ライフサイエンスコーパス: plasma membrane

1 th activated RACB largely takes place at the plasma membrane.

2 s-Golgi network within small vacuoles to the plasma membrane.

3 with the virion that impeded binding to the plasma membrane.

4 revent the translocation of PAF26 across the plasma membrane.

5 rans-Golgi network/early endosome and at the plasma membrane.

6 ts to enhance Spike-mediated fusion with the plasma membrane.

7 ge factor, Son of Sevenless 1 (SOS1), to the plasma membrane.

8 involving transport from the nucleus to the plasma membrane.

9 titutive localization of this protein at the plasma membrane.

10 as, Rac and the phosphoinositide PIP3 in the plasma membrane.

11 hasis on Rho-family GTPases operating at the plasma membrane.

12 SBT S-acylation, function, and levels on the plasma membrane.

13 omes and is not present or functional on the plasma membrane.

14 eractions within the HIV-1 MA lattice at the plasma membrane.

15 ons with phosphoinositides (PtdInsPs) in the plasma membrane.

16 fusion protein selectively localizes to the plasma membrane.

17 oisotopes depends on its localization to the plasma membrane.

18 rine is enriched relative to the bulk of the plasma membrane.

19 t has the extraordinary ability to cross the plasma membrane.

20 a switch by modulating its residence in the plasma membrane.

21 rt cGAMP and cyclic dinucleotides across the plasma membrane.

22 resulting in increased F-actin levels at the plasma membrane.

23 xposed to the strong electrical field of the plasma membrane.

24 ytoskeleton by transmitting signals from the plasma membrane.

25 ion by interacting with ECT2 on the adjacent plasma membrane.

26 tal vesicles along the ribbon and toward the plasma membrane.

27 eval of vesicle components from the synaptic plasma membrane.

28 n terminal where they fuse with the neuronal plasma membrane.

29 ized insulin proreceptors from the ER to the plasma membrane.

30 MHC II, promoting its traffic away from the plasma membrane.

31 fusion of FgSnc1-associated vesicles to the plasma membrane.

32 on of distinct diacylglycerol species in the plasma membrane.

33 ns of tight junction proteins located in the plasma membrane.

34 A receptors are rapidly recycled back to the plasma membrane.

35 YP121(DeltaC), which blocks secretion at the plasma membrane.

36 pholipids found on the cytosolic side of the plasma membrane.

37 olgi apparatus, prevacuolar compartment, and plasma membrane.

38 mide, which was located predominantly at the plasma membrane.

39 s a cytotoxic cation channel in the parasite plasma membrane.

40 f Ca(2+)-activated potassium channels to the plasma membrane.

41 nteraction is replaced when Gag binds to the plasma membrane.

42 easing the level of VDAC-1 expression in the plasma membrane.

43 its dynamic localization and function in the plasma membrane.

44 by lowering the expression of VDAC-1 in the plasma membrane.

45 reduced trafficking of bestrophin-1 into the plasma membrane.

46 fulfill a function in susceptibility at the plasma membrane.

47 king the endo-/sarcoplasmic reticulum to the plasma membrane.

48 3 (L-Arrdc3 KO) have increased IR protein in plasma membrane.

49 ndance and modifying lipid rafts in neuronal plasma membranes.

50 iptional and post-transcriptional (increased plasma membrane affiliation) regulation of TNFR1 in the

51 However, F-actin next to the plasma membrane also tethers the membrane and thus resis

52 etween enhancement of viral infection at the plasma membrane and amphipathicity-based mechanisms used

53 ble basis for the origin of structure in the plasma membrane and an illuminating picture of the organ

54 integrally associated with both the parasite plasma membrane and an intermediate filament cytoskeleto

55 These are formed by ORAI proteins in the plasma membrane and are activated by stromal interaction

56 the NGF- and NT-3-bound Trk-A dimers in the plasma membrane and at neutral pH.

57 ereby promotes rapid permeabilization of the plasma membrane and bacterial cell death.

58 ALA4-GFP displayed plasma membrane and endomembrane localization patterns w

59 Getting large macromolecules through the plasma membrane and endosomal barriers remains a major c

60 n between CD63(+) vesicle trafficking to the plasma membrane and focal adhesion dynamics.

