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1 g the formation of non-secreting, aggressive pituitary tumours.
2 ubtypes of ACTH-secreting and possibly other pituitary tumours.
3 ent in human corticotrophin (ACTH)-secreting pituitary tumours.
4 sult in young-onset growth hormone secreting pituitary tumours.
5 (P-NETs) in association with parathyroid and pituitary tumours.
12 orticotropic hormone (ACTH) secretion from a pituitary tumour (Cushing disease (CD)) or non-pituitary
13 tuitary tumour (Cushing disease (CD)) or non-pituitary tumour (ectopic ACTH secretion), which stimula
14 In this study we have examined 32 sporadic pituitary tumours for expression of the DAP kinase prote
15 lary craniopharyngioma (PCP), a benign human pituitary tumour harbouring BRAF p.V600E also contains S
16 r protein products are a frequent finding in pituitary tumours; however, genetic changes associated w
18 ow that silencing of the GADD45gamma gene in pituitary tumours is primarily associated with methylati
20 phenotype associated with the development of pituitary tumours, specifically those of the intermediat
23 yclin A and CDC2 and novel targets including pituitary tumour transforming gene 1, Polo-like kinase 1
25 In adult cohorts separately, rs1344110 in pituitary tumour-transforming 1 interacting protein (PTT
26 TTG1IP showed significant co-expression with pituitary tumour-transforming 1, the binding factor of P
27 re an approximate 10-fold down-regulation of pituitary tumour-transforming gene-1 (pttg1) and an appr
28 clinical characteristics of 84 patients with pituitary tumour who had troublesome headache were inves