ライフサイエンスコーパス: pigment epithelium

1 retinal, 38% subretinal, and 36% sub-retinal pigment epithelium).

2 , separating photoreceptors from the retinal pigment epithelium.

3 lso throughout the retina and in the retinal pigment epithelium.

4 s degeneration of photoreceptors and retinal pigment epithelium.

5 nduced pluripotent stem cell-derived retinal pigment epithelium.

6 of bisretinoid formation within the retinal pigment epithelium.

7 dative stress in the choroid and the retinal pigment epithelium.

8 P < 10(-9)), even in areas of intact retinal pigment epithelium.

9 drusen, atrophy, and changes to the retinal pigment epithelium.

10 ased ICP, and perhaps changes in the retinal pigment epithelium.

11 with perturbed melanogenesis in the retinal pigment epithelium.

12 ppress consumption of glucose by the retinal pigment epithelium.

13 ith PVR, and reactive changes in the retinal pigment epithelium.

14 c nerve head pallor, and mottling of retinal pigment epithelium.

15 e interdigitation zone and an intact retinal pigment epithelium.

16 the cone outer segment closer to the retinal pigment epithelium.

17 ent cells, including melanocytes and retinal pigment epithelium.

18 nt progressive migration and loss of retinal pigment epithelium.

19 lls, and the basolateral side of the retinal pigment epithelium.

20 st, but only specifically around the retinal pigment epithelium.

21 nsport between the neural retina and retinal pigment epithelium.

22 eye stroma and epithelial cells from retinal pigment epithelium.

23 cative of pathological events in the retinal pigmented epithelium.

24 s against 27 genotoxic agents in the retinal pigment epithelium-1 (RPE1) cell line.

25 rtransmission, (2) disruption of the retinal pigment epithelium, (3) photoreceptor degeneration, and

26 otographs were graded for drusen and retinal pigment epithelium abnormalities and were evaluated for

27 (n = 37) in those 80 years or older, retinal pigment epithelium abnormalities from 4.1% (n = 85) to 7

28 Retinal pigment epithelium abnormalities, AVLs, neovascularizati

29 ler cells, endothelial cells, and in retinal pigment epithelium; agonism of PPARalpha with genetic or

30 The 2 biomarkers were RPE65 for retinal pigment epithelium and CD163 for histiocytes, each tagge

31 l time points and from the periphery, fovea, pigment epithelium and choroid of light-responsive adult

32 eration of POM cells surrounding the retinal pigment epithelium and decreases the expression of Foxc1

33 helium, iris sphincter pupillae muscle, iris pigment epithelium and dilator muscle complex, nonpigmen

34 n accumulate in the lysosomes of the retinal pigment epithelium and display cytotoxic effects.

35 on microscopy showed thinning of the retinal pigment epithelium and disruption of the external limiti

36 Quantifying preserved retinal pigment epithelium and EZ areas on FAF and OCT images, r

37 n export the lactate as fuel for the retinal pigment epithelium and for neighboring Muller glial cell

38 ultimodal imaging supports an intact retinal pigment epithelium and inner retina but an abnormal phot

39 cient to alter Fpn levels within the retinal pigment epithelium and Muller cells, but may limit iron

40 choriocapillaris, Bruch's membrane, retinal pigment epithelium and occasionally neurosensory retina.

41 e choriocapillaris in the absence of retinal pigment epithelium and outer retinal abnormalities suppo

42 ng 2 distinct criteria: (1) complete retinal pigment epithelium and outer retinal atrophy (cRORA) and

43 chieved a more rapid recovery of the retinal pigment epithelium and photoreceptor cells.

44 choriocapillaris, which supplies the retinal pigment epithelium and photoreceptors, and the ciliary b

45 onents of individual granules in the retinal pigment epithelium and present their analytical characte

46 tagged with different chromogens, yellow for pigment epithelium and purple for CD163-positive (CD163(

47 adelta T cells in protection against retinal pigment epithelium and retinal injury.