61 e CNTFRalpha by GPI-anchor cleavage from the plasma membrane and from extracellular vesicles (EVs) af

62 OPr endocytosis for activation of PKC at the plasma membrane and in the cytosol and ERK in the nucleu

63 es, resulting in increased in F-actin at the plasma membrane and increased release of sEVs, with alte

64 ng of the complex it forms with ClC-K to the plasma membrane and is involved in activating this chann

65 ur data suggest that EVE is localized in the plasma membrane and is involved in potassium uptake of d

66 ession of OsPIP2;2 recruited OsPIP1;3 to the plasma membrane and led to a significant enhancement of

67 p facilitate DNA plasmids to cross both cell plasma membrane and nuclear membrane quickly to promote

68 tion of the MT cytoskeleton confined between plasma membrane and nucleus during T cell polarization.

69 sts of specific proteins present both at the plasma membrane and on insulin SGs.

70 bcellular location to interact with both the plasma membrane and peroxisomal membrane and has the str

71 ne, nonreceptor tyrosine kinase (SRC) to the plasma membrane and promotes activation of an SRC-depend

72 s nuclear transit before accumulating at the plasma membrane and recruiting nucleocapsids to the budd

73 ediates binding to phospholipids in both the plasma membrane and synthetic membranes, and is sufficie

74 are the proximity of insulin granules to the plasma membrane and their anchoring or docking to the pl

75 ven crystal-like structures in the bacterial plasma membrane and thereby promotes rapid permeabilizat

76 ompartment, the cytosol for transport at the plasma membrane and tonoplast, are intrinsically connect

77 ein located in the endoplasmic reticulum and plasma membranes and has been shown to play important ro

78 golipid residing on the outer leaflet of the plasma membrane, and acetylcholine receptor (AChR), whic

79 ) complexes trigger clathrin assembly on the plasma membrane, and biochemical and structural studies

80 its biogenesis, trafficking to and from the plasma membrane, and how the regulation of these process

81 ulin acetylation in whole-tissue homogenate, plasma membrane, and lipid-raft membrane domains in tiss

82 ing the endoplasmic reticulum (ER) membrane, plasma membrane, and nanodomains induced by cholera toxi

83 osomes in close proximity to the basolateral plasma membrane, and phosphoinositide-binding residues o

84 specifically targeting the cytosol, vacuole, plasma membrane, and wall of plant cells.

85 nsmembrane conductance regulator (CFTR) is a plasma membrane anion channel that plays a key role in c

86 including Arf6, normally associated with the plasma membrane, are recruited to the replication organe

87 A plasma membrane-associated polarity crescent defined by

88 non-HLA antibodies (against agrin, adipocyte plasma membrane-associated protein, Rho GDP-dissociation

89 pressed suicides, and depressed nonsuicides, plasma membrane-associated tubulin showed significant de

90 MT-4 cells express higher levels of plasma-membrane-associated Env than nonpermissive cells,

91 docytosis inhibitor, Dyngo-4a, re-stored the plasma membrane association of beta1-p.C162A to WT level

92 generated by actin and myosin linked to the plasma membrane at cell-cell and cell-matrix junctions.

93 ld methylate lipid substrates located in the plasma membrane at membrane contact sites.

94 dent of the exocyst tethering complex on the plasma membrane at the division site.

95 Instead, the plasma-membrane-attached PLDgamma1 protein colocalized a

96 omere-cytoskeleton filament interactions and plasma membrane attachment.

97 by the presence of amphipathic components of plasma membrane because they may serve as interaction, c

98 nsible for docking secretory vesicles to the plasma membrane before exocytosis.

99 that reduced Gag-ESCRT binding increased Gag-plasma membrane binding and Gag release.

100 ding studies and the characterization of Gag-plasma membrane binding and Gag release.

101 strin homology-like domain are important for plasma membrane binding.

102 Mutations in the Gag-plasma membrane-binding matrix domain that reduced Gag-E

103 nsistency by performing in-depth analysis of plasma membrane budding, a cellular process that has pre

104 ffect the recruitment of PTPN22 R620W to the plasma membrane but protected this mutant from degradati

105 may have limited mobility in the endothelial plasma membrane, but no biophysical investigation of the

106 s, mainly because of its effects on the cell plasma membrane, but the molecular origins of this actio

107 y oligomeric beta3-subunits, not just at the plasma membrane, but throughout the secretory pathway.

