ライフサイエンスコーパス: placental growth factor

1 elease of T cells, a process that depends on placental growth factor.

2 GFR family, and binds to VEGF-A, VEGF-B, and placental growth factor.

3 lt1), an antiangiogenic protein, relative to placental growth factor.

4 -1alpha, the angiopoietin receptor Tie2, and placental growth factor.

5 st of vascular endothelial growth factor and placental growth factor.

6 r, vascular endothelial growth factor165, or placental growth factor.

7 r stimulated with a VEGFR-1-specific ligand, placental growth factor 1.

8 ve VEGF receptors, binds VEGF-A, VEGF-B, and placental growth factors 1 and 2 with high affinity.

9 eceptor-2 (R2), whereas the VEGFR1 agonists, placental growth factors 1 and 2, had no effect on FABP4

10 ndlimb ischemia and to examine the effect of placental growth factor-1 (PlGF-1) on this process.

11 The VEGFR-1-specific ligand placental growth factor-1 (PlGF-1), but not VEGF-E, whic

12 Placental growth factor-1 (PlGF-1), which exclusively bi

13 arged peptide derived from the C-terminus of placental growth factor-2 (PLGF-2) was selected to enhan

14 proposed prediction model was compared with placental growth factor, a predictive biomarker for preg

15 VEGF and placental growth factor affect the early stages of myelo

16 difference in the treatment effects on serum placental growth factor and soluble fms-like tyrosine ki

17 Uterine artery pulsatility index and serum placental growth factor and soluble fms-like tyrosine ki

18 s of the VEGF family, VEGF-A through VEGF-E, placental growth factor and the newly described, tissue-

19 , and Ki-67 using immunohistochemistry; with placental growth factor and vascular endothelial growth

20 Using selective ligands (VEGF-E and placental growth factor) and a receptor-specific inhibit

21 human chorionic gonadotropin, progesterone, placental growth factor, and placental lactogen.

22 X-C motif chemokine 10), interleukin-1alpha, placental growth factor, and platelet-derived growth fac

23 nic factors angiopoietin-1 (ANGPT1), ANGPT2, placental growth factor, and platelet-derived growth fac

24 easurements of mean arterial pressure, serum placental growth factor, and serum soluble fms-like tyro

25 ine chromogranin A, neuron-specific enolase, placental growth factor, and soluble vascular endothelia

26 opoietin, platelet-derived growth factor-BB, placental growth factor, and vascular endothelial growth

27 1, ANGPT2, platelet-derived growth factor-B, placental growth factor, and VEGF mRNA in AdCA5-injected

28 s in the diagnosis of pre-eclampsia, such as placental growth factor based testing, have been shown t

29 Changes in plasma placental growth factor, basic fibroblast growth factor,

30 ctors soluble fms-like tyrosine kinase 1 and placental growth factor, both biomarkers associated with

31 vascular endothelial growth factor (VEGF) or placental growth factor-critical regulators of tumour an

32 ed SSL had a higher performance than that of placental growth factor for total study population (AUC,

33 nta into the maternal circulation, including placental growth factor, growth/differentiation factor 1

34 fusion protein that scavenges both VEGF and placental growth factor in patients with recurrent malig

35 tor (not bound to bevacizumab) and increased placental growth factor in plasma.

36 Placental growth factor increased, whereas endoglin and

37 ta growth factor levels are high in SCD, and placental growth factor induces the release of the vasoc

38 eased vascular endothelial growth factor and placental growth factor levels and intrauterine growth r

39 Placental growth factor negatively regulates endothelial

40 Neither placental growth factor nor an Flt-1-selective variant w

41 .046), hepatocyte growth factor (P = .046), placental growth factor (P < .001), stromal-derived fact

42 correlated significantly with an increase in placental growth factor (P = .003) and a decrease in sol

43 , monocyte chemotactic protein-1 (P = .046), placental growth factor (P = .027), and transforming gro

44 more, prion-like protein doppel ( Prnd ) and placental growth factor ( Pgf ) were upregulated in Alk1

45 BCCs and MSCs expressed placental growth factor (PGF) and its cognate receptor V

46 ify the pro-proliferative VEGF family member placental growth factor (PGF) as an aldosterone-regulate

