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1 ates (six cynomolgus, five rhesus, and three pigtail macaques).
2 Zika virus (ZIKV) inoculation in a pregnant pigtail macaque.
3 dose that is required for engraftment in the pigtail macaque.
4 udy rhesus macaques to also be used to study pigtail macaques.
5 owed us to expand the host range of HIV-1 to pigtail macaques.
6 gus monkey isolates, and (iii) isolates from pigtail macaques.
7 intrarectal SIV(mac251) challenge in outbred pigtail macaques.
8 and produced an acute disease in rhesus and pigtail macaques.
9 te vaccines with tattoo ink in both mice and pigtail macaques.
10 l model of HIV-1 infection, SIV infection of pigtailed macaques.
11 mutation K165R in HAART-treated SIV-infected pigtailed macaques.
12 d AIDS following experimental inoculation of pigtailed macaques.
13 ivity with NK cells from rhesus macaques and pigtailed macaques.
14 roducibly induced a rapidly fatal disease in pigtailed macaques.
15 membrane and envelope proteins (VSV-KFDV) in pigtailed macaques.
16 ens (66%), 30 of 40 rhesus macaques, 5 of 11 pigtail macaques, 2 of 4 sooty mangabeys, and 0 of 1 chi
17 modify viral and host cell genetics to make pigtail macaques a suitable, clinically relevant model f
20 ned, expressed, and analyzed rhesus macaque, pigtailed macaque, and murine DC-SIGN and made a panel o
21 udy defines sites of immune escape in SIV in pigtailed macaques, and this enables a more refined leve
24 sistance to the host cell inhibitor Mx2 from pigtail macaques, but that complete resistance is associ
29 transcriptase inhibitor tenofovir protected pigtailed macaques depending on the timing of viral chal
30 dent functions) for their ability to protect pigtail macaques from an i.v. high-dose cell-associated
33 elay viral infection in rotavirus-challenged pigtailed macaques has important implications for the de
34 the C-C chemokine receptor 5 (CCR5) locus in pigtailed macaque HSPCs by zinc finger nucleases (ZFNs)
36 on in vivo, was enhanced in lymph nodes from pigtailed macaques infected with simian immunodeficiency
38 macrophage infection in vivo, we inoculated pigtailed macaques intravenously or intrarectally with t
40 peutics in an appropriate model, such as the pigtail macaque (Macaca nemestrina; Mn), is crucial.
41 hed conditions for efficient transduction of pigtailed macaque (Macaca nemestrina) long-term repopula
42 pic virus type I/II (STLV-I/II) seronegative pigtailed macaque (Macaca nemestrina) with a cutaneous T
43 iency virus (SIV) model in which over 90% of pigtailed macaques (Macaca nemestrina) coinoculated with
44 escribe a comprehensive analysis of two male pigtailed macaques (Macaca nemestrina) experimentally in
48 lyzed the renal pathology and function of 27 pigtailed macaques (Macaca nemestrina), infected intrave
49 ut' experiments on a large, captive group of pigtailed macaques (Macaca nemestrina), we show that a p
51 ee dominance-related signal used by monkeys (pigtailed macaques: Macaca nemestrina) means submission
57 ociated neurologic disorders, we used an SIV pigtailed macaque model to study innate immune responses
58 cterization of NKG2A and NKp80 in rhesus and pigtailed macaque NK cells provides a new approach in th
59 pheral blood were isolated from SIV-infected pigtailed macaques on days 4, 14, and 114 postinoculatio
61 of TRIMCyp has, in fact, occurred twice, and pigtailed macaques (pgt) express an independently genera
62 Among nonhuman primates, SIV-infected Asian pigtailed macaques (PM) are relatively more susceptible
63 XMRV replication and spread were limited in pigtailed macaques, predominantly by APOBEC-mediated hyp
67 rican green monkey (AGM), to a new host, the pigtailed macaque (PTM), viral adaptation and increased
68 d, lymph nodes, and intestine collected from pigtailed macaques (PTMs) and African green monkeys (AGM
69 n in pathogenic SIV infections of rhesus and pigtailed macaques (PTMs) and in nonpathogenic SIV infec
70 exposed adult and juvenile AGMs, as well as pigtailed macaques (PTMs) as a nonnatural host control,
74 ve neuropathologic review of 30 SIV-infected pigtailed macaques receiving combination antiretroviral
80 from simian immunodeficiency virus-infected pigtailed macaques showed an increase in YKL-40 concentr
83 D169 was observed in lymph nodes of infected pigtailed macaques, suggesting productive infection of C
84 ophage infection in blood and lymph nodes of pigtailed macaques that did or did not develop simian im
85 ransmission, we passaged a modified HIV-1 in pigtailed macaques that were transiently depleted of CD8
89 uence diversity in 44 MHC-typed SIV-infected pigtail macaques, we defined over 70 sites within SIV wh
97 p CNS disease by 3 months after inoculation, pigtailed macaques were treated with a regimen of tenofo
98 GI tract physiology, we treated SIV-infected pigtail macaques with ARVs, probiotics, and prebiotics o
100 ive immune response following vaccination of pigtailed macaques with envelope protein(s) derived from
101 rated that treatment of acutely SIV-infected pigtailed macaques with the drug sevelamer, which binds