108 The control of calcium influx at the plasma membrane by endoplasmic reticulum (ER) calcium st

109 R ICD is recruited from the cytoplasm to the plasma membrane by light, followed by its subsequent act

110 the C-terminal GOLD domain directs it to the plasma membrane by recognizing specific phosphoinositide

111 Imported across the plasma membrane by SLC1A5, glutamine has emerged as a me

112 fish larvae, we have studied the role of the plasma membrane Ca(2+) channel ORAI1 in this process.

113 the Ca(2+)/H(+) antiporting activity of the plasma membrane Ca(2+)-ATPase at the postsynaptic membra

114 escribe roles for the mechanically activated plasma membrane calcium channels Piezo1 and transient re

115 t the extent of upstream damage to mammalian plasma membranes, calibrated by severity of nanopore-med

116 Metformin inhibits the expression of the plasma membrane citrate transporter NaCT in HepG2 cells

117 by phosphatidylinositol 3-kinase C2alpha at plasma membrane clathrin-coated pits is spatially segreg

118 he interaction of amyloid intermediates with plasma membrane components.

119 3 deletion, strain Inp1-GFP localizes to the plasma membrane, concentrated in patches near the bud ne

120 EXO70A2 also localized to the PT tip plasma membrane, consistent with a role in exocyst-media

121 L1/RFA family interact with ABA receptors at plasma membrane, cytosol, and nucleus, targeting them fo

122 condensation (87.5% of nuclei analyzed) and plasma membrane damage (in 100% of treated hyphae).

123 This dual plasma membrane damage seems to be a common mechanism fo

124 tion (potentiators) and the other increasing plasma membrane density (correctors)-have provided signi

125 ntain the plasma membrane potential and that plasma membrane depolarization blocks cellular uptake of

126 calcium entry (SOCE) pathway and accompanied plasma membrane depolarization.

127 We find that IRK-GFP localizes to the outer plasma membrane domain in endodermal cells but localizes

128 lighting the importance of these specialized plasma membrane domains in cellular feedback via the Hip

129 densities are in general lower at the basal plasma membrane due to partial limited accessibility for

130 an unprecedented role for AP-1B at the basal plasma membrane during collective cell migration of epit

131 nslocation of MT1-MMP-laden endosomes to the plasma membrane, enabling both invadopodia outgrowth and

132 l stages and its protein is localized to the plasma membrane, endoplasmic reticulum (ER) and Golgi.

133 lude that infection-induced signals from the plasma membrane epigenetically regulate Wnt signaling.

134 ster specifically with Gag assembling at the plasma membrane even in cells that do not form uropods.

135 of heterogeneous force distributions on the plasma membrane for RBC shape maintenance may also have

136 et pre-existing PIP2-enriched domains of the plasma membrane for viral assembly, and that Gag multime

137 we find that naive stem cells release their plasma membrane from the underlying actin cortex when tr

138 , submicrometer-sized vesicles released from plasma membrane, from MetS patients were shown to induce

139 Besides carrying out this plasma membrane function, PLCbeta1 has a cytosolic popul

140 fically, density can have adverse impacts on plasma-membrane functions, cytoplasmic viscosity, protei

141 Overexpression of TMPRSS2, which increases plasma membrane fusion versus endosome fusion of SARS-Co

142 atal projection neurons can be determined by plasma membrane GABA uptake transporters (GATs) located

143 n which TORC2-dependent signals control both plasma membrane growth and endocytosis, which would ensu

144 homeostasis requires coordinated control of plasma membrane growth and endocytosis.

145 of cipk23 mutants, whereas activation of the plasma membrane H(+) -ATPase was not.

146 the decades-old acid growth theory invoking plasma membrane H(+)-ATPase activation is still useful.

147 (IBperi) were formed, but inclusions at the plasma membrane (IBPM), which probably represent the vir

148 acuoles without degradative xenophagy to the plasma membrane.IMPORTANCE The long-term goal of this re

149 Internally tagged Rho3 is restricted to the plasma membrane in a gradient corresponding to cell pola

150 techniques and show they are tethered to the plasma membrane in a highly specialized arrangement.

151 nsported from the trans-Golgi network to the plasma membrane in an AP-1- and Arf1/4-dependent manner.

152 B-coated structures at or close to the basal plasma membrane in cell protrusions.

153 er type 4 (GLUT4)-containing vesicles to the plasma membrane in response to insulin stimulation.

