ライフサイエンスコーパス: plasmacytoid dendritic cell

1 of pathogenic immune cells (plasmablasts and plasmacytoid dendritic cells).

2 nduction of type 1 interferon responses from plasmacytoid dendritic cells.

3 cell (DC) differentiation, predominantly of plasmacytoid dendritic cells.

4 ed CLEC4C or CD303) is uniquely expressed on plasmacytoid dendritic cells.

5 monocytes, conventional dendritic cells, and plasmacytoid dendritic cells.

6 s, with lower expression on conventional and plasmacytoid dendritic cells.

7 nocytes of substances inhibitory or toxic to plasmacytoid dendritic cells.

8 of endothelial and myeloid cells, as well as plasmacytoid dendritic cells.

9 thought to initiate disease exacerbation via plasmacytoid dendritic cells.

10 y in mature B cells, T-cell progenitors, and plasmacytoid dendritic cells.

11 Ly49Q functions as a pan-MHC-Ia receptor on plasmacytoid dendritic cells.

12 ve CD4 T cells and an increased frequency of plasmacytoid dendritic cells.

13 , in promoting IFN-alpha production by human plasmacytoid dendritic cells.

14 s TLR7 can stimulate them in the presence of plasmacytoid dendritic cells.

15 ith the growing arsenal of markers for human plasmacytoid dendritic cells.

16 ived dendritic cells, or in blood CD1c(+) or plasmacytoid dendritic cells.

17 d interferon-alpha (IFN-alpha) production by plasmacytoid dendritic cells.

18 s again dependent on type I IFNs produced by plasmacytoid dendritic cells.

19 e expression, IFNalpha levels, and activated plasmacytoid dendritic cells; 2) higher proinflammatory

20 Depletion of plasmacytoid dendritic cells, a major cellular source of

21 vitro upon priming naive CD4(+) T cells with plasmacytoid dendritic cells activated with oxidized mit

22 S-1/MAVS signaling and induced type I IFN in plasmacytoid dendritic cells and macrophages, with the l

23 by unique members of the microbiota regulate plasmacytoid dendritic cells and monocytes via TLR7 and

24 neutrophils promoted cleavage of FcgRIIA on plasmacytoid dendritic cells and monocytes, resulting in

25 ces of type I IFNs during IOE infection were plasmacytoid dendritic cells and monocytes.

26 by inflammatory monocytes, dendritic cells, plasmacytoid dendritic cells and NK cells in all tissues

27 ion by monocytes required TLR7 activation of plasmacytoid dendritic cells and secretion of type I IFN

28 extracellular trap (NET) formation activates plasmacytoid dendritic cells and serves as a source of a

29 ne caused by TLR7 induction of type I IFN by plasmacytoid dendritic cells and subsequent IFN stimulat

30 Upon adoptive transfer of wild-type plasmacytoid dendritic cells and subsequent VSV infectio

31 03 treatment led to pronounced activation of plasmacytoid dendritic cells and substantial increases i

32 ous levels, including marginal zone B cells, plasmacytoid dendritic cells and T cell populations, and

33 mmune cells such as myeloid dendritic cells, plasmacytoid dendritic cells, and B cells in vitro.

34 script origin analysis identified monocytes, plasmacytoid dendritic cells, and B lymphocytes as prima

35 al infections, especially by memory T cells, plasmacytoid dendritic cells, and CD56(bright) NK cells.

36 expose immunostimulatory molecules, activate plasmacytoid dendritic cells, and may participate in org

37 mmune factors, such as natural killer cells, plasmacytoid dendritic cells, and the expression pattern

38 Because Tregs and plasmacytoid dendritic cells are known to modulate T-eff

39 nd established systemic lupus erythematosus, plasmacytoid dendritic cells are not effector cells, hav

40 We demonstrated that myeloid and plasmacytoid dendritic cells are recruited during the es

41 -hematopoietic cellular sources, rather than plasmacytoid dendritic cells, are responsible for interf

42 ent type I interferons (IFN-alpha/beta) from plasmacytoid dendritic cells as well as the production o

43 onventional, monocyte-derived, lymphoid, and plasmacytoid dendritic cells, as well as activated T-cel

44 of MHV68 in a subset of immune cells called plasmacytoid dendritic cells, as well as on the control

45 ells, natural killer cells, conventional and plasmacytoid dendritic cells, B cells and especially pla

46 endent mechanisms through TLR7 activation of plasmacytoid dendritic cells, B cells, and monocytes.