48 The normal retinal pigment epithelium and uveal melanocytes did not stain f

49 (dystrophies primarily involving the retinal pigment epithelium), and H35.50 (unspecified macular deg

50 n involving the neurosensory retina, retinal pigment epithelium, and choroid in 4 eyes (44%).

51 Total retinal, retinal pigment epithelium, and choroid layer thickness were sig

52 rees of atrophy of the outer retina, retinal pigment epithelium, and choroid, with outer retinal tubu

53 tive characteristics of the choroid, retinal pigment epithelium, and retina were compared between the

54 ely by promoting inflammation of the retinal pigment epithelium, and validate TLR2 as a novel therape

55 ty and genotoxicity studies in human retinal pigment epithelium (ARPE-19) cells.

56 The presence of histiocytoid pigment epithelium at the Bruch membrane probably also h

57 the small irregular elevation of the retinal pigment epithelium at the site of the quiescent CNV visu

58 athology revealed the following: (1) retinal pigment epithelium atrophy (with or without residual les

59 Retinal pigment epithelium atrophy and photoreceptor layer thinn

60 Retinal pigment epithelium atrophy and photoreceptor loss on OCT

61 Retinal pigment epithelium atrophy was delineated on the basis o

62 Retinal pigment epithelium-BM thickness, as measured by SD OCT s

63 ckening of the nerve fiber layer and retinal pigment epithelium-Bruch's membrane, with thinning in ot

64 er semiautomatic segmentation of the retinal pigment epithelium/Bruch membrane complex.

65 ge-related macular degeneration, the retinal pigment epithelium can be damaged by light acting on pho

66 ession of glucose consumption in the retinal pigment epithelium can increase the amount of glucose th

67 analysis of LC/MS data from a human retinal pigment epithelium cell line (ARPE-19) grown on normal a

68 ncluding Bruchs membrane thickening, retinal pigment epithelium cell loss, retinal functional deficit

69 erimeter (a measure of the number of retinal pigment epithelium cells exposed at the lesion border).

70 zation and a decrease in mesenchymal retinal pigment epithelium cells in alphaB-crystallin knockout m

71 Retinal pigment epithelium cells were in the centre, photorecept

72 ls revealed molecular resemblance to retinal-pigment epithelium cells, and anatomic analysis shows th

73 Finally, in primary human fetal retinal pigment epithelium cells, ligand binding to TLR2 induced

74 olar, horizontal, photoreceptor, and retinal pigment epithelium cells, thus exposing the anatomical s

75 toreceptor-specific markers in human retinal pigment epitheliums cells.

76 n visual system (retina, macula, and retinal pigment epithelium/choroid) reveals features of regulato

77 s therapeutic levels of sunitinib in retinal pigmented epithelium/choroid and retina for more than si

78 loss of pigment and thinning of the retinal pigment epithelium, choroidal thinning, undifferentiated

79 resembling congenital hypertrophy of retinal pigment epithelium (CHRPE) lesions.

80 combined hamartoma of the retina and retinal pigment epithelium (CHRRPE) involving the macula.

81 io (3.8%), vessel tortuosity (2.5%), retinal pigment epithelium degeneration (2.5%), myelinated nerve

82 Retinal pigment epithelium degeneration followed by retinal and

83 , collected from outside the zone of retinal pigment epithelium degeneration) were evaluated using Ul

84 correlating with different stages of retinal pigment epithelium degeneration.

85 tudy, we tested the effect of treatment with pigment epithelium-derived factor (PEDF) in combination

86 Pigment epithelium-derived factor (PEDF) is a multifunct

87 a1 transcription factor and are dependent on pigment epithelium-derived factor (PEDF) on the outer su

88 ation of the known neuroprotective molecule, pigment epithelium-derived factor (PEDF) plus docosahexa

89 We have previously demonstrated that pigment epithelium-derived factor (PEDF) plus docosahexa

90 One of these molecules, pigment epithelium-derived factor (PEDF), is a broadly e

91 Null mutations in for pigment epithelium-derived factor (PEDF), the protein pr

92 elial growth factor while increasing that of pigment epithelium-derived factor in diabetic retinas.

