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1 form, an exponential elongation phase, and a plateau phase.
2 entified performance deterioration after the plateau phase.
3 , inflammation in control mice remained at a plateau phase.
4 on titre, followed by a distinct switch to a plateau phase.
5 s relative to the original template into PCR plateau phase.
6 owing a rapid burst phase followed by a slow plateau phase.
7 during the cardiac action potential waveform plateau phase.
8 burst phase and 1.0178 +/- 0.0003 during the plateau phase.
9 he burst phase and 1.0022 +/- 0.0003 for the plateau phase.
10 3-mo expansion phase followed by a long-term plateau phase.
11 g two distinct time intervals separated by a plateau phase.
12 ells, but only for fractionated radiation in plateau phase.
13 of stimulation but not during the subsequent plateau phase.
14 ill present with reduced efficacy during the plateau phase.
15 attenuated inactivation, primarily in the AP plateau phase.
16 hyperpolarizing for pulses during the early plateau phase.
17 nts' growth rates either slowed or reached a plateau phase.
18 cer cells growing either exponentially or in plateau phase.
19 erably between days, particularly during the plateau phase.
20 ) and [Ca(2+)](i) remained higher during the plateau phase.
21 diabetic corneas lost the normal rising and plateau phases.
22 fibrils with characteristic lag, growth, and plateau phases.
23 respective of PrP(C) expression level, and a plateau (phase 2), which continues until clinical onset
24 epolarizing currents during action potential plateau phase, (2) increasing intracellular Na(+) (Na(i)
25 resurgence of outward current terminates the plateau phase and is thus a key regulator of action pote
26 re typically older lung indexes entering the plateau phase and, therefore, unlikely to show spirometr
27 -furanosyl) uracil (FMAU) were determined in plateau-phase and exponentially growing cultures of 3 hu
28 inetics was sigmoidal, with lag, growth, and plateau phases as shown by thioflavin T and turbidity me
29 of initial peak and the remaining 40% of the plateau phase, as administration of TEA in combination w
30 RTTs also displayed the initial increase and plateau phase, but the third region was virtually absent
32 ment to decrease the telomerase activity (in plateau phase cells of RKO, HeLa; and growing cells of R
33 a mitotic shake-off technique as well as in plateau-phase cells arrested by growing to confluency.
34 ould, likewise, induce mutations specific to plateau-phase cells, aprt mutations induced by amsacrine
36 of 74 aprt mutations induced by treatment of plateau phase Chinese hamster ovary CHO cells with the r
38 ) after treatment with 1 microM amsacrine in plateau phase, compared with a spontaneous frequency of
40 TK(1) activities and S-phase fractions under plateau-phase conditions, consistent with a loss of norm
41 ter was controllable in both exponential and plateau phase cultures and support the plausibility of u
42 not be induced until 48 h after infection of plateau phase cultures but could still be induced 180 h
43 n daily fractions of 2.0 Gy were examined in plateau phase cultures of human tumor cells of varying i
48 , the fibrils formed in the lag, growth, and plateau phases have different structures, with new forms
49 ression profiling analyses were conducted on plateau phase human lung cancer (A549) cell cultures tre
51 currents with a pronounced nondesensitizing plateau phase in cells that co-expressed both subunits.
53 ted a transient [Ca(2+)](i) signal without a plateau phase in the presence of extracellular Ca(2+) an
54 ch apoptosis rarely occurred, followed by a "plateau" phase in which cell loss by apoptosis was compe
55 e action potential shape during the peak and plateau phases is determined primarily by transient outw
59 igh PCLI in patients with apparently stable, plateau phase MM is an adverse parameter that may predic
60 lly patients have been observed with stable, plateau phase MM with minimal numbers of residual light-
62 t 5% of the log-phase mutants and 16% of the plateau-phase mutants were +1 frameshifts, and all but o
65 ibrillation shocks and shocks applied at the plateau phase of a normal action potential produced by v
66 (EAD), or abnormal depolarization during the plateau phase of action potentials, is a hallmark of lon
68 ll single Ca(2+) channel currents during the plateau phase of broad action potentials raise local Ca(
69 nitial mobilization peak had dissipated, the plateau phase of calcium entry was unchanged (92 +/- 9 n
70 l-gating modifier capable of lengthening the plateau phase of cardiac action potential (without incre
71 current (ICaL) is a major determinant of the plateau phase of cardiac action potential and has a crit
72 ectively, the rising phase and the prolonged plateau phase of cerebellar Purkinje cell (PC) action po
76 monstrate that the establishment of the high plateau phase of infection is dependent on non-antibody-
78 icantly increased the firing rate during the plateau phase of stretch but not during the dynamic phas
79 In populations of quiescent myocytes, the plateau phase of the [Ca2+]i signal evoked by 2-M-S-ATP
80 l number of LCCs gating in mode 2 during the plateau phase of the action potential (AP) can trigger E
82 ch induces additional Ca2+ influx during the plateau phase of the action potential, further slowing t
83 ADs) are abnormal depolarizations during the plateau phase of the action potential, which are known t
87 across subjects and motor units) during the plateau phase of the submaximal isometric contractions (
89 ere consistent with a model wherein a normal plateau phase of variable length was followed by initiat
92 ions (EADs) that can occur either during the plateau phase or repolarizing phase of the action potent
94 To the degree that these factors diminished plateau phase pulmonary function, they may be important
95 progressively suppressed the voltage of the plateau phase (r=-0.90) with increasing Ito1 density.
100 loads >500 nM, recovery showed an additional plateau phase that was abolished i nm-chlorophenylhydraz
101 oads > 500 nM, recovery showed an additional plateau phase that was abolished in m-chlorophenylhydraz
102 emoved during the stable, CCh-induced Ca(2+) plateau phase, the decline of cytosolic Ca(2+) is much f
104 ivity changed little as cells moved from the plateau phase to exponential growth, suggesting that in
105 g the worst levels at week 24, followed by a plateau phase until week 48, and then an improvement of
106 idine-resistant Ca(2+) channels, whereas the plateau phase was attributed to Ca(2+) release from inos
108 by ATP and/or thapsigargin was lower but the plateau phase was similar in the PMCA1a expressing cells
109 ase (median duration of the installation and plateau phases was 6 [IQR 4-9] and 4 days [3-10], respec
112 f an initial peak followed by a steady state plateau phase which was observed for stimulation duratio
113 ng upstroke phase that initiated a sustained plateau phase, which was associated with Ca(2+) spikes i
114 burst phase and 1.0019 +/- 0.0007 during the plateau phase, while those for the hydroxylamine leaving