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1 -induced thrombocytopenia (HIT) is caused by platelet-activating anti-platelet factor 4 (PF4)/heparin
2                                              Platelet-activating anti-protamine-heparin antibodies sh
3                           Seven patients had platelet-activating, anti-protamine-heparin antibodies a
4 prothrombotic adverse drug effect induced by platelet-activating antibodies against multimolecular co
5 eparin reexposure can be safely performed if platelet-activating antibodies are no longer detectable
6 thrombocytopenia (HIT) can be appropriate if platelet-activating antibodies are no longer detectable.
7  a novel, unexpected, PS backbone-dependent, platelet-activating effect of nucleotide-based drug cand
8  mechanisms may contribute to the unexpected platelet-activating effects of pharmaceutical integrin a
9     The mechanism by which UFH elicits these platelet-activating effects, however, is not well unders
10 ceptor glycoprotein VI (GPVI) mediates these platelet-activating effects.
11 els for 1 hour and intraluminal injection of platelet activating factor (50 micro/kg).
12                                          The platelet activating factor (PAF) acetyl hydrolase 1b (Pa
13 alyzes the hydrolytic inactivation of plasma platelet activating factor (PAF) and is also known as PA
14                                              Platelet activating factor (PAF) and PAF-like lipids ind
15 as well as resveratrol and quercetin, on the platelet activating factor (PAF) biosynthetic enzymes, a
16 plexed with the physiological ligand GM2 and platelet activating factor (PAF) have shown binding at t
17                                              Platelet activating factor (PAF) interacts with cell sur
18 tenuate the permeability increase induced by platelet activating factor (PAF) or bradykinin.
19 th BN52021, WEB-2170, and WEB-2086 (specific platelet activating factor (PAF) receptor antagonists),
20 Both LPS and BLP stimulated the synthesis of platelet activating factor (PAF), a potent lipid mediato
21 rved that tumor necrosis factor (TNF)-alpha, platelet activating factor (PAF), and lipopolysaccharide
22 rmyl-methionyl-leucyl-phenylalanine (fMLP)), platelet activating factor (PAF), leukotriene B4 (LTB(4)
23 mbination with uridine 5'-diphosphate (UDP), platelet activating factor (PAF), or lysophosphatidic ac
24  incubated in H2O2 (70 micromol/L, 2 hours), platelet activating factor (PAF), prostaglandin E2 (PGE2
25 ion and chemoattraction, Mig potently blocks platelet activating factor (PAF)- and leukotriene B4 (LT
26 ed for lysophosphatidylcholine (lysoPCh) and platelet activating factor (PAF).
27 A crystal structure of GM2-AP complexed with platelet activating factor (PAF).
28 se, neutrophil activation in the presence of platelet activating factor (PAF).
29                                          Low platelet activating factor acetyl hydrolase activity was
30 oluble tumor necrosis factor receptor I, and platelet activating factor acetyl hydrolase.
31 with the HDL-associated enzymes paraoxonase, platelet activating factor acetylhydrolase (PAF), and gl
32 nts, the most potent effect was observed for platelet activating factor acetylhydrolase alpha (Paf-AH
33  mesangial cell line induced upregulation of platelet activating factor and the fibrotic marker TGF-b
34 d to surrogate PMNs respond to activation by platelet activating factor by initiating translation and
35 regation was blocked using a monoclonal anti-platelet activating factor receptor (PafR) antibody and
36                Here, we examined the role of platelet activating factor receptor (PAFR) in colitis-as
37 ine, an H1R antagonist, blocked EAE, and the platelet activating factor receptor antagonist CV6209 re
38 creased adherence mediated by binding to the platelet activating factor receptor on epithelial cells.
39 d receptor-1, protease activated receptor-3, platelet activating factor receptor, and factor V.
40 D synthase, histamine receptor type 1 (H1R), platelet activating factor receptor, Ig Fc epsilon recep
41 rotein is rapidly synthesized in response to platelet activating factor under the control of a specia
42 ng receptors for chemokines, PGs, histamine, platelet activating factor, and anaphylatoxin.
43 ith N-formyl-methionyl-leucyl-phenylalanine, platelet activating factor, and leukotriene B4 was phosp
44 d immunoglobulins (IgG, IgA), cytokines with platelet activating factor, calcium ionophore, or phorbo
45         NET formation after stimulation with platelet activating factor, ionomycin, or phorbol 12-myr
46 ogen synthase kinase 3beta inhibitor, blocks platelet activating factor-induced activation of glycoge
47 ulation of a G-protein coupled receptor with platelet activating factor.
48  significantly inhibited the biosynthesis of platelet activating factor.
49 hingomyelin (SM) to ceramide and inactivates platelet activating factor.
50 y alcohols, which are possible precursors of platelet activating factor.
