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1 or bilateral functional overload (FO) of the plantaris muscle.
2 ntaris motoneurons to innervate a fast quail plantaris muscle.
3 e primary myosin isoform was observed in the plantaris muscle.
4  of PoWeR-induced myonuclei accretion in the plantaris muscle.
5 paired PoWeR-induced fibre-type shift in the plantaris muscle.
6 hing pattern normally observed in chick slow plantaris muscles.
7 ate synthase activity in both the soleus and plantaris muscles (26.2 +/- 1.6 versus 30.7 +/- 3.4 and
8 ical ablation of the synergic muscles of the plantaris muscle, a fast muscle susceptible to contracti
9 satellite cell and myonuclei abundance of PU plantaris muscle after PoWeR was not observed in PT.
10 n-stimulated p38 MAPK phosphorylation in the plantaris muscle and Akt phosphorylation in both muscles
11  and old animals, but less consistent in old plantaris muscle and liver.
12 and morphological analyses of the soleus and plantaris muscles, and Northern analyses of muscle contr
13 ive stress associated with muscle atrophy in plantaris muscle, but not in soleus.
14                          Overloading the rat plantaris muscle by synergist muscle ablation, which pro
15                Overload was induced in mouse plantaris muscle by unilateral synergist ablation.
16 rease of Pgc-1alpha mRNA expression in mouse plantaris muscle, concurrent with an activation of the p
17 binding increased with nuclear extracts from plantaris muscle exposed to mechanical overload, a stimu
18 phosphorylation as main affected pathways in plantaris muscle from tumor-bearing rats, while the same
19                     Like the soleus muscles, plantaris muscles from Nfkb1(-/-) and Bcl3(-/-) mice als
20                                           In plantaris muscle, glycolytic type IId/x and IIb, but not
21 On the seventh day, the gastrocnemius-soleus-plantaris muscle group was isolated and snap frozen, or
22 e used a functional overload model to induce plantaris muscle hypertrophy by surgically removing the
23 nctional overload-induced hypertrophy of the plantaris muscle in mice and during differentiation of p
24 ear moduli for the lateral gastrocnemius and plantaris muscles in a 7-T MR imager, from which the mec
25  type-specific hypertrophy in the soleus and plantaris muscles in response to progressive weighted wh
26 nsity, and endplate morphology in denervated plantaris muscles in wild-type and MMP3 null mice.
27  ablation, which promotes hypertrophy of the plantaris muscle, increased Ser(2448) phosphorylation.
28             In the predominantly fast-twitch plantaris muscle, insulin-stimulated NKCC activity becam
29 cessary for skeletal muscle hypertrophy, the plantaris muscle of adult Pax7-DTA mice was subjected to
30 eases in autophagy protein expression in the plantaris muscle of sedentary muscle-specific Pgc-1alpha
31 mentous tendon from the superior part of the plantaris muscle, the posterior surface of the femur and
32 curs around the popliteal region between the plantaris muscle, the posterior surface of the femur, an
33                   The fibre-type shift of PU plantaris muscle to a more oxidative type 2a fibre compo
34 hereas fast glycolytic tibialis anterior and plantaris muscles underwent atrophy (11.6 and 13.3%, res
35 eletal muscle hypertrophy was induced in the plantaris muscle using the functional overload (FO) mode
36 ired growth in 1 week overloaded fast-twitch plantaris muscles (via unilateral gastrocnemius ablation
37 up Ia muscle afferents from triceps surae or plantaris muscles were labeled intraaxonally with horser
38 sms regulating NKCC activity, rat soleus and plantaris muscles were stimulated ex vivo by insulin or
39 ific activity measured in control soleus and plantaris muscles when compared with wild type transgene
40 rload induced progressive hypertrophy of the plantaris muscle which was associated with significant i
41 e used RNA-seq to analyse gene expression in plantaris muscles while monitoring respiration, arterial
42 3 in parallel to mitochondrial biogenesis in plantaris muscle with mixed fiber types.
43 the soleus muscle and type IIa fibers in the plantaris muscle, with corresponding increases in interm
44 revealed muscle atrophy in type II fibers in plantaris muscle, with no changes in plantaris type I fi