47 Plasmacytoid dendritic cells, B lymphocytes, and eosinop

48 1 receptor-associated kinase 1 expression in plasmacytoid dendritic cells both in vivo and in vitro,

49 s, including a series of 45 cases of blastic plasmacytoid dendritic cell (BPDC) neoplasms and their p

50 ytokine production, including IFN-alpha from plasmacytoid dendritic cells, but only in the presence o

51 We reveal that some cells, including plasmacytoid dendritic cells, can express both CCR2 and

52 nate and innate-like cell subsets, including plasmacytoid dendritic cells; CD1c + myeloid DCs; neutro

53 tion status, the numbers of conventional and plasmacytoid dendritic cells (cDCs and pDCs) were higher

54 the developmental origin of conventional and plasmacytoid dendritic cells (cDCs and pDCs, respectivel

55 Plasmacytoid dendritic cells (DCs [pDCs]) develop from p

56 his study, we show that conventional but not plasmacytoid dendritic cells (DCs) are required for anti

57 significant loss of circulating myeloid and plasmacytoid dendritic cells (DCs) during acute IM, a lo

58 that a highly enriched population of bovine plasmacytoid dendritic cells (DCs) produced IFN in respo

59 factors as well as an increased frequency of plasmacytoid dendritic cells (DCs) that corresponded wit

60 unopathology; others, including basophil and plasmacytoid dendritic cell depletion, correlate strongl

61 ell contact, and induction was suppressed by plasmacytoid dendritic cell depletion.

62 Thus, upon VSV infection, plasmacytoid dendritic cell-derived IFN-I primarily prot

63 eage specification and induced monocytic and plasmacytoid dendritic cell differentiation instead.

64 We isolated plasmacytoid dendritic cells from healthy persons and fr

65 ification of CD38 as a critical regulator of plasmacytoid dendritic cell function in response to infl

66 his study, we report that DMF inhibits human plasmacytoid dendritic cell function through a mechanism

67 ess IFN-beta and CXCL10, and macrophages and plasmacytoid dendritic cells have a deficiency in activa

68 In addition, plasmacytoid dendritic cells have a transcriptional sign

69 Plasmacytoid dendritic cells have been implicated in the

70 tiation of these cytokines is not clear, but plasmacytoid dendritic cells have been thought to contri

71 require specific TLR stimulation, T-cell and plasmacytoid dendritic cell help for distinct activation

72 ways induce IFN-I production in CMV-infected plasmacytoid dendritic cells; however, the initial burst

73 F-alpha secretion and suppressed CpG-induced plasmacytoid dendritic cell IFN-alpha gene expression.

74 MyD88-independent manner, while we confirmed plasmacytoid dendritic cell IFN-I had inverse requiremen

75 cted a higher frequency and proliferation of plasmacytoid dendritic cells in BAL fluid at 3 days post

76 assical dendritic cells and CD11c+ low)B220+ plasmacytoid dendritic cells in both the heart and splee

77 numbers of tolerogenic CD103(+) and PDCA1(+) plasmacytoid dendritic cells in GITR(-/-) mice.

78 n, and increased classic dendritic cells and plasmacytoid dendritic cells in peripheral blood and bon

79 to induce interferon-alpha in primary human plasmacytoid dendritic cells in response to hepatitis C

80 CXCL4 is the predominant protein secreted by plasmacytoid dendritic cells in systemic sclerosis, both

81 Plasmacytoid dendritic cells influenced by microbes migr

82 w that type I interferons (IFNs) produced by plasmacytoid dendritic cells inhibit the clearance of ap

83 -dependent recruitment and transformation of plasmacytoid dendritic cells into killer cells; this occ

