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1 and prednisone) alone or in conjunction with photopheresis.
3 titox, mycophenolate mofetil, extracorporeal photopheresis, and several monoclonal antibodies have be
4 odality fashion with combined extracorporeal photopheresis, bexarotene, interferon-y, and low-dose TS
5 tosis in patients clearing on extracorporeal photopheresis but persisted in nonresponsive patients.
6 ted with clinical response to extracorporeal photopheresis (ECP) and mortality after ECP in lung allo
7 ty is preserved in mice after extracorporeal photopheresis (ECP) but reduced with glucocorticosteroid
8 The safety and efficacy of extracorporeal photopheresis (ECP) for 12 to 24 weeks together with sta
16 gulatory T (Treg) cells after extracorporeal photopheresis (ECP) is thought to contribute to how ECP
23 27), and significantly fewer patients in the photopheresis group had two or more rejection episodes (
25 etected significantly less frequently in the photopheresis group than in the standard-therapy group (
26 bexarotene, interferon alpha, extracorporeal photopheresis, histone deacetylase inhibitors, and antib
27 y study to assess the safety and efficacy of photopheresis in the prevention of acute rejection of ca
30 nti-LFA-3-IgG fusion protein, extracorporeal photopheresis, mesenchymal stem cells and regulatory T c
32 ts for chronic GVHD including extracorporeal photopheresis, rituximab, sirolimus, mycofenolate mofeti
33 loaded dendritic cells as well as the use of photopheresis to generate an anti-idiotype cytotoxic T-c
35 e photopheresis group received a total of 24 photopheresis treatments, each pair of treatments given