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1 eleosts, tac3 expression was absent from the pituitary.
2  in the retina, hindbrain, hypothalamus, and pituitary.
3 ronal tracer DiI following implants into the pituitary.
4 ently high circulating levels of LH from the pituitary.
5 de in the developing and postnatal mammalian pituitary.
6 nctional role for the lncRNA in the anterior pituitary.
7 one and prolactin production in the anterior pituitary.
8 e and follicle-stimulating hormone) from the pituitary.
9 regulator of TGFbeta/Activin pathways in the pituitary.
10 tegrated development of the hypothalamus and pituitary.
11 nfundibulum - the precursor of the posterior pituitary.
12 ndocrine lobe [adenohypophysis (ADH)] of the pituitary, a master gland controlling growth, metabolism
13                   Drivers of sporadic benign pituitary adenoma growth are largely unknown.
14 like growth factor I (IGF1), most often by a pituitary adenoma.
15 22.2%]), trigeminal neuralgia (565 [11.5%]), pituitary adenomas (641 [13.1%]), haemangioblastoma (29
16 /secretion, cell viability and apoptosis) in pituitary adenomas (n = 74), and to compare with the res
17                              Non-functioning pituitary adenomas (NFPAs) are the most frequent pituita
18                                              Pituitary adenomas (PAs) are benign growths arising from
19 Pituitary histopathology did not uncover any pituitary adenomas or somatotroph hyperplasia in either
20                                Patients with pituitary adenomas should be identified at an early stag
21 ome sequencing of 159 prospectively resected pituitary adenomas showed that somatic copy number alter
22  markers to predict the ultimate response of pituitary adenomas to BIM-23A760.
23 benign schwannomas, WHO grade 1 meningiomas, pituitary adenomas, and haemangioblastoma.
24 lomo et al. performed a whole-exome study of pituitary adenomas.
25 lowing decompression, with ADCYAP1 (encoding pituitary adenylate cyclase activating peptide, PACAP) b
26                                              Pituitary adenylate cyclase activating polypeptide (PACA
27 stress (CVS) has been shown to increase BNST pituitary adenylate cyclase activating polypeptide (PACA
28                            The activation of pituitary adenylate cyclase-activating peptide (PACAP) s
29  revealed strong homology with the mammalian pituitary adenylate cyclase-activating peptide (PACAP).
30 e present structures of peptide and Gs-bound pituitary adenylate cyclase-activating peptide, PAC1 rec
31 gated one such brain stress response system, pituitary adenylate cyclase-activating polypeptide (PACA
32                        The G protein-coupled pituitary adenylate cyclase-activating polypeptide recep
33 own and beige gene programs and illustrate a pituitary-adipose signaling axis in the control of therm
34 tion between functioning of the hypothalamic pituitary adrenal (HPA) axis and cognitive capability at
35 and competition may involve the hypothalamic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonad
36 tream molecular consequences of hypothalamic-pituitary-adrenal (HPA) axis activation by exogenous adr
37 independent of their effects on hypothalamic pituitary-adrenal (HPA) axis activation, aversive condit
38 aviors as well as regulation of hypothalamic-pituitary-adrenal (HPA) axis activity.
39                                 Hypothalamic-pituitary-adrenal (HPA) axis dysfunction contributes to
40 fective disturbance and promote hypothalamic-pituitary-adrenal (HPA) axis dysregulation, a key featur
41 ell as emotional reactivity and hypothalamic-pituitary-adrenal (HPA) axis functioning.
