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1  at intercellular junctions of arachnoid and pia mater cells that form the leptomeninges and border t
2 s into the SAS bordered by the arachnoid and pia mater during health and neuroinflammation, and detec
3 ture and function of the dura, arachnoid and pia mater in the context of conventional views, recent p
4                      The dura, arachnoid and pia mater, as the constituent layers of the meninges, al
5 ssels, and in the dura mater, arachnoid, and pia mater of the meningeal sheath surrounding the optic
6 s, classically known as dura, arachnoid, and pia mater.
7 blasts in the embryonic dura, arachnoid, and pia.
8 at affect arteries and veins in the dura and pia, we determined the extent to which CGRP contributes
9 haracterized: dura-arachnoid enhancement and pia-subarachnoid space enhancement.
10 ected in the cerebral cortex, pia mater, and pia-ensheathed leptomeningeal vessels in two GBCA-expose
11 rane located between the neuroepithelium and pia-meninges.
12 l blood vessels and later within neurons and pia arachnoid (> or =3 hours), particularly within pirif
13 moniae iron uptake ABC transporters, piu and pia, resulted in a strain with impaired growth in two ty
14 treptonigrin of the individual pit, piu, and pia mutant strains and comparison of the growth in iron-
15 e astrocytes interact with blood vessels and pia, we suggest that such contact represents an early st
16 ates comprise three layers (dura, arachnoid, pia mater), representing an important barrier surroundin
17                Gap junction coupling between pia-arachnoid cells and astrocytes was shown by dye tran
18  that are not observed in young animals, but pia is unexpectedly hypertrophied in a mouse model of Al
19 tention was detected in the cerebral cortex, pia mater, and pia-ensheathed leptomeningeal vessels in
20  of overmigrated neurons into the developing pia-arachnoid, scattering its mesenchymal cells througho
21 t in basal laminae of the retina, epidermis, pia, cardiac and striated muscle, kidney, blood vessels,
22 recorded cell was estimated by distance from pia to the layer VI/white matter boundary, and verified
23 in layer VI when vertical cuts extended from pia mater through layers IV or V, but were no longer syn
24                   cPLA2 mRNA was detected in pia mater, both at the brain surface and inner core of t
25 cells, and in the brain, it was expressed in pia matter and in neuronal tissues.
26 n subjects revealed IFITM mRNA expression in pia mater and blood vessels.
27                             Calcium waves in pia-arachnoid cells could invade contiguous astrocytes,
28 es appear as the meninges differentiate into pia, arachnoid, and dura.
29                                    Meningeal pia mater reportedly forms the outermost boundary that c
30 ubarachnoid space (enclosed by the meningeal pia and arachnoid layers), and the outmost meningeal dur
31           NMO-IgG outlines CNS microvessels, pia, subpia, and Virchow-Robin space.
32 oreceptor outer segments and the optic nerve pia and dura.
33 unohistochemical staining of the optic nerve pia mater.
34 ing density was high in the olfactory nerve, pia mater, and aspects of the ventricular ependyma and w
35 that interlaminar processes contact neurons, pia, and capillaries, suggesting a potential role for IL
36 iable and that the structure and function of pia suggests a previously unrecognized role in regulatin
37 rmation and further suggest a unique role of pia in cerebellar cortex histogenesis.
38 l influx of CSF tracer, suggesting a role of pia in CSF filtration, but not flow restriction.
39 urogenesis and translocate to the overlaying pia, forming the indusium griseum.
40 nd emigrated neurons disrupted the overlying pia mater.
41 issue-resident leukocytes in CNS parenchyma, pia-enriched subdural meninges, dura mater, choroid plex
42 echanical stimulation of adult human and rat pia-arachnoid cell cultures (loaded with calcium indicat
43 pes were observed in layer I: Cajal Retzius, pia surface, vertical axon, and horizontal axon cells.
44             Furthermore, we demonstrate that pia is comprised of vascular and cerebral layers that co
45                            Yet, we show that pia is perforated and permissive to PVS fluid flow.
46 ingle and double mutant strains suggest that pia is the dominant iron transporter during in vitro and
47                                          The pia-arachnoid waves were blocked by either octanol or ap
48 landmarks, i.e., the barrels in Layer 4, the pia and white matter surfaces and the blood vessel patte
49                                  Because the pia mater and Purkinje cells sandwich the cerebellar EGL
50 ction-limited imaging up to 850 um below the pia in awake mice.
