戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 of the disease in the presence of inadequate plaque control.
2         Patients were seen every 2 weeks for plaque control.
3 n of primary dentition, diastemata, and poor plaque control.
4 aintenance periods were designed to optimize plaque control.
5 h standard periodontal treatment emphasizing plaque control.
6  be a useful herbal formulation for chemical plaque control agents and improvement in plaque and ging
7                  Both groups showed improved plaque control and BOP scores.
8 ngivitis was then induced, with cessation of plaque control and institution of a soft diet over 8 wee
9 groups nearly always used patient education, plaque control and mechanical debridement when treating
10 treatment group (ETG) (n = 82) that received plaque control and root planing plus amoxicillin and met
11 oot planing and systemic antibiotics or with plaque control and subgingival scaling significantly red
12                                              Plaque control did not modulate this pathogenic pattern,
13 bgingival margins of composites present poor plaque control, enhanced biofilm accumulation, and cause
14 al cleaning is an integral component of home plaque control for periodontally involved patients, limi
15 sociated with defect morphology, compliance, plaque control, inflammation, bacterial colonization, an
16 treatment group (CTG) (n = 83) that received plaque control instructions, supragingival scaling, and
17 e used as an effective adjunct to mechanical plaque control is needed.
18 to prevent attachment loss (AL) when optimal plaque control is present.
19                                  However, if plaque control is suboptimal, a minimum of 2 mm of KT is
20                                     Chemical plaque control is the most commonly recommended means of
21  periodicity is inadequate, or if mechanical plaque control is unrealistically good--that is, unlikel
22                       A combined approach of plaque control measures along with anti-inflammatory med
23 next placed around the healing-abutments and plaque control measures were abandoned.
24                                              Plaque control measures were followed with anti-inflamma
25 e inserted in each socket, and postoperative plaque control measures were undertaken.
26  was to evaluate the effect of supragingival plaque control on the recurrence of periodontitis (RP) a
27                                   Inadequate plaque control, peri-implant inflammation, history of pr
28 dement of the second molar and a three-visit plaque control program may have a beneficial effect on P
29 after healing-abutment connection, a 2-month plaque control program was initiated.
30                                          The plaque control record (O'Leary index) appears to be a co
31 g depth (PD), bleeding on probing (BOP), and plaque control record (PlaCR) were recorded.
32                   Bleeding on probing (BOP), plaque control record, body weight (BW), visceral fat (F
33 n probing (primary outcome), gingival index, plaque control record, probing attachment level, and pro
34                                  A stringent plaque control regimen was enforced in all the patients
35                                  A stringent plaque-control regimen was enforced in all subjects duri
36 vels compared to the study subjects' regular plaque control routines (1.3 +/- 0.04; P < 0.05).
37 cted at more ergonomic and economic means of plaque control that have the efficacy of traditional met
38  dental implants are performed to facilitate plaque control, to improve patient comfort, to prevent f