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1 at resulted in reduced specific force of the plantaris.
2 wing an acute hypertrophic stimulus in mouse plantaris.
3 ensor digitorum longus (13-fold over basal), plantaris (5.8-fold), red gastrocnemius (4.7-fold), whit
4  a basal defect in total fiber number in the plantaris and a mild secondary reduction in growth, cons
5 OCS-3 mRNA expression by 80% and 154% in the plantaris and soleus muscle, respectively.
6 hat IL-6 mRNA expression was elevated in the plantaris and soleus muscles of the trained animals comp
7 gression altered the Ox-PTMs profile in both plantaris and soleus.
8  45%, 43%, and 58% in the tibialis anterior, plantaris, and diaphragm muscles, respectively.
9 tion, and diabetes led to atrophy of soleus, plantaris, and gastrocnemius muscles, but only unloaded
10  bait in a yeast one-hybrid screen of an MOV-plantaris cDNA library, we identified nominal transcript
11 A) from nerve and muscle during normal quail plantaris development dramatically changed the normal fa
12  phosphorylation were measured in overloaded plantaris from both WT and MKR mice, respectively.
13 tinction of the musculotendinous unit of the plantaris from the remaining muscles of the lower extrem
14 eine Ox-PTMs profile in the proteome of both plantaris (glycolytic) and soleus (oxidative) muscles in
15 duction (P < 0.05) in absolute growth of the plantaris in response to overload in HFD mice vs. LFD mi
16 structure has been found, for which the name plantaris ligamentous tendon is proposed.
17 viously undetected anatomical structure, the plantaris ligamentous tendon, and to determine its frequ
18 increases of 100% and 122%, respectively, in plantaris mass (P < 0.05).
19 after 4 days of reloading, during which time plantaris mass also returned to control values.
20                          After 7 days of FO, plantaris mass increased significantly by 26% and 62% in
21      In control or unchallenged animals, the plantaris mass was 11% greater in WT compared to the MKR
22 s of unloading resulted in a 16% decrease in plantaris mass, a 110% increase in myostatin mRNA, and a
23       In contrast, removal of PSA from chick plantaris motoneurons and muscle fibers had little effec
24 -quail hindlimb chimeras to force slow chick plantaris motoneurons to innervate a fast quail plantari
25 /T-rich element with mechanically overloaded plantaris (MOV-P) nuclear extract detected two proteins
26 satellite cell and myonuclei abundance of PU plantaris muscle after PoWeR was not observed in PT.
27 n-stimulated p38 MAPK phosphorylation in the plantaris muscle and Akt phosphorylation in both muscles
28  and old animals, but less consistent in old plantaris muscle and liver.
29                          Overloading the rat plantaris muscle by synergist muscle ablation, which pro
30                Overload was induced in mouse plantaris muscle by unilateral synergist ablation.
31 binding increased with nuclear extracts from plantaris muscle exposed to mechanical overload, a stimu
32 phosphorylation as main affected pathways in plantaris muscle from tumor-bearing rats, while the same
33 On the seventh day, the gastrocnemius-soleus-plantaris muscle group was isolated and snap frozen, or
34 e used a functional overload model to induce plantaris muscle hypertrophy by surgically removing the
35 nctional overload-induced hypertrophy of the plantaris muscle in mice and during differentiation of p
36 cessary for skeletal muscle hypertrophy, the plantaris muscle of adult Pax7-DTA mice was subjected to
37 eases in autophagy protein expression in the plantaris muscle of sedentary muscle-specific Pgc-1alpha
38                   The fibre-type shift of PU plantaris muscle to a more oxidative type 2a fibre compo
39 eletal muscle hypertrophy was induced in the plantaris muscle using the functional overload (FO) mode
40 rload induced progressive hypertrophy of the plantaris muscle which was associated with significant i
41 3 in parallel to mitochondrial biogenesis in plantaris muscle with mixed fiber types.
42 ical ablation of the synergic muscles of the plantaris muscle, a fast muscle susceptible to contracti
43 ive stress associated with muscle atrophy in plantaris muscle, but not in soleus.