154 ns confer a steady-state organization of the plasma membrane in resting cells that is poised to orche

155 EGF induces sustained ERK activity near the plasma membrane in sharp contrast to the transient activ

156 isms controlling the abundance of PIP in the plasma membrane in these cells.

157 and shifts the localization of ClVST1(97) to plasma membranes in sweet watermelons.

158 icolor imaging of lipid organization of cell plasma membranes in the presence of fluorescently tagged

159 n live cells, it stains exclusively the cell plasma membranes, in contrast to Laurdan and its carboxy

160 ern and describe a ZBP1-initiated nucleus-to-plasma membrane "inside-out" death pathway with potentia

161 hat central neurons possess to integrate the plasma membrane into nuclear signaling in neurons.

162 p defective for recruitment of Ste20p to the plasma membrane, intramolecular interactions, and intera

163 ular domain of TrkB in the cytosol or on the plasma membrane is able to induce the activation of down

164 sive cells, and Env internalization from the plasma membrane is less efficient than that from another

165 The yeast plasma membrane is unusual in that it may have a high li

166 en the cell and the outside environment, the plasma membrane is well-positioned to be the first respo

167 Molecular movement in biological plasma membranes is often characterized by anomalous dif

168 signaling via G proteins is delimited to the plasma membrane, it is now recognized that GPCRs signal

169 Laurdan, which is redistributed within both plasma membrane leaflets and intracellular membranes.

170 d movement of insulin vesicles away from the plasma membrane limits insulin secretion.

171 etermined the molecular composition of yeast plasma membrane lipids located within a defined diameter

172 an cell membranes, constituting up to 50% of plasma membrane lipids.

173 (Kir) Kir2.2 has multiple interactions with plasma membrane lipids: Phosphatidylinositol (4, 5)-bisp

174 In the PVN, TNFR1 expression and plasma membrane localization are upregulated during hype

175 The plasma membrane localization of GPI-anchored proteins wa

176 both exercise-induced activation of AMPK and plasma membrane localization of the GLUT4 glucose transp

177 intracellular distribution of the otherwise plasma membrane-localized mGluR2.

178 We identified the plasma membrane-localized palmitoyl acyltransferase DHHC

179 educing the ability of cells to repair their plasma membrane makes them more sensitive to the peptide

180 xosomes) or as a result of shedding from the plasma membrane (microvesicles, oncosomes and apoptotic

181 Dispatched1 uses the energy of the plasma membrane Na(+) gradient, thus functioning as an S

182 Further, electron microscopy reveals that plasma membrane "nanopore-like" structures (~100-nm diam

183 e depleted "accessible cholesterol" from the plasma membrane of [(13)C]cholesterol-loaded macrophages

184 The plasma membrane of a cell is characterized by an asymmet

185 e ClC-2 chloride channel is expressed in the plasma membrane of almost all mammalian cells.

186 ermed CHAT, was non-cytotoxic, traversed the plasma membrane of breast and prostate cancer cell lines

187 functional K(v)11.1 potassium channel at the plasma membrane of cardiomyocytes prolongs action potent

188 e molecular organization of receptors in the plasma membrane of cells is paramount for their function

189 The plasma membrane of eukaryotic cells is asymmetric with r

190 ritical to the maintenance of the asymmetric plasma membrane of eukaryotic cells.

191 e involved in ER export and transport to the plasma membrane of GPI-anchored proteins.

192 d Alternaria-evoked Ca(2+) uptake across the plasma membrane of HBE cells and interleukin (IL)-33 rel

193 s, the Nav1.5 alpha-subunit assembles on the plasma membrane of HEK293F cells into spatially localize

194 tion of pore-forming toxins that disrupt the plasma membrane of host cells is a common virulence stra

195 , PKA and Ca(V)1.2 into nanocomplexes at the plasma membrane of human and mouse arterial myocytes.

196 nent synthetic membranes (liposomes) and the plasma membrane of human erythrocytes to investigate the

197 ion of a specific pool of cholesterol in the plasma membrane of macrophages without changes to total

198 ly reconstitute the PC-1/PC-2 complex in the plasma membrane of mammalian cells and show that it func

199 mediates its transient association with the plasma membrane of neurosecretory cells by binding to ph

200 act by binding to specific receptors in the plasma membrane of target cells.

201 actin meshwork that uniformly underlies the plasma membrane of the entire cell.

202 ally sequestered to the inner leaflet of the plasma membrane of the healthy eukaryotic cells.