93 Pigment epithelium-derived factor restoration increases

94 Inhibitor, Clade F) gene which encodes PEDF (pigment epithelium-derived factor), a potent inhibitor o

95 Drusenoid pigment epithelium detachment (DPED) is a known precurso

96 ), hyperreflective foci (HRF), fibrovascular pigment epithelium detachment (fvPED), and serous PED (s

97 The majority (n = 4) showed a drusenoid pigment epithelium detachment (PED) preceding the lesion

98 ent epithelium (RPE) layer in serous retinal pigment epithelium detachment (PED) with multi-contrast

99 er-reflective intraretinal foci; dome-shaped pigment epithelium detachment (PED); hyper-reflective fl

100 h nonneovascular DPED and 2 with neovascular pigment epithelium detachment [PED]) and 49 eyes of 33 c

101 worse vision, presence of atrophy/fibrosis, pigment epithelium detachment, and geographic atrophy/fi

102 atients with chronic CSCR and flat irregular pigment epithelium detachments (FIPEDs).

103 ifferent retinal layers, such as the retinal pigment epithelium-drusen complex (RPEDC), were assessed

104 ad thickening of Bruchs membrane and retinal pigment epithelium dysfunction.

105 typical macular cherry-red spot with retinal pigment epithelium dystrophy in the middle periphery, in

106 the automatically provided boundary "retinal pigment epithelium fit" positioned at the level of Bruch

107 , more subretinal fluid, and less subretinal pigment epithelium fluid (all P < 0.01).

108 eye, hemorrhage, and absence of sub-retinal pigment epithelium fluid at baseline were associated wit

109 achment, intraretinal fluid, and sub-retinal pigment epithelium fluid were predictive of FS at that l

110 nal fluid, subretinal fluid, and sub-retinal pigment epithelium fluid.

111 of intraretinal, subretinal, and subretinal pigment epithelium fluid; thickness at the foveal center

112 as associated with protection of the retinal pigmented epithelium from damage caused by disease.

113 NIR-AF over the island of surviving retinal pigment epithelium: Group 1 (preserved NIR-AF centrally)

114 Retinal pigmented epithelium has plentiful melanosomes, signifyi

115 human induced pluripotent stem cell-retinal pigment epithelium (hiPSC-RPE) derived from patients wit

116 An in vitro model of human retinal pigment epithelium (HRPEsv) cells was treated with diffe

117 ages in 2 (17%) and 3 (18%); loss of retinal pigment epithelium in 1 (8%) and 4 (24%); and drusen in

118 ed at a variable distance before the retinal pigment epithelium in 56.67% of eyes.

119 and hyperreflectivity underlying the retinal pigment epithelium in 9 eyes (100%), retinal thinning in

120 were correlated with changes in the retinal pigment epithelium in eyes with age-related macular dege

121 TLR2 was robustly expressed by the retinal pigment epithelium in mouse and human eyes, both normal

122 incidence of atrophic lesions of the retinal pigment epithelium in patients with Stargardt disease as

123 eatures like microvasculature in a brain and pigmented epithelium in an eye field.

124 showed a hyporeflective band anterior to the pigment epithelium indicating the presence of dysfunctio

125 layer thickness: inner nuclear layer-retinal pigment epithelium (INL-RPE) and the specific sublayers

126 nduced pluripotent stem cell-derived retinal pigment epithelium (iPSC-RPE) to test the potential of g

127 several treatments available, if the retinal pigment epithelium is damaged, we have to cope with the

128 hologies arranged in layers, forming retinal pigment epithelium, is a synapomorphy of vertebrates.

129 OS); and inner segment outer segment-retinal pigment epithelium (ISOS-RPE).

130 ssive geographic atrophy (GA) of the retinal pigment epithelium leads to loss of central vision.

