modification of LC3 and GABARAP proteins to phosphatidylethanolamine in the double-membrane phagopho

119 ine in the outer leaf and in cholesterol and phosphatidylethanolamine in the inner leaf.

120 t enzyme that converts phosphatidylserine to phosphatidylethanolamine in the inner mitochondrial memb

121 namics of Atto646N-labeled sphingomyelin and phosphatidylethanolamine in the plasma membrane.

122 In contrast, ABCA4 transported phosphatidylethanolamine in the reverse direction.

123 GTPase-positive endosomes and enrichment of phosphatidylethanolamine in the viral replication compar

124 tidylserine and other phospholipids, such as phosphatidylethanolamine, in ISG could play important ro

125 pholipids, especially phosphatidylserine and phosphatidylethanolamine, in TBSV replication.

126 -associated protein 1 light chain 3 (LC3) to phosphatidylethanolamine, including Atg7, Atg3, and the

127 impact resulted in accumulation of oxidized phosphatidylethanolamine, increased expression of 15-lip

128 ryl hemisuccinate, linoleamide/oleamide, and phosphatidylethanolamine inhibit and phosphatidic acid a

129 Phosphatidylethanolamine is a glycerophospholipid that,

130 The presence of phosphatidylethanolamine is however required for correct

131 Phosphatidylethanolamine is one of the most abundant mem

132 Phosphatidylserine and N,N-dimethyl phosphatidylethanolamine isomers in a bovine brain total

133 accessibility we undertook quantification of phosphatidylethanolamine levels and species in patient a

134 mine fatty acyl species in patients, overall phosphatidylethanolamine levels were broadly unaffected

135 kinase, leading to a severe decrease in the phosphatidylethanolamine levels within P. falciparum, wh

136 e reaction products such as Amadori product, phosphatidylethanolamine-linked pyrrolecarbaldehyde and

137 avorable for the postulated binding of E7 to phosphatidylethanolamine lipid headgroups.

138 single compound level, 168 sphingolipids, 36 phosphatidylethanolamine lipids, and 5 tobacco-related c

139 in the bilayer, through addition of lamellar phosphatidylethanolamine lipids, lowers lactose permease

140 perties, with particularly high affinity for phosphatidylethanolamine lipids.

141 old by liver PC or 1,2-dioleoyl-sn-glycero-3-phosphatidylethanolamine lipids.

142 e incorporated ApoE into phosphatidylcholine/phosphatidylethanolamine liposomes and found that the pr

143 K contained 4-10-fold higher amounts of lyso phosphatidylethanolamine (LPE) and 2-4-fold higher amoun

144 essential for maintaining adequate levels of phosphatidylethanolamine, LPE, and LPC in the cells.

145 TRPV1 embedded in a phosphatidylcholine and phosphatidylethanolamine membrane provides insight into

146 sphatidylcholine, phosphatidylserine, and/or phosphatidylethanolamine metabolism had a major effect o

147 d at least partially to higher expression of phosphatidylethanolamine methyl transferase (PEMT).

148 al ligands of CD300c, phosphatidylserine and phosphatidylethanolamine, modulate IgE-mediated basophil

149 death upon treatment with muramyl tripeptide phosphatidylethanolamine (MTP-PE), a synthetic ligand fo

150 e-derived methyl groups for PC synthesis via phosphatidylethanolamine N-methyltransferase (PEMT) as i

151 pathway for PC biosynthesis is catalyzed by phosphatidylethanolamine N-methyltransferase (PEMT) that

152 en choline-derived methyl groups are used by phosphatidylethanolamine N-methyltransferase (PEMT)] was

153 and serving as the primary substrate for the phosphatidylethanolamine N-methyltransferase pathway.

