ライフサイエンスコーパス: pilocytic

1 re needed to identify genetic alterations in pilocytic and low-grade astrocytomas, which account for

2 nges that drive the initiation and growth of pilocytic and other low-grade astrocytomas beyond the as

3 work should focus on elucidating features of pilocytic astocytomas that will identify prospectively c

4 The most common histology was pilocytic astrocytoma (46.3%).

5 Juvenile pilocytic astrocytoma (JPA) is one of the most common br

6 endymal giant cell astrocytomas (n = 4) or a pilocytic astrocytoma (n = 1) were treated with oral rap

7 e two major genomic alterations in pediatric pilocytic astrocytoma (PA) are NF1 loss and KIAA1549:BRA

8 The pilocytic astrocytoma (PA) is the most common childhood

9 Pilocytic astrocytoma (PA) is the most common glial cell

10 ncing (WGS) analysis of three NF1-associated pilocytic astrocytoma (PA) tumors.

11 The initial study cohort consisted of 7 pilocytic astrocytoma (PA), 19 ependymoma (EPN), 5 gliob

12 medulloblastoma (MB), which is malignant, to pilocytic astrocytoma (PA), a primarily benign tumor, an

13 ignated MG-Act in BRAF-fused, MAPK-activated pilocytic astrocytoma (PA), but not in high-grade glioma

14 Pilocytic astrocytoma (PA), the most common childhood br

15 of the most common brain tumor in children (pilocytic astrocytoma [PA]), we identify glutamatergic p

16 Thirty-five samples of pilocytic astrocytoma and 10 control samples of cerebell

17 on a 4-yr-old male with a diagnosis of acute pilocytic astrocytoma and global cerebral hypoxic ischem

18 nses were observed (one in thalamic juvenile pilocytic astrocytoma and one in optic pathway glioma) a

19 iffuse glioma showed molecular similarity to pilocytic astrocytoma and relatively favorable survival.

20 o differentiate medulloblastoma cancers from pilocytic astrocytoma and two molecular subtypes of epen

21 a case of a patient diagnosed with a grade 1 pilocytic astrocytoma at the age of 2 years, approximate

22 how that glutamate promotes proliferation of pilocytic astrocytoma cells.

23 ive in recurrent, refractory, or progressive pilocytic astrocytoma harbouring common BRAF aberrations

24 tratum 1 comprised patients with WHO grade I pilocytic astrocytoma harbouring either one of the two m

25 Pilocytic astrocytoma is the most common glioma in child

26 cells, and PFA/PFB ependymoma and cerebellar pilocytic astrocytoma resemble the prenatal gliogenic pr

27 ic nerve gliomas and in human NF1-associated pilocytic astrocytoma tumors.

28 ave measurable non-enhancing disease (except pilocytic astrocytoma) at baseline using the Response As

29 er, uterine, urothelial carcinoma, recurrent pilocytic astrocytoma, and non-small-cell lung cancer (e

30 y, EBV was the most frequent HHV detected in pilocytic astrocytoma, but at very low levels.

31 rs including oligodendroglioma, astrocytoma, pilocytic astrocytoma, oligoastrocytoma, anaplastic astr

32 ths after a subtotal excision of a brainstem pilocytic astrocytoma.

33 considered responsible for tumorigenesis of pilocytic astrocytoma.

34 multiforme (GBM) to the indolent, low-grade pilocytic astrocytoma.

35 - predominantly low-grade glial tumours like pilocytic astrocytoma.

36 craniopharyngioma, ependymoma, and juvenile pilocytic astrocytoma.

37 Pilocytic astrocytomas (PAs) are the most common glioma

38 Pilocytic astrocytomas (PAs) are WHO grade I brain tumor

39 Adult pilocytic astrocytomas (PAs) have been regarded as indis

40 Pilocytic astrocytomas (PAs), WHO malignancy grade I, ar

41 Pilocytic astrocytomas (PAs, WHO grade I) are the most c

42 a tumours (including 55 Medulloblastomas, 36 Pilocytic Astrocytomas and 26 Ependymomas) were scanned

43 or predisposition syndrome develop low-grade pilocytic astrocytomas at an increased frequency.

44 Pilocytic astrocytomas of the optic nerve (optic nerve g

45 These data illustrate that pilocytic astrocytomas overexpress specific NF1 gene tra

46 Pilocytic astrocytomas showed a marked predominance of t

47 5 astrocytic tumors including 21 GBMs and 19 pilocytic astrocytomas using oligonucleotide-based micro

48 Low-grade brain tumors (pilocytic astrocytomas) arising in the neurofibromatosis

49 increased risk of brain tumors (particularly pilocytic astrocytomas) independently of gestational age

50 There were 37 pilocytic astrocytomas, 34 medulloblastomas (23 classic,

51 e discrete cluster of gliomas identified the pilocytic astrocytomas, a second grouped the 1p/19q code

52 of seven oligodendrogliomas, three of three pilocytic astrocytomas, and one of five glioblastoma mul

53 NF1 gene acts as a tumor suppressor gene in pilocytic astrocytomas, and that NF1 gene expression wou

54 n with high fetal growth appeared to involve pilocytic astrocytomas, but not other astrocytomas, medu

55 presents tumors with molecular similarity to pilocytic astrocytomas, class II tumors are similar to 1

56 ole of the NF1 gene as a tumor suppressor in pilocytic astrocytomas, however, remains to be proven.

57 were most frequent in supratentorial midline pilocytic astrocytomas, in patients with progressive/rec

58 tween the ADC metrics and cellularity of the pilocytic astrocytomas, medulloblastomas, and ependymoma

59 Pilocytic astrocytomas, which contain abnormal glial cel

60 its central importance to the development of pilocytic astrocytomas.

61 s system tumors, including neurofibromas and pilocytic astrocytomas.

62 ignature that distinguished between GBMs and pilocytic astrocytomas.

63 pects of NF1 gene expression in six sporadic pilocytic astrocytomas.

64 e region on chromosome 17q occur in sporadic pilocytic astrocytomas.

65 viously, TDP-43 was detected in RFs of human pilocytic astrocytomas; however, involvement of TDP-43 i

66 sk of neurofibromas, schwannomas, low grade, pilocytic optic pathway gliomas, as well as malignant pe

67 ed human astrocytic tumors, but not sporadic pilocytic or other low-grade astrocytomas, specifically

68 Histology (juvenile pilocytic v astrocytoma not otherwise specified [NOS]) w