51                                              Platelet-activating factor (1-O-alkyl-2-acetyl-sn-glycer
52                                              Platelet-activating factor (PAF [1-O-alkyl-2-acetyl-sn-g
53                           The plasma form of platelet-activating factor (PAF) acetylhydrolase (PAF-AH
54                                 Human plasma platelet-activating factor (PAF) acetylhydrolase functio
55  low density lipoprotein and comigrates with platelet-activating factor (PAF) acetylhydrolase on KBr
56                                          The platelet-activating factor (PAF) acetylhydrolase SsE pro
57 the PAFAH1B2 and PAFAH1B3 subunits of type I platelet-activating factor (PAF) acetylhydrolase, a phos
58              UV-induced keratinocyte-derived platelet-activating factor (PAF) activates mast cell mig
59 idized glycerophosphocholines (Ox-GPCs) with platelet-activating factor (PAF) activity produced non-e
60 uction of glycerophosphocholines that act as platelet-activating factor (PAF) agonists.
61 er short-chained phospholipids--inflammatory platelet-activating factor (PAF) and apoptotic oxidative
62 genesis and photoimmune suppression, such as platelet-activating factor (PAF) and serotonin (5-HT) re
63                                              Platelet-activating factor (PAF) and structurally-relate
64 perties of the yoghurts PL fractions against platelet-activating factor (PAF) and thrombin-induced pl
65                           Species related to platelet-activating factor (PAF) and/or lyso-phosphatidy
66                               Treatment with platelet-activating factor (PAF) antagonists also reduce
67         Over the past few years, the role of platelet-activating factor (PAF) as a potent mediator in
68                                      We used platelet-activating factor (PAF) as a proinflammatory ag
69 r wild-type control as experimental animals, platelet-activating factor (PAF) at 10(-7) m as the test
70                                              Platelet-activating factor (PAF) causes wheal and flare
71 at cigarette smoking leads to an increase in platelet-activating factor (PAF) content and PAF recepto
72 ured spleen and liver cytokine responses and platelet-activating factor (PAF) content in mice given C
73 -2) regulate surfactant protein-A (SP-A) and platelet-activating factor (PAF) expression, which incre
74 ittle, if any, role in neutrophil priming by platelet-activating factor (PAF) for OpZ-dependent respo
75                                              Platelet-activating factor (PAF) has been shown to affec
76            In the current study, the role of platelet-activating factor (PAF) in combination with tum
77 t studies have implicated the lipid mediator platelet-activating factor (PAF) in UVB-mediated systemi
78  models of LE that the phospholipid mediator platelet-activating factor (PAF) increases in LE and tha
79                                              Platelet-activating factor (PAF) increases permeability
80                             It is known that platelet-activating factor (PAF) induces severe endothel
81                       Evidence suggests that platelet-activating factor (PAF) is a mediator in inflam
82                                              Platelet-activating factor (PAF) is a mediator of inflam
83                                              Platelet-activating factor (PAF) is a naturally occurrin
84                                              Platelet-activating factor (PAF) is a phospholipid media
85           In this study, we demonstrate that platelet-activating factor (PAF) is a potent inducer of
86                                              Platelet-activating factor (PAF) is a potent lipid media
87                                              Platelet-activating factor (PAF) is a potent phospholipi
88                                              Platelet-activating factor (PAF) is a potent proinflamma
89                                              Platelet-activating factor (PAF) is a potent proinflamma
90                                              Platelet-activating factor (PAF) is a potent proinflamma
91                                              Platelet-activating factor (PAF) is a potent, bioactive
92                                              Platelet-activating factor (PAF) is a powerful proinflam
93                                              Platelet-activating factor (PAF) is a proinflammatory ph
94                                      Because platelet-activating factor (PAF) is a proximal mediator
95                             The phospholipid platelet-activating factor (PAF) is an important mediato
96                                              Platelet-activating factor (PAF) is an important mediato
97 tors of inflammation are not identified, but platelet-activating factor (PAF) is implicated as a pote
98                                              Platelet-activating factor (PAF) is implicated in the ca
99  Because the lipid mediator of inflammation, platelet-activating factor (PAF) is released by UV-irrad
100     Animal models show that the receptor for platelet-activating factor (PAF) is responsible for many
101                  Although the lipid mediator platelet-activating factor (PAF) is synthesized in respo
102                  Although the lipid mediator platelet-activating factor (PAF) is synthesized in respo
103                                              Platelet-activating factor (PAF) mediates an array of bi
104              Animal and human data show that platelet-activating factor (PAF) mediates the life-threa
105 ry potential of the proinflammatory mediator platelet-activating factor (PAF) on the subcellular dist
106 urvival after anaphylaxis, administration of platelet-activating factor (PAF) or histamine, and expos
107 th Kindlin-2 siRNA showed enhanced basal and platelet-activating factor (PAF) or lipopolysaccharide-s
108 esulted from inhibitory signals generated by platelet-activating factor (PAF) or oxidized LDL that ac
109 ncreased AA and PGE2 release, accompanied by platelet-activating factor (PAF) production.
