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1 in deposition of the perfused fibronectin in platelet thrombi.
2 L), is involved in thrombosis by stabilizing platelet thrombi.
3 llower platelets onto vanguard platelets and platelet thrombi.
4 WF multimers participate in the formation of platelet thrombi.
5 et glycoprotein (GP)-IIIa to identify fibrin-platelet thrombi.
6 vel approach for the dissolution of existing platelet thrombi.
7 or the PAR4-P2Y12 dependent stabilization of platelet thrombi.
8 ascular migration," specifically mediated by platelet thrombi.
9              It is manifested classically by platelet thrombi and localized microvascular endothelial
10 vasoconstriction, followed by dislodgment of platelet thrombi and restoration of blood flow at the si
11                                              Platelet thrombi and vascular inflammation are prominent
12 rombus instability, through fragmentation of platelet thrombi and/or enhanced endogenous fibrinolysis
13 cluding von Willebrand factor release, small platelet thrombi, and complement deposition.
14  have suggested that activated platelets and platelet thrombi can contribute significantly to the ini
15 th blocking P-selectin antibodies, developed platelet thrombi containing minimal tissue factor and fi
16 response to anti-CD40L ICs and had pulmonary platelet-thrombi devoid of fibrin.
17 ut not D117E-sCD40L-coated surfaces, induced platelet thrombi formation under arterial shear rate.
18 led a complex organization of the "caps." In platelet thrombi formed in whole blood on collagen under
19 ions (mesangiolysis, subendothelial lucency, platelet thrombi in glomerular capillary lumina).
20 so increased the size and persistence of VWF-platelet thrombi in ionophore-treated mesenteric microve
21 injury stimulates the formation of occlusive platelet thrombi in mice but not in the size- and flow-m
22 ly fatal disease characterized by widespread platelet thrombi in the microcirculation.
23                             The formation of platelet thrombi is determined by the integrin alphaIIbb
24 d that the formation of gap junctions within platelet thrombi is required for the control of clot ret
25 ild-type mice, low TF mice formed very small platelet thrombi lacking TF or fibrin.
26 of Blood, Welsh and colleagues determine how platelet thrombi limit the loss of plasma-borne proteins
27 ng xenograft rejection, we hypothesized that platelet thrombi might contribute to graft dysfunction d
28 ukocyte recruitment and migration induced by platelet thrombi occurred most prominently in veins but
29 dhesion events leading to the development of platelet thrombi on collagen fibrils.
30                     VWF-mediated increase in platelet thrombi on collagen was not enhanced and indeed
31 aques with or without superimposed transient platelet thrombi or altered vasomotor tone may be respon
32                                              Platelet thrombi that form in coronary and carotid arter
33                However, subsequent growth of platelet thrombi was markedly diminished in Par4(-/-) mi
34                                              Platelet thrombi were even greater when plasma fibronect
35                            Fibrin-containing platelet thrombi were observed at the LVV and in the ter
36                        Fibrin deposition and platelet thrombi were seen in both groups of recipients.