203 eased transcription and translocation to the plasma membrane of TLR4 in donor TRAMs.

204 Plasma membranes of animal cells are enriched for choles

205 release of nascent virus particles from the plasma membranes of infected cells.

206 and the transmembrane proteins reside on the plasma membranes of live Xenopus muscle cells.

207 rticles, suggesting that localization to the plasma membrane, oligomerization into a matrix layer, an

208 hat relies on the physical disruption of the plasma membrane once the peptide targets specific phosph

209 We consider a model lipid plasma membrane, one that describes the outer leaf as co

210 to the recycling endosome rather than to the plasma membrane, our findings reveal the complexity of r

211 Therefore, we tested how the yeast plasma membrane P4-ATPase, Dnf2, responds to changes in

212 causing increased binding to an alternative plasma membrane partner, ITSN1.

213 The model hints that the plasma membrane permeability of the cells is not radiall

214 ed that certain aminoglycosides induce rapid plasma membrane permeabilization and that this nonriboso

215 on of the receptor in lipid rafts, which are plasma membrane platforms replete with cholesterol and s

216 ery low steady-state levels of PI within the plasma membrane (PM) and endosomes.

217 b with consistent curvature connected to the plasma membrane (PM) by a neck region with opposing curv

218 cumulated cholesterol in LEL and had reduced plasma membrane (PM) cholesterol.

219 or the formation of polarized domains at the plasma membrane (PM) during the morphogenesis of cotyled

220 K-Ras must interact primarily with the plasma membrane (PM) for its biological activity.

221 The permeability of the plasma membrane (PM) for small molecules is low and late

222 ate and transitory protein engagement at the plasma membrane (PM) is crucial to a broad range of cell

223 OCE by increasing endoplasmic reticulum (ER)-plasma membrane (PM) junctions and STIM1 translocation t

224 STIM1 translocates to the ER-plasma membrane (PM) junctions to interact with ORAI1, t

225 The plasma membrane (PM) provides a critical interface betwe

226 N-GSDMD then oligomerizes in the plasma membrane (PM) to form pores that increase membran

227 rafficking and endoplasmic reticulum (ER) to plasma membrane (PM) transport.

228 dynamic assembly of the octameric TPC at the plasma membrane (PM), we performed state-of-the-art dual

229 are regulated through crosstalk between the plasma membrane (PM), where most cellular cholesterol re

230 ORAI1 is a calcium channel in the plasma membrane (PM).

231 olipid constituent of the inner layer of the plasma membrane (PM).

232 ulation of the biophysical properties of the plasma membrane (PM).

233 mbrane and their anchoring or docking to the plasma membrane (PM).

234 d biophysical analysis of isolated mammalian plasma membranes (PMs).

235 that glycolysis is required to maintain the plasma membrane potential and that plasma membrane depol

236 ependent on orchestrated fluctuations in the plasma membrane potential or voltage, which are mediated

237 mbrane (PVM), collaborates with the parasite plasma membrane (PPM) to support the transport of protei

238 clock regulation of endoplasmic reticulum-to-plasma membrane procollagen transport by the sequential

239 The Na(+)/I(-) symporter (NIS), the plasma membrane protein that actively transports I(-) (s

240 They are associated with plasma membrane protrusions, such as primary cilia, as w

241 Plasma membranes provide a highly selective environment

242 Stimulation of plasma membrane receptor tyrosine kinases (RTKs), such a

243 roteins from the endosome via retrograde and plasma membrane recycling pathways.

244 , which assemble in the cytoplasm and at the plasma membrane, respectively.

245 vide a mechanism by which signaling-mediated plasma membrane resurfacing of SSTR2 can fine-tune pitui

246 by activating Galphaq to localize it to the plasma membrane returns differentiated PC12 and SK-N-SH

247 lability of surface ORAI1 and increasing the plasma membrane ruffling.

248 terminal Slp homology domain, interacts with plasma membrane SNARE complex proteins via a central lin

249 XO70 subunits bind preferentially to cognate plasma membrane SNAREs, notably SYP121 and VAMP721.

250 w that wounding provokes a reorganisation of plasma membrane subdomains.

