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1 y reduced by preabsorption with pooled human platelet concentrate.
2 levels of GF suggested to be associated with platelet concentrate.
3 atter requires transfusion of HLA-compatible platelet concentrates.
4 nological functions of leukocytes present in platelet concentrates.
5 g viruses, bacteria, and leukocytes in human platelet concentrates.
6 eloped to inactivate bacteria and viruses in platelet concentrates.
7 ed platelet-rich plasma to apheresis-derived platelet concentrates.
9 telet-rich fibrin (PRF), a second-generation platelet concentrate, and atorvastatin (ATV), a potent m
10 derivative, platelet-derived growth factor, platelet concentrates, and fibroblast-growth factor-2.
11 PLA2 was measured in packed red blood cells, platelet concentrates, and fresh frozen plasma over the
12 tokines in the plasma fraction of transfused platelet concentrates, and leukodepletion prior to stora
13 powerful marker to determine the quality of platelet concentrates, because it reflects metalloprotei
15 study, it can be concluded that PRF and PRGF platelet concentrate failed to augment clinical effects
16 nt feasibility of using UVB-irradiated human platelet concentrates for prevention of HLA alloimmuniza
17 ns were found in transfused patients, pooled platelet concentrates, fresh frozen plasma, and packed r
20 rial were to assess the clinical efficacy of platelet concentrate grafts (PCG) in the treatment of Mi
22 telet-rich fibrin (PRF), a second-generation platelet concentrate, has been the focus of intensive re
23 anufacture of buffy coat whole-blood-derived platelet concentrate have convinced the Canadian Blood S
28 eloped countries, bacterial contamination of platelet concentrates is the highest infectious risk in
34 40L were measured in blood components and in platelet concentrates (PCs) implicated in TRALI or contr
35 ve therapy for furcation defects, the use of platelet concentrates (PCs) in addition to open flap deb
36 hemorrhagic complications, administration of platelet concentrates, plasma, or coagulation factor con
37 or limitation, however, was the finding that platelet concentrates prepared from blood anticoagulated
39 a mouse model of platelet transfusion, aged platelet concentrates resulted in augmented inflammation
41 ferentiation after treatment with TGFbeta or platelet concentrate supernatant, assessed by alpha smoo
42 let-rich fibrin (PRF) is a second-generation platelet concentrate that releases various growth factor
43 healthy donor blood, but did not increase in platelet concentrates that caused adverse transfusion re
44 sfusion of 2 fresh ABO blood group-identical platelet concentrates (therapeutic units), ongoing plate
45 nal alterations of aging platelets extend to platelet concentrates, this may hold important implicati
46 Previous studies have shown that exposure of platelet concentrates to riboflavin and light (Mirasol P
48 he detection of a bacterial contamination of platelet concentrates was assessed using samples spiked
49 crease with filter A after the first 5 mL of platelet concentrates was filtered that returned to pref
51 randomized to receive prophylactic apheresis platelet concentrates when the platelet count was either