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1 als but is a key step in the biosynthesis of plant sterols.
2  hereditary phytosterolemia and were rich in plant sterols.
3 disease is characterized by increased plasma plant sterols.
4 uts, while northern detritus was enriched in plant sterols.
5 erol absorption because it is independent of plant sterols.
6  ABCG5/G8 knockout mice a diet enriched with plant sterols.
7  absorption and promote excretion of dietary plant sterols.
8 f odds (LOD) score of 9.9, designated plasma plant sterol 14 (Plast14).
9 supplementation with orange juice containing plant sterols (2 g/d) significantly reduced LDL choleste
10 -binding cassette transporters G5/8 regulate plant sterol absorption and also the secretion into bile
11 The aim of this study was to examine whether plant sterols affect CRP concentrations and the lipoprot
12 y sterols and decreased hepatic excretion of plant sterols and cholesterol.
13 Eskimo woman had consumed a diet very low in plant sterols and moderate to low in cholesterol content
14 ol absorption is a selective process in that plant sterols and other non-cholesterol sterols are abso
15  by very high levels of sitosterol and other plant sterols and premature atherothrombotic vascular di
16 itro and ex vivo studies have suggested that plant sterols and stanols can shift the T helper (Th) 1/
17                                              Plant sterols and steroid hormones, the brassinosteroids
18 micronutrients such as copper and magnesium, plant sterols, and phytochemicals.
19 acebo, fish oil, cinnamon, garlic, turmeric, plant sterols, and red yeast rice.
20 e these new data support the conclusion that plant sterols are excluded from the body because they ar
21                                 In contrast, plant sterols are relatively poorly esterified by ACAT,
22                          It is not known why plant sterols are so assiduously excluded from the body.
23                                              Plant sterols are structural components of cell membrane
24                                              Plant sterols are synthesized via the isoprenoid pathway
25 -400 mg of non-cholesterol sterols, of which plant sterols are the major constituents.
26 timibe inhibits the intestinal absorption of plant sterols as well as cholesterol, leading to reducti
27 omplex (G5G8) that opposes the absorption of plant sterols but is also expressed in liver where it pr
28 d hepatobiliary secretion of cholesterol and plant sterols by 1.5-2-fold, increased the amount of int
29            The reason for poor absorption of plant sterols by the body is still unknown.
30  altered substrate specificity: transport of plant sterols by the heterodimer was preserved, whereas
31    Respectively, cholestanol/cholesterol and plant sterols campesterol/cholesterol and sitosterol/cho
32 nate, p < 0.001; linolenate, p < 0.001), and plant sterols (campesterol, p < 0.001; brassicasterol, p
33                        One theory holds that plant sterols can directly promote atherosclerosis, but
34                            The side chain in plant sterols can have either a methyl or ethyl addition
35 ificant and progressive reductions in plasma plant sterol concentrations in patients with sitosterole
36  effective in milligram doses, lowers plasma plant sterol concentrations in sitosterolemic subjects,
37                                        Serum plant sterol concentrations increased from baseline (0.4
38                                Reductions in plant sterol concentrations were similar irrespective of
39 complexity of grapevine, we investigated the plant sterol content of berry and seed tissues at pre-ve
40 s to reduce pro-inflammatory fatty acids and plant sterol content, can lower the risk of IFALD, rever
41 phenolics, carotenoids, coumarins, saponins, plant sterols, curcumins, and phthalides have been ident
42 mo ligands previously identified include the plant sterol cyclopamine (and its therapeutically useful
43 the output of Americans consuming 250-400 mg plant sterols/d.
44 n Abcg5- and Abcg8-deficient mice fed a high plant sterol diet resulted in accumulation of free stero
45    These data indicate that selected dietary plant sterols disrupt cholesterol homeostasis by affecti
46  results are discussed within the context of plant sterol distribution and metabolism.
47                                     However, plant sterols do not exchange with UC and are secreted i
48 ied the degradation of cholesterol and three plant sterols during a 360 min heating treatment (180 de
49 e demonstrate that both stereoisomers of the plant sterol, (E)- and (Z)-GS, bind to the steroid recep
50                                      Dietary plant sterols effectively reduce LDL cholesterol when in
51                              The interest in plant sterols enriched foods has recently enhanced due t
52                                              Plant sterol-enriched functional foods are widely used f
53 f stearidonic acid (SDA) and 0.65 g/100mL of plant sterol esters (PSE) were prepared without or with
54              A reduced-fat spread containing plant sterol esters incorporated into a low-fat diet is
55                                              Plant sterol esters reduce cholesterol absorption and lo
56         When challenged with stigmasterol, a plant sterol found in intravenous soy lipids, lipid accu
57 type (WT) littermates were challenged with a plant sterol-free low fat or high fat (HF) diet.
58 p promoting active efflux of cholesterol and plant sterols from enterocytes back into the intestinal
59  heterodimer that transports cholesterol and plant sterols from hepatocytes into bile.
60 e appear to hyperabsorb both cholesterol and plant sterols from the intestine.
61                                          The plant sterol guggulsterone (GS) is the active agent in t
62                                          The plant sterol guggulsterone [4,17(20)-pregnadiene-3,16-di
63 ults for the first time demonstrate that the plant sterol guggulsterone suppresses ocular inflammatio
64  either VLDL or LDL there was no increase in plant sterols in bile, but with perfusion of HDL, the se
65 and G8 are known to cause an accumulation of plant sterols in blood and tissues (sitosterolemia).
66                                       Plasma plant sterols in F(2)s displayed a unimodal distribution
67 ped a phenotype that included high levels of plant sterols in many tissues, liver abnormalities, and
68            Here we show that accumulation of plant sterols in mice lacking ABCG5 and ABCG8 (G5G8-/- m
69 up to 27%, and decreased the accumulation of plant sterols in plasma by approximately 25%.
70  protect against the accumulation of dietary plant sterols in plasma.
71      There was a significant accumulation of plant sterols in the brains of the Pdgfb(ret/ret) mice.
72 in which patients accumulate cholesterol and plant sterols in the circulation and develop premature C
73 s with this disease have very high levels of plant sterols in the plasma and develop tendon and tuber
74 s with this disease have very high levels of plant sterols in the plasma, and develop tendon and tube
75            Increased serum concentrations of plant sterols, including stigmasterol, during parenteral
76  Components of PN lipid emulsions, including plant sterols, interact with hepatic innate immune activ
77                             Intercalation of plant sterols into the plasma membrane therefore results
78 ced-calorie orange juice beverage containing plant sterols is effective in reducing CRP and LDL chole
79 hich serve numerous biochemical functions in plants: sterols (isoprenoids with a C30 backbone) are es
80 fected individuals presented elevated plasma plant sterol levels (mean beta-Sitosterol and campestero
81 nd ABO have been associated with circulating plant sterol levels and CVD, thereby suggesting atheroge
82  from this disease have very elevated plasma plant sterol levels and develop tendon and tuberous xant
83 er genes besides ABCG5ABCG8 influence plasma plant sterol levels and now become candidates to explain
84  candidates to explain differences in plasma plant sterol levels between humans.
85                                       Plasma plant sterol levels differ among humans due to genetic a
86 inhibitor of cholesterol absorption, reduces plant sterol levels in patients with sitosterolemia.
87 cross between F(1)s was performed and plasma plant sterol levels measured in 102 male and 99 female F
88 take yields a 2-fold increase in circulating plant sterol levels that equally represent markers of ch
89       A disease characterized by high plasma plant sterol levels, beta-sitosterolemia, was recently f
90  analysis of these mutants has revealed that plant sterols may be key signaling molecules influencing
91 evated cholesterol absorption rather than by plant sterols may therefore mediate the relationships of
92 no acid identity to recently isolated higher-plant sterol methyltransferases from soybean and Arabido
93 anges, we examined the effects of individual plant sterols on cholesterol metabolism in cultured adre
94  there is a paucity of data on the effect of plant sterols on CRP concentrations.
