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1 y Score, prehospital Glasgow Coma Scale, and plasma catecholamines.
2 potension was associated with an increase in plasma catecholamines.
3                           Elevated levels of plasma catecholamines accompany ischemic AKI, possibly c
4 e setting of insulinopenia through increased plasma catecholamine and corticosterone concentrations s
5 in the chromaffin granule; and (g) increased plasma catecholamine and Npy levels.
6 first 2 hrs after traumatic brain injury and plasma catecholamine and serum fentanyl concentrations m
7  bradycardic response was prolonged, and the plasma catecholamine and vasopressin responses were dimi
8 sone treatment, femoral vasoconstriction and plasma catecholamine and vasopressin responses were enha
9 lar resistances, ECG, serum cardiac enzymes, plasma catecholamines and atrial natriuretic factor, and
10 ng traumatic brain injury, and elevations in plasma catecholamines and cortisol portend a poor outcom
11 flex impairment, and sympathetic activation (plasma catecholamines and heart rate variability indexes
12 acute hypoxaemia-induced elevations in fetal plasma catecholamines and suppression of glucose-stimula
13  evaluated the acute drug-induced changes in plasma catecholamines and used these results as a basis
14       KO mice displayed hypertension, higher plasma catecholamine, and adipokine levels and lower IL-
15 easured postural blood pressure, heart rate, plasma catecholamines, and systemic norepinephrine spill
16                     Indirect indices such as plasma catecholamines are transiently elevated after unc
17 rences in baseline hemodynamic parameters or plasma catecholamines between the 2 groups.
18 eart rate, systemic arterial blood pressure, plasma catecholamine, clonidine, interleukin-6 concentra
19                               We manipulated plasma catecholamines (combined adrenaline and noradrena
20 ons in BP during GB were related to baseline plasma catecholamine concentrations (r=-0.31 to -0.41, P
21  of this study is that increases in MSNA and plasma catecholamine concentrations did not differ betwe
22 muscle sympathetic nerve activity (MSNA) and plasma catecholamine concentrations in healthy young and
23                                              Plasma catecholamine concentrations increased 3-fold dur
24                                              Plasma catecholamine concentrations increased in both gr
25                             Manipulations of plasma catecholamine concentrations influence outcome fr
26 Thus, heart rate power spectral analysis and plasma catecholamine concentrations may prove to be usef
27             Women had lower baseline SBP and plasma catecholamine concentrations than men (P<0.05).
28                                              Plasma catecholamine concentrations were determined by h
29 w blood oxygen concentrations increase fetal plasma catecholamine concentrations, which lower fetal i
30 ry bypass (CPB) is associated with increased plasma catecholamine concentrations, which might worsen
31 fetuses have chronic hypoxaemia and elevated plasma catecholamine concentrations.
32 ensitization despite comparable elevation of plasma catecholamines during the development of heart fa
33 uscle sympathetic nerve activity (MSNA), and plasma catecholamines evoked by upright tilt in recurren
34                                              Plasma catecholamine, glucose, and insulin levels were m
35 onary thrombosis suggests that elevations of plasma catecholamines, high shear forces acting on the p
36 ed with the postural heart rate and abnormal plasma catecholamine homeostasis.
