ライフサイエンスコーパス: plasma sodium

1 esuscitation results in smaller increases in plasma sodium.

2 and their severity, as well as the level of plasma sodium.

3 (HR, 2.04 per 1 mmol/L per hour decrease in plasma sodium; 95% CI, 1.19 to 3.51; P=0.01).

4 [HR], 1.73 per 1 mmol/L per hour decrease in plasma sodium; 95% confidence interval [95% CI], 1.23 to

5 f important biochemical variables, including plasma sodium and chloride.

6 tic patients, cachectic patients had reduced plasma sodium and increased norepinephrine, epinephrine

7 Decreases in plasma sodium and potassium and increases in urea and cr

8 nel (ENaC) participates in the regulation of plasma sodium and volume, and gain of function mutations

9 In plasma, sodium and chloride are the principal strong ion

10 fidence interval, 0.72 to 0.88) mmol/L lower plasma sodium, but we observed no gene-environment inter

11 primary end point was the absolute change in plasma sodium concentration after 4 days of treatment.

12 of isosal on hemodynamics, brain edema, and plasma sodium concentration after head injury associated

13 the role of genetic variation in regulating plasma sodium concentration and highlight the importance

14 the effects on the cerebellum of the rise of plasma sodium concentration and the emergence of thirst

15 tion between a polygenic score developed for plasma sodium concentration and thiazide exposure on sod

16 Here we identify plasma sodium concentration as a factor that modulates b

17 1, we identified eight loci associated with plasma sodium concentration at P<5.0 x 10(-6) Of these,

18 ad a significantly higher increase of median plasma sodium concentration compared with those receivin

19 nome-wide association study meta-analysis on plasma sodium concentration in 45,889 individuals of Eur

20 Exclusion criteria included a plasma sodium concentration of less than 130 mmol/L or g

21 Abnormal plasma sodium concentration represents an imbalance of t

22 A polygenic score for plasma sodium concentration was associated with 0.43 (95

23 Clinically, also in vivo, rise in plasma sodium concentration within the physiological ran

24 Cardiovascular hemodynamics, plasma sodium concentration, plasma colloid osmotic pres

25 ed genome-wide association study analyses of plasma sodium concentration, thiazide-induced decrease i

26 ysate sodium >138 mmol/L after adjusting for plasma sodium concentration.

27 igate the relationship between dialysate and plasma sodium concentrations and mortality among mainten

28 idence interval, 1.25 to 1.98), adjusted for plasma sodium concentrations and other confounding varia

29 level as in the rats on solid chow; however, plasma sodium concentrations and plasma osmolality remai

30 A continuous decrease in plasma sodium concentrations from baseline values (140 t

31 tic study identified 31 loci associated with plasma sodium concentrations in individuals of European

32 Plasma sodium concentrations in the group receiving hype

33 show any evidence of effect modification by plasma sodium concentrations or other patient-specific v

34 HRP also induced a reduction in plasma sodium concentrations relative to control (P=0.02

35 Baseline plasma sodium concentrations were similar for the two gr

36 Low plasma sodium concentrations, initially resistant to tre

37 is associated with abnormal fluid status and plasma sodium concentrations.

38 dent associated signals at P < 5x10(-8) with plasma sodium concentrations.

39 Plasma sodium content was lower in the isosal group than

40 ney transplant recipients failed to maintain plasma sodium during water loading (plasma sodium slope

41 sociation between time-varying dialysate and plasma sodium exposure and all-cause mortality, adjusted

42 (2.02 +/- 0.96 L) was FW loss, and increased plasma sodium from 139 (range: 135-143 mmol/L) to 143 (r

43 HS challenge, healthy volunteers displayed a plasma sodium increase of [Formula: see text] and [Formu

44 had decreased from 79.7 to 78.8 kg, and mean plasma sodium increased from 140.4 to 142.6 mmol/L (both

45 es did not correlate with either the initial plasma sodium level (r=0.05, P>.12) or the rate of corre

46 n of plasma oxytocin or copeptin levels with plasma sodium level at 180 minutes (peak concentration o

47 Change in plasma sodium level from baseline to 180 minutes demonst

48 The mean (+/-SD) plasma sodium level was 121 +/- 3 mmol/L, and oxygen sat

49 At baseline, the mean (SD) plasma sodium level was 140 (3) mEq/L and decreased in r

50 with intravenous NaCl, 514 mmol/L, increased plasma sodium levels by 10 mmol/L in 12 hours.

51 eived empagliflozin had a larger increase in plasma sodium levels compared with those who received pl

52 ean decrease (+/- one half of the 95% CI) in plasma sodium levels of 0.9 +/- 0.9 mmol/L from a mean o

53 restriction cycle, significant decreases in plasma sodium levels of 1.23 +/- 0.5 mmol/L (from values

54 m chloride before respiratory insufficiency, plasma sodium levels were increased by 22 (10) mmol/L in

55 ong patients who had fluid restriction only, plasma sodium levels were increased by 3 (2) mmol/L in 4

56 um chloride after respiratory insufficiency, plasma sodium levels were increased by 30 (6) mmol/L in

57 Plasma sodium levels were lower in sodium-depleted rats

58 Plasma sodium levels, chest radiograph, electrocardiogra

59 Plasma sodium levels, urinary sodium excretion, and plas

60 alysate sodium concentrations, regardless of plasma sodium levels.

61 o evidence of genetic predisposition for the plasma sodium-lowering effect of thiazides.

62 dialysate sodium (<=138 or >138 mmol/L) and plasma sodium (<135, 135-142, >142 mmol/L) concentration

63 Time-updated GCS and plasma sodium measurements improved predictions based so

64 ng time-updated Glasgow Coma Scale (GCS) and plasma sodium measurements, together with patient baseli

65 in 30 (37%) (P = .002) with a difference in plasma sodium of 4 (95% CI, 2-5) mEq/L (P < .001) betwee

66 The possibility that increased plasma sodium/osmolality in AV3V-lesion rats down-regula

67 al positive psychotic symptoms (P < .09) and plasma sodium (P < .18) were even marginally associated

68 fidence interval, 0.39 to 0.46) mmol/L lower plasma sodium per SD decrease, and thiazide use was asso

69 Steeper plasma sodium reduction during the test independently as

70 In addition, a steeper plasma sodium slope 3 months after transplantation indep

71 maintain plasma sodium during water loading (plasma sodium slope of -0.6+/-0.4 mmol/L per hour in tra

72 ases the strong ion difference by increasing plasma sodium, tris-hydroxymethyl aminomethane acts by i

73 In HIV-infected patients, higher plasma sodium was uniformly associated with good prognos

74 ed from body composition (WD(4)), the actual plasma sodium (WD(5)), the substitution of plasma osmola