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1 the Arabidopsis 5-FCL gene (At5g13050) under photorespiratory (30 and 370 micromol of CO2 mol(-1)) an
2 (30 and 370 micromol of CO2 mol(-1)) and non-photorespiratory (3200 micromol of CO2 mol(-1)) conditio
3                                        Under photorespiratory active conditions, the mutant accumulat
4                                          Two photorespiratory amino acids, glycine and serine, and my
5 enzyme responsible for the reassimilation of photorespiratory ammonia as well as for primary nitrogen
6 iscuss the significance of NADH-GOGAT in non-photorespiratory ammonium assimilation and in glutamate
7 s of photorespiratory mutants indicates that photorespiratory ammonium released in mitochondria is re
8 to chloroplastic GS2 for the assimilation of photorespiratory ammonium.
9 ndependent serine biosynthetic pathways, the photorespiratory and glycolytic phosphoserine (PS) pathw
10 per thousand ppm(-1) is largely explained by photorespiratory and mesophyll effects.
11 model of g(m) that describe the potential of photorespiratory and respiratory CO(2) released within t
12 plants also exhibit an increase in the CO(2) photorespiratory burst and an increase in levels of phot
13 g mechanism protein M) mutant, the metabolic photorespiratory burst triggered by shifting to low CO2
14               Consistent with this view, the photorespiratory by-product, H(2)O(2), induced the pyren
15             In this study, we designed a new photorespiratory bypass (called GOC bypass), characteriz
16 ssed cycle is designed to function as both a photorespiratory bypass and an additional CO2-fixing pat
17                    We engineered a synthetic photorespiratory bypass based on the 3-hydroxypropionate
18 t engineering a newly designed chloroplastic photorespiratory bypass could increase photosynthetic ef
19 ons to confirm and understand the drivers of photorespiratory bypass crop yield increases at various
20 tics; increased knowledge of the behavior of photorespiratory bypass metabolomic fluxes; exposure of
21 tional BHAC in plant peroxisomes to create a photorespiratory bypass that is independent of 3-phospho
22               Over the past few years, three photorespiratory bypasses have been introduced into plan
23 e native pathway through the introduction of photorespiratory bypasses offers a potential route to in
24 ntrations) show that excess glycine from the photorespiratory C(2) cycle (i.e. glycine not part of th
25 ost CO(2) sustains a higher flux through the photorespiratory C2 cycle that fully meets the glycine a
26 lase, catalyzes an essential sequence of the photorespiratory C2 cycle, namely, the conversion of two
27 centration and temperature, to determine how photorespiratory capacity acclimates to environmental tr
28 ents indicate that there is no plasticity in photorespiratory capacity in B. papyrifera, and that a f
29 climation toward conditions requiring a high photorespiratory capacity.
30 torespiratory metabolites, of enzymes of the photorespiratory carbon cycle, and of corresponding tran
31 e-assimilation of ammonia resulting from the photorespiratory carbon cycle.
32                                              Photorespiratory carbon flux reaches up to a third of ph
33 duced 66% by MPA, while intermediates of the photorespiratory carbon oxidation cycle showed a 3-fold
34 odeling indicate that the establishment of a photorespiratory carbon pump (termed C2 photosynthesis)
35 y Gly decarboxylase, affects the recovery of photorespiratory carbon, and this appears to be critical
36 as identified a number of mutants exhibiting photorespiratory chlorosis at ambient CO(2), including s
37 synthesis is characterised using recapturing photorespiratory CO(2) by RuBisCo in Kranz-like cells an
38 olution of C(4) photosynthesis, leading to a photorespiratory CO(2) concentrating mechanism.