154 ese analyses indicated that Opi3 (as well as phosphatidylethanolamine N-methyltransferase) has an N-o

155 topology of Opi3 (and its human counterpart, phosphatidylethanolamine N-methyltransferase, expressed

156 cytidine diphosphocholine (CDP-choline) and phosphatidylethanolamine-N-methyltransferase (PEMT) path

157 rofolate reductase 1 (MTHFD1) rs2236225, and phosphatidylethanolamine-N-methyltransferase (PEMT) rs12

158 tion, possibly via impaired folate-dependent phosphatidylethanolamine-N-methyltransferase (PEMT)-PC s

159 ficantly more likely to have the G allele of phosphatidylethanolamine-N-methyltransferase (PEMT; V175

160 N-acyl phosphatidylethanolamines (NAPEs) are precursors of N-ac

161 purely zwitterionic (phosphatidylcholine or phosphatidylethanolamine) ones.

162 ed at opposite voltage polarities, either in phosphatidylethanolamine or phosphatidylcholine membrane

163 Baicalein decreased ferroptotic phosphatidylethanolamine oxidation and improved outcome

164 nhibited by cholesterol (CHOL) as well as by phosphatidylethanolamine (PE) and -glycerol (PG) lipids.

165 The non-bilayer-forming phospholipids phosphatidylethanolamine (PE) and cardiolipin (CL) are r

166 cells or reconstituted in liposomes lacking phosphatidylethanolamine (PE) and containing only anioni

167 hingomyelin (dhSM) levels, and a decrease in phosphatidylethanolamine (PE) and ether phosphatidylchol

168 hosphatidylserine as well as 1-alkyl, 2-acyl phosphatidylethanolamine (PE) and phosphatidycholine wer

169 chromatography (TLC) analysis indicated that phosphatidylethanolamine (PE) and phosphatidylcholine (P

170 we show that neutral phospholipids, namely, phosphatidylethanolamine (PE) and phosphatidylcholine (P

171 constituents of cellular membranes, such as phosphatidylethanolamine (PE) and phosphatidylcholine.

172 Phosphatidylethanolamine (PE) and phosphatidylserine (PS

173 inding of CD300a and CD300c to their ligands phosphatidylethanolamine (PE) and phosphatidylserine (PS

174 he inner mitochondrial membrane, synthesizes phosphatidylethanolamine (PE) and, in some cells, synthe

175 esses.IMPORTANCE Phosphatidylserine (PS) and phosphatidylethanolamine (PE) are usually sequestered to

176 oline or cholesterol but encodes enzymes for phosphatidylethanolamine (PE) biosynthesis; however, exo

177 hatidylcholine (PC) and significantly higher phosphatidylethanolamine (PE) compared to healthy contro

178 reased expression (2- to 3-fold) of genes in phosphatidylethanolamine (PE) de novo biosynthesis resul

179 We found that PgpB was inhibited by phosphatidylethanolamine (PE) in a competitive mode in v

180 ymes in phospholipid metabolism that produce phosphatidylethanolamine (PE) in bacteria, protists, pla

181 the greatest decrease found in the level of phosphatidylethanolamine (PE) in root hairs and stripped

182 aazacyclododecane tetraacetic acid (Gd-DOTA) phosphatidylethanolamine (PE) into the surfactant were m

183 Phosphatidylethanolamine (PE) is a major cellular phosph

184 Phosphatidylethanolamine (PE) is a major phospholipid sp

185 Here we demonstrate that phosphatidylethanolamine (PE) is also a ligand for PS re

186 nfections, and synthesis of the phospholipid phosphatidylethanolamine (PE) is required for virulence.

187 and cerebellar phosphatidylcholine (PC) and phosphatidylethanolamine (PE) levels, while hippocampal

188 e TMD is predominantly alpha-helical, but in phosphatidylethanolamine (PE) membranes, the TMD changes

189 genetic inactivation of de novo synthesis of phosphatidylethanolamine (PE) mitigates OPA cytotoxicity

190 Phosphatidylethanolamine (PE) N-methyltransferase (PEMT)

191 phatidylcholine (PC) synthesis by way of the phosphatidylethanolamine (PE) N-methyltransferase (PEMT)

192 ubiquitin-like protein LC3 is conjugated to phosphatidylethanolamine (PE) on the inner and outer mem

193 ted by the conjugation of cytosolic LC3-I to phosphatidylethanolamine (PE) on the surface of nascent

194 ere demethylated either by substitution with phosphatidylethanolamine (PE) or chemically by using mon

195 lipid composition where the primary lipid is phosphatidylethanolamine (PE) or its methyl derivatives.

