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1 bution of label from (14) C-glycerol between phosphoglycerides.
2 earoyl-2-oleoyl- and sn-1-palmitoyl-2-oleoyl phosphoglycerides.
3 f comparable docosahexaenoic acid-containing phosphoglycerides.
4 sed from 0 to 10 mol % of the total membrane phosphoglycerides.
5 d, the precursor for AA, rises in the plasma phosphoglycerides 3-fold.
6              Docosahexaenoic acid-containing phosphoglycerides accumulate preferentially in membranes
7                    Also, none of the anionic phosphoglycerides affected transfer action by human glyc
8 in is linked via phosphodiester linkage to a phosphoglyceride aglycone.
9 f MgCl2) when the vesicles contained anionic phosphoglycerides alone.
10 alkenyl chain lowers the molecular area of a phosphoglyceride and, concomitantly, makes it less compr
11 ut bound to membranes that contained anionic phosphoglycerides and could be stabilized by these membr
12 onal regulation of ACD11 and CPTP by anionic phosphoglycerides and found that 1-palmitoyl-2-oleoyl-ph
13 ic and lateral packing properties of related phosphoglycerides and found that: 1), The dipole moment-
14 he molecular packing properties of polyenoic phosphoglycerides and raise important questions about th
15          Plasma triacylglycerols and choline phosphoglycerides and red blood cell (RBC) choline and e
16  did bind to vesicles that contained anionic phosphoglycerides, and maximal binding occurred (in the
17                  4), Ethanolamine-containing phosphoglycerides are generally less compressible than t
18 activity of the bound enzyme toward membrane phosphoglycerides, assayed in the presence of albumin, i
19 ahexaenoyl- and sn-1-stearoyl-2-arachidonoyl phosphoglycerides, but the structural significance of th
20 onoyl group increases the molecular areas of phosphoglycerides by 3.8 A(2) (7%) relative to the prese
21 g of 160 lipids drawn from the glyceride and phosphoglyceride classes.
22                          F-DPPC is the first phosphoglyceride containing sn-1 and sn-2 ester-linked f
23 ned to the triacylglyceride (TG) and choline phosphoglyceride (CPG) pools.
24 pe 1 diabetes group had lower plasma choline phosphoglyceride DHA (3.7 +/- 0.9%; P < 0.01) than did t
25 ed blood cell (RBC) choline and ethanolamine phosphoglyceride FAs were assessed.
26 s provide the first evidence for a potential phosphoglyceride headgroup-specific regulatory interacti
27  important questions about the role of these phosphoglycerides in synapses.
28 ation of the polysaccharide chain lengths of phosphoglyceride-linked ECA (ECA(PG)).
29                                              Phosphoglyceride-linked enterobacterial common antigen (
30                          The assembly of the phosphoglyceride-linked form of enterobacterial common a
31 nd time-resolved anisotropy decay induced by phosphoglyceride membranes lacking or containing glycoli
32 P < 0.01) and RBC (type 2: P < 0.05) choline phosphoglycerides of the diabetics than of the control s
33 ess stabilized by the addition of an anionic phosphoglyceride or stearoyl-coenzyme A; and they show s
34 s that contained various mixtures of anionic phosphoglycerides, phosphatidylcholine, phosphatidyletha
35 of homogeneous monolayers of these and other phosphoglyceride species to obtain insights into their p
36 s, some going to the serine and ethanolamine phosphoglyceride (SPG and EPG) pools.
37 hil membranes consistent with utilization of phosphoglyceride substrate and release of free fatty aci
38 affecting its preference for specific diacyl phosphoglyceride substrates, (b) the activator promoted
39 sferase (DGAT) but can also be produced from phosphoglycerides such as phosphatidylcholine (PC) by th
40 entration of cellular fatty acids as well as phosphoglycerides, the components of cellular membranes.
41  is transferred from an sn-1-stearoyl-2-acyl phosphoglyceride to coenzyme A, and a relatively non-acy
42 referential transfer of stearoyl groups from phosphoglycerides to sn-2-acyl molecular species of lyso
43 nimals respond to chronic cold by increasing phosphoglyceride unsaturation to restore the fluidity of
44  control subjects, and cord RBC ethanolamine phosphoglycerides were lower in DHA (P < 0.05) in both d
45 d cholesterol provided evidence that anionic phosphoglycerides were positive effectors of binding, ph
46                             When the surface phosphoglycerides were replaced with either 16:0-18:2 ci
47  lipid bilayers allowed us to determine that phosphoglycerides with glycerol or inositol on the extra