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1 showed reduced binding (64 to 87% of that of piliated AAr176) to 16HBE14o(-) and ME180 cells, hifA an
2 transposon insertions in these ORFs were non-piliated and failed to express pilus-associated phenotyp
3 eraged in larger populations exposed to both piliated and non-piliated pathogens, presumably via the
4  events predominated, which differed between piliated and non-piliated progeny.
5 l in situ structure of a T4bP machine in its piliated and non-piliated states.
6 ng of biofilm development differ between the piliated and non-piliated strains.
7                 T1P are phase variable (both piliated and nonpiliated bacteria exist in a clonal popu
8 D66c) were detected 4 h after infection with piliated and nonpiliated gonococci.
9 6 was induced equally by the interactions of piliated and nonpiliated meningococci, whereas lipopolys
10 pilus machine, or T4PM) in situ, in both the piliated and nonpiliated states, at a resolution of 3 to
11  optimized to each other, and that they give piliated bacteria significant advantages in rapidly chan
12 vors of RecA expression are enriched for non-piliated bacteria that carry large deletions of the pilE
13 a general feature of FimA proteins of type 1-piliated bacteria.
14  pilus expression and independently of fully piliated bacteria.
15 des a protein found in the outer membrane of piliated bacteria; and cooB.
16  shown that pilT and pilU mutants, which are piliated but defective in twitching motility, display re
17                                              Piliated but not nonpiliated gonococci adhered to cells
18                                              Piliated, but not nonpiliated, N. gonorrhoeae strain F62
19  protein shown previously to localize to the piliated cell pole before and during pilus assembly, con
20 nly one pole that most likely identifies the piliated cell pole.
21                                 We show that piliated cells mostly contain a single pilus that is not
22 In addition, the phage were unable to infect piliated cells overexpressing MS2 coat protein, a resist
23  phase variation, reducing the proportion of piliated cells, reduced mannose binding 8-fold, and decr
24               The interaction between type 1 piliated E. coli and bladder epithelial cells leads to t
25                        Infection with type 1-piliated E. coli can trigger a number of host responses,
26 cultures compared to infection with a type 1 piliated E. coli K-12 strain.
27 sults demonstrate that the binding of type 1-piliated E. coli to vaginal epithelial cells correlates
28  uroepithelial cells are activated by type 1 piliated E. coli.
29 hat CooC is an outer membrane protein of CS1-piliated E. coli.
30    Acute UTIs are typically caused by type 1-piliated Escherichia coli and result in urothelial apopt
31 lls, and human tracheal explants with type 1-piliated Escherichia coli mediated a marked (25-50-fold)
32                          Adherence of type 1-piliated Escherichia coli to carbohydrate structures of
33 lex cell cycle involving sessile-stalked and piliated, flagellated swarmer cells.
34 zation approach can reduce interactions of a piliated form of this opportunistic pathogen with respir
35 sponse was limited to cultures infected with piliated gonococci and was more vigorous in the endocerv
36 ilus gene cluster had decreased adherence of piliated H. influenzae to mucins, and Fab fragments of a
37 ng NF-kappaB activity increased the level of piliated HB101-induced apoptosis to the level of apoptos
38                                              Piliated hemolytic Moraxella bovis is recognized as the
39 HifD and HifE proteins, respectively, of all piliated Hib and NTHI strains tested.
40 fD or HifE of strain Eagan also bound to all piliated Hib strains but did not bind to the piliated NT
41 d most of the mature protein reacted more to piliated Hib than to nonpiliated Hib or to a mutant cont
42                                          The piliated isolates formed either spreading/corroding or n
43                            Furthermore, such piliated L. lactis cells evoked a higher TNF-alpha respo
44                                              Piliated MS11Deltaopa cells formed dispersed microcoloni
45 quired for biofilm formation as evident by a piliated mutant of S. elongatus that develops biofilms.
46                     Intriguingly, one of the piliated mutants that does not exhibit spreading retains
47                                Attachment of piliated Neisseria gonorrhoeae or Neisseria meningitidis
48            Expression of the ChoP epitope on piliated neisseriae displayed phase variation, both link
49 predator and prey; contact is always via the piliated, non-flagellar pole of the predator, involving
50 pproximately 5000 sialylated or unsialylated piliated, non-opaque (P+Opa-, transparent) colony type g
51 piliated Hib strains but did not bind to the piliated NTHI strains.
52 was induced within 3 h of exposure to type 1 piliated NU14 and was dependent upon interactions mediat
53 s which were visually distinct from those of piliated Opa-expressing MS11 cells.
54 earrangements reported here are triggered by piliated, Opa- and Opc- strains and also by nonpiliated
55 ype of the inoculation strain to the P+Opa+ (piliated, opaque) phenotype 12-60 h before onset of dise
56 ildren whose nasopharynges were colonized by piliated organisms, the corresponding middle ear isolate
57                         This assay employs a piliated parental Gc variant with a recA allele whose pr
58 populations exposed to both piliated and non-piliated pathogens, presumably via the exchange of immun
59 s in epithelial cells after uptake of type 1-piliated pathogens.
60                                              Piliated PilT mutants and a panel of pilus assembly muta
61 as observed to localize predominantly to the piliated pole.
62 ces susceptibility to T6SS compared to a non-piliated prey, by preventing segregation from a T6SS-wie
63 analysis of piliated variants arising from a piliated progenitor.
64 quencing the pilE locus in randomly selected piliated progeny of both MS11 and FA1090 in recB and rec
65 ted, which differed between piliated and non-piliated progeny.
66  small percentage of pneumococci appeared as piliated, RrgA-expressing, DivIVA-negative single cocci,
67                            In peritrichously piliated species, it is unclear how multiple pili are co
68 armer cell with several polar pili and a non-piliated stalked cell.
69 re of a T4bP machine in its piliated and non-piliated states.
70  slow in vitro, it is rapid in vivo as a non-piliated strain lacking the other fim recombinases rapid
71   Using microarray analysis, we found that a piliated strain showed increased expression of the gene
72 s higher growth rate compared to that of the piliated strain.
73 sed protection against challenge by the four piliated strains that we have examined (PAK, PAO, KB7 an
74  CooD minor pilin (when overexpressed in CS1-piliated strains) was detected in periplasmic intermolec
75 IgA from prior episodes of colonization with piliated strains.
76 elopment differ between the piliated and non-piliated strains.
77 in found exclusively in the periplasm of CS1-piliated strains.
78 n involving the irreversible transition from piliated to nonpiliated variants.
79 n involving the irreversible transition from piliated to nonpiliated variants.
80                     Here we show that type 1-piliated uropathogens can invade the superficial epithel
81 roviding the first comprehensive analysis of piliated variants arising from a piliated progenitor.
82                             On solid medium, piliated variants form small (S-phase), corroding coloni
83 tory and nonendocarditis blood isolates were piliated, while the majority of joint fluid, bone, and e