84 IFN Regulatory Factor 5 (IRF5) in the human plasmacytoid dendritic cell line Gen2.2 prevented IFNbet

85 ypes of conventional dendritic cells (cDCs), plasmacytoid dendritic cells, macrophages (MPhis), B lym

86 Additionally, plasmacytoid dendritic cells maintained higher levels of

87 Activation of plasmacytoid dendritic cells, modulation of B-cell respo

88 e, memory and activated subsets, myeloid and plasmacytoid dendritic cells, monocytes, B lymphocytes,

89 Next, we showed that, in contrast to plasmacytoid dendritic cells, myeloid dendritic cells, c

90 We characterized plasmacytoid dendritic cell, natural killer (NK), and T-

91 dent oncogenic regulatory network in blastic plasmacytoid dendritic cell neoplasm (BPDCN) and demonst

92 Patients with blastic plasmacytoid dendritic cell neoplasm (BPDCN) have a poor

93 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare m

94 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is an aggre

95 Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is an uncom

96 et al describe a novel treatment of blastic plasmacytoid dendritic cell neoplasm (BPDCN) using an en

97 study of treatment of patients with blastic plasmacytoid dendritic cell neoplasm (BPDCN), an aggress

98 ing acute myeloid leukemia (AML) and blastic plasmacytoid dendritic cell neoplasm (BPDCN).

99 Blastic plasmacytoid dendritic-cell neoplasm (BPDCN) is an aggre

100 s of CD4 and memory B lymphocytes, decreased plasmacytoid dendritic cell numbers, and increased repre

101 on of the gut-homing receptor alpha4beta7 on plasmacytoid dendritic cells (p < 0.01) and the magnitud

102 HIV modulates plasmacytoid dendritic cell (pDC) activation via Toll-li

103 We identified that transient plasmacytoid dendritic cell (pDC) depletion during prima

104 nscription factor is required for B cell and plasmacytoid dendritic cell (pDC) development, but its m

105 HIV-2 favored plasmacytoid dendritic cell (pDC) differentiation into c

106 The plasmacytoid dendritic cell (pDC) is vital to the coordi

107 expression of the monocyte marker, CD14; the plasmacytoid dendritic cell (pDC) marker, BDCA4, identif

108 Blastic plasmacytoid dendritic cell (PDC) neoplasm (BPDCN) is an

109 induced limited type I interferon (IFN) and plasmacytoid dendritic cell (pDC) responses.

110 immune complexes that amplify TLR9-mediated plasmacytoid dendritic cell (pDC)-hyperactivation and in

111 of CD8alpha(+)beta(+) and CD8alpha(+)beta(-) plasmacytoid dendritic cells (pDC) and increased recruit

112 Human plasmacytoid dendritic cells (pDC) are important modulat

113 Plasmacytoid dendritic cells (pDC) are key regulators of

114 Plasmacytoid dendritic cells (pDC) are sentinels prepare

115 Plasmacytoid dendritic cells (pDC) are specialized in se

116 Plasmacytoid dendritic cells (pDC) are the major produce

117 Plasmacytoid dendritic cells (pDC) are the producers of

118 vesicular stomatitis virus (VSV) particles, plasmacytoid dendritic cells (pDC) are triggered to moun

119 Plasmacytoid dendritic cells (pDC) are type I interferon

120 Plasmacytoid dendritic cells (pDC) constitute the body's

121 Here, we report that AGM plasmacytoid dendritic cells (pDC) express extremely low

122 Plasmacytoid dendritic cells (pDC) have been regarded as

123 s, cytokines produced in large quantities by plasmacytoid dendritic cells (pDC) in response to engage

124 edge, nothing is known about the survival of plasmacytoid dendritic cells (pDC) in such situation.