39 ermediate Flaveria species revealed that the photorespiratory CO(2) pump was not established in one s
40 revealed an increase in the stoichiometry of photorespiratory CO(2) release and impaired Gly-to-Ser t
41  decarboxylase (GDC) is the key component of photorespiratory CO(2) release in plants and is active i
42  the wild type, suggesting that the ratio of photorespiratory CO(2) release to Rubisco oxygenation wa
43 ted 2% of gross CO(2) uptake (v(c)), whereas photorespiratory CO(2) release was approximately 20% of
44 ase (GDC)-dependent) manner, and recuperates photorespiratory CO2 as oxaloacetate (OAA).
45 rees C, indicating that the stoichiometry of photorespiratory CO2 formation per glycolate oxidized no
46 a involves the establishment of a two-celled photorespiratory CO2 pump, termed C2 photosynthesis.
47 ments can be used as a proxy to identify the photorespiratory component of light-induced photosynthet
48 eversed when these plants are grown in a non-photorespiratory condition (i.e. 1% CO2 atmosphere), dem
49 ed when the aae13-1 mutant is grown in a non-photorespiratory condition (i.e. a 1% CO2 atmosphere), d
50       Mutants with large glycine pools under photorespiratory conditions (5-formyl THF cycloligase an
51        Under high light, CEF increased under photorespiratory conditions (high oxygen and low CO2), c
52 demonstrated that mutant plants under active photorespiratory conditions accumulated high levels of s
53                  Maltose was increased under photorespiratory conditions in both wild type and plants
54 tarch phosphorylase enzyme were placed under photorespiratory conditions, G6P levels remained constan
55                                           In photorespiratory conditions, leaf glycine levels were up
56 HO-THF level under all conditions and, under photorespiratory conditions, quadrupled the pool of 10-f
57                                        Under photorespiratory conditions, transitory starch breakdown
58  carbon sources increased was observed under photorespiratory conditions, while photosynthetic condit
59 transferred for different lengths of time to photorespiratory conditions.
60       This enzyme complex is involved in the photorespiratory cycle and is inhibited by victorin, wit
61                                          The photorespiratory cycle begins with ribulose-1,5-bisphosp
62 ytosol, no transporter required for the core photorespiratory cycle has been identified at the molecu
63                                          The photorespiratory cycle is complex and occurs in three or
64  associated with subtle perturbations in the photorespiratory cycle of HPR1-T335D-complemented hpr1-1
65 plants, glycolate oxidase is involved in the photorespiratory cycle, one of the major fluxes at the g
66 ion of SHMT1 boosted the Gly flux out of the photorespiratory cycle, which increased the levels of th
67 r, which is required for the function of the photorespiratory cycle.
68                           Our data show that photorespiratory delays cause offsets in predicted CO(2)
69 ism, changes in cell organization, increased photorespiratory enzyme activity, induction of periplasm
70 depleted lipoylation of the H subunit of the photorespiratory enzyme glycine decarboxylase, increased
71                              Mutation of the photorespiratory enzyme Ser-hydroxymethyltransferase 1 (
72 clude that Arabidopsis AGT1 is a peroxisomal photorespiratory enzyme that catalyzes transamination re
73  both the amounts of messenger RNAs encoding photorespiratory enzymes and the respective protein cont
74                                              Photorespiratory enzymes are encoded by nuclear genes, t
75 egulate the expression of the genes encoding photorespiratory enzymes is coordinated temporal control
76 is is consistent with its involvement in the photorespiratory export of glycolate from Arabidopsis ch
77   Microscopic analysis revealed that altered photorespiratory flux also affected GC starch accumulati
78 resource limitation in conjunction with high photorespiratory flux as a selective pressure relevant t
79 opsis thaliana) and in a mutant with altered photorespiratory flux due to the absence of the peroxiso
80 te a doubling in the carboxylation rate, the photorespiratory flux increased from 17 to 28% of net CO
81 erning the regulatory mechanisms that adjust photorespiratory flux is rare.
82 pathway and is especially critical when high photorespiratory fluxes are required and when the major
83 these family members, including one probable photorespiratory gene (SHM1) and a second gene expressed
84 ntal evidence for a coordinate regulation of photorespiratory genes over time.