131 A loss of the choriocapillaris/retinal pigment epithelium left a "window-defect", where the ves

132 ly coupled, the results suggest that retinal pigment epithelium loss is more extensive than photorece

133 nsmission of light into the choroid, retinal pigment epithelium loss, and loss of outer retinal layer

134 peripheral monocyte infiltration and retinal pigment epithelium migration, and their depletion result

135 Mild disease presented with retinal pigment epithelium mottling, a patchy pattern of hypoaut

136 TLR2 signaling, was detected in the retinal pigment epithelium of human eyes, particularly in eyes w

137 erreflective foci represent cells of retinal pigment epithelium origin that are similar to those foun

138 ischemic infarction of the choroid, retinal pigment epithelium, outer part of the retina and the opt

139 vein, congenital hypertrophy of the retinal pigment epithelium, pars plana, ora serrata pearl, typic

140 receptor layer (PRL) outer segments, retinal pigment epithelium plus drusen (RPE+drusen) complex, and

141 gical function of photoreceptors and retinal pigment epithelium requires precise regulation to mainta

142 Interestingly, the retinal pigment epithelium, responsible for normal phagocytosis

143 notypes, including the occurrence of retinal pigment epithelium (RPE) abnormalities, choroidal neovas

144 e epigenetic plasticity of amphibian retinal pigment epithelium (RPE) allows them to regenerate the e

145 Frank atrophy of the retinal pigment epithelium (RPE) and a neovascular complex were

146 hat begins around the age of 30 with retinal pigment epithelium (RPE) and Bruch's membrane changes re

147 hyperreflective deposits between the retinal pigment epithelium (RPE) and Bruch's membrane on SD-OCT,

148 The human retinal pigment epithelium (RPE) and choroid are complex tissues

149 increased deposition of C5b-9 in the retinal pigment epithelium (RPE) and choroid.

150 remains elusive, dysfunction of the retinal pigment epithelium (RPE) and dysregulation of complement

151 ents is an important function of the retinal pigment epithelium (RPE) and it is essential for retinal

152 ophin1 (BEST1) is expressed in human retinal pigment epithelium (RPE) and mutations in the BEST1 gene

153 n support functions performed by the retinal pigment epithelium (RPE) and on oxygen and nutrients del

154 disruption), components of complete retinal pigment epithelium (RPE) and outer retinal atrophy (e.g.

155 ted Abca4(-/-) mouse model presented retinal pigment epithelium (RPE) and photoreceptor degeneration

156 by neighboring epithelial cells, the retinal pigment epithelium (RPE) and podocytes, respectively.

157 AMD) is a progressive disease of the retinal pigment epithelium (RPE) and the retina leading to loss

158 f cone photopigments may require the retinal pigment epithelium (RPE) and/or retinal Muller glia.

159 n the morphology and function of the retinal pigment epithelium (RPE) are common features shared by m

160 outer boundaries of the choroid and retinal pigment epithelium (RPE) as well as the inner retinal su

161 etinal disruption and atrophy of the retinal pigment epithelium (RPE) associated with ORT on spectral

162 toreceptor atrophy can occur without retinal pigment epithelium (RPE) atrophy and that atrophy can un

163 horiocapillaris in correspondence of retinal pigment epithelium (RPE) atrophy in 80% (n = 16) of case

164 ected visual acuity (BCVA), age, and retinal pigment epithelium (RPE) atrophy were recorded and corre

165 ltimodal imaging and ocular history: retinal pigment epithelium (RPE) atrophy with treatment-naive qu

166 s localized to the apical aspects of retinal pigment epithelium (RPE) cells and contributes to a dela

167 lls, mitochondria, Muller cells, and retinal pigment epithelium (RPE) cells and were visualized using

168 Retinal pigment epithelium (RPE) cells are cultured on top of cu

169 te the intraretinal migration of the retinal pigment epithelium (RPE) cells in age-related macular de

170 Cholesterol accumulation beneath the retinal pigment epithelium (RPE) cells is supposed to contribute

171 s cadherin-3, a protein expressed in retinal pigment epithelium (RPE) cells that may have a key role

172 In human retinal pigment epithelium (RPE) cells, the primary site for the

173 culture media was incorporated into retinal pigment epithelium (RPE) cells.

