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1 hymal heterogeneity in first-trimester human placentae.
2 mitochondrial fusion in both male and female placentae.
3 istinct from both each other and from normal placentae.
4 s, isolated from villous tissue of full-term placentae.
5  and blunted STB differentiation seen in T21 placentae.
6 12/55 EPLs (21.8%), and 0/15 healthy control placentae.
7 ratio 1.35-1.79) was detected in all healthy placentae.
8  inner fruit tissue containing the seeds and placentae.
9 ssociation with hypotrophic and hypovascular placentae.
10 ) from female placentae than those from male placentae.
11 ssion in response to maternal diet than male placentae.
12 cotyledon VEGF was decreased (P<0.05) in FGR placentae.
13 HIV-1-suppressive factor produced locally in placentae.
14 m H3K4me3 and increased CTCF binding in male placentae.
15 human trophoblast cells and mouse Phd2(-)(/-)placentae.
16 ed in PE placentae when compared with normal placentae.
17 le nMitoQ increased fusion in hypoxic female placentae.
18 improved complex IV activity in hypoxic male placentae.
19 ead of one observed in all other hemochorial placentae.
20 total transthyretin compared to normotensive placentae (2352 +/- 2949 ng/mL vs. 3250 +/- 1864 ng/mL,
21   We detected microorganisms in 10.6% of 535 placentae (443 were delivered late preterm and 92 were d
22 etic DNA-methylation pattern in pathological placentae affecting placentogenesis, but also the develo
23                    The Gd content of excised placentae and fetuses was measured, using inductively co
24          PinT pregnancies resulted in larger placentae and heavier foals relative to PinP (P < 0.05).
25  gene expression was also observed in Pkd2-/-placentae and in cystic kidneys of Pkd1cond/-; Meox2cre/
26                             TinP had smaller placentae and lighter foals relative to TinT (P < 0.05).
27 ohydrate, lipid and amino acid metabolism of placentae and maternal hearts.
28  production of transthyretin by preeclamptic placentae and whether transthyretin is carried into the
29 ler cell activation, impaired recruitment to placentae, and altered maternofetal cytokines.
30 ogy samples with known CN, including tonsil, placentae, and FFPE melanoma cell lines.
31                                          The placentae are smaller but do not reveal any evidence of
32 proteins in trophoblast cell lines and human placentae at different gestational ages.
33 ompared with control and infectious villitis-placentae by immunoSEQ.
34 n abundance was measured by western blots in placentae collected from 133 neonates born to adolescent
35  BPGM, were lower in cord serum of human FGR placentae compared to control.
36 ere present in higher levels in preeclamptic placentae compared to normotensive placentae (p < 0.05,
37              FPN protein was detected in all placentae delivered between 25 and 42 wk GA.
38                                       Female placentae demonstrated more striking alterations in gene
39 e find global changes in CTCF binding within placentae derived from the male offspring of alcohol-exp
40 , the choline/lipid ratio was </=0.02 in all placentae, despite preservation of the lipid peak (p<0.0
41 chain profile in human T cells isolated from placentae diagnosed with VUE compared with control and i
42                                Although male placentae do not display overt changes in placental hist
43 or characterization of vascular perfusion in placentae during pregnancy to identify placental insuffi
44                                      Hypoxic placentae exhibited higher hemoglobin-oxygen affinity co
45                       In contrast, human FGR placentae exhibited reduced BPGM levels in the syncytiot
46  and moderate apoptosis, was associated with placentae expressing intermediate levels of HB-EGF.
47 s pathway and showed increased expression in placentae from a small sample of SARS-CoV-2-positive pre
48 al mitochondrial function in male and female placentae from fetuses exposed to prenatal hypoxia, whic
49  methylation analysis was performed on human placentae from first, second and third trimesters to det
50 in the syncytiotrophoblast layer compared to placentae from healthy uncomplicated pregnancies.
51 pressing cells were significantly greater in placentae from HIV-1-infected women who did not transmit
52 cific isoform of TP (TPbeta) is increased in placentae from IUGR pregnancies, compared to healthy pre
53  tissues, was not observed in blastocysts or placentae from later stage clones, although fetuses reca
54        Analysis of MAOA expression in bovine placentae from natural reproduction revealed imprinted X
55  by loci harboring genes highly expressed in placentae from normal and complicated pregnancies [G.
