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1                                 Cones may be phototropic, actively orientating themselves towards lig
2                Young rhizoids are negatively phototropic, and NPA also inhibits rhizoid phototropism.
3 acterized in the purple nonsulfur anoxygenic phototropic bacterium Rhodopseudomonas palustris.
4  displayed elongated hypocotyls but retained phototropic behavior and the ability to fully deetiolate
5 utant, in which phyA remains in the cytosol, phototropic bending is slower than in the wild type.
6 ocotyl growth rate, apical hook opening, and phototropic bending with high spatiotemporal resolution.
7 ropins are flavoprotein kinases that control phototropic bending, light-induced chloroplast movement,
8 ich encodes phototropin, a photoreceptor for phototropic bending.
9  optical performance is further optimized by phototropic chromatophores that regulate the dose of ill
10     Modulatory increases in the magnitude of phototropic curvature have been termed "enhancement." He
11  functionality consistent with a model where phototropic curvature is established by signalling outpu
12                           The development of phototropic curvature of etiolated seedlings of Arabidop
13 c responsiveness, accounting for the greater phototropic curvature of the nph2 and nph4 mutants to UV
14                             The amplitude of phototropic curvature to blue light is enhanced by a pri
15 owth rates were equal for both genotypes and phototropic curvature was only slightly inhibited in NS
16 d required for phot1-directed first-positive phototropic curvature.
17 ts overexpressing CRY1 or CRY2 show enhanced phototropic curvature.
18 dephosphorylation of NPH3 and development of phototropic curvatures by protein phosphatase inhibitors
19                             We conclude that phototropic enhancement by canopy shade results from the
20 een termed "enhancement." Here, we show that phototropic enhancement is primarily a phytochrome A (ph
21 ultimate target(s) of phyA action during the phototropic enhancement response is a rate-limiting ARF-
22 4 expression increases, which contributes to phototropic enhancement.
23                  Consistent with its lack of phototropic function in Arabidopsis, Otphot does not ass
24 l evolution of the lamellar structures under phototropic growth conditions.
25 ateral auxin gradient, ultimately leading to phototropic growth in shoots.
26     These inorganic nanostructures exhibited phototropic growth in which lamellar stripes grew toward
27                                    Inorganic phototropic growth is analogous to biological systems su
28 iments and simulations are consistent with a phototropic growth mechanism in which the optical near-f
29 d in the second step of a two-step inorganic phototropic growth process depends on a preexisting stru
30 ical systems such as palm trees that exhibit phototropic growth wherein physical extension of the pla
31 C HYPOCOTYL 3 (NPH3) is a key determinant of phototropic growth which is regulated by phototropin (ph
32               Genes implicated in control of phototropic growth, but not clock genes, are differentia
33 -A/blue-light activated kinases that trigger phototropic growth.
34 r spontaneous order generation via inorganic phototropic growth.
35 nanoscale self-organization during inorganic phototropic growth.
36  The plasma-membrane associated protein, NON-PHOTOTROPIC HYPOCOTYL 3 (NPH3) is a key determinant of p
37  family of proteins and is homologous to NON-PHOTOTROPIC HYPOCOTYL 3 (NPH3), a BTB/POZ protein that r
38 res blue light, is blocked by removal of NON-PHOTOTROPIC HYPOCOTYL 3 (NPH3), and is recapitulated by
39 onjunction with the signalling component Non-Phototropic Hypocotyl 3 (NPH3).
40  of the phototropic signalling component Non-Phototropic Hypocotyl 3 (NPH3).
41 ecently demonstrated that members of the NON-PHOTOTROPIC HYPOCOTYL 3 (NPH3)/RPT2-like (NRL) family in
42 eins identified to date, only one, NPH3 (non-phototropic hypocotyl 3), is essential for all phot1-dep
43                                              Phototropic hypocotyl bending in response to blue light
44                     In Arabidopsis thaliana, phototropic hypocotyl bending is initiated by the blue l
45                           Here, we show that phototropic hypocotyl bending is strongly dependent on t
46 everal signaling components that include NON-PHOTOTROPIC HYPOCOTYL3, PHYTOCHROME KINASE SUBSTRATE, RO
47 hromes and phytochromes are not required for phototropic induction, these photoreceptors do modulate
48 hese stromatolites were probably accreted by phototropic microbes that, from their habitat in shallow
49 series of twisting, bending, photophobic and phototropic motions.