44 rease of Pgc-1alpha mRNA expression in mouse plantaris muscle, concurrent with an activation of the p
45                                           In plantaris muscle, glycolytic type IId/x and IIb, but not
46  ablation, which promotes hypertrophy of the plantaris muscle, increased Ser(2448) phosphorylation.
47             In the predominantly fast-twitch plantaris muscle, insulin-stimulated NKCC activity becam
48 mentous tendon from the superior part of the plantaris muscle, the posterior surface of the femur and
49 curs around the popliteal region between the plantaris muscle, the posterior surface of the femur, an
50 the soleus muscle and type IIa fibers in the plantaris muscle, with corresponding increases in interm
51 revealed muscle atrophy in type II fibers in plantaris muscle, with no changes in plantaris type I fi
52 or bilateral functional overload (FO) of the plantaris muscle.
53 ntaris motoneurons to innervate a fast quail plantaris muscle.
54 e primary myosin isoform was observed in the plantaris muscle.
55  of PoWeR-induced myonuclei accretion in the plantaris muscle.
56 paired PoWeR-induced fibre-type shift in the plantaris muscle.
57 ate synthase activity in both the soleus and plantaris muscles (26.2 +/- 1.6 versus 30.7 +/- 3.4 and
58 ired growth in 1 week overloaded fast-twitch plantaris muscles (via unilateral gastrocnemius ablation
59                     Like the soleus muscles, plantaris muscles from Nfkb1(-/-) and Bcl3(-/-) mice als
60 ear moduli for the lateral gastrocnemius and plantaris muscles in a 7-T MR imager, from which the mec
61  type-specific hypertrophy in the soleus and plantaris muscles in response to progressive weighted wh
62 nsity, and endplate morphology in denervated plantaris muscles in wild-type and MMP3 null mice.
63 hereas fast glycolytic tibialis anterior and plantaris muscles underwent atrophy (11.6 and 13.3%, res
64 up Ia muscle afferents from triceps surae or plantaris muscles were labeled intraaxonally with horser
65 sms regulating NKCC activity, rat soleus and plantaris muscles were stimulated ex vivo by insulin or
66 ific activity measured in control soleus and plantaris muscles when compared with wild type transgene
67 e used RNA-seq to analyse gene expression in plantaris muscles while monitoring respiration, arterial
68 and morphological analyses of the soleus and plantaris muscles, and Northern analyses of muscle contr
69 hing pattern normally observed in chick slow plantaris muscles.
70 the betaA/T-rich element is reacted with MOV-plantaris nuclear extract.
71 of intramuscular nerve branching in the fast plantaris of these chimeras closely resembled the slow b
72 s highly enriched only when using either MOV plantaris or control soleus nuclear extract.
73 roteins within the LMC when using either MOV plantaris or control soleus nuclear extracts were antige
74 reased, whereas in the shortening soleus and plantaris (PLN) muscles the increase was significantly l
75 trocnemius (GA), but not the soleus (SOL) or plantaris (PLT) muscles, of D14 mice.
76 ation of fat herniation (P =.051) and of the plantaris tendon (P =.098) demonstrated marginal correla
77 pture in 30 patients (21.3%), rupture of the plantaris tendon in two patients (1.4%), and partial rup
78 study evaluated the longitudinal response to plantaris tendon overload using the synergist ablation m
79 al, structural and cellular responses due to plantaris tendon overload using the synergistic ablation
80 ry in response to synergist ablation-induced plantaris tendon overuse.
81 in adult mice via mechanical overload of the plantaris tendon.
82 e appear to be more common than those of the plantaris tendon.
83 id mediators were detected in homogenates of plantaris tendons from ambulatory control rats.
84 pertrophy of soleus type 1 and 2a fibres and plantaris type 2b/x fibres compared to PU.
85 bers in plantaris muscle, with no changes in plantaris type I fibers and no differences in both soleu
86 n vivo Myc-controlled gene expression in the plantaris was defined using a genetic muscle fiber-speci
87 st muscles places functional overload on the plantaris, was used to stimulate robust hypertrophy.
88 hindlimb muscles (gastrocnemius, soleus, and plantaris) were evaluated in mice after completing a 6-w