47 mbers VEGFA, VEGFB, VEGFC, VEGFD (FIGF), and placental growth factor (PGF); VEGF receptors VEGFR1 (FL

48 cluding angiopoietin 2, angiopoietin-like 4, placental growth factor, platelet-derived growth factor

49 in the expression of the angiogenic factors placental growth factor, platelet-derived growth factor

50 These findings reveal that placental growth factor plays a critical role in the con

51 VEGF-B and placental growth factor (PlGF) activate VEGFR-1 selectiv

52 evels of placental genes and in secretion of placental growth factor (PlGF) and human chorionic gonad

53 We sought to evaluate placental growth factor (PlGF) and soluble Fms-like tyro

54 like tyrosine kinase 1 (sFlt-1), which binds placental growth factor (PlGF) and vascular endothelial

55 Placental growth factor (PlGF) and vascular endothelial

56 ascular endothelial growth factor (VEGF) and placental growth factor (PlGF) are increased in sepsis.

57 ascular endothelial growth factor (VEGF) and placental growth factor (PLGF) are increased in the mate

58 tyrosine kinase 1 (sFlt1) and proangiogenic placental growth factor (PlGF) at presentation and exami

59 dverse events (AEs), and association between placental growth factor (PLGF) change and OS.

60 r endothelial growth factor (VEGF) but lower placental growth factor (PLGF) compared to controls.

61 Angiogenic placental growth factor (PlGF) concentrations rise durin

62 h as fms-like tyrosine kinase-1 (sFlt-1) and placental growth factor (PlGF) from urine as it was than

63 Placental growth factor (PLGF) has previously been repor

64 ell growth factor (VEGF) homodimers and VEGF/placental growth factor (PlGF) heterodimers.

65 ascular endothelial growth factor (VEGF) and placental growth factor (PlGF) in animal and human model

66 d the mRNA expression of VEGF receptor-1 and placental growth factor (PLGF) in HEECs.

67 The role of placental growth factor (PlGF) in modulation of tumor an

68 ed transgenic, conditional overexpression of placental growth factor (PlGF) in murine cardiac tissues

69 ution of isoforms of VEGF and of the related placental growth factor (PlGF) in the body and resulting

70 athway, decreased the expression of VEGF and Placental growth factor (PLGF) in TNBCs, and inhibited t

71 Placental growth factor (PlGF) is a critical mediator of

72 Placental growth factor (PlGF) is a member of the proang

73 Placental growth factor (PlGF) is a member of the vascul

74 Placental growth factor (PlGF) is an angiogenic factor i

75 Placental growth factor (PlGF) is an angiogenic factor,

76 Placental growth factor (PLGF) is an important arterioge

77 luble fms-like tyrosine kinase 1 (sFlt-1) to placental growth factor (PlGF) is elevated in pregnant w

78 Here, we report that placental growth factor (PlGF) is expressed in the major

79 We report that placental growth factor (PlGF) negatively affects the en

80 and treatment study was set up, blocking the placental growth factor (PlGF) or vascular endothelial g

81 ascular endothelial growth factor (VEGF) and placental growth factor (PlGF) plays a crucial role in b

82 Placental growth factor (PlGF) remodels tumor vasculatur

83 al circuit to enhance splenic SNS and induce placental growth factor (PlGF) secretion.

84 labeling with anti-PECAM1 antibody and anti-placental growth factor (PlGF) showed high levels of PlG

85 flk-1 kinase inhibitor SU1498 and failure of placental growth factor (PlGF) to up-regulate DAF confir

86 uble fms-like tyrosine kinase-1 (sFlt-1) and placental growth factor (PlGF) were measured at enrolmen

87 Up-regulation of placental growth factor (PlGF), a member of the vascular

88 evidence that a monoclonal antibody against placental growth factor (PlGF), a member of the VEGF fam

89 se 1 (sFlt1), an antiangiogenic protein, and placental growth factor (PlGF), a proangiogenic protein,

90 mbined with microarray analysis, to identify Placental Growth Factor (PlGF), a Vascular Endothelial G

91 pported by our finding that tumor-associated placental growth factor (PlGF), a VEGFR1-specific agonis