125 We show that plasmacytoid dendritic cells (pDC) of natural hosts disp

126 ns, focusing on effects of IFN-alphabeta and plasmacytoid dendritic cells (pDC) on Th2 immune respons

127 Following IAV infection, plasmacytoid dendritic cells (pDC) or CD8alpha(+) DC reg

128 Plasmacytoid dendritic cells (pDC) produce IFN-I in resp

129 se to the Toll-like receptor-9 agonist CpGA, plasmacytoid dendritic cells (pDC) produced type 1 IFNs,

130 Plasmacytoid dendritic cells (pDC) rapidly and massively

131 8(+) Tregs, total and IFN-alpha synthesizing plasmacytoid dendritic cells (pDC) relative to plasma vi

132 the alpha interferon (IFN-alpha) response of plasmacytoid dendritic cells (pDC) to MHV68 was reduced

133 Enzymatic IDO1 activity, TGF-beta, and plasmacytoid dendritic cells (pDC) were neutralized by 1

134 Unexpectedly, plasmacytoid dendritic cells (pDC) were not recruited to

135 7 triggers rapid sensing of nucleic acids by plasmacytoid dendritic cells (pDC).

136 ion against cancer using naturally occurring plasmacytoid dendritic cells (pDC).

137 on concomitantly trigger activation of human plasmacytoid dendritic cells (pDC).

138 ports suggest involvement of CD4 T cells and plasmacytoid dendritic cells (pDC).

139 nt on ICOS-L costimulation provided by tumor plasmacytoid dendritic cells (pDC).

140 he microbiome alters TLR7-MyD88 signaling in plasmacytoid dendritic cells (pDCs) and blunts systemic

141 "auto"-regulatory feedback mechanism between plasmacytoid dendritic cells (pDCs) and Breg cells.

142 derived from mice and humans, as well as in plasmacytoid dendritic cells (pDCs) and CD141(+) DCs fro

143 dendritic cells (DCs) can be distinguished, plasmacytoid dendritic cells (pDCs) and CD16(+), CD1c(+)

144 In this study we determined whether plasmacytoid dendritic cells (pDCs) and CD8 T cells from

145 We show that chronic activation of plasmacytoid dendritic cells (pDCs) and elevated type-I

146 hypothesized that cell-cell contact between plasmacytoid dendritic cells (pDCs) and HCV-infected cel

147 d gnotobiotic conditions, notably increasing plasmacytoid dendritic cells (pDCs) and interferon signa

148 factor alpha (TNF-alpha) production by human plasmacytoid dendritic cells (pDCs) and monocytes, and i

149 The numbers of plasmacytoid dendritic cells (pDCs) and naive T cells (T

150 Following the discovery of plasmacytoid dendritic cells (pDCs) and of their extraor

151 1 infection induces persistent activation of plasmacytoid dendritic cells (pDCs) and production of ty

152 Plasmacytoid dendritic cells (pDCs) and type I IFN drive

153 Plasmacytoid dendritic cells (pDCs) are a dendritic cell

154 During microbial infections, plasmacytoid dendritic cells (pDCs) are a main source of

155 Plasmacytoid dendritic cells (pDCs) are a major source o

156 Plasmacytoid dendritic cells (pDCs) are a unique sentine

157 Plasmacytoid dendritic cells (PDCs) are central in antiv

158 Plasmacytoid dendritic cells (pDCs) are considered poten

159 Plasmacytoid dendritic cells (pDCs) are considered to be

160 Plasmacytoid dendritic cells (pDCs) are important early

161 Plasmacytoid dendritic cells (pDCs) are important in ant

162 In vitro evidence suggests that plasmacytoid dendritic cells (pDCs) are intimately invol

163 Plasmacytoid dendritic cells (pDCs) are key components o

164 Plasmacytoid dendritic cells (pDCs) are key players in t

165 Plasmacytoid dendritic cells (pDCs) are major type-I int

166 Plasmacytoid dendritic cells (pDCs) are potent producers

167 Plasmacytoid dendritic cells (pDCs) are primary producer

168 Airway myeloid dendritic cells (mDCs) and plasmacytoid dendritic cells (pDCs) are professional ant

169 Plasmacytoid dendritic cells (pDCs) are professional typ

170 In chronic diseases, such as HIV infection, plasmacytoid dendritic cells (pDCs) are rendered dysfunc

171 Plasmacytoid dendritic cells (pDCs) are robust producers

172 Plasmacytoid dendritic cells (pDCs) are the major produc

173 Plasmacytoid dendritic cells (pDCs) are the major source

174 Plasmacytoid dendritic cells (pDCs) are the major type I

175 During infections and inflammation, plasmacytoid dendritic cells (pDCs) are the most potent

176 Plasmacytoid dendritic cells (pDCs) are the most powerfu

177 Plasmacytoid dendritic cells (pDCs) are vital to antivir

178 of antigen-presenting cells, in particular, plasmacytoid dendritic cells (pDCs) around each MTZ form