56 easuring mRNA and protein expression in term placentae from normotensive (NT) and PE women who delive
57                                      We used placentae from normotensive and preeclampsia pregnancy c
58  P = 0.002) were all significantly higher in placentae from NT women at altitude, despite mRNA expres
59                 Importantly, our analyses of placentae from pathological pregnancies show that extrem
60                                              Placentae from preeclampsia pregnancies contain increase
61                                              Placentae from preeclamptic and normal pregnancies were
62 ion preserved placental architecture whereas placentae from untreated infected mice had widespread he
63 phoblast cells up to week 35 of gestation in placentae from women who delivered preterm.
64    Using primary human trophoblast cells and placentae from women with PE, various aspects of cholest
65 ts in both the trophoblast hypoxia model and placentae from women with PE.
66 xposure during early-mid gestation, when pig placentae grow heavily, on placental and fetal developme
67                                          IVF placentae, however, displayed hypomethylation of imprint
68 rs and trophoblast proliferation in human PE placentae, implying translational relevance.
69 estational day 19 and metabolomic profile of placentae, maternal and fetal hearts analysed using high
70            We performed TWAS in healthy term placentae (N = 147) to derive candidate placental causal
71  preterm infants (n = 477) and their infants/placentae (n = 535) were recruited, and swab specimens o
72 ntas from HIV-infected women, we studied the placentae of 30 HIV-positive and 13 control gravidae.
73                                Additionally, placentae of both groups of manipulated concepti exhibit
74                          By day 90, however, placentae of conceptuses from the high PE group expresse
75  exhibits pronounced sexual dimorphism, with placentae of females more sensitive to nutritional pertu
76 ue factor was significantly increased in the placentae of infected C57BL/6 mice but was reduced in mi
77  sensitive to nutritional perturbations than placentae of males.
78 lish whether HB-EGF expression is altered in placentae of pre-eclamptic women.
79          In contrast, we found random XCI in placentae of the deceased clones but completely skewed X
80 t they were not significantly different from placentae of uninfected mothers.
81 e the microbes most frequently isolated from placentae of women who deliver preterm.
82 r levels of HB-EGF protein were found in the placentae of women who were not in labour.
83 sibly related to mitochondrial biogenesis in placentae of women with diabetes (n = 23) and controls (
84                   HB-EGF is downregulated in placentae of women with preeclampsia, a disorder associa
85  on GD 20, and total Cr was estimated in the placentae; ovaries were removed from the F1 offspring on
86 amnionitis in moderate/late preterm and term placentae (P < .001).
87 eclamptic placentae compared to normotensive placentae (p < 0.05, n = 7), however the levels of trans
88                                    In female placentae, prenatal hypoxia decreased respiration throug
89  complications can occur, including abruptio placentae, renal failure, subcapsular hematomas, and hep
90 se to chorio-allantoic and visceral yolk sac placentae, respectively.
91 ntiated TS cell cultures and dissected mouse placentae resulted in proliferating colonies that expres
92                                 Preeclamptic placentae secreted similar levels of total transthyretin
93               Microarray analysis of Pkd1-/- placentae showed upregulation of a set of apolipoprotein
94 on of BPGM was found in both human and mouse placentae syncytiotrophoblast, with higher expression fa
95 higher in genes (651 out of 700) from female placentae than those from male placentae.
96 ng (PAS-Seq) of RNA from normal and PE human placentae to interrogate transcriptome-wide gene express
97 ncy, resulting in compensatory growth of the placentae to maintain fetal development.
98 14-65 days of gestation) and healthy control placentae (various gestational ages) were assessed using
99                      Human TfR isolated from placentae was used to characterize the structure of the
100                                          The placentae were analyzed via immunohistochemistry and in
101 day (GD) 9.5 to 14.5 through drinking water, placentae were removed on GD 20, and total Cr was estima
102                                        Human placentae were selected from FGR and matched controls an
103 = 3), PE/IUGR (n = 3) and HELLP/IUGR (n = 2) placentae were used to determine the mean methylation le
104 sed mitochondrial damage were observed in PE placentae when compared with normal placentae.
105 duced complex IV activity and fusion in male placentae, while nMitoQ improved complex IV activity in
106  mice resulted in growth restricted pups and placentae with poor syncytialisation and diminished grow
107 acity is significantly lower in preeclampsia placentae, with decreased glutathione content, and GPx4

 
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