42    Genetic variation within the hypothalamic-pituitary-adrenal (HPA) axis has been linked to risk for
43            Dysregulation of the hypothalamic-pituitary-adrenal (HPA) axis occurs early in Alzheimer's
44                             The hypothalamic-pituitary-adrenal (HPA) axis orchestrates the physiologi
45                             The hypothalamic-pituitary-adrenal (HPA) axis regulates responses to inte
46                             The hypothalamic-pituitary-adrenal (HPA) axis, a neuroendocrine network t
47 s, predation risk activates the hypothalamic-pituitary-adrenal (HPA) axis, and there is growing evide
48 at is the primary output of the hypothalamus-pituitary-adrenal (HPA) axis, has been hypothesized to b
49 s affected by senescence of the hypothalamic-pituitary-adrenal (HPA) axis, leading to progressive dys
50 s, leading to activation of the hypothalamic-pituitary-adrenal (HPA) axis, the sympathetic nervous sy
51 derived cytokines stimulate the hypothalamic-pituitary-adrenal (HPA) axis, triggering endogenous gluc
52 in persistent alteration of the hypothalamic-pituitary-adrenal (HPA) axis.
53 s from adrenergic input and the hypothalamus-pituitary-adrenal (HPA) axis.
54 xis, the fish equivalent of the hypothalamic-pituitary-adrenal (HPA) axis.
55 mmalian stress response via the hypothalamic-pituitary-adrenal (HPA) axis.
56 rent increases in immobility and hypothalamo-pituitary-adrenal (HPA) output in male rats during tail
57 on immune, cardiometabolic, and hypothalamic-pituitary-adrenal (HPA) systems) in first-episode psycho
58  describe the physiology of the hypothalamic-pituitary-adrenal (HPA), hypothalamic-pituitary thyroid
59                                 Hypothalamic-pituitary-adrenal (HPA)-axis hyperactivity and inflammat
60 he newborn decreases the limbic-hypothalamic-pituitary-adrenal (LHPA) axis activity in the offspring.
61 ne concentrations (a measure of hypothalamic-pituitary-adrenal [HPA] axis activation and the stress r
62  promotes the activation of the hypothalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axe
63          Instead of consecutive hypothalamus-pituitary-adrenal axis activation, we report that acute
64 regulate cognition, anxiety and hypothalamic-pituitary-adrenal axis activation.
65 ed the hypothesis that maternal hypothalamic-pituitary-adrenal axis activity, measured by hair cortis
66 ic inflammation, or exacerbated hypothalamic-pituitary-adrenal axis activity.
67                           Lower hypothalamic-pituitary-adrenal axis and autonomic reactivity to stres
68 t, including alterations in the hypothalamic-pituitary-adrenal axis and inflammatory cytokines, which
69 kely mediated by inhibiting the hypothalamic-pituitary-adrenal axis and inflammatory responses to str
70 luence of the microbiota on the hypothalamic-pituitary-adrenal axis and neuroimmune system.
71 s through the activation of the hypothalamus-pituitary-adrenal axis and the sympathetic nervous syste
72 y effects of opioid drugs on the hypothalamo-pituitary-adrenal axis and their negative effects on bon
73   Second, we review the role of hypothalamic-pituitary-adrenal axis dysfunction in the neurobiology o
74                   Specifically, hypothalamic-pituitary-adrenal axis dysfunction, enhanced inflammatio
75 hanistically, we found impaired hypothalamic-pituitary-adrenal axis feedback, blunted sympathetic res
76 t in GABAergic, neurons induced hypothalamic-pituitary-adrenal axis hyperactivity and reduced fear- a
77                             The hypothalamic-pituitary-adrenal axis is a key regulatory pathway in th
78 vation of catecholaminergic and hypothalamic-pituitary-adrenal axis leads to splenic atrophy and cont
79                             The hypothalamic-pituitary-adrenal axis modulates immunity in response to
80  health, such as by influencing hypothalamic-pituitary-adrenal axis regulation and cortisol dynamics,
81  important central component of hypothalamic-pituitary-adrenal axis regulation that prepares the orga
82 ted Glp1r knockdown had reduced hypothalamic-pituitary-adrenal axis responses to both acute and chron
83 cess glucocorticoid release via hypothalamus-pituitary-adrenal axis stimulation.
