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1 y reduced by preabsorption with pooled human platelet concentrate.
2 levels of GF suggested to be associated with platelet concentrate.
3 atter requires transfusion of HLA-compatible platelet concentrates.
4 nological functions of leukocytes present in platelet concentrates.
5 g viruses, bacteria, and leukocytes in human platelet concentrates.
6 eloped to inactivate bacteria and viruses in platelet concentrates.
7 ed platelet-rich plasma to apheresis-derived platelet concentrates.
8 ation (18 patients), and PPV plus autologous platelet concentrate (22 patients).
9 telet-rich fibrin (PRF), a second-generation platelet concentrate, and atorvastatin (ATV), a potent m
10  derivative, platelet-derived growth factor, platelet concentrates, and fibroblast-growth factor-2.
11 PLA2 was measured in packed red blood cells, platelet concentrates, and fresh frozen plasma over the
12 tokines in the plasma fraction of transfused platelet concentrates, and leukodepletion prior to stora
13  powerful marker to determine the quality of platelet concentrates, because it reflects metalloprotei
14                             Similarly, human platelet concentrates decreased their hemostatic functio
15 study, it can be concluded that PRF and PRGF platelet concentrate failed to augment clinical effects
16 nt feasibility of using UVB-irradiated human platelet concentrates for prevention of HLA alloimmuniza
17 ns were found in transfused patients, pooled platelet concentrates, fresh frozen plasma, and packed r
18                                            A platelet concentrate graft (PCG) was applied underneath
19                                          The platelet concentrate graft may be an alternative graft m
20 rial were to assess the clinical efficacy of platelet concentrate grafts (PCG) in the treatment of Mi
21 n of red blood cells and whole blood-derived platelet concentrates has been reported.
22 telet-rich fibrin (PRF), a second-generation platelet concentrate, has been the focus of intensive re
23 anufacture of buffy coat whole-blood-derived platelet concentrate have convinced the Canadian Blood S
24              Platelet-rich fibrin (PRF) is a platelet concentrate having sustained release of various
25                                   Storage of platelet concentrates, however, is associated with a red
26       Growing evidence shows the efficacy of platelet concentrates in periodontal therapy.
27                           The topical use of platelet concentrates is recent, and its efficiency rema
28 eloped countries, bacterial contamination of platelet concentrates is the highest infectious risk in
29                                              Platelet concentrates may be prepared from whole blood o
30                                              Platelet concentrates (p = .004) were transfused less in
31                                              Platelet concentrate (PC) is known to contain growth fac
32                                         Both platelet concentrates (PC) derived from whole blood or s
33 ng bacteria, parasites, and viruses, in both platelet concentrates (PCs) and plasma.
34 40L were measured in blood components and in platelet concentrates (PCs) implicated in TRALI or contr
35 ve therapy for furcation defects, the use of platelet concentrates (PCs) in addition to open flap deb
36 hemorrhagic complications, administration of platelet concentrates, plasma, or coagulation factor con
37 or limitation, however, was the finding that platelet concentrates prepared from blood anticoagulated
38                                 In addition, platelet concentrates prepared from F-LR + MPR whole blo
39  a mouse model of platelet transfusion, aged platelet concentrates resulted in augmented inflammation
40 gatively affect availability and activity of platelet concentrate-suggested growth factors (GF).
41 ferentiation after treatment with TGFbeta or platelet concentrate supernatant, assessed by alpha smoo
42 let-rich fibrin (PRF) is a second-generation platelet concentrate that releases various growth factor
43 healthy donor blood, but did not increase in platelet concentrates that caused adverse transfusion re
44 sfusion of 2 fresh ABO blood group-identical platelet concentrates (therapeutic units), ongoing plate
45 nal alterations of aging platelets extend to platelet concentrates, this may hold important implicati
46 Previous studies have shown that exposure of platelet concentrates to riboflavin and light (Mirasol P
47      Extracellular mitochondria are found in platelet concentrates used for transfusion and are prese
48 he detection of a bacterial contamination of platelet concentrates was assessed using samples spiked
49 crease with filter A after the first 5 mL of platelet concentrates was filtered that returned to pref
50               One hundred sixty-six of these platelet concentrates were then transfused to 120 patien
51 randomized to receive prophylactic apheresis platelet concentrates when the platelet count was either
52 ed by PCFIA, occurs during leukodepletion of platelet concentrates with either filter A or B.