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1 ted with the Syk-selective kinase inhibitor, piceatannol.
2 as inhibited by the Syk-selective inhibitor, piceatannol.
3  a rapid accumulation of E-resveratrol and E-piceatannol.
4 onal bipyramidal geometry in the presence of piceatannol.
5 n be modulated by apigenin, resveratrol, and piceatannol.
6  the spleen tyrosine kinase (Syk) inhibitor, Piceatannol.
7 he latter was prevented by the Syk inhibitor piceatannol.
8 esults are obtained using the Syk inhibitor, piceatannol.
9 t function caused by kinase-deficient Syk or piceatannol.
10 d ERK1 and ERK2 phosphorylation inhibited by piceatannol.
11  3-O-glucoside (0.28 +/- 0.10 mg/100 g), and piceatannol (0.23 +/- 0.12 mg/100 g) predominated in Cor
12                       Group 1 (n=8) received piceatannol 30 mg/kg per day intravenously and cyclospor
13                                              Piceatannol 4'-galloylglucoside, epicatechin, 8-hydroxyl
14                    Pharmacologic exposure to piceatannol, a known Syk family kinase inhibitor, inhibi
15                                              Piceatannol, a natural stilbene, is an analog and a meta
16                                              Piceatannol, a promising health-promoting stilbene compo
17                                              Piceatannol, a stilbene analogue to resveratrol with hig
18  with our recent discovery of high amount of piceatannol, a stilbene with potent biological activitie
19 phatidylinositol-3 kinase inhibitor, or with piceatannol, a Syk inhibitor, consistent with uptake thr
20                     Pretreatment of PMN with piceatannol, a Syk-selective inhibitor, blocked EIgG pha
21                    Pretreatment of PMNs with piceatannol, a Syk-selective inhibitor, blocked Syk acti
22 yrphostin A9, a Pyk2-specific inhibitor, and piceatannol, a Syk-specific inhibitor.
23 yl groups of stilbenes are critical and that piceatannol, a tetrahydroxystilbene, suppresses NF-kappa
24 geting and in human platelets incubated with piceatannol, a tyrosine kinase inhibitor reportedly sele
25                                              Piceatannol abrogated the expression of TNF-induced NF-k
26 in the natural polymer and demonstrates that piceatannol acts as an authentic monomer participating i
27                   Apigenin, resveratrol, and piceatannol all induced Nrf2 translation.
28                       Group 3 (n=4) received piceatannol alone as in group 1.
29                                              Piceatannol also abrogated Ly49A-dependent inhibition of
30                                              Piceatannol also inhibited NF-kappaB activated by H(2)O(
31  and pharmacological evidence that, although piceatannol also inhibits alphaIIbbeta3 signaling, it do
32                                Additionally, piceatannol, an inhibitor of STAT3 activation, down-regu
33                                         Both piceatannol and a dominant negative behaving Stat3 adeno
34    When Syk phosphorylation was blocked with piceatannol and cells were similarly challenged, phospha
35 induced Syk phosphorylation was inhibited by piceatannol and dominant negative but not wild type Syk
36 study reveals an anti-adipogenic function of piceatannol and highlights IR and its downstream insulin
37 opy of products from the copolymerization of piceatannol and monolignols confirms the structures in t
38           During maturation, the contents in piceatannol and other stilbenes, ellagitannins, and flav
39                                              Piceatannol and pinostilbene showed activity comparable
40 with the selective small molecule inhibitors Piceatannol and PRT062607 markedly protected against tox
41  we studied the effect of the Syk inhibitors piceatannol and R406 on S1PR1 expression and function.
42 f lignin and implying that compounds such as piceatannol and resveratrol are potentially available in
43 lavonoid derivatives and stilbenes, as trans-piceatannol and resveratrol, as main secondary metabolit
44 ystilbenes, including the valuable compounds piceatannol and resveratrol.
45 rans-stilbene structure of the PTK inhibitor piceatannol and the cis-stilbene structure of the tubuli
46        Interestingly, two kinase inhibitors, Piceatannol and Tyrphostin 23, appeared to exert synthet
47 hesis was detected, whereas with oligomycin, piceatannol, and aurovertin (inhibitors of F(1)F(0)-ATP
48 he corresponding aglycones isorhapontigenin, piceatannol, and resveratrol, along with glucose, were r
49 inase inhibitor genistein, the Syk inhibitor piceatannol, and the RhoA inhibitor C3 exoenzyme had no
50  noncompetitive manner to ATP, through which piceatannol appears to inhibit adipogenesis.
51  crassa carotenoid oxygenase 1 (CAO1), using piceatannol as a substrate.
52 -coupled DHPs confirmed both resveratrol and piceatannol as authentic monomers participating in the o
53 n 3-glucoside and the stilbenes resveratrol, piceatannol, astringin and isorhapontin were discovered,
54  models may be due to lack of specificity of piceatannol, because this compound inhibited the activit
55 ere pretreated with the Syk kinase inhibitor Piceatannol before ligation, which abrogated the previou
56   Pretreatment of mDC with the Syk inhibitor piceatannol blocked B7-DC XAb-induced Ag uptake with a c
57                        The Syk/Zap inhibitor piceatannol blocked CD28, and TCR induced tyrosine phosp
58 n of Syk function by kinase-deficient Syk or piceatannol blocked target cell-induced PI3K, Rac1, PAK1
59                                              Piceatannol blocks Syk and ZAP-70, tyrosine kinases invo
60 ation of STAT1 and STAT3 is not inhibited by piceatannol but is sensitive to the Src kinase-specific
61                                      Whether piceatannol can also suppress NF-kappaB activation was i
62 tion in apparent catalytic efficiency toward piceatannol compared with the wildtype enzyme.