84 howed a significantly increased hypothalamic-pituitary-adrenal axis stress response and impaired sens
85  sympathetic nervous system and hypothalamic-pituitary-adrenal axis) transcription factor activation.
86 with targets in the adrenergic, hypothalamic-pituitary-adrenal axis, and neuropeptide Y systems.
87 rs physiological changes in the hypothalamic-pituitary-adrenal axis, reward processing in the brain,
88 luding on the regulation of the hypothalamic-pituitary-adrenal axis, thereby affecting an individual'
89 ress response by activating the hypothalamic-pituitary-adrenal axis.
90 have a regulatory effect in the hypothalamic-pituitary-adrenal axis.
91 in the context of a hyperactive hypothalamic-pituitary-adrenal axis.
92  mediated and controlled by the hypothalamic-pituitary-adrenal axis.
93 nd endocannabinoid systems; the hypothalamus-pituitary-adrenal axis; and adenosine and nitric oxide s
94 pathetic nervous system and the hypothalamus-pituitary-adrenal endocrine axis.
95 natal growth, cardiovascular development and pituitary-adrenal function of isolated chronic developme
96 that avBST inhibition augmented posttraining pituitary-adrenal output and enhanced the memory for inh
97 aint abolished their heightened hypothalamic-pituitary-adrenal responsivity and reduced stress-induce
98  the expression of genes in the hypothalamic-pituitary-adrenal/stress system (e.g., Crhr1) is one of
99 n animal models reveal that the hypothalamic-pituitary-adrenocortical (HPA) axis calibrates to the ha
100  step, namely activation of the hypothalamic-pituitary-adrenocortical (HPA) axis.
101                                    The small pituitary and eyes are associated with reduced cell prol
102 neurons, which simultaneously project to the pituitary and forebrain regions involved in social behav
103 em translate to vesicular release toward the pituitary and identify how dopamine dynamics are control
104  both in thyrotropes and gonadotropes of the pituitary and in Leydig and germ cells in the testes, bu
105  < 0.05) in the tilapia cerebellum, thalamus-pituitary and liver, respectively.
106 lls co-localized with dendritic cells in the pituitary and produced increased levels of interferon-ga
107 ntrations, transcript abundance in the fetal pituitary and testes and circulating steroids, at day 75
108 al progesterone administration altered fetal pituitary and testicular function in ovine male fetuses.
109 TRH-immunoreactive elements in the brain and pituitary, and explored its role in regulation of hypoph
110 n factor BMAL2, in the pars tuberalis of the pituitary, and triggers summer biology through the eyes
111   The fish condition supports the vertebrate pituitary arising through interactions of an ancestral e
112  methods to measure the hypothalamic-adrenal-pituitary axis (N = 173), immune and inflammatory marker
113              Salivary cortisol (hypothalamic pituitary axis), heart rate variability (sympathetic adr
114 ess are largely mediated by the hypothalamic-pituitary axis, a highly conserved neurohormonal cascade
115                                   In tumoral pituitaries, BIM-23A760 also inhibited Ca(2+) concentrat
116  been reported on the safety and efficacy of pituitary biopsy in the pediatric population for suspect
117                                 In the adult pituitary, BMPs participate in the control of hormone se
118 ocellular neurons that do not project to the pituitary but synapse onto magnocellular neurons, is pre
119    In situ hybridization showed two distinct pituitary cell populations, tshbetaa cells in the anteri
120                              Single anterior pituitary cell RNA sequencing and immunocytochemical ana
121 se RP progenitors and for differentiation of pituitary cell types.
122                        Here, we used primary pituitary cell-cultures from two normal nonhuman-primate
123  regulatory circuit that relevantly modulate pituitary cell-function.
124 dipokines on the functioning of all anterior-pituitary cell-types.
125 C) genes Asic1, Asic2, and Asic4 in anterior pituitary cells and Asic1 and Asic2 in immortalized GH3
126 also found that the expression of GH-C53S in pituitary cells deviates from that of GH-WT.