179 Plasmacytoid dendritic cells (pDCs) bridge innate and ad

180 the production of type I interferon (IFN) in plasmacytoid dendritic cells (pDCs) by binding to endoso

181 Plasmacytoid dendritic cells (pDCs) constitute a unique

182 rythematosus, type I IFN (IFN-I) produced by plasmacytoid dendritic cells (pDCs) critically promotes

183 Plasmacytoid dendritic cells (pDCs) efficiently produce

184 Plasmacytoid dendritic cells (pDCs) express Toll like re

185 rferon (IFN-I) signaling and IFN-I-producing plasmacytoid dendritic cells (pDCs) facilitate the diffe

186 Plasmacytoid dendritic cells (pDCs) from females produce

187 LR7 and TLR9 agonists was greatly delayed in plasmacytoid dendritic cells (pDCs) from IRAK1[D359A] mi

188 f the JCI, Mitchell and authors investigated plasmacytoid dendritic cells (pDCs) from the blood of in

189 nocytes, myeloid dendritic cells (mDCs), and plasmacytoid dendritic cells (pDCs) from three individua

190 The developmental origin of IFN-producing plasmacytoid dendritic cells (pDCs) has been uncertain.

191 Plasmacytoid dendritic cells (pDCs) have been proposed a

192 Although plasmacytoid dendritic cells (pDCs) have been shown to p

193 Plasmacytoid dendritic cells (pDCs) have long been impli

194 Plasmacytoid dendritic cells (pDCs) highly populate lung

195 bsets, we found that BCMA was transcribed in plasmacytoid dendritic cells (pDCs) in both blood and ly

196 Bone marrow plasmacytoid dendritic cells (pDCs) in patients with mul

197 The physiologic role played by plasmacytoid dendritic cells (pDCs) in the induction of

198 The potential contribution of plasmacytoid dendritic cells (pDCs) in the presentation

199 of metaflammation by recruiting circulating plasmacytoid dendritic cells (pDCs) into VAT through che

200 ryptococcus neoformans; however, the role of plasmacytoid dendritic cells (pDCs) is unknown.

201 cluding IFN-alpha, and sortilin depletion in plasmacytoid dendritic cells (pDCs) led to a reduction o

202 myeloid dendritic cells (MDDCs/mDCs), and by plasmacytoid dendritic cells (pDCs) of human and murine

203 Plasmacytoid dendritic cells (pDCs) play a crucial role

204 Plasmacytoid dendritic cells (pDCs) play a crucial role

205 Plasmacytoid dendritic cells (pDCs) play a crucial role

206 Human plasmacytoid dendritic cells (pDCs) play a major role in

207 type I IFN response by a small percentage of plasmacytoid dendritic cells (pDCs) present in the monoc

208 Plasmacytoid dendritic cells (pDCs) produce abundant typ

209 Plasmacytoid dendritic cells (pDCs) produce large amount

210 Plasmacytoid dendritic cells (pDCs) produce type I inter

211 Plasmacytoid dendritic cells (pDCs) produced FasL in res

212 Plasmacytoid dendritic cells (pDCs) rapidly produce larg

213 Plasmacytoid dendritic cells (pDCs) rapidly produce type

214 g protective antiviral immune responses, and plasmacytoid dendritic cells (pDCs) represent a major so

215 Plasmacytoid dendritic cells (pDCs) require a particular

216 ell TLR7 expression or temporal depletion of plasmacytoid dendritic cells (pDCs) slow progression; ho

217 stem due to unceasing IFN-alpha release from plasmacytoid dendritic cells (pDCs) stimulated by nuclei

218 Plasmacytoid dendritic cells (pDCs) that secrete large a

219 Ab response against HIV-1 p24 that activates plasmacytoid dendritic cells (pDCs) through FcgammaRIIa

220 terized the immune response of monocytes and plasmacytoid dendritic cells (pDCs) to influenza A virus