63                                              Piceatannol could be released from these lignins upon de
64                          We also showed that piceatannol directly binds to IR and inhibits IR kinase
65                                              Piceatannol dose-dependently inhibited differentiation m
66 a strong accumulation of E-resveratrol and E-piceatannol during the first six weeks of storage.
67 d gallic acids from pineapple and mango, and piceatannol from passion fruit.
68 , stilbene or rhaponticin (another analog of piceatannol) had no effect, suggesting the critical role
69 s (oligomycin, IC(50) approximately 1.8 muM; piceatannol, IC(50) approximately 1.05 muM; and angiosta
70 Here, we sought to determine the function of piceatannol in adipogenesis and elucidate the underlying
71 n of the development of obesity, the role of piceatannol in the development of adipose tissue and rel
72 tream insulin signaling as novel targets for piceatannol in the early phase of adipogenesis.
73                                              Piceatannol inhibited NF-kappaB in cells with deleted Sy
74                          Both wortmannin and piceatannol inhibited PI 3-kinase, but only piceatannol
75                                We found that piceatannol inhibited protein kinase Cdelta (PKCdelta) a
76                                 The stilbene piceatannol inhibited Syk preferentially over Lyn and ot
77  piceatannol inhibited PI 3-kinase, but only piceatannol inhibited Syk.
78 en examined for the mechanism, we found that piceatannol inhibited TNF-induced IkappaBalpha phosphory
79                                 We show that piceatannol inhibits adipogenesis of 3T3-L1 preadipocyte
80 rating the hydroxystilbenes, resveratrol and piceatannol, into monolignol polymerization in vitro, us
81                                              Piceatannol is an anti-inflammatory, immunomodulatory, a
82                     Moreover, we showed that piceatannol is an inhibitor of IR kinase activity and ph
83 grown sim fruits showed significantly higher piceatannol levels than sun-exposed fruits.
84 of kinase-deficient Syk or pretreatment with piceatannol markedly suppressed IL-2-stimulated activati
85 -glucoside) and, at lower levels, astringin (piceatannol-O-glucoside) and piceid (resveratrol-O-gluco
86 lue for ATP of 57.8 mum and a K(i) value for piceatannol of 28.9 mum.
87                       Inhibition of Syk with piceatannol or PI3K with wortmannin inhibited LPS-induce
88                                              Piceatannol+oxyresveratrol was tentatively identified in
89                                              Piceatannol, previously reported as a selective inhibito
90                                              Piceatannol, previously reported as a Syk/ZAP70-specific
91                          We examined whether piceatannol prolongs kidney allograft survival in the st
92            Similarly, the p72(syk) inhibitor piceatannol promoted PMN transmatrix migration, whereas
93 trol, dienestrol, hexestrol, oxyresveratrol, piceatannol, pterostilbene, and resveratrol-3-beta-gluco
94 atonin, chloroquine, imiquimod, resveratrol, piceatannol, quercetin, and other flavonoids.
95  the spleen tyrosine kinase family inhibitor piceatannol reduced their ability to migrate across the
96               Inhibiting Syk activation with piceatannol results in the inhibition of PIP(3) producti
97                                        trans-piceatannol showed particularly sensitive to temperature
98     Using a combination of the Syk inhibitor piceatannol, SILAC quantification, peptide fractionation
99 an Syk phosphorylation and is inhibited with piceatannol, suggesting that paxillin is a substrate for
100    The treatment of human myeloid cells with piceatannol suppressed TNF-induced DNA binding activity
101                             Furthermore, the piceatannol-suppressed mitotic clonal expansion was acco
102  TNF-induced Syk activation was abolished by piceatannol (Syk-selective inhibitor), which led to the
103                           A concentration of piceatannol that inhibited the phosphorylation and kinas
104 o prenylates pinosylvin to chiricanine A and piceatannol to arachidin-5, a prenylated stilbenoid iden
105           Both Syk null murine platelets and piceatannol-treated human platelets exhibited a partial,
106          In the early phase of adipogenesis, piceatannol-treated preadipocytes displayed a delayed ce
107                    Down-regulation of Syk by piceatannol treatment impaired NK cellular viability and
108                                 In contrast, piceatannol treatment of human platelets completely inhi
109 ortantly, we demonstrate that sensitivity to piceatannol/tyrphostin 23 epistatically relies on a HIF1
110                                  Ultimately, piceatannol was confirmed as a novel inhibitor of ADAMTS
111             This anti-adipogenic property of piceatannol was largely limited to the early event of ad
112                                The effect of piceatannol was not restricted to myeloid cells, as TNF-
113                                    At day 8, piceatannol was reduced to 10 mg/kg per day and the comb
114     These results are the first to show that piceatannol, when combined with subtherapeutic dosages o
115 d with the spleen tyrosine kinase inhibitor, piceatannol, which blocks 'outside-in' beta2 integrin si
116  reaction of resveratrol, pterostilbene, and piceatannol with 4-methylcatechol quinone and hydroxytyr
117                               In contrast to piceatannol, wortmannin did not inhibit PKCdelta and Raf
118 milar requirements and was also sensitive to piceatannol, wortmannin, and LY294002, indicating additi
119 nase) activity and Syk phosphorylation using piceatannol, wortmannin, and LY294002, inhibitors of PI

 
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