127                                 Culturing of pituitary cells in GnRH-free conditions downregulated Fs
128 sive element, we sought to determine whether pituitary cells secrete BMPs or BMP antagonists.
129                  Further, treatment of mouse pituitary cells with cAMP pathway agonists in vitro and
130 ents increased the excitability of secretory pituitary cells, consistent with their potential physiol
131  somatostatin receptor subtype 2 (SSTR2), in pituitary cells.
132 ells and Asic1 and Asic2 in immortalized GH3 pituitary cells.
133 icantly increased formation of hGH dimers in pituitary cells.
134  disulfide bond linking two hGH molecules in pituitary cells.
135 ronic stress, hypothalamic activation of the pituitary changes from corticotropin-releasing hormone-d
136                 Interestingly, we found that pituitary-conditioned medium contains a factor that inhi
137 ar whether other factors besides an enlarged pituitary contribute to the hypersecretion.
138  secretion, releasing the thyroid gland from pituitary control.
139 Stimulation of cAMP in C57BL/6 mouse primary pituitary cultures using forskolin or a long-acting GH-r
140             Of the 94 patients, 80 (85%) had pituitary Cushing's syndrome.
141 red for growth, puberty onset, and potential pituitary deficiency.
142 on spaceflight was associated with increased pituitary deformation, augmented aqueductal cerebrospina
143 R-7 family, miR-7a2, is essential for normal pituitary development and hypothalamic-pituitary-gonadal
144 estigate the function of this pathway during pituitary development and in the regulation of the SOX2
145                    Knockout mice for Tpit, a pituitary differentiation factor, show that Creb3l2 expr
146 is not successful and can be classified into pituitary-directed drugs, steroid synthesis inhibitors,
147 h an increasing number of suspected cases of pituitary diseases, there has been a paradigm shift in t
148 vivo long-acting GHRH treatment also induced pituitary DNA damage in mice.
149 ) astronauts developed or showed exacerbated pituitary dome depression compared with baseline.
150 icting a mutant protein known to cause human pituitary dwarfism.
151 guidance for the screening and management of pituitary dysfunction in adult patients with TBI.
152  persist throughout life, giving rise to all pituitary endocrine lineages.
153                                    Moreover, pituitary enlargement may lead to compressive symptoms,
154 meningeal enhancement, sagging of the brain, pituitary enlargement, and subdural fluid collections.
155                       Hence, circadian clock-pituitary epigenetic pathway interactions form the basis
156                Our observations suggest that pituitary ERalpha is involved in the estrogen negative f
157                Dysregulation of hypothalamic-pituitary estrogen receptor alpha-mediated signaling cau
158                     Primate and human normal pituitaries exhibited similar sst2/sst5/D2 expression pa
159               Finally, we found that primate pituitaries expressed leptin/adiponectin/resistin.
160  dimeric glycoprotein hormones that suppress pituitary FSH production.
161        While ovarian feedback on hypothalamo-pituitary function is a well-established concept, the pr
162 no recurrence and partial restoration of the pituitary function was seen.
163 e changes in circulating GH levels to adjust pituitary GH secretion within a narrow physiological ran
164 protein and localized a specific area of the pituitary gland (i.e., adenohypophysis) known to secrete
165 actin from lactotropic cells in the anterior pituitary gland and thus play a central role in prolacti
166 ocin (OT) originates from secretion from the pituitary gland into the circulation and from absorption
167 wing to organic disease of the hypothalamus, pituitary gland or testes has been treated with testoste
168 he hormone arginine vasopressin (AVP) in the pituitary gland or the hypothalamus, whereas nephrogenic
169 acellular compartment to the cell surface in pituitary gland somatotropes, concomitant with increasin
170                       Endocrine cells in the pituitary gland typically display either spiking or burs
171     We observed that a reduced volume in the pituitary gland was associated with the slope of neuroti
172 ated by the pars tuberalis (PT) of the fetal pituitary gland, before the fetal circadian system and a
173                                       In the pituitary gland, hormones are stored in a functional amy
174                                       In the pituitary gland, peptide hormones can be stored as amylo
175  (GH), a pleiotropic hormone secreted by the pituitary gland, regulates immune and inflammatory respo
176  Prolactin is a major hormone product of the pituitary gland, the central endocrine regulator.
177  similar across brain regions except for the pituitary gland, which is not protected by the BBB.