174 method of subretinal fluid drainage, retinal pigment epithelium (RPE) changes, and choroidal thicknes

175 V4 expression in the endothelium and retinal pigment epithelium (RPE) components of the BRB, and that

176 ieu in wild-type mouse, we triggered retinal pigment epithelium (RPE) damage/PRC death by subretinall

177 ar degeneration and atypical central retinal pigment epithelium (RPE) defects not attributable to geo

178 ophy, characterised by extensive sub-retinal pigment epithelium (RPE) deposits, RPE atrophy, choroida

179 ry cohort of 4 eyes revealed that in retinal pigment epithelium (RPE) elevations with a greatest tran

180 d subretinal fluid (SRF), 36% had subretinal pigment epithelium (RPE) fluid, and 66% had subretinal h

181 d (IRF), subretinal fluid (SRF), sub-retinal pigment epithelium (RPE) fluid, and subretinal tissue co

182 d pluripotent stem cells to generate retinal pigment epithelium (RPE) from an individual suffering fr

183 in three-dimensional optic cups and retinal pigment epithelium (RPE) from iPSCs with this common CEP

184 mRNA levels in both neuroretina and retinal pigment epithelium (RPE) from mouse and baboon over a se

185 CM) microenvironment surrounding the retinal pigment epithelium (RPE) has been implicated in the etio

186 To report on the presence of retinal pigment epithelium (RPE) humps in high myopia, and to de

187 spectral-domain OCT with respect to retinal pigment epithelium (RPE) in 836 spectral-domain OCT slic

188 ofiles specifically localized to the retinal pigment epithelium (RPE) in Abca4 (-/-) Stargardt model

189 ascular/neural network in DR and the retinal pigment epithelium (RPE) in AMD.

190 growth and maturation phases of the Retinal Pigment Epithelium (RPE) in-vitro at the cell layer leve

191 Daily, the retinal pigment epithelium (RPE) ingests a bolus of lipid and pr

192 While AMD histopathology involves retinal pigment epithelium (RPE) injury associated with immune c

193 The retinal pigment epithelium (RPE) is a highly polarized epithelia

194 The retinal pigment epithelium (RPE) is a key site of injury in inhe

195 The retinal pigment epithelium (RPE) is a monolayer of cobblestone-l

196 The retinal pigment epithelium (RPE) is a monolayer of pigmented cel

197 The retinal pigment epithelium (RPE) is a particularly vulnerable ti

198 ce between the neural retina and the retinal pigment epithelium (RPE) is critical for several process

199 Lipofuscin in the retinal pigment epithelium (RPE) is the major source of fundus a

200 term natural history of the residual retinal pigment epithelium (RPE) is unclear, with reported RPE a

201 0.03) were: hyperreflective foci and retinal pigment epithelium (RPE) layer atrophy or absence, follo

202 was to evaluate focal damage in the retinal pigment epithelium (RPE) layer in serous retinal pigment

203 ated from the SD-OCT and the area of retinal pigment epithelium (RPE) loss from the FAF.

204 ase 1/2 (ERK1/2) is increased in the retinal pigment epithelium (RPE) of age-related macular degenera

205 ical phenotypes were observed in the retinal pigment epithelium (RPE) of individuals with butterfly-s

206 at autophagy is dysfunctional in the retinal pigment epithelium (RPE) of the AMD donor eyes (AMD RPE)

207 P3 inflammasome activation mainly in retinal pigment epithelium (RPE) or rather in non-RPE cells prom

208 D, estimate of coverage by different retinal pigment epithelium (RPE) phenotypes in the DPED surface;

209 increased activation of Rap1a in the retinal pigment epithelium (RPE) reduces oxidative signaling to

210 height of 45.3 (36.1) mum above the retinal pigment epithelium (RPE) reference plane that was preset

211 e measurements of vitreous (VIT) and retinal pigment epithelium (RPE) signal intensities, which were

212 ere obtained from en face SS-OCT sub-retinal pigment epithelium (RPE) slab images.

213 mpact of miR-204 loss on retinal and retinal pigment epithelium (RPE) structure and function.

214 To investigate when retinal pigment epithelium (RPE) tears occur and their associate

215 including central macular mean EZ to retinal pigment epithelium (RPE) thickness (2q4: 26.6 mum to 31.