50 gurations, programmable motion patterns, and phototropic movement where the material moves in respons
51                                            A phototropic mutant of P. blakesleeanus with a heterozygo
52  We show, as examples, that the gravitropic, phototropic, nutational, and thigmotropic dynamic respon
53 ese loci, NPH1, encodes the apoprotein for a phototropic photoreceptor.
54 he sporangiophore remain constant during the phototropic response (bending toward unilateral light) a
55  all three assumptions are incorrect for the phototropic response and probably incorrect for the avoi
56 ation into a longitudinal network during the phototropic response in Arabidopsis thaliana depends on
57                  In etiolated seedlings, the phototropic response is enhanced by the red/far-red (R/F
58                                          The phototropic response of Arabidopsis (Arabidopsis thalian
59                                 The enhanced phototropic response of cry1 mutants in the lab and in r
60 pin is a blue-light receptor involved in the phototropic response of higher plants.
61 ant were essentially agravitropic, but their phototropic response was robust.
62 ss to direct sunlight becomes important, the phototropic response was strong.
63 A, we show that nuclear phyA accelerates the phototropic response, whereas in the fhy1 fhl mutant, in
64 ue-light photoreceptors are deficient in the phototropic response.
65 L 3 (NPH3), a BTB/POZ protein that regulates phototropic responses along with the protein kinase PHOT
66                                              Phototropic responses also require auxin transport and w
67 he predominant photoreceptor of UV-B-induced phototropic responses in Arabidopsis (Arabidopsis thalia
68                        Despite their altered phototropic responses in blue and green light as etiolat
69 phoric flavoprotein photoreceptor regulating phototropic responses in higher plants.
70 ylating flavoprotein photoreceptor mediating phototropic responses in higher plants.
71 PphnRNP-H1 is involved in red light-mediated phototropic responses in P. patens and that it binds wit
72 1-5 mutant exhibited enhanced phot2-mediated phototropic responses like those of the phot1-5 rcn1-1 d
73                           Examination of the phototropic responses of a mutant deficient in biologica
74                                    Thus, the phototropic responses of fungi through madA and plants t
75 ngly, both auxin-regulated organogenesis and phototropic responses require an auxin response factor (
76  cells, and mutant hypocotyls display strong phototropic responses to lateral light stimulation.
77 iated signaling pathways have been linked to phototropic responses under various conditions.
78 utants are all altered with respect to their phototropic responses, only the nph4 mutants are also al
79 tyl 3), is essential for all phot1-dependent phototropic responses, yet little is known about how pho
80 dules into a single gene, thereby optimizing phototropic responses.
81 ner that is physiologically similar to plant phototropic responses.
82 her, our results support the hypothesis that phototropic responsiveness is modulated by inputs that i
83 mation by UV-A light mediates an increase in phototropic responsiveness, accounting for the greater p
84 g pathways have also been shown to influence phototropic responsiveness, and these pathways are influ
85 kinase domain, which is tightly coupled with phototropic responsiveness.
86 regulation of this E3 is required for normal phototropic responsiveness.
87 rassinosteroids and auxin signaling modulate phototropic responsiveness.
88  CASSETTE subfamily B19 (ABCB19) by phot1 in phototropic seedlings suggests that phot1 may directly r
89 nd PP2A activity is reduced, showed enhanced phototropic sensitivity and enhanced blue light-induced
90             Though several components of the phototropic signal response pathway have been identified
91  significance of the A'alpha helix region in phototropic signaling of tomato.
92 ate with or trigger dephosphorylation of the phototropic signalling component Non-Phototropic Hypocot
93 d auxin signaling in the hypocotyl and, upon phototropic stimulation, a steeper auxin signaling gradi
94                 Here we report an artificial phototropic system based on nanostructured stimuli-respo
95 ts the simplest, and possibly most abundant, phototropic system requiring only a retinal-bound transm