178 el role for 4.1N in the axis of hypothalamus-pituitary gland-reproductive system.
179 udy is to identify proton sensors in the rat pituitary gland.
180 ithelial cells in the adenohypophysis of the pituitary gland.
181 ximity to the optic chiasm, hypothalamus and pituitary gland.
182 y infiltration of T and B lymphocytes in the pituitary gland.
183 ion of autoreactive T and B cells within the pituitary gland.
184 d by a 50% threshold for all lesions and the pituitary gland; and for (18)F-FDG (C)-RD of SUVs of the
185 ory granules were significantly decreased in pituitary glands of 4.1N(-/-) compared to 4.1N(+/+).
186                               Examination of pituitary glands revealed that the secretory granules we
187 atasets (brain; blood; thyroid, adrenal, and pituitary glands).
188 imilarity in the formation of the pineal and pituitary glands, and suggests that all CNS neuroendocri
189  known to regulate the mammalian hypothalamo-pituitary gonadal axis, may be involved in the seasonal
190 ic-pituitary thyroid (HPT), and hypothalamic-pituitary-gonadal (HPG) axes and review the evidence for
191 mic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonadal (HPG) axes in underlying some of these
192 al hormones are produced by the hypothalamic-pituitary-gonadal (HPG) axis and have been shown to dete
193 stic ovary syndrome (PCOS) is a hypothalamic-pituitary-gonadal (HPG) axis disorder.
194 ormal pituitary development and hypothalamic-pituitary-gonadal (HPG) function in adulthood.
195    In women, suppression of the hypothalamic-pituitary-gonadal axis appears to be a principal contrib
196        Conditions impairing the hypothalamic-pituitary-gonadal axis during paediatric or pubertal lif
197  of metabolic information to the hypothalamo-pituitary-gonadal axis is mediated by leptin receptors o
198                             The hypothalamic-pituitary-gonadal axis is of relevance in many processes
199  based on the activation of the hypothalamic-pituitary-gonadal axis is unclear.
200  2 regulate many aspects of the hypothalamic-pituitary-gonadal axis.
201 RH to activate and maintain the hypothalamic-pituitary-gonadal axis.
202  by affective comorbidities and hypothalamic-pituitary-gonadal dysregulation.
203 lucose utilization and GLUT1 expression in a pituitary gonadotrope cell model and in primary gonadotr
204 lated the Fshb expression levels obtained in pituitary gonadotrope cells perifused with varying GnRH
205                           GnRH regulates the pituitary gonadotropin's follicle-stimulating hormone (F
206 show normal sexual behavior but hypothalamic-pituitary-gonadotropin (HPG) axis dysregulation, likely
207 ) leads to a marked decrease in secretion of pituitary gonadotropins LH and FSH and impairment of rep
208                                Sex-dependent pituitary growth hormone (GH) secretory profiles-pulsati
209 e a small hypothalamus and low expression of pituitary growth hormone and prolactin (prl).
210                              Average midline pituitary height decreased from 5.9 to 5.3 mm (P < .001)
211                                     Detailed pituitary histological and immunohistochemical studies w
212                                              Pituitary histopathology did not uncover any pituitary a
213 ciency on mouse phenotype and, specifically, pituitary histopathology.
214 iates L-DOPA-induced dyskinetic movements in pituitary homeobox 3 (Pitx3)-deficient mice that lack of
215 ked to induction of MC2R, a receptor for the pituitary hormone ACTH.