216 CT measurement parameters, including retinal pigment epithelium (RPE) thickness, central macular thic

217 obust gene editing in vivo in murine retinal pigment epithelium (RPE) tissue and skeletal muscle afte

218 ent genome editing in vivo in murine retinal pigment epithelium (RPE) tissue via subretinal injection

219 tifying potassium ion channel in the retinal pigment epithelium (RPE) to maintain ionic homeostasis a

220 transport is a major function of the retinal pigment epithelium (RPE) to support the neural retina.

221 biologically and genetically defined retinal pigment epithelium (RPE) to the diseased human eye.

222 Retinectomies expose the retinal pigment epithelium (RPE) to the vitreous cavity; the dir

223 onstrated no adverse perturbation of retinal pigment epithelium (RPE) transcriptional programs in any

224 ctron microscopy of the obelix(td15) retinal pigment epithelium (RPE) uncovered reduced phagosome cle

225 s in regions with normally appearing retinal pigment epithelium (RPE) were the loss of the POS and el

226 Proteins in the retinal pigment epithelium (RPE), a cell layer adjacent to the p

227 cular degeneration (AMD) affects the retinal pigment epithelium (RPE), a cell monolayer essential for

228 retinal projection through SHRM onto retinal pigment epithelium (RPE), and (4) masking of choriocapil

229 ) channel predominantly expressed in retinal pigment epithelium (RPE), and over 250 genetic mutations

230 nner and outer segments (IS/OS), the retinal pigment epithelium (RPE), and the choriocapillaris (CC)

231 sulting from loss of photoreceptors, retinal pigment epithelium (RPE), and underlying choriocapillari

232 eceptor support system involving the retinal pigment epithelium (RPE), Bruch's membrane, and the chor

233 isomerization occurs in the adjacent retinal pigment epithelium (RPE), but because ipRGCs are far fro

234 haracterized pathway in cells of the retinal pigment epithelium (RPE), cone visual pigments are thoug

235 ound in the photoreceptor-supporting retinal pigment epithelium (RPE), especially in a zone correspon

236 ordinated terminal maturation of the retinal pigment epithelium (RPE), fenestrated choroid endothelia

237 n accumulation of material below the retinal pigment epithelium (RPE), have long been established as

238 nal hyperreflective material (SHRM), retinal pigment epithelium (RPE), hyperreflective foci (HRF), fi

239 ontaining lipofuscin pigments in the retinal pigment epithelium (RPE), increased oxidative stress, au

240 nt membrane-associate protein in the retinal pigment epithelium (RPE), is a key retinoid isomerase of

241 eta signaling in the entire eye, the retinal pigment epithelium (RPE), or the vascular endothelium.

242 On B-scans, the choroid, retinal pigment epithelium (RPE), photoreceptor (PR) layer, and

243 in red cone photoreceptors, while in retinal pigment epithelium (RPE), TH regulates expression of a c

244 photoreceptor layers, despite intact retinal pigment epithelium (RPE), to approximately 70% of baseli

245 The retinal pigment epithelium (RPE), which lies between the retina

246 met by Muller glia and cells of the retinal pigment epithelium (RPE), which provide essential metabo

247 of ocular and systemic factors with retinal pigment epithelium (RPE)-Bruch's membrane (BM) complex t

248 The volumes of retinal pigment epithelium (RPE)-drusen complex, RPE-drusen comp

249 P = .001; SE -2.27 D [SD 4.65]), and retinal pigment epithelium (RPE)-related dystrophies (OR low myo

250 A nonvisual opsin known as retinal pigment epithelium (RPE)-retinal G-protein-coupled recep

251 ns of matured photoreceptors and the retinal pigment epithelium (RPE).

252 oper interaction with the underlying retinal pigment epithelium (RPE).

253 model of chronic degeneration of the retinal pigment epithelium (RPE).

254 na, the ciliary margin (CM), and the retinal pigment epithelium (RPE).

255 lial mesenchymal transition (EMT) of retinal pigment epithelium (RPE).

256 ulate the phagocytosis of OSs by the retinal pigment epithelium (RPE).

257 s, especially the involvement of the retinal pigment epithelium (RPE).

