ライフサイエンスコーパス: plasma renin activity

1 supplemental use of the in vitro stimulated plasma renin activity.

2 r PA using the ratio of serum aldosterone to plasma renin activity.

3 ifferences between aldosterone excretion and plasma renin activity.

4 rkedly elevated with a relatively suppressed plasma renin activity.

5 d on day 3 despite significant reductions in plasma renin activity.

6 nsin I and II despite continuously increased plasma renin activity.

7 TS, there was not a compensatory increase in plasma renin activity (0.79+/-0.58 versus 0.79+/-0.74 ng

8 Postinfusion, the decrease in plasma renin activity (-0.9+/-0.2 versus -0.6+/-0.2 ng/m

9 vident in pool walking than in land walking (plasma renin activity, -1.27 vs. 0.81 ng/mL/h, p = 0.002

10 values <15 ng/dL) but contrasting values of plasma renin activity (15.00 versus 0.56 ng/mL/h; P<0.00

11 Ang 1-7 treatment were associated with lower plasma renin activity (-40%) and serum aldosterone (-48%

12 (+ 78%, P = 0.014) and PRA-S (surrogate for plasma renin activity; + 58%, P = 0.040).

13 32 +/- 24 versus 24 +/- 15 mg/dl, P = 0.06), plasma renin activity (7.1 +/- 9.9 versus 3.4 +/- 5.6 ng

14 lability to produce sustained suppression of plasma renin activity after oral administration.

15 Plasma renin activity, aldosterone and arginine vasopres

16 e mean arterial pressure, renal plasma flow, plasma renin activity, aldosterone, urine sodium, and ba

17 Plasma renin activity also increased appropriately when

18 Losartan, EXP3174 and captopril elevated plasma renin activities and comparably and significantly

19 early-morning ratio of plasma aldosterone to plasma renin activity and 24-hour urinary aldosterone an

20 ith ACEI+D, whereas OMA+D resulted in higher plasma renin activity and a delayed increase in aldoster

21 station fetal sheep have minor influences on plasma renin activity and ACTH in normovolaemic fetuses,

22 cardiac nerves and a sustained rise in fetal plasma renin activity and ACTH.

23 The LS diet increased plasma renin activity and aldosterone concentration, whe

24 onism; controlled posture studies to measure plasma renin activity and aldosterone concentrations, fo

25 Plasma renin activity and aldosterone immediately increa

26 th healthy controls and explored the role of plasma renin activity and aldosterone in the regulation

27 fore and after dexamethasone administration, plasma renin activity and aldosterone level.

28 OMA alone did not increase plasma renin activity and aldosterone, but resulted in a

29 Plasma renin activity and angiotensin levels were reduce

30 activity, height, weight, sodium excretion, plasma renin activity and heart rate were examined.

31 Plasma renin activity and insulin concentrations were bo

32 ients with POTS have paradoxically unchanged plasma renin activity and low aldosterone given their ma

33 At 2 wk, plasma renin activity and plasma aldosterone levels were

34 -four hour urinary sodium was increased, and plasma renin activity and plasma aldosterone levels were

35 Plasma renin activity and plasma angiotensin II levels w

36 Furthermore, both plasma renin activity and plasma ET-1 increased with ET-

37 Despite greater intra-group improvements in plasma renin activity and serum aldosterone levels in th

38 The decreases in plasma renin activity and serum aldosterone levels were

39 king: the mean starting and ending values of plasma renin activity and serum aldosterone were 6.8 vs.

40 al models characterized by various levels of plasma renin activity and significantly potentiated urin

41 Heart rate (HR), blood pressure (BP), plasma renin activity, and aldosterone were measured wit

42 ratio of plasma aldosterone concentration to plasma renin activity, and higher urine aldosterone leve

43 osteronism may cause hypokalemia, suppressed plasma renin activity, and hypertension.