216                                     Combined pituitary hormone deficiency (CPHD) is a genetically het
217 growth pathogenesis associated with combined pituitary hormone deficiency.
218 ntibody that targets the beta-subunit of the pituitary hormone follicle-stimulating hormone (Fsh) inc
219 he concentration of circulating prolactin, a pituitary hormone influencing maternal care.
220          Galanin is a peptide that regulates pituitary hormone release, feeding, and reproductive and
221  membrane resurfacing of SSTR2 can fine-tune pituitary hormone release.
222 amp analyses and are associated with reduced pituitary hormone secretion from AtT-20 cells.
223 idism, and the role of IGSF1 as regulator of pituitary hormone secretion.
224 e (TIDA) neurons that control release of the pituitary hormone, prolactin, which triggers key materna
225 nhibited the expression/secretion of several pituitary hormones (specially GH/PRL), which was accompa
226 olour, possibly reflecting common effects of pituitary hormones on puberty and pigmentation.
227 bernation, and metabolism, are controlled by pituitary hormones released in response to annual enviro
228 ropin subunit and GnRH receptor genes in rat pituitary in vitro and in vivo to clarify their expressi
229                  In conclusion, we show that pituitary-infiltrating lymphocytes proliferate in situ d
230 g., heterotopia, Dandy-Walker malformation), pituitary insufficiency, and/or synpolydactyly.
231 ehaviour and functioning of the hypothalamic-pituitary-interrenal (HPI) axis, the fish equivalent of
232  by seasonal stimulation of the hypothalamic-pituitary-interrenal axis, and that relaxed selection on
233 on scores; RR, 6.74 [95% CI, 4.61 to 9.86]), pituitary irradiation (5-20 Gy: RR, 4.24 [95% CI, 1.98 t
234 (>= 15 Gy on >= 90% of their volume) without pituitary irradiation increased the RR of SAH by 4.62 (9
235 tment, mitotane therapy within 6 months, and pituitary irradiation within 10 years.
236                If patients had also received pituitary irradiation, this increased the RR by an addit
237          However, the role of ERalpha in the pituitary is still controversial because of the varied p
238 levels at e10.5 and e12.5 were comparable to pituitary levels from adult female mice at proestrus and
239 n the differential diagnosis of solid-cystic pituitary masses along with clinical correlation, which
240 tative changes in pre- to postflight (day 1) pituitary morphologic structure were determined.
241 anges in intracranial volumetric parameters, pituitary morphologic structure, and aqueductal cerebros
242 th the responses of normal primate and human pituitaries (n = 3-5).
243                                          The pituitary neuropeptide oxytocin promotes social behavior
244 ntroduced to be exclusively expressed in the pituitary on the background of a global ERalpha-null (Pi
245  mediators of the HPA stress response in the pituitary or adrenal glands.
246           Consecutive patients who underwent pituitary or parasellar tumor resection between January
247 g regulation occurs within the hypothalamus, pituitary, or gonads.
248 onate to adulthood, we investigated a common pituitary origin for hypothyroidism and macroorchidism,
249 o participation of cells in the hypothalamus-pituitary-ovary feedback control loop.
250 d B cells underwent proliferation within the pituitary parenchyma.
251 ential component of PSC regulation in normal pituitary physiology and tumourigenesis.
252 rve transfer and highly purified lyophilized pituitary porcine GH treatment (0.6 mg/day); Group-3 (po
253 n, and their fibers innervated the brain and pituitary profusely.
254 ption in neurochemical identity, however, as pituitary prolactin secretion is primarily under monoami
255 and female reproductive tracts, hypothalamic-pituitary regulation of reproduction and prenatal develo
256  effects of BIM-23A760 in normal and tumoral pituitaries remains incomplete.
