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1 istration of (153)Sm-EDTMP on the developing physeal and articular cartilage of healthy, juvenile rab
5 62 x 10(-3) mm2/sec +/- 0.38) than it was in physeal cartilage (1.28 x 10(-3) mm2/sec +/- 0.31) (P <.
6 only source compartments contributing to the physeal cartilage doses of 50.0 mGy/MBq for the proximal
7 ls expand in response to trauma, and produce physeal cartilage for growth plate tissue regeneration.
8 on of the effects of 153Sm-EDTMP on immature physeal cartilage is warranted to develop optimized trea
9 nically significant damage to the developing physeal cartilage may occur as a result of the intraveno
10 clinically significant damage to developing physeal cartilage might occur as a result of systemic 15
12 als were sacrificed at 16 wk of age, and the physeal cartilage of multiple bones was evaluated by use
14 nd distal femoral structures, epiphyseal and physeal cartilage, and epiphyseal and metaphyseal marrow
20 anged with age, with individuals approaching physeal closure having shorter tracts in a random arrang
21 bbit proximal tibial physis results in early physeal closure, which is accompanied by a transient ele
27 and level of injury within the cartilage in physeal fracture-separations can be defined with MR imag
30 rmal and may be due to infections, traumatic physeal injuries, genetic predisposition, metabolic fact
32 th and bone remodeling potential, minimizing physeal injury, and aggressive treatment of open fractur
33 sed to yield both 3D rendered and projection physeal maps that are particularly useful in preoperativ
37 maging findings differentiate epiphyseal and physeal regions and correlate with histologic findings.
40 acement of the femoral head epiphysis with a physeal step was seen on the longitudinal section obtain