44 l, pro B-type natriuretic peptide, increased plasma renin activity, and increased blood urea nitrogen

45 drome, with normal blood pressure, increased plasma renin activity, and reduced NCC expression and ph

46 Aldosterone excretion rate, plasma renin activity, and size and location of adenomas

47 atremia, hyperkalemia, hypovolemia, elevated plasma renin activity, and sometimes shock and death.

48 Weekly blood pressure, plasma renin activity, Ang II, ET, and catecholamines we

49 at achieved by CA alone, while also reducing plasma renin activity, angiotensin II, aldosterone and v

50 radiol, PAI-1, tissue plasminogen activator, plasma renin activity, angiotensin II, and aldosterone w

51 Plasma renin activity, angiotensin II, and aldosterone w

52 erone levels, and also significantly reduced plasma renin activity, angiotensin II, and vasopressin c

53 There were no changes in plasma renin activity, angiotensin II, or aldosterone.

54 Obesity, higher salt-sensitivity and low plasma renin activity are possible reasons of this poor

55 chloremia, metabolic acidosis and suppressed plasma renin activity are variable associated findings.

56 rcalciuria, increased serum aldosterone, and plasma renin activity, are the two major diseases linked

57 Plasma renin activity at five pre-determined time points

58 independent loci displayed associations with plasma renin activity at genome-wide significance (P<5x1

59 hich is associated with a markedly depressed plasma renin activity because of sodium retention.

60 art rate, blood pressure, serum aldosterone, plasma renin activity, blood volume, and plasma norepine

61 ane anaesthesia and surgery caused a rise in plasma renin activity but was associated with a suppress

62 tamin D levels and the blood pressure and/or plasma renin activity, but the mechanism is not understo

63 Neither urinary cGMP nor plasma renin activity changed.

64 Plasma renin activity decreased from 80 +/- 88 ng/dL at

65 Plasma renin activity drawn immediately before angiotens

66 days of treatment with NTG patches increased plasma renin activity for the entire treatment period.

67 ion rates appear to be accompanied by higher plasma renin activities in mice, compared with rats, rab

68 ects of ACE inhibition on blood pressure and plasma renin activity in both normotensive and hypertens

69 nal kallikrein and renal renin activity, and plasma renin activity in control and diabetic rats and d

70 Hemorrhage increased heart rate and plasma renin activity in intact sheep, but these respons

71 , but it significantly altered the change in plasma renin activity in response to ACE inhibition (-0.

72 Concomitantly, plasma renin activity increased from 3.08 +/- 0.68 (mean

73 Plasma renin activity increased significantly in the cap

74 plemental measurement of in vitro stimulated plasma renin activity insignificantly (p > 0.10) and imp

75 tension with hypokalaemia and suppression of plasma renin activity is known as mineralocorticoid hype

76 nt BP reduction and prolonged suppression of plasma renin activity is observed.

77 volume, creatinine clearance, and change in plasma renin activity levels between each activity were

78 ta-analyzed genome-wide association data for plasma renin activity (n=5275), plasma renin concentrati

79 seline cycle (P = 0.001), and an increase in plasma renin activity of 0.14 +/- 0.08 ng/(L . s) from a

80 a combination of the two, compared with the plasma renin activity of the controls.

81 We find no difference in plasma renin activity or plasma aldosterone concentratio

82 multivariate analysis, LVMI correlated with plasma renin activity (p < 0.001) and plasma norepinephr

83 Insulin infusion increased plasma renin activity (P < 0.01) and renal plasma flow (

84 ration produced a dose-dependent decrease in plasma renin activity (P=0.004), with similar trends obs

85 The reduction of plasma renin activity, plasma aldosterone, and arginine

86 Plasma renin activity, plasma aldosterone, plasma and 24

87 Preoperative plasma renin activity, plasma and urinary aldosterone co

88 ours after hospitalization adrenal function, plasma renin activity, plasma noradrenaline and vasopres

89 +/- 0.23 vs. 4.14 +/- 0.27 L; P = 0.72) and plasma renin activity (PRA) (20.5 +/- 7.03 vs. 23.2 +/-