37                                              Plasma catecholamines in NET(-/-) and NET(+/+) mice were
38                                              Plasma catecholamines in newborn rats (0-2 hr old) were
39                                          All plasma catecholamines increased after ephedrine administ
40                                              Plasma catecholamines increased fourfold in the CHF grou
41 ts had faster heart rate (P < 0.001), higher plasma catecholamine levels (P = 0.020), lower end-tidal
42 However, the impact of exercise intensity on plasma catecholamine levels among HCM patients has not b
43 ration of recombinant human renalase reduced plasma catecholamine levels and ameliorated ischemic AKI
44 diac hypertrophy is associated with elevated plasma catecholamine levels and an increase in cardiac m
45             Gallein also reduced circulating plasma catecholamine levels and catecholamine production
46 ocorticoid-induced fetal hypertension, fetal plasma catecholamine levels and changes in fetal femoral
47                                              Plasma catecholamine levels at presentation were markedl
48            In conclusion, large increases in plasma catecholamine levels cause hyperthermia during ex
49                                              Plasma catecholamine levels in 13 patients with stress-r
50                                   During SI, plasma catecholamine levels increased progressively and
51 key contributors to the chronically elevated plasma catecholamine levels observed in HF, where adrena
52                                 We show that plasma catecholamine levels remain stably low at exercis
53   In patients with mild non-obstructive HCM, plasma catecholamine levels remain stably low at exercis
54                In +/+ but not in fa/fa rats, plasma catecholamine levels rose, and both P-STAT3 and P
55 tained plasma glucose levels above 11 mM and plasma catecholamine levels were 5.0-5.5 pmol ml-1 lower
56                   Heart rate variability and plasma catecholamine levels were assessed as proxies of
57 infused (AI) at 0.1 microgram kg-1 min-1 and plasma catecholamine levels were elevated 6 pmol ml-1 ab
58 necrosis, and apoptosis were more severe and plasma catecholamine levels were higher in renalase-defi
59                                              Plasma catecholamine levels were measured at baseline an
60 alyzed heart rate variability parameters nor plasma catecholamine levels were significantly different
61                                              Plasma catecholamine levels within and between groups di
62 indicated by elevated CB neural activity and plasma catecholamine levels, and elevated reactive oxyge
63 e, respiratory rate, mean arterial pressure, plasma catecholamine levels, and heart rate power spectr
64 ockade (with trimethaphan) on supine SBP and plasma catecholamine levels, and the effect of alpha(1)-
65 ation in heart failure, resulting in lowered plasma catecholamine levels, improved cardiac betaAR sig
66             Based on MDMA-induced changes in plasma catecholamine levels, rats were subjected to the
67 ot accompanied by corresponding increases in plasma catecholamine levels.
68 ardiomyopathy," is associated with increased plasma catecholamine levels.
69  relationship between exercise intensity and plasma catecholamine levels.
70                                              Plasma catecholamines levels 30 mins after MDMA (40 mg/k
71  diphenhydramine on depression, anxiety, and plasma catecholamine metabolites were assessed.
72 of depression, plasma tryptophan levels, and plasma catecholamine metabolites.
73 iovascular disease associated with increased plasma catecholamines, overactivation of the sympathetic
74 late device specifically designed to extract plasma catecholamines prior to their quantification by a
75                                              Plasma catecholamines provide a reliable biomarker of sy
76 ectrochemical detection method for measuring plasma catecholamines (R(2) > 0.85).
77 iated with a more than four-fold increase in plasma catecholamines, renin activity, and endothelin co
78 to others factors besides attenuation of the plasma catecholamine response to exercise.
79 , reduced baroreflex sensitivity, diminished plasma catecholamine responses to acute stress, and incr
80 nhanced baroreflex sensitivity and augmented plasma catecholamine responses to acute stress.
81 nts the effect of hypoglycemia to reduce the plasma catecholamine responses to subsequent hypoglycemi
82 e hypoxia (both P < 0.05), without affecting plasma catecholamine responses.
83  occur in the context of increased levels of plasma catecholamines, some critical mechanisms that gov
84                                            A plasma catecholamine surge was observed in every animal
85  resting and mental stress-induced levels of plasma catecholamines, tissue plasminogen activator anti
86                                              Plasma catecholamines (unchanged during euglycemia) rose
87 lasma cortisol levels exceeded 17.5 ng ml-1, plasma catecholamines were a major influence on fetal gl
88                                     Arterial plasma catecholamines were assessed at basal (-30 and 0
89                                     Arterial plasma catecholamines were assessed at basal (-30 and 0
90 ance, muscle sympathetic nerve activity, and plasma catecholamines were measured at baseline and at 3
91            Increased adrenal medulla-derived plasma catecholamines were necessary and sufficient to i
92                                   Changes in plasma catecholamines were not different between the CRT
93 al hormonal investigations include urine and plasma catecholamines with their metabolites, plasma cor