221 ntaining nucleoprotein autoantigens activate plasmacytoid dendritic cells (PDCs) to produce type I in

222 RNA from human Huh-7 hepatoma cells to human plasmacytoid dendritic cells (pDCs) triggers pDC alpha/b

223 gp96 preferentially engages conventional and plasmacytoid dendritic cells (pDCs) under low and high d

224 denitis tissues but localization in CD123(+) plasmacytoid dendritic cells (pDCs) was found only in ly

225 Plasmacytoid dendritic cells (pDCs) were considered to b

226 Plasmacytoid dendritic cells (pDCs) were identified as t

227 Plasmacytoid dendritic cells (pDCs) were isolated from w

228 Aberrant activation of plasmacytoid dendritic cells (pDCs) with excessive produ

229 Plasmacytoid dendritic cells (pDCs), a primary source of

230 ic for double-stranded DNA (dsDNA) activated plasmacytoid dendritic cells (pDCs), a type of cell of t

231 f PTB in macaque lungs include the influx of plasmacytoid dendritic cells (pDCs), an Interferon (IFN)

232 BM demonstrated increased CD9(-)Siglec H(hi) plasmacytoid dendritic cells (pDCs), and depletion of pD

233 N-alpha production was primarily mediated by plasmacytoid dendritic cells (pDCs), and was significant

234 Ialpha, including mast cells, basophils, and plasmacytoid dendritic cells (pDCs), are regulated by Ig

235 Plasmacytoid dendritic cells (pDCs), B cells and NK cell

236 due to differences in number or function of plasmacytoid dendritic cells (pDCs), because these cells

237 both conventional dendritic cells (cDCs) and plasmacytoid dendritic cells (pDCs), but whereas cDCs pr

238 HIV-1-dependent reduction of ILC3s required plasmacytoid dendritic cells (pDCs), IFN-I, and the CD95

239 , particularly type I interferons (IFNs) and plasmacytoid dendritic cells (pDCs), in the pathogenesis

240 Plasmacytoid dendritic cells (pDCs), prominent cells of

241 This was associated with an influx of plasmacytoid dendritic cells (pDCs), regulatory T cells,

242 ice enhances Fas ligand (FasL) expression on plasmacytoid dendritic cells (pDCs), resulting in apopto

243 Using isolated plasmacytoid dendritic cells (pDCs), we demonstrated tha

244 oduction upon incubation in vitro with human plasmacytoid dendritic cells (pDCs), whereas lentivirus

245 2 was sufficient for recognition of TgPRF by plasmacytoid dendritic cells (pDCs), whereas TLR11 and T

246 Here, we demonstrate that plasmacytoid dendritic cells (pDCs), which are prototypi

247 ritical virus-induced IFN-alpha responses of plasmacytoid dendritic cells (pDCs), which can be defici

248 This occurs via selective expansion of plasmacytoid dendritic cells (pDCs), which further augme

249 ce, we analyzed the role of this receptor in plasmacytoid dendritic cells (pDCs), which preferentiall

250 responses; therefore, we questioned whether plasmacytoid dendritic cells (pDCs), which produce IFN w

251 ive immunity and are massively produced from plasmacytoid dendritic cells (pDCs).

252 Type I IFN is mainly produced by plasmacytoid dendritic cells (pDCs).

253 able hematologic malignancy originating from plasmacytoid dendritic cells (pDCs).

254 ompanied by increases in CD80(+) and CD86(+) plasmacytoid dendritic cells (pDCs).

255 synergistic response in both mouse and human plasmacytoid dendritic cells (pDCs).

256 mplex- and TLR7-mediated activation of human plasmacytoid dendritic cells (pDCs).

257 A major source of IFN-alphabeta is plasmacytoid dendritic cells (pDCs).

258 miR-126 had high and specific expression by plasmacytoid dendritic cells (pDCs).

259 T cells (CD8+CD40Ig Tregs) interacting with plasmacytoid dendritic cells (pDCs).

260 face molecule selectively expressed by human plasmacytoid dendritic cells (pDCs).