90 Ly markedly raised plasma renin activity (PRA) and aldosterone, and exerted

91 weight loss on Iso-induced water intake and plasma renin activity (PRA) and found that weight loss d

92 A BEARS-based method was developed for plasma renin activity (PRA) and was evaluated on fifty-t

93 this study, it was hypothesized that the low plasma renin activity (PRA) is misleading, masking and p

94 nt BP reduction and prolonged suppression of plasma renin activity (PRA) is observed after aliskiren

95 Endothelin, catecholamine, and plasma renin activity (PRA) responses to 24-hour sodium

96 Plasma renin activity (PRA) was similar in young versus

97 els, 24-h urinary aldosterone excretion, and plasma renin activity (PRA) were determined in all group

98 ion of sodium, potassium, and creatinine and plasma renin activity (PRA) were measured in 2937 mildly

99 Supine blood pressure and plasma renin activity (PRA) were measured, and daily sod

100 The plasma hormones, AVP, ANP, plasma renin activity (PRA), angiotensin II, and aldoste

101 rats in the highest quartiles of both BP and plasma renin activity (PRA).

102 eart Study participants with aldosterone and plasma renin activity (PRA).

103 ) with measurements of serum aldosterone and plasma renin activity (PRA).

104 ween race and rehospitalization according to plasma renin activity (PRA).

105 this site relate to salt sensitivity and low plasma renin activity (PRA).

106 Plasma renin activity (PRA; >4 ng/mL/hour) was used as a

107 - 0.23 versus 4.14 +/- 0.27 L; P = 0.72) and plasma renin activity (PRA; 20.5 +/- 7.03 versus 23.2 +/

108 3 participants with suppressed plasma renin (plasma renin activity [PRA] <=1.0 ng/mL/h) and elevated

109 <0.0001), and ratio of plasma aldosterone to plasma renin activity (r=-0.43, P<0.0001) but was indepe

110 tassium and plasma aldosterone concentration-plasma renin activity ratio for patients with hypertensi

111 evel and plasma aldosterone concentration to plasma renin activity ratio.

112 tly alter the magnitude of the ACTH, AVP, or plasma renin activity response to haemorrhage.

113 tients' work-up included plasma aldosterone, plasma renin activity, serum cortisol, and estimation of

114 Salt depletion for 1 wk increased plasma renin activity seven-fold in wild-type mice but o

115 iotensin II receptors with losartan elevated plasma renin activity some 29-fold (P < 0.001) and cause

116 mia also completely eliminated the increased plasma renin activity that accompanied restraint in cont

117 and 5.4 +/- 4.3 pg/mL, respectively) despite plasma renin activity under 0.6 ng/mL/hr.

118 Plasma renin activity was assessed by radioimmunoassay o

119 llowing reduction of RPP to 60 mmHg for 3 h, plasma renin activity was increased more than 7-fold (P

120 Plasma renin activity was markedly suppressed (0.2 +/- 0

121 Plasma renin activity was measured at specific time inte

122 ary cGMP was 40% greater than in N rats, but plasma renin activity was not significantly greater in C

123 Plasma aldosterone but not plasma renin activity was significantly elevated after 2

124 Plasma renin activity was significantly elevated by losa

125 Plasma renin activity was significantly increased during

126 Plasma renin activity was significantly lower in 13 hype

127 rrhage (20% loss of blood volume), including plasma renin activity, was assessed at 2 and 5 months po

128 Renal tissue kallikrein levels and plasma renin activity were decreased, whereas renal reni

129 aldosterone, atrial natriuretic peptide, and plasma renin activity were drawn at baseline and 2 hours

130 sodium levels, urinary sodium excretion, and plasma renin activity were measured for five time period

131 Renal plasma flow and plasma renin activity were measured serially.

132 activity, the starting and ending levels of plasma renin activity were measured.

133 Plasma Ang II, ET, catecholamines, and plasma renin activity were unchanged during the progress

134 xcretion, urinary aldosterone excretion, and plasma renin activity were unchanged.