257 higher expression of the Fshb subunit in the pituitary, resulting in elevated serum estrogen and high
258      Single-cell RNA sequencing of the adult pituitary reveals similar competency of endodermal and e
259 a scaling factor for translation capacity in pituitary secretory cells and that it directly binds ~75
260 nclude that the gonadotropes of the anterior pituitary sense glucose availability and integrate this
261 s expressed in hypothalamic GHRH neurons and pituitary somatotropes.
262                                  Tissue from pituitary-specific Aip-knockout (Aip(Flox/Flox);Hesx1(Cr
263 because of the varied phenotypes reported in pituitary-specific ERalpha KO mouse models.
264 ESRRG), which results in stronger binding of pituitary-specific positive transcription factor 1 (Pit-
265                          Biopsy for isolated pituitary stalk thickening for suspected germinoma is ge
266  endonasal vs. open craniotomy) for isolated pituitary stalk thickening were identified.
267 nohypophysis, and tshbetab cells near to the pituitary stalk, a location comparable to the pars tuber
268                                SOX2 positive pituitary stem cells (PSCs) are specified embryonically
269 nd regulated in other tissues, including the pituitary, suggesting that locally- and AT-produced adip
270            Applied to tissue sections of rat pituitary, the platform demonstrated improved spatial re
271 alamic-pituitary-adrenal (HPA), hypothalamic-pituitary thyroid (HPT), and hypothalamic-pituitary-gona
272 h elevated leptin maintains the hypothalamic pituitary thyroid axis, despite leptin resistance.
273                             The hypothalamus-pituitary-thyroid (HPT) axis plays a crucial role in the
274 othalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axes, as well as a rise in systolic bl
275 ing hormone (TRH) regulates the hypothalamic-pituitary-thyroid axis in mammals and also regulates pro
276                    Although the hypothalamic-pituitary-thyroid axis is under the control of the circa
277 beta leads to disruption of the hypothalamic-pituitary-thyroid axis with resistance to TH, while muta
278                                       In the pituitary, TRH-immunoreactive fibers were seen in the ne
279 f surgically resected tumors from forty five pituitary tumor patients [gonadotropic (LH/FSH-secreting
280                                    Our human pituitary tumor transcriptome data revealed the "epithel
281              The mechanisms of AIP-dependent pituitary tumorigenesis are still under investigation an
282      Growth hormone-secreting (GH-secreting) pituitary tumors are driven by oncogenes that induce cAM
283                                              Pituitary tumors are frequently associated with mutation
284 essive, young-onset growth hormone-secreting pituitary tumors are not fully understood.
285 iagnosis of various histological subtypes of pituitary tumors is made using serum based hormone panel
286  Prolactinomas are the most frequent type of pituitary tumors, which represent 10-20% of all intracra
287 anel in the diagnosis of various subtypes of pituitary tumors.
288 itary adenomas (NFPAs) are the most frequent pituitary tumors.
289  tumorigenesis in Rb1-deficient prostate and pituitary tumors.
290    In adult cohorts separately, rs1344110 in pituitary tumour-transforming 1 interacting protein (PTT
291 g the formation of non-secreting, aggressive pituitary tumours.
292 lation of the gene and protein expression of pituitary-type GHRH-receptor in prefrontal cortex of mic
293 ngth ELA enabled a distinctive regulation of pituitary vasopressin release.
294                  T cell proliferation in the pituitary was confirmed in patients affected by autoimmu
295 that regulate GH secretion from the anterior pituitary were determined.
296               The effects of MIR205HG on the pituitary were independent of miR-205.
297  Expression profiles of both paralogs in the pituitary were measured by qPCR throughout smoltificatio
298 ound that miR-7a2 is highly expressed in the pituitary, where it suppresses golgi glycoprotein 1 (GLG
299 he rat hypothalamus project to the posterior pituitary, where they secrete their products into the bl
300 tions of an ancestral endoderm-derived proto-pituitary with newly evolved placodal ectoderm.

 
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