92 Placental growth factor (PlGF), abundantly produced from

93 nowledge of the circulating concentration of placental growth factor (PlGF), an angiogenic factor, in

94 EGF), basic fibroblast growth factor (bFGF), placental growth factor (PlGF), and monocyte chemoattrac

95 ved, but the precise role of VEGF-A, VEGF-B, placental growth factor (PlGF), and their receptors VEGF

96 estigated vitreous protein levels, including placental growth factor (PlGF), angiopoietin-2 (ANG2) an

97 factor family (VEGFA, VEGFB, and VEGFC), and placental growth factor (PlGF), in addition to their rec

98 kinase 1 (sFlt1), an antagonist of VEGF and placental growth factor (PlGF), is upregulated in preecl

99 whereas B16F10 cells secrete high levels of placental growth factor (PLGF), LLC cells produce high l

100 showed that cardiac angiogenesis induced by placental growth factor (PlGF), promotes myocardial hype

101 tion factor (GDF)-15, myeloperoxidase (MPO), placental growth factor (PlGF), soluble fms-like tyrosin

102 ated dilatation, and serum concentrations of placental growth factor (PlGF), soluble fms-like tyrosin

103 in early gestation expressed VEGF-A, VEGF-C, placental growth factor (PlGF), VEGFR-1, and VEGFR-3 and

104 Placental growth factor (PlGF), which signals through VE

105 thelial growth factor (VEGF)-related factor, placental growth factor (PlGF),has been shown recently t

106 Placental growth factor (PlGF)-based testing has high di

107 family that includes VEGFB, VEGFC, VEGFD and placental growth factor (PLGF).

108 ascular endothelial growth factor (VEGF) and placental growth factor (PlGF).

109 diates cell migration in response to VEGF or placental growth factor (PLGF).

110 luble fms-like tyrosine kinase 1 (sFlt1) and placental growth factor (PlGF)] in predicting short-term

111 EGF-C, soluble VEGF receptor [sVEGFR]-3, and placental growth factor [PlGF]) levels were measured.

112 tio of soluble fms-like tyrosine kinase 1 to placental growth factor positively correlated with blood

113 known as Flt1)-cognate receptor for VEGF and placental growth factor-prevented BMDC infiltration in l

114 On cytokine profiling, LDA increased placental growth factor production and selectively resto

115 esponse 1, soluble guanylate cyclase 1A3 and placental growth factor protein.

116 otein/creatinine ratio >0.30 g/g, and sFlt-1/placental growth factor ratio >85).

117 omes, and soluble fms-like tyrosine kinase 1/placental growth factor ratio, an index of placental dys

118 comes nor soluble fms-like tyrosine kinase 1/placental growth factor ratio.

119 1 (soluble fms-like tyrosine kinase 1)/PlGF (placental growth factor) ratio.

120 xogenous VEGF or the VEGFR1-specific ligand, placental growth factor, revealed distinct roles of VEGF

121 he plasma soluble fms-like tyrosine kinase-1/placental growth factor (sFlt-1/PlGF) ratio.

122 r(7), the soluble fms-like tyrosine kinase 1:placental growth factor (sFLT1:PlGF) ratio (AUC 0.78 ver

123 ngiopoietin 2, interleukin 6, interleukin 8, placental growth factor, soluble TIE-1, soluble VEGFR1,

124 onic anhydrase IX, hepatocyte growth factor, placental growth factor, stromal cell-derived factor 1al

125 ury molecule 1, tyrosine-protein kinase Mer, placental growth factor, thrombomodulin, and TNF recepto

126 We found one gene, PGF (placental growth factor), to be differentially expressed

127 sVEGFR2 showed the largest decrease, whereas placental growth factor underwent the largest increase.

128 r; platelet-derived growth factor AA and BB; placental growth factor; vascular endothelial growth fac

129 t only VEGF-A but also VEGF homologues (e.g. placental growth factor, VEGF-B, and VEGF-C), which may

130 tio of soluble fms-like tyrosine kinase 1 to placental growth factor was independently associated wit

131 Neither placental growth factor, which activates Flt1, epidermal

132 In contrast, overexpression of placental growth factor, which signals through Flt1 but

133 that binds all isoforms of VEGF-A as well as placental growth factor with high affinity, was administ