51 that lie as far as 600 micrometers below the pia mater of primary somatosensory cortex in rat; this d
52  intensity is greatest ~300-400 um below the pia, not at the brain surface.
53 pressing neurons that lie 700 um beneath the pia, calcium dynamics of layer 5b projection neurons and
54 2(-/-) cell lines were implanted between the pia and arachnoid meninges as well as in the sciatic ner
55 n mast cells (or their precursors) enter the pia and access the thalamus by traveling along the ablum
56  then along blood vessels extending from the pia into the telencephalon on posthatch day 4-5, and in
57 he number of cells and microvessels from the pia to the white matter, show a significant correlation
58 shared by neuron ensembles spanning from the pia to the white matter.
59 nctionally similar neurons spanning from the pia to the white matter.
60 intense mGluR1 alpha immunoreactivity in the pia mater and blood vessels in the subarachnoid space an
61 t, in newborn mice, Ink4c is detected in the pia mater and in an adjacent layer of rapidly dividing c
62  new synthesis of elastin was present in the pia mater but not in astrocytes in the glial scar.
63 on, and creates small discontinuities in the pia mater overlying the tectum.
64 ymphatic marker staining was detected in the pia mater, in the arachnoid, in venous sinuses, and amon
65            Staining was also observed in the pia matter, on a subpopulation of ependymal cells lining
66               Mast cells first appear in the pia on embryonic day (E)13-14 in ovo, then along blood v
67                        The population in the pia reaches a maximum at postnatal (PN) day 11, and decl
68 disturbances that coincide with holes in the pia, and the caudomedial tectum exhibits prominent folds
69  uptake mechanisms, GABA transporters in the pia-arachnoid may help to regulate the amount of GABA av
70        The fraction of LC inserting into the pia (2.2%-29.6%) had a significant decrease with age (P
71 ecrease in the area of LC insertion into the pia (P = 0.55).
72                     The LC inserted into the pia in all eyes.
73 ar implications of the LC insertion into the pia mater are not well understood and should be investig
74                    The LC insertion into the pia mater is common in middle-aged and older eyes, and d
75 he peripapillary scleral flange and into the pia, and computed the total area of insertion and fracti
76 ertion and fraction of LC inserting into the pia.
77 that abnormalities in a region involving the pia mater and subpial cord occur early in the course of
78 he meninges, a triple layer of membranes-the pia mater, arachnoid mater, and dura mater-surround the
79 ast cells occur in two locations, namely the pia and the brain parenchyma.
80 tolysin gene) and piaB (permease gene of the pia ABC transporter) are currently used as the gold-stan
81 lted in widespread infection of cells of the pia and arachnoid mater of the leptomeninges over large
82  adhesion is critical for maintenance of the pia and proper cerebellar morphogenesis.
83             Leptomeninges, consisting of the pia mater and arachnoid, form a connective tissue invest
84 orresponding to the expected location of the pia mater and subpial region-and in spinal cord white an
85 ions requires a concomitant expansion of the pia mater.
86 m to anatomically distinct cell types of the pia, inner arachnoid, outer arachnoid barrier, and dural
87 gration defects leading to disruption of the pia-arachnoid, ectopia of fibroblasts in the cortex, and
88                                 Removing the pia resulted in the formation of a physical connection b
89 ral fibers issued collateral branches to the pia at the frontal part of the brain.
90 , extend processes from the ventricle to the pia in regular parallel arrangements.
91 minar process that runs perpendicular to the pia into deeper cortical layers.
92 ortical parenchyma may be transmitted to the pia-arachnoid and might then serve in the induction of n
93  and 3 was almost entirely restricted to the pia-arachnoid, whereas mGATs 1 and 4 were present only i
94 spinal cord, from the ependymal layer to the pia-glial limitans, and from oligodendrocytes surroundin
95 aminae are predominantly oriented toward the pia.
96 surrounding blood vessels and underlying the pia.
97 lls in the more internal connective tissues (pia-arachnoid and endoneurium-perineurium) was also foun
98 ry itself does not bias activity flow toward pia.