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1 CM), thereby promoting premature skin aging (photoaging).
2 a major determinant of premature skin aging (photoaging).
3 olar UV irradiation-induced premature aging (photoaging).
4 the pathophysiology of premature skin aging (photoaging).
5  and may contribute to premature skin aging (photoaging).
6 omeostasis and premature skin aging, such as photoaging.
7 g major interest for treatments against skin photoaging.
8 ollagen degradation and photoinflammation in photoaging.
9 t skin from ultraviolet rays (UVR) and delay photoaging.
10  skin patterning is a sign of both aging and photoaging.
11 ich may provide potential prevention against photoaging.
12 E(2)) on HA-rich extracellular matrix during photoaging.
13 and anti-inflammatory effects of E(2) during photoaging.
14 ytes and fibroblast-based in vitro models of photoaging.
15 c target for the prevention and treatment of photoaging.
16 risk for skin cancer-a phenomenon defined as photoaging.
17  skin wrinkle formation in a murine model of photoaging.
18  possibility of using ERbeta as a target for photoaging.
19 tion associated with chronological aging and photoaging.
20 lammation, and tumorigenesis associated with photoaging.
21  nm) (UVA) may cause photocarcinogenesis and photoaging.
22 raviolet (UV) light to human skin results in photoaging.
23 olecular mechanism in the pathophysiology of photoaging.
24 ies are directed at improving the results of photoaging.
25 ts in an old and wrinkled appearance, called photoaging.
26 let AI light (340 to 400 nm) associated with photoaging.
27 y and/or antioxidant activities, may prevent photoaging.
28 s-links might be useful markers of aging and photoaging.
29 s toxic intermediates in the pathogenesis of photoaging.
30 f compounds that protect against UVA-induced photoaging.
31  degrade skin collagen and may contribute to photoaging.
32  enzymes, as mediators of collagen damage in photoaging.
33 protection against skin cancer, sunburn, and photoaging, a genome-wide perspective of gene expression
34  changes that correlate with improvements in photoaging after laser therapy.
35  alterations represent acute events, whereas photoaging and carcinogenesis are long-term consequences
36 trix proteins play a functional role in skin photoaging and carcinogenesis by sensitization of photo-
37            Results indicate that accelerated photoaging and contact with water result in faster trans
38    Radiation-induced skin damage ranges from photoaging and cutaneous carcinogenesis caused by UV exp
39 gnal transduction ultimately leading to skin photoaging and even skin cancer.
40 ene expression program associated with human photoaging and intrinsic skin aging (collectively termed
41 ved to underlie changes associated with both photoaging and natural aging, we determined whether natu
42 hlight the protective effects of NAM against photoaging and oxidative stress in the human epidermis a
43 ers of oxidative stress underlying cutaneous photoaging and photocarcinogenesis, but the molecular id
44 n of reactive oxygen species contributing to photoaging and photocarcinogenesis.
45                                              Photoaging and skin cancer have been extensively studied
46  TGFB provide possible treatment options for photoaging and skin cancer.
47 e clinically useful to prevent and/or reduce photoaging and skin cancer.
48 V light-based damage to skin cells can cause photoaging and skin cancer.
49  and should be useful for protection against photoaging and skin cancer.
50 a suggest a previously unreported pathway of photoaging and support the concept that photoaging is at
51 ology for premature skin aging that leads to photoaging and UV-induced skin cancers.
52 tudy, the contribution of a typical abiotic (photoaging) and biotic (biodegradation) process and the
53  skin due to ultraviolet light from the sun (photoaging) and damage occurring as a consequence of the
54 solar UV radiation leads to premature aging (photoaging) and skin cancer.
55 rix may serve as markers for skin aging, for photoaging, and for immediate assessment of exposure to
56 e appropriate for mild, moderate, and severe photoaging, and in educating patients on the risks and b
57 iation (760-1440 nm) in dermal inflammation, photoaging, and photocarcinogenesis.
58  for skin cancer, prevent sunburns, mitigate photoaging, and treat photosensitive dermatoses.
59 or 2 mo on alternate days) following in vivo photoaging animal protocol.
60                      Photocarcinogenesis and photoaging are established consequences of chronic expos
61    This study aimed at exploring the role of photoaging, as well as other well-known epidemiological
62 quations to estimate change in the degree of photoaging associated with increasing total antioxidant
63 quations to estimate change in the degree of photoaging associated with increasing total antioxidant
64 erienced in daily life, potentially promotes photoaging by affecting breakdown, rather than synthesis
65                                    Aging and photoaging cause distinct changes in skin cells and extr
66 n from sunlight causes premature skin aging (photoaging), characterized in part by wrinkles, altered
67                  Solar UV irradiation causes photoaging, characterized by fragmentation and reduced p
68  vitamin D production but also leads to skin photoaging, damage, and malignancies.
