ライフサイエンスコーパス: plasma glucagon

1 ypoglycemia produced a threefold increase in plasma glucagon.

2 ively, P < 0.05 for both) and no increase in plasma glucagon.

3 increased ( approximately 10-fold) levels of plasma glucagon.

4 nd yet associates with dramatic increases in plasma glucagon.

5 ever, produced a more pronounced blunting of plasma glucagon, ACTH, and hepatic glucose production co

6 There was a prompt rise in plasma glucagon after intravenous arginine in all groups

7 as associated with a significant increase in plasma glucagon and a decrease in the plasma insulin con

8 ng antibody to healthy individuals increased plasma glucagon and amino acid levels, but did not chang

9 sociated with decreased food intake, reduced plasma glucagon and corticosterone concentrations, and d

10 only slightly, it fully normalized elevated plasma glucagon and corticosterone levels and reversed t

11 ose, plasma lipids, liver triglycerides, and plasma glucagon and enhanced pancreatic insulin content,

12 eduction in blood glucose and an increase in plasma glucagon and glucagon-like peptide (GLP-1).

13 OXA infusion increases plasma glucagon and glucose levels and decreases plasma

14 lerance in Pemt(-/-) mice through modulating plasma glucagon and its action in liver.

15 -/-) mice have elevated ( approximately 60%) plasma glucagon and reduced ( approximately 20%) plasma

16 lucose and was accompanied by lowered HbA1c, plasma glucagon, and triglyceride concentrations and exp

17 stion resulted in elevated serum insulin and plasma glucagon compared with FRU and MF (P < 0.001 for

18 fatty acid was accompanied by a fall in the plasma glucagon concentration from 122 to 85 pmol/L (P =

19 nd was accompanied by an increase in fasting plasma glucagon concentration.

20 results in higher plasma glucose and higher plasma glucagon concentrations after a mixed meal and af

21 sfunction, characterized by elevated fasting plasma glucagon concentrations and inadequate postprandi

22 In humans, postprandial lipemia increased plasma glucagon concentrations and led to an inadequate

23 We measured plasma glucagon concentrations in patients with type 1 d

24 and mice, chronic physiological increases in plasma glucagon concentrations increased mitochondrial o

25 gon was infused at rates designed to achieve plasma glucagon concentrations spanning the physiologica

26 Similarly, serum insulin and plasma glucagon concentrations were markedly higher with

27 Plasma glucagon concentrations were suppressed by -13 +/

28 fusion (4 ng/kg/min), achieving steady-state plasma glucagon concentrations, followed by a 1-hour was

29 However, with increasing plasma glucagon concentrations, while SGE remained uncha

30 - 77 pmol/l), but there was no difference in plasma glucagon concentrations.

31 hours and were independent of any changes in plasma glucagon concentrations; these effects were abrog

32 Arterial plasma glucagon decreased from 54 +/- 7 to 32 +/- 3 ng/l

33 nd the hepatic portal-arterial difference in plasma glucagon decreased slightly from 41 +/- 7 and 4 +

34 whether macronutrient infusion can suppress plasma glucagon during critical illness and study the ro

35 Both groups had significant rises in plasma glucagon during the hypoglycemic clamp similar to

36 In patients with diabetes, elevations in plasma glucagon, epinephrine, and norepinephrine were bl

37 /- 6 pM), suppression of glucagon secretion (plasma glucagon, I:31 +/- 4, II: 63 +/- 8 pg/ml) doubled

38 nd the hepatic portal-arterial difference in plasma glucagon increased from 43 +/- 5 and 4 +/- 2 to 5

39 Plasma glucagon increased in the control study from 44 +

40 his pattern of change mirrored precisely the plasma glucagon/insulin ratio.

41 tor monoclonal antibody undergo elevation of plasma glucagon levels and alpha-cell expansion similar

42 effect associated with normalization of both plasma glucagon levels and hepatic expression of glucone

43 The arterial plasma glucagon levels and the hepatic portal-arterial d

44 plus xenin-25 transiently increased ISR and plasma glucagon levels in subjects with NGT and IGT but

45 lasma triglyceride levels, but did not alter plasma glucagon levels or alpha-cell mass.

46 As expected, plasma glucagon levels were elevated in STZ-DM rats, and

47 were associated with protein breakdown, and plasma glucagon levels were inversely correlated with pr

48 body mass index) or other biomarkers (e.g., plasma glucagon levels).

49 Xenin-25 alone had no effect on ISRs or plasma glucagon levels, but the combination of GIP plus

50 e-stimulated insulin secretion, and elevated plasma glucagon levels.

51 -double positive bihormonal cells and excess plasma glucagon levels.

52 In contrast, AR231453 increased plasma glucagon-like peptide 1 (GLP-1) and insulin level

53 Antropyloroduodenal pressures, plasma glucagon-like peptide 1 (GLP-1), cholecystokinin,

54 Fasting and postprandial plasma glucagon-like peptide 1 (GLP-1), peptide YY (PYY)

55 Sitagliptin increased plasma glucagon-like peptide-1 (7-36) levels and, at pea

56 sulin secretion (P < .01), and 3-fold larger plasma glucagon-like peptide-1(7-36) (GLP-1(7-36)) excur

57 nd the hepatic portal-arterial difference in plasma glucagon (ng/l) did not change significantly (43

58 nd the hepatic portal-arterial difference in plasma glucagon (ng/l) did not rise significantly (40 +/

59 nd the hepatic portal-arterial difference in plasma glucagon (ng/l) rose from 43 +/- 5 and 5 +/- 2 to

60 content, increased proinsulin, and augmented plasma glucagon observed in KATP-GOF mice were normalize

61 n EGP cannot be explained by the increase in plasma glucagon or decrease in plasma insulin or glucose

62 As expected, plasma glucagon (P = 0.0094), epinephrine (P = 0.0063),

63 tradiol also attenuated hypoglycemia-induced plasma glucagon, pituitary proopiomelanocortin (POMC), a

64 he great potential that normalization of the plasma glucagon profile may have for the treatment of T2

65 Arterial plasma glucagon rose to a similar maximum in CON (73 +/-

66 We applied novel analytical methods of plasma glucagon (sandwich ELISA and mass spectrometry-ba

67 both FRU and MF (P < 0.05 for all), although plasma glucagon was also higher when compared with PRO (

68 Plasma glucagon was elevated after exercise in control m

69 Plasma glucagon was elevated in Pemt(-/-) mice fed the H

70 Serum insulin was decreased, and plasma glucagon was increased.

71 ticosteroid levels were altered as expected, plasma glucagon was reduced markedly in the mutant anima

72 were greater with ID4 than ID2, as was GIGD; plasma glucagon was suppressed by ID2, but not ID4.