251 orphologically defined structures within the plasma membrane, such as cellular junctions, focal adhes

252 of magnitude more elastic than the classical plasma membrane suggesting a physical explanation for th

253 and two sites of gamma-ENaC adjacent to the plasma membrane, suggesting direct interactions of PIP(2

254 vement kinetics were not clearly affected in plasma membrane-targeted cbl1/4/5/8/9 quintuple mutant p

255 e mechanistic differences of the MA-mediated plasma membrane targeting of the B-type mouse mammary tu

256 e variants impaired heterologous expression, plasma membrane targeting, and/or signaling, whereas hyp

257 ated missense variants increased expression, plasma membrane targeting, and/or signaling.

258 ns affect both of these pathways by changing plasma membrane tension and thus the morphology and comp

259 RhoA activity and subsequent decrease in the plasma membrane tension.

260 phingolipid-enriched microdomains within the plasma membrane that play important roles in many pathop

261 ctural link between the cytoskeleton and the plasma membranes that maintain cellular integrity.

262 endothelial cortex, 50 to 150 nm beneath the plasma membrane, the endothelial glycocalyx (eGC) is rec

263 ls, followed by accumulation of Kv1.2 in the plasma membrane, thereby depressing NAcSh MSNs firing.

264 From measurements on the plasma membrane, this scale is on the order of 100 nm.

265 adherens junctions, which link actin to the plasma membrane through alpha-, beta- and p120 catenins.

266 etion, some transmembrane proteins reach the plasma membrane through alternative routes.

267 as GTPase-activating protein (RasGAP) to the plasma membrane through dephosphorylation of docking sit

268 osphoinositides 1 (Grp1) is recruited to the plasma membrane through its pleckstrin homology (PH) dom

269 ate filaments (IFs) rather than actin to the plasma membrane through protein complexes comprising rel

270 ins are targeted to the inner leaflet of the plasma membrane through their N-terminal matrix (MA) dom

271 e initiated by BDNF and its receptors at the plasma membrane to modulate BDNF-dependent gene expressi

272 The journey from plasma membrane to nuclear pore is a critical step in th

273 , over 50 different proteins assemble on the plasma membrane to reshape it into a cargo-laden vesicle

274 ough depleting accessible cholesterol on the plasma membrane to suppress virus-cell fusion.

275 cells rely on linker proteins to connect the plasma membrane to the actin network.

276 n of the C2AB domain of granuphilin from the plasma membrane to the cytosol.

277 specific long-range signal from LTCCs in the plasma membrane to the nucleus that is required for acti

278 man bone osteosarcoma epithelial cells, with plasma membrane transporter 1 (MCT1) tagged with an enha

279 A plasma membrane transporter, the solute carrier (SLC) hu

280 n particular, the identity of unidirectional plasma membrane transporters that mediate D-serine reupt

281 nd generate the second messenger ceramide at plasma membranes, triggering apoptosis in specific cells

282 Giant plasma membrane vesicles (GPMVs) are a widely used exper

283 distinguishes better lipid domains in giant plasma membrane vesicles (GPMVs) than Laurdan.

284 Here, giant plasma membrane vesicles (GPMVs) were employed to quanti

285 ence for stable oligomers of RHBDL2 in giant plasma membrane vesicles of human cells even at concentr

286 n, MLKL oligomerizes and integrates into the plasma membrane via its executioner domain.

287 RAS proteins concentrate in the plasma membrane via lipid-tethers and hypervariable regi

288 trafficked directly from the cytosol to the plasma membrane, we discovered that the oncogenic avian

289 ic trafficking and retromer recycling to the plasma membrane, we were able to reduce the virus's abil

290 Nanoparticles activated DOPr at the plasma membrane, were preferentially endocytosed by DOPr

291 hen cells are large, and by signals from the plasma membrane when they are small.

292 The small GTPase KRAS is localized at the plasma membrane where it functions as a molecular switch

293 havior of PIP(2) on the inner leaflet of the plasma membrane, where it is involved in attaching the m

294 ed near domains that interact with T-tubular plasma membrane, where LTCCs are housed.

295 Wnt ligand receptors, Fzd and LRP5/6 at the plasma membrane, which then recruits the destruction com

296 recruitment of the ARF-GEF cytohesins to the plasma membrane, which, in turn, bind and activate the s

297 striatum, mainly by lateral diffusion in the plasma membrane with only a limited contribution of vesi

298 Starting from the plasma membrane with the recognition of microbe-associat

299 understanding of the lateral organization in plasma membranes with higher compositional complexity.

300 a spatially restricted signal to repair the plasma membrane wound.