196 choline supplementation for the synthesis of phosphatidylethanolamine (PE) or phosphatidylcholine (PC

197 Of these, cardiolipin (CL) and phosphatidylethanolamine (PE) participate in apoptosis a

198 In metabolite set enrichment analyses, phosphatidylethanolamine (PE) plasmalogens were positive

199 rophospholipids phosphatidylcholine (PC) and phosphatidylethanolamine (PE) show distinct reactivities

200 Here, we show that phosphatidylethanolamine (PE) synergizes with PS to enha

201 S transfer to mitochondria and mitochondrial phosphatidylethanolamine (PE) synthesis.

202 o bushy stunt virus (TBSV) are enriched with phosphatidylethanolamine (PE) through a previously unkno

203 rved in phospholipid content as the ratio of phosphatidylethanolamine (PE) to phosphatidylcholine (PC

204 rict most of the phosphatidylserine (PS) and phosphatidylethanolamine (PE) to the inner cytosolic lea

205 ious lipids, we have developed an artificial phosphatidylethanolamine (PE) vesicle-based Tomato bushy

206 Recently, higher quantities of phosphatidylethanolamine (PE) were also found at these c

207 e enrichments of phosphatidylserine (PS) and phosphatidylethanolamine (PE) were characterized with UP

208 phospholipids (phosphatidylcholine (PC) and phosphatidylethanolamine (PE)) and enhance the signals f

209 Phosphatidylethanolamine (PE), a cone-shaped phospholipi

210 the conversion of phosphatidylserine (PS) to phosphatidylethanolamine (PE), a critical step in membra

211 ing head groups of phosphatidylcholine (PC), phosphatidylethanolamine (PE), and cholesterol were crea

212 atidylserine (PS), phosphatidylcholine (PC), phosphatidylethanolamine (PE), and phosphatidylinositol

213 e was dominated by phosphatidylcholine (PC), phosphatidylethanolamine (PE), and triacylglycerol (TAG)

214 phatidylcholine (PC) and non-bilayer-forming phosphatidylethanolamine (PE), are not clearly defined.

215 Phosphatidylcholine (PC), phosphatidylethanolamine (PE), cardiolipin (CL) and sphi

216 s, particularly the nonbilayer-forming lipid phosphatidylethanolamine (PE), in mitochondrial function

217 we show that methylation of a phospholipid, phosphatidylethanolamine (PE), is a major consumer of SA

218 polyunsaturated phosphatidylcholine (PC) or phosphatidylethanolamine (PE), known to stimulate Na,K-A

219 of the Kennedy pathway, the N-methylation of phosphatidylethanolamine (PE), or the remodeling of exog

220 pid methylation of phosphatidylcholine (PC), phosphatidylethanolamine (PE), phosphatidic acid (PA), a

221 e ion/ion reactions of (13)C-TrEnDi-modified phosphatidylethanolamine (PE), phosphatidylserine (PS),

222 (13)C-TrEnDi-modified phosphatidylethanolamine (PE), phosphatidylserine (PS),

223 idylinositol (PI), and C43H80O8PN (C38:3), a phosphatidylethanolamine (PE), reached the Bonferroni co

224 Specifically, elevation in phosphatidylethanolamine (PE), sphingomyelin (SM), phosp

225 at ABCA4 can transport N-11-cis-retinylidene-phosphatidylethanolamine (PE), the Schiff-base conjugate

226 demonstrate that Cu(2+) binds bivalently to phosphatidylethanolamine (PE), the second most abundant

227 A previously unknown MmpL3 ligand, phosphatidylethanolamine (PE), was discovered inside thi

228 cript, we investigate synergy between PS and phosphatidylethanolamine (PE)-a lipid of much higher abu

229 We found that CTD specifically affects phosphatidylethanolamine (PE)-associated functions that

230 ylserine decarboxylase Psd1, which generates phosphatidylethanolamine (PE).