85               A coordinate repression of the photorespiratory genes was observed in the mutant backgr
86  are in phase with those described for other photorespiratory genes.
87                            In higher plants, photorespiratory Gly oxidation in leaf mitochondria yiel
88 mitochondrial proteins, hyperaccumulation of photorespiratory Gly, and reduced accumulation of many i
89                     This work indicates that photorespiratory glycine accumulation during the early l
90 ration due to the reduced lipoylation of the photorespiratory glycine decarboxylase.
91  with a decrease in the rate of oxidation of photorespiratory glycine in the mitochondrion.
92 rboxylase (GDC), necessary for the high-flux photorespiratory glycine-into-serine conversion.
93 ry route mediated by HPR2, which we name the photorespiratory glyoxylate shunt.
94  point toward divergent functions of the two photorespiratory GOX isoforms in Arabidopsis and contrib
95 pe HPR1 or HPR1-T335A fully complemented the photorespiratory growth phenotype of hpr1-1 in ambient a
96   Our findings reveal a link between SHR and photorespiratory H2O2 production that has implications f
97                              In peroxisomes, photorespiratory HYDROXYPYRUVATE REDUCTASE1 (HPR1) catal
98                       This involves altering photorespiratory influx by manipulating the growth envir
99 ty in the pathway that manages unpredictable photorespiratory influx in dynamic environments.
100                  Feeding [2-(13)C]glycine (a photorespiratory intermediate) stimulated emissions of [
101 lso caused the cells to excrete glycolate, a photorespiratory intermediate, but did not change the ap
102                       However, levels of the photorespiratory intermediates 2-phosphoglycolate and gl
103 eased steady state contents of TCA cycle and photorespiratory intermediates as well as elevated NAD(P
104 drial proteins, and the hyperaccumulation of photorespiratory intermediates, glycine and glycolate.
105 spiratory burst and an increase in levels of photorespiratory intermediates, suggesting changes in ph
106 e, as TPP riboswitch mutants accumulate less photorespiratory intermediates.
107 ar efficiency to glycolate; in contrast, the photorespiratory isoforms GOX1 and GOX2, which share sim
108                                        Thus, photorespiratory losses of CO2 were significantly reduce
109                                              Photorespiratory losses were similar to other C3 species
110 l isoform of aspartate aminotransferase, and photorespiratory markers, while the C-CP and P-CP have h
111 cycled by a series of biochemical reactions (photorespiratory metabolism).
112 hondrial-localized thioredoxin o1 (TRXo1) on photorespiratory metabolism.
113 vailability: water-replete trees export more photorespiratory metabolites to lignin whereas water-lim
114 ut little is known about the contribution of photorespiratory metabolites to the regulation of gene e
115 ts the toxic accumulation of non-metabolized photorespiratory metabolites, and (iii.) photorespiratio
116 we examined diurnal changes in the levels of photorespiratory metabolites, of enzymes of the photores
117 e and irradiance on leaf respiration (R, non-photorespiratory mitochondrial CO(2) release) of snow gu
118  we have isolated genetic suppressors of the photorespiratory mutant hpr1 (hydroxypyruvate reductase
119 tants also partially rescue hpr1 and another photorespiratory mutant, catalase 2.
120                                  Analysis of photorespiratory mutants indicates that photorespiratory
121                 This is in contrast to other photorespiratory mutants, which have severely reduced ra
122 m, revealing onward metabolism of Asn by the photorespiratory nitrogen cycle and accumulation of arom
123 at chloroplast linear electron transport and photorespiratory O(2) uptake were similar between genoty
124 stomatal conductance, photosynthetic CO2 and photorespiratory O2 fixation, and starch synthesis in re
125     This study determines photosynthetic and photorespiratory parameters for leaves in a natural stan
126 dels of C(3) photosynthesis by including the photorespiratory pathway (PCOP) and metabolism to starch
127         This shunt complements the canonical photorespiratory pathway and is especially critical when
128 an up-regulation of the Calvin cycle and the photorespiratory pathway in peroxisomes and mitochondria
129 cally the lack of transient increases in the photorespiratory pathway intermediates 2-phosphoglycolat
130                                          The photorespiratory pathway is comprised of enzymes localiz
131 atory fluxes are required and when the major photorespiratory pathway is deficient.