261 (dsDNA) viruses in the endosome to stimulate plasmacytoid dendritic cells (pDCs).

262 reased the steady state number of intestinal plasmacytoid dendritic cells (pDCs).

263 IFN) produced by dendritic cells, especially plasmacytoid dendritic cells (pDCs).

264 SLE, a major source of type I IFNs being the plasmacytoid dendritic cells (pDCs).

265 ffectors predominantly produced by activated plasmacytoid dendritic cells (pDCs).

266 LL37 licenses autoreactivity by stimulating plasmacytoid dendritic cells-(pDCs)-Type I interferon (I

267 0.0001) and increased regulatory T-cell and plasmacytoid dendritic cell percentages.

268 circuit blocks E-protein activity to exclude plasmacytoid dendritic cell potential and explains the s

269 enotype and gene expression revealed a novel plasmacytoid dendritic cell precursor preferentially mob

270 nfluenza virus, the patient's leukocytes and plasmacytoid dendritic cells produced very little type I

271 and IFN-alpha production from monocytes and plasmacytoid dendritic cells, respectively, retaining th

272 nd type I interferons from human B cells and plasmacytoid dendritic cells, respectively.

273 It is known that plasmacytoid dendritic cells sense herpesvirus DNA in en

274 n of pro-inflammatory immune cells including plasmacytoid dendritic cells, T helper cells and plasma

275 that rCl-13(GPC/VGKS) infected fewer splenic plasmacytoid dendritic cells than rCl-13, yet the two vi

276 tologic cancer that is caused by transformed plasmacytoid dendritic cells that overexpress interleuki

277 gressive hematologic malignancy derived from plasmacytoid dendritic cells that typically involves the

278 munity, in which the differential ability of plasmacytoid dendritic cells to produce interferon alpha

279 cells, monocyte-derived dendritic cells, and plasmacytoid dendritic cells to vIL-10 suppresses multip

280 ment were also related to the proportions of plasmacytoid dendritic cells, to distinct expression pat

281 ed endothelial cells can promote myeloid and plasmacytoid dendritic cell transmigration across endoth

282 ontrast, the absence of Ly49Q did not affect plasmacytoid dendritic cell-triggering receptor expresse

283 human and murine models between neutrophils, plasmacytoid dendritic cells, type I IFNs, and endotheli

284 atures reflect the extent of activation in a plasmacytoid dendritic cell-type I IFN-T/B lymphocyte ne

285 gered the production of type I interferon by plasmacytoid dendritic cells via activation of Toll-like

286 term IDO expression in both conventional and plasmacytoid dendritic cells via autocrine or paracrine

287 une lupus patients can stimulate B cells and plasmacytoid dendritic cells via Toll-like receptors 7 a

288 cer, previously thought to be active only in plasmacytoid dendritic cells, was found to also be trans

289 e, Ly49Q, the single known MHC-I receptor on plasmacytoid dendritic cells, was shown to bind H-2D(b)

290 uction of B cells, natural killer cells, and plasmacytoid dendritic cells were blocked.

291 TLR7.1 plasmacytoid dendritic cells were cell-intrinsically act

292 T cells, B cells, type I IFN, IFN-gamma, and plasmacytoid dendritic cells were contributed to efficie

293 cells (MDDCs), myeloid dendritic cells, and plasmacytoid dendritic cells were examined.

294 ses, frequencies of tolerogenic CD103(+) and plasmacytoid dendritic cells were increased.

295 In addition, infiltrated plasmacytoid dendritic cells were the primary IL-10-secr

296 s, demonstrating that IL-36 acts directly on plasmacytoid dendritic cells, where it potentiates toll-

297 ted Treg cell boost involved TNF, OX40L, and plasmacytoid dendritic cells, whereas in a condition of

298 Moreover, CCL22 induces an influx of plasmacytoid dendritic cells, which correlates with high

299 interplay of Tregs, invariant NKT cells, and plasmacytoid dendritic cells, which results in suppressi

300 primary human epithelial cells, B cells, and plasmacytoid dendritic cells with dsDNA viruses induces