258 ith Ca(V) 1.3 Ca(2+) channels in the retinal pigment epithelium (RPE).

259 were higher in neural retina than in retinal pigment epithelium (RPE).

260 ptor outer segments (POS) within the retinal pigment epithelium (RPE).

261 and mild transient thickening of the retinal pigment epithelium (RPE)/Bruch's complex (Bc).

262 y IL-1beta was markedly increased in retinal pigment epithelium (RPE)/choroid and positively correlat

263 egulated manner in chicken embryonic retinal pigment epithelium (RPE)/choroid in the absence of light

264 y penetrates the retina to reach the retinal pigment epithelium (RPE)/choroid with minimal subsequent

265 ative regulator of the ER-associated retinal pigment epithelium (RPE)65 isomerase necessary for recyc

266 toreceptor zone, ellipsoid zone, and retinal pigment epithelium (RPE, P < 0.001 and P = 0.005-0.045,

267 th majority of the expression in the retinal pigmented epithelium (RPE) and limited expression in the

268 nd accumulation of lipofuscin in the retinal-pigmented epithelium (RPE) and of lipoproteins at the Br

269 but delivery of viral vectors to the retinal pigmented epithelium (RPE) and retina can be challenging

270 py revealed focal alterations in the retinal pigmented epithelium (RPE) and yellow retinal dots corre

271 acular degeneration characterized by retinal pigmented epithelium (RPE) death; the RPE also exhibits

272 The retinal pigmented epithelium (RPE) forms the outer blood-retinal

273 he major biological functions of the retinal pigmented epithelium (RPE) is the clearance of shed phot

274 we delivered a wild-type Mfrp to the retinal pigmented epithelium (RPE) of Mfrp (rd6) /Mfrp (rd6) mic

275 tem cells for neural retina (NR) and retinal pigmented epithelium (RPE) of the teleost medaka (Oryzia

276 nt eyes show substantially increased retinal pigmented epithelium (RPE) proliferation in the fissure

277 consisting of vision loss, increased retinal pigmented epithelium (RPE) stress, and increased basal l

278 abolism involving dysfunction of the retinal pigmented epithelium (RPE) underlies the pathogenesis of

279 evealed predominant up-regulation of retinal pigmented epithelium (RPE)-specific genes associated wit

280 reactive oxygen species (ROS) in the retinal pigmented epithelium (RPE).

281 FH is expressed in the retinal pigmented epithelium (RPE).

282 ocessing enzyme DICER1 in the mature retinal pigmented epithelium (RPE).

283 ed phagocytes: Sertoli cells and the retinal pigmented epithelium (RPE).

284 sign (separation of hyperreflective retinal pigment epithelium [RPE] from Bruch's membrane, with the

285 /=196350 mum2) and depigmentation of retinal pigment epithelium (slope of -19.17 for the NEI-VFQ-25 c

286 In normal visual physiology, the pigment epithelium supports photoreceptors and participa

287 onocytes, together with RPE65 in the retinal pigment epithelium, supports differentiation toward hist

288 Retinal pigment epithelium tears act differently depending on wh

289 dy events (atrophy status, fibrosis, retinal pigment epithelium tears).

290 ding to a loss of photoreceptors and retinal pigment epithelium that manifests clinically as pigmenta

291 Loss of retinal pigment epithelium, the presence of a thin choroid, a pe

292 utant lacks pigmentation also in the retinal pigment epithelium, therefore enabling optical access to

293 e choroidal blood passes through the retinal pigment epithelium to the retina where photoreceptors co

294 cement of the temporal peripapillary retinal pigment epithelium (tRPE) from its position in central g

295 Retinal pigment epithelium undulations and vascular dilations at

296 er layer, outer plexiform layer, and retinal pigmented epithelium using image guidance and segmented

297 Damage of the pigment epithelium was also confirmed.

298 al soft drusen and depigmentation of retinal pigment epithelium was associated with substantially low

299 lete atrophy of the outer retina and retinal pigment epithelium were observed in both split-detection

300 ternal limiting membrane and loss of retinal pigment epithelium with hypertransmission of OCT signal