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1 ack caveolae, have increased permeability to plasma albumin.
2 nduction, with corresponding improvements in plasma albumin.
3 ehicle-treated mice, p < 0.05; and increased plasma albumin-adjusted calcium to 2.03 +/- 0.02 mmol/l
4 ng to HUVECs 100% and 50%, respectively, and plasma albumin and other proteins prevent a sufficient l
5 ased plasma albumin and the relation between plasma albumin and the edema of protein-energy malnutrit
6 he kinetic changes responsible for decreased plasma albumin and the relation between plasma albumin a
7 owed that dialysate electrolyte composition, plasma albumin, and plasma total CO2 accurately predict
8  that at physiological NO(.) concentrations, plasma albumin becomes saturated with NO(.) and accelera
9 enhances the endocytosis and transcytosis of plasma albumin by podocytes, which may eventually impair
10                                    In HUVEC, plasma albumin caused a sustained decrease in [Ca2+]i fr
11 ood-to-tissue albumin transport, measured as plasma albumin clearance corrected for intravascular vol
12 ally thought to stem from the prevailing low plasma albumin concentration and the decreased transcapi
13 oncentration, which represents the fact that plasma albumin concentration does not reflect its functi
14 ortened in FcRn-deficient mice, and that the plasma albumin concentration of FcRn-deficient mice is l
15 n increased plasma, BAL albumin, and the BAL:plasma albumin concentration ratio.
16 etabolite were associated with drug dose and plasma albumin concentration, and were lower among men a
17             Albumin administration increased plasma albumin concentrations (p <.05 compared with plac
18 y 1 there were no significant differences in plasma albumin concentrations in nonedematous and edemat
19 PN, for which Hpn knockout mice manifest low plasma albumin concentrations.
20                                              Plasma albumin, for example, divides into 15-17 clusters
21 e septic rats and three controls after their plasma albumin had been labeled with Evans blue dye.
22 opathy, and proteinuria, along with improved plasma albumin in the puromycin aminonucleoside glomerul
23            The decrease in [Ca2+]i caused by plasma albumin is due to an uptake into intracellular st
24 lucocorticoid doses, and in those with lower plasma albumin level.
25                     The effects of serum and plasma albumin on [Ca2+]i in human endothelial cells wer
26                      Because modification of plasma albumin on tyrosine residues generates nitrated a
27                                              Plasma albumin permeation into the joint cavity was also
28 as assessed using the cerebrospinal fluid-to-plasma albumin ratio (CPAR).
29 xy-4-hydroxyphenylethyleneglycol (MHPG), CSF/plasma albumin ratio (Q-alb), AB, phosphorylated tau, an
30 xy-4-hydroxyphenylethyleneglycol (MHPG), CSF/plasma albumin ratio (Q-alb), Abeta, phosphorylated tau,
31 004), protein (r = 0.59; P < .0001), and CSF/plasma albumin ratio (r = 0.60; P < .0001).
32 BBB dysfunction (plasma PDGFRbeta and CSF-to-plasma albumin ratio).
33 duration, all P < .05), and an increased CSF:plasma albumin ratio, a marker of blood-brain barrier br
34 t is mediated by covalent binding of iRGD to plasma albumin through a disulfide bond.
35                                 No effect of plasma albumin was observed in ECV304 cells.
36 docytopathy, podocytopenia, proteinuria, and plasma albumin were measured.
37                 The results show that normal plasma albumin, which carries few lipids, lowers [Ca2+]i