231 so be applied for cross-linking proteins and phosphatidylethanolamine (PE).

232 ntly reduced levels of PS and its derivative phosphatidylethanolamine (PE).

233 he phospholipids phosphatidylserine (PS) and phosphatidylethanolamine (PE).

234 pholipids, particularly cardiolipin (CL) and phosphatidylethanolamine (PE).

235 t bilayers representing models of bacterial (phosphatidylethanolamine (PE)/phosphatidylglycerol) and

236 Phosphatidylethanolamines (PE) and phosphatidylcholines

237 executed via oxygenation of polyunsaturated phosphatidylethanolamines (PE) by 15-lipoxygenases (15-L

238 spholipid classes, phosphatidylcholine (PC), phosphatidylethanolamines (PE), and phosphatidylinositol

239 n the total levels of complex lipids such as phosphatidylethanolamines (PE), lactosylceramides (LCER)

240 rdiolipins (CL), phosphatidylglycerols (PG), phosphatidylethanolamines (PE), phosphatidic acids (PA),

241 Herein, we show that distearyl-phosphatidylethanolamine-PEG (DSPE-PEG), which forms 12-

242 living systems: phosphatidylcholines (PCs), phosphatidylethanolamine (PEs), and phosphatidylserines

243 hatidylcholines (C18:2, C20:5, C18:1), and 3 phosphatidylethanolamines (PEs) (C38:3 PE plasmalogen) a

244 Phosphatidylcholines (PCs) and phosphatidylethanolamines (PEs) are usually the most abu

245 f a series of phosphatidylcholines (PCs) and phosphatidylethanolamines (PEs) in nitrogen using a drif

246 reference sites; phosphatidylcholines (PCs), phosphatidylethanolamines (PEs), and their lyso and ethe

247 ophosphatidylcholines, phosphatidylcholines, phosphatidylethanolamines (PEs), sphingomyelins, and tri

248 pholipids commonly found in lipid membranes, phosphatidylethanolamine, phosphatidylcholine, and phosp

249 in mutants defective in the biosynthesis of phosphatidylethanolamine, phosphatidylcholine, or cardio

250 prises sphingomyelin, cerebroside, ceramide, phosphatidylethanolamine, phosphatidylcholine, phosphati

251 ds in phosphatidylserine increased; and most phosphatidylethanolamines, phosphatidylcholines, sphingo

252 ix major lipid classes: phosphatidylcholine, phosphatidylethanolamine, phosphatidylglycerol, phosphat

253 choline/phosphatidylglycerol (PC/PG) and 1:1 phosphatidylethanolamine/phosphatidylglycerol (PE/PG) bi

254 ar mixtures of magainin 2 (MG2a) and PGLa in phosphatidylethanolamine/phosphatidylglycerol mimics of

255 note, Gpc1 loss did not significantly affect phosphatidylethanolamine, phosphatidylinositol, and phos

256 ing phosphatidylcholine, phosphatidylserine, phosphatidylethanolamine, phosphatidylinositol, and the

257 rs2, and Neo1) are implicated in flipping of phosphatidylethanolamine, phosphatidylserine, and phosph

258 Similarly, phosphatidylethanolamine, phosphatidylserine, and sphing

259 oline, and cholesterol and the inner leaf of phosphatidylethanolamine, phosphatidylserine, phosphatid

260 osphatidylcholine, lyso-phosphatidylcholine, phosphatidylethanolamine, phosphatidylserine, triacylgly

261 ma metastasis, including phosphatidic acids, phosphatidylethanolamines, phosphatidylserines, and phos

262 N-Acyl-phosphatidylethanolamine phospholipase D (NAPE-PLD) (EC

263 ne dynamics, allosteric modulation of N-acyl phosphatidylethanolamine phospholipase D).