132                                          The photorespiratory pathway is highly compartmentalized, in
133 nd 14CO2 in a 1:1 ratio, suggesting that the photorespiratory pathway is otherwise normal in the muta
134 h CO(2) conditions; and all glycine from the photorespiratory pathway is routed to proteins within ph
135                                 Although the photorespiratory pathway is well characterized, little i
136 serine catabolism of vertebrates, and in the photorespiratory pathway of oxygenic phototrophs.
137 d differences in the use of Calvin cycle and photorespiratory pathway reactions.
138 ypothesis that facilitating flux through the photorespiratory pathway stimulates photosynthetic CO2 a
139 coding for the core metabolic enzymes of the photorespiratory pathway that allows plants with C3-type
140 lase complex (GDC) is a key component of the photorespiratory pathway that occurs in all photosynthet
141 nes encoding mitochondrial components of the photorespiratory pathway, we characterized a family of A
142 notransferases play central roles within the photorespiratory pathway.
143 and peroxisomal (catalase) components of the photorespiratory pathway.
144  contribute to a better understanding of the photorespiratory pathway.
145 that increase the metabolic flux through the photorespiratory pathway.
146 on of the fixed carbons lost in anabolic and photorespiratory pathways in conjunction with flux rerou
147 phosphate carboxylase/oxygenase kinetics and photorespiratory pathways.
148 s buffer plant growth and metabolism against photorespiratory perturbations.
149 tion, glycolate oxidase (GOX) mutants with a photorespiratory phenotype have not been described yet i
150 one of the two genes for Fd-GOGAT leads to a photorespiratory phenotype in the gls1 mutants.
151 LYCOLATE OXIDASE1 (GOX1) that attenuated the photorespiratory phenotype of cat2-2 Interestingly, knoc
152                                          The photorespiratory phenotype of cat2-2 mutants did not dep
153 or the SHM1 promoter in shm1-1 abrogated the photorespiratory phenotype of the shm mutant, whereas ov
154 hondrial SHMT activity and displays a lethal photorespiratory phenotype when grown at ambient CO2, bu
155  in the cat2-2 background did not affect the photorespiratory phenotype, indicating that GOX1 and GOX
156 glycerate regeneration or decarboxylation of photorespiratory precursors.
157 lic GS1 in leaves may act via photosynthetic/photorespiratory process.
158  diffusional limitations to gas exchange and photorespiratory rates.
159  leaves with closed stomata, indicating that photorespiratory recycling of CO(2) provided little phot
160 ic capacity, highlighting a role for the non-photorespiratory release of CO(2) in heat tolerance.
161              The Calvin-Benson cycle and its photorespiratory repair shunt are in charge of nearly al
162 een cytosolic glyoxylate and a non-canonical photorespiratory route mediated by HPR2, which we name t
163                     Sequence analysis of the photorespiratory sat mutants revealed a single nucleotid
164 otein interactions and complex formation for photorespiratory SHMT activity demonstrates more complic
165 SHMT1, and this interaction is necessary for photorespiratory SHMT activity.
166 across gradients of drought (r(2) > 0.8) and photorespiratory stress (r(2) > 0.9).
167 nd specialized metabolites and help mitigate photorespiratory stress under elevated temperature and d
168 a cat2-2 genetic background upon exposure to photorespiratory stress.
169 imit emission rates under severe drought and photorespiratory stresses.
170 of the control loops that sense the ratio of photorespiratory to photosynthetic carbon flux and in tu
171 e translocator 1 (PLGG1) as a candidate core photorespiratory transporter.

 
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