95  study evaluated the influence of esterified plant sterols on serum lipid concentrations in adults wi
96 D, thereby suggesting atherogenic effects of plant sterols or of cholesterol uptake.
97                                              Plant sterols, or phytosterols, are very similar in stru
98 acebo, fish oil, cinnamon, garlic, turmeric, plant sterols, or red yeast rice.
99                                          The plant sterol output in the coprolite was only 0.4% of th
100                                              Plant sterols (phytosterols) and the drug ezetimibe redu
101                                           In plants, sterols play fundamental roles as membrane const
102                           We analyzed plasma plant sterol (PPS) levels, a surrogate measure of choles
103                              Cholestanol and plant sterols provide a measure of cholesterol absorptio
104                                              Plant sterol (PS) oxidation products (POP) derived from
105                                              Plant sterol (PS) supplementation is increasingly accept
106 ion method on sterol bioaccessibility from a plant sterol (PS)-enriched beverage.
107                                        Three plant sterol (PS)-enriched beverages, milk based fruit j
108                                  Benefits of plant sterols (PS) for cholesterol lowering are compromi
109 letate and linolenate) on a mixture of three plant sterols (PS: campesterol, stigmasterol and beta-si
110                              The benefits of plant sterols (PSs) for cholesterol lowering are hampere
111                                              Plant sterols (PSs) lower LDL cholesterol, an establishe
112                     The consumption of 2 g/d plant sterols (PSs) reduces circulating LDL cholesterol
113 inal domain similar in sequence to yeast and plant sterol reductases.
114 porter cause accumulation of cholesterol and plant sterols, resulting in premature coronary atheroscl
115                           To determine which plant sterol(s) caused the metabolic changes, we examine
116                             However, biliary plant sterol secretion was markedly different: with the
117 cid ursodeoxycholic acid (p = 0.033) and the plant sterol sitostanol (p = 0.003) were significantly d
118                       Using the unesterified plant sterol, sitostanol (SIT), as a nonexchangeable ana
119 CAT) 2 to differentiate cholesterol from the plant sterol, sitosterol, was compared with that of the
120 surrogate markers of cholesterol absorption (plant sterols: sitosterol, campesterol) and synthesis (c
121 l-related molecules (cholesterol precursors, plant sterols, some oxysterols, cholesterol sulfate, cho
122                                    The major plant sterol species is sitosterol; hence the name of th
123 ools of cholesterol and very elevated plasma plant-sterol species and frequently develop tendon and t
124 C22 in the isoprenoid side chain such as the plant sterol stigmasterol.
125                                        Serum plant sterols (stigmasterol, avenasterol, sitosterol, an
126                             Phytosterols are plant sterols structurally similar to cholesterol that a
127                              The GenePredict Plant Sterol study (GPS) was designed to validate associ
128 lemented by exogenous stigmasterol, the main plant sterol, suggesting that sterols are required for T
129                                              Plant sterol supplementation produces minimal increases
130  sterols but seems to have lower affinity to plant sterols than cholesterol.
131 similarly large for each of three individual plant sterols that was tracked.
132           The ratio of plasma campesterol (a plant sterol) to lathosterol (a cholesterol precursor) w
133 lacebo Bev) or with (1 g/240 mL; Sterol Bev) plant sterols twice a day with meals for 8 wk.
134                      The increase in biliary plant sterols was detected 5-10 min after HDL was added
135  but with perfusion of HDL, the secretion of plant sterols was increased two- to threefold (P = 0.000
136                                        Serum plant sterols were higher in patients who were currently
137                    In Sr-bI/Abcg5 dko plasma plant sterols were highest, while hepatic plant sterols
138 ma plant sterols were highest, while hepatic plant sterols were lower compared with Abcg5 ko (P < 0.0
139 lating perfusions, hepatic concentrations of plant sterols were not different after different lipopro
140 ease in the fractional absorption of dietary plant sterols, which was associated with an approximatel

 
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