69 UVA1 induction of MMP12 mediates some of its photoaging effects, particularly by contributing to elas
70 levance of using fibrillin-1 as a marker for photoaging, facial skin was biopsied at baseline and aft
71  determined whether natural skin aging, like photoaging, gives rise to increased matrix metalloprotei
72     With a quantified relative importance of photoaging (>55%) vs biodegradation, the crucial contrib
73 on spectra of the physiological responses of photoaging has remained elusive.
74    This Commentary reports on the effects of photoaging in an experimental model of ultraviolet radia
75                         To study UVA-induced photoaging in an in vivo system, we utilized a line of t
76 fect signaling pathways and genes related to photoaging in human dermal fibroblasts; (2) to investiga
77 onses to ultraviolet light that eventuate in photoaging in human skin in vivo.
78 t, may also contribute to signs of premature photoaging in skin.
79  the use of mtDNA mutations as biomarkers of photoaging in the skin.
80 study, we investigated the effects of NAM on photoaging in two-dimensional human primary keratinocyte
81 amples to outdoor and accelerated artificial photoaging, in dry and wet conditions over 12 weeks.
82 xhibit ocular changes secondary to premature photoaging, including ocular surface tumors and pterygiu
83 eriod, the overall prevalence of severe skin photoaging increased from 42% at baseline to 88%.
84 mechanisms by which ultraviolet light causes photoaging involve activation of growth factor and cytok
85 y of photoaging and support the concept that photoaging is at least in part a process of damage-accel
86                                              Photoaging is caused in part by damage to skin connectiv
87                               Telangiectatic photoaging is characterized by less transient and nontra
88                                              Photoaging is the most common form of skin damage and is
89  exposure to the etiology of skin cancer and photoaging is undisputed.
90 r ultraviolet (UV) to induce skin cancer and photoaging is well recognized.
91                           Clinically chronic photoaging may result in fine wrinkles, texture abnormal
92 r, baldness, genetic risk score, and digital photoaging measures (digitally assessed pigmented spots,
93 n conclusion, genetic risk score and digital photoaging measures showed associations with increased l
94 of these processes in cancer, we discuss how photoaging might render the skin microenvironment permis
95                    This study focused on the photoaging of atmospheric particulate matter smaller tha
96 tiates chemical processes that result in the photoaging of skin.
97  the optical properties as a function of the photoaging of the extracts.
98         Because retinoic acid (RA) can alter photoaging of the skin and repeated ultraviolet (UV)-ind
99                                The premature photoaging of the skin is mediated by the sensitization
100 ccelerated by UV and with that contribute to photoaging of the skin.
101  over PAH(even) in the presence of EC during photoaging of the whole soot.
102 oxidant capacity of foods people eat and the photoaging of their skin.
103              Analyzing and understanding the photoaging of these compounds are essential for assessin
104  biodegradation, the crucial contribution of photoaging on MP aging was highlighted.
105 elanoma, and the tumor-permissive effects of photoaging on the skin microenvironment remain largely u
106 umeric scales that include manifestations of photoaging other than rhytids, such as lentigines, hyper
107  capacity experienced approximately 10% less photoaging over 15 years than those who ate foods with l
108  female participants with moderate-to-severe photoaging received nonablative fractional laser treatme
109 nalysis revealed progressive accumulation of photoaging-related changes and increased chronic inflamm
110 rick skin type classification and the Glogau photoaging scale.
111 ts' diets in 1992, 1994, and 1996 and graded photoaging severity using microtopography in 1992, 1996,
112 t in skin of color, both intrinsic aging and photoaging significantly impact skin function and compos
113 s may contribute to the clinical features of photoaging, such as wrinkle formation and loss of elasti
114 te multiple pathways underlying the cause of photoaging suggests ERbeta to be a novel therapeutic tar
115 etinoids are widely used in the treatment of photoaging to stimulate dermal repair.
116  of photoaging, which we term telangiectatic photoaging (TP).
117 effect of topical fluorouracil, 5%, cream on photoaging using validated photonumeric scales.
118 melanoma skin cancer (NMSC) and in cutaneous photoaging was explored using a genetic approach.
119 und to increase with aging and decrease with photoaging we investigated the characteristics of this d
120 mmunologic injury may play a role in chronic photoaging, we asked whether RA alters the acute photoim
121 of age-related changes in the skin known as "photoaging." We are in the preliminary stages of underst
122  arm of 16 subjects with a clinical range of photoaging were examined for fibrillin-1 and fibrillin-2
123  Individuals with clinically assessed severe photoaging were recruited to the study (n = 8).
124 idation in vivo in mouse skin, a hallmark of photoaging, when analyzed biochemically, by immunoblotti
125 oreover, MPs undergo chemical changes due to photoaging, which are worth investigating since they can
126                                              Photoaging, which is premature skin aging caused by long
127 ultraviolet A radiation (UVA) contributes to photoaging, which results in the accumulation of massive
128 ngs can also be associated with a subtype of photoaging, which we term telangiectatic photoaging (TP)
129 his study did not demonstrate improvement in photoaging with a standard course of topical fluorouraci

 
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