264 Concentrations of 2 phosphatidylethanolamine plasmalogens, PE(18:2/P-18:0) a

265 tidylcholine (POPC); 2) 1-palmitoyl 2-oleoyl-phosphatidylethanolamine (POPE); and 3) a mixture of 75%

266 me required for the mitochondrial pathway of phosphatidylethanolamine production, is closely monitore

267 species in the CL, phosphatidylcholine, and phosphatidylethanolamine profiles, was reduced in PGC-1a

268 be selective, since phosphatidylcholine and phosphatidylethanolamine promoted ISVP* formation, where

269 Phosphatidylethanolamine (PtdEtn) is the second major ph

270 ow the expected decreased methyl-pool and PC/phosphatidylethanolamine ratio and are resistant to the

271 sults in a deleterious decrease in the PC-to-phosphatidylethanolamine ratio.

272 of pro-ferroptotic hydroperoxy-arachidonoyl-phosphatidylethanolamine, reduces cardiomyocyte cell dea

273 ndant phospholipids, phosphatidylcholine and phosphatidylethanolamine, respectively, in mammalian mem

274 Maillard type reaction products of phosphatidylethanolamine showed potent antioxidant activ

275 s) and depletion of phosphatidylinositol and phosphatidylethanolamine species containing unsaturated

276 presumed to include phosphatidylcholine and phosphatidylethanolamine species with two intact acyl ch

277 such as diacylglycerol lipase alpha, N-acyl-phosphatidylethanolamine-specific phospholipase D, and 1

278 ed Pcyt2alpha phosphorylation, activity, and phosphatidylethanolamine synthesis by 50-90%.

279 netic studies have validated the pathway for phosphatidylethanolamine synthesis from phosphatidylseri

280 ed by PCYT2, is the rate-limiting enzyme for phosphatidylethanolamine synthesis via the CDP-ethanolam

281 of EPT1, thereby hindering the final step in phosphatidylethanolamine synthesis.

282 ner membrane-localized metabolic enzymes for phosphatidylethanolamine synthesis.

283 e alphaE(C)/J and vesicles containing dansyl-phosphatidylethanolamine that depended on the native J s

284 rations in triacylglycerols and cardiolipins-phosphatidylethanolamines that precede the clinical outc

285 leoyl phosphatidylglycerol added to dioleoyl phosphatidylethanolamine, the proteins induced a dose-re

286 Phosphatidylethanolamine, the second most abundant eukar

287 ine N-methyltransferase (PEMT) that converts phosphatidylethanolamine to PC.

288 o2, which is required for the methylation of phosphatidylethanolamine to produce phosphatidylcholine,

289 lyzes the transfer of the sn-1-acyl chain of phosphatidylethanolamine to this N-terminal cysteine, ge

290 hat CD300a recognizes phosphatidylserine and phosphatidylethanolamine, two aminophospholipids exposed

291 brane insertion of LC3 protein modified with phosphatidylethanolamine using multiple coarse-grain sim

292 ynthesize phosphatidylcholine by methylating phosphatidylethanolamine using S-adenosylmethionine as a

293 ing of human phosphatidylserine to bacterial phosphatidylethanolamine was identified as CT699.

294 mples longer chain phosphatidylglycerols and phosphatidylethanolamines were more abundant.

295 es and cholesterol esters and an increase in phosphatidylethanolamines were observed in NASH patients

296 Monogalactosyldiacylglycerols and N-acyl phosphatidylethanolamines were the most accessible lipas

297 phospholipid ligands, phosphatidylserine and phosphatidylethanolamine, which are not exposed on the i

298 rrent was generated with the lipid substrate phosphatidylethanolamine, which carries no or little cha

299 en prion conversion and lipids, specifically phosphatidylethanolamine, which is a critical cofactor i

300 y that attaches AUTOPHAGY-RELATED8 (ATG8) to phosphatidylethanolamine, which then coats emerging auto