110                          We hypothesize that platelet-activating factor (PAF) receptor (PAFR) ligatio
111                            LAU-0901, a novel platelet-activating factor (PAF) receptor antagonist, is
112 regation was inhibited by antagonists of the platelet-activating factor (PAF) receptor but not inhibi
113 tor and that antagonists of 5-HT(2A) and the platelet-activating factor (PAF) receptor can block cis-
114 f intracellular Leishmania, we surmised that platelet-activating factor (PAF) receptor had a signific
115                            Activation of the platelet-activating factor (PAF) receptor leads to a dec
116   Corneal stromal myofibroblasts express the platelet-activating factor (PAF) receptor, but its role
117 ba extracts and are known antagonists of the platelet-activating factor (PAF) receptor.
118     Ginkgolides are known antagonists of the platelet-activating factor (PAF) receptor.
119 igated for their ability to bind to a cloned platelet-activating factor (PAF) receptor.
120       These studies examined the role of the platelet-activating factor (PAF) signaling system in UVB
121                 Previously, we reported that platelet-activating factor (PAF) stimulates higher G pro
122 ne suppression are the binding of UV-induced platelet-activating factor (PAF) to its receptor and the
123  and the proinflammatory glycerophospholipid platelet-activating factor (PAF) were markedly reduced i
124 cell types with respect to the metabolism of platelet-activating factor (PAF), a biologically active
125 s by exploiting molecular mimicry to degrade platelet-activating factor (PAF), a host-derived inflamm
126                                              Platelet-activating factor (PAF), a phospholipid second
127                    We analyzed the effect of platelet-activating factor (PAF), a potent phospholipid
128    Recent studies suggest that the action of platelet-activating factor (PAF), a potent phospholipid
129                                              Platelet-activating factor (PAF), a potent phospholipid
130 ated endocytosis (CME), we hypothesized that platelet-activating factor (PAF), an effective chemoattr
131  in corneal epithelial cells stimulated with platelet-activating factor (PAF), and interferes with th
132  UVB radiation generates the lipid mediator, platelet-activating factor (PAF), as well as oxidized ph
133 nergistic signaling induced by serotonin and platelet-activating factor (PAF), but not histamine.
134                            The phospholipid, platelet-activating factor (PAF), exerts multiple physio
135 Leu-Phe (fMLP), adenosine diphosphate (ADP), platelet-activating factor (PAF), interleukin-8 (IL-8),
136  shown that the lipid inflammatory mediator, platelet-activating factor (PAF), synthesized by activat
137 mbrane inhibits hyperpermeability induced by platelet-activating factor (PAF), VEGF in ECV-CD8eNOSGFP
138 e calcium entry (SOCE) following the initial platelet-activating factor (PAF)-induced release of Ca(2
139                                    Degrading platelet-activating factor (PAF)-like lipids in L5 by a
140 ults in the production of the lipid mediator platelet-activating factor (PAF).
141 roduction of prostaglandin I(2) (PGI(2)) and platelet-activating factor (PAF).
142  is mediated, in part, by the lipid mediator platelet-activating factor (PAF).
143 rat small intestine and is down-regulated by platelet-activating factor (PAF).
144  secrete the lipid mediator of inflammation, platelet-activating factor (PAF).
145 P) protects mice from lethal challenges with platelet-activating factor (PAF).
146  mediators, including adhesion molecules and platelet-activating factor (PAF).
147 rete the inflammatory phospholipid mediator, platelet-activating factor (PAF).
148  phospholipids and the inflammatory mediator platelet-activating factor (PAF).
149  reduces neutrophil recruitment by targeting platelet-activating factor (PAF).
150         Among the proinflammatory mediators, platelet-activating factor (PAF, 1-O-alkyl-2-acetyl-sn-g
151 holine (ChoP), a ligand for the receptor for platelet-activating factor (rPAF).
152 gulation of expression of the plasma form of platelet-activating factor acetylhydrolase (PAF AH) in t
153                                              Platelet-activating factor acetylhydrolase (PAF-AH) is a
154                                   The enzyme platelet-activating factor acetylhydrolase (PAF-AH) rapi
155 or was inactivated by alkaline hydrolysis or platelet-activating factor acetylhydrolase (PAF-AH) trea
156 the possibility that the activity represents platelet-activating factor acetylhydrolase (PAF-AH), an
157                               We report that platelet-activating factor acetylhydrolase (PAFAH) Ib, c
158 t that the cytoplasmic PLA(2) enzyme complex platelet-activating factor acetylhydrolase (PAFAH) Ib, w
159 fective milk containing diminished levels of platelet-activating factor acetylhydrolase (PAFAH).
160                                              Platelet-activating factor acetylhydrolase (PLA2G7) is a
161 roteins such as alpha-N-catenin (CTNNA2) and platelet-activating factor acetylhydrolase 1b regulatory
162                                              Platelet-activating factor acetylhydrolase 1B1 (LIS1), a
163 eration by ectopic expression of PL-specific platelet-activating factor acetylhydrolase 2 (PAFAH2) ma
164  lycopene), and antioxidant enzyme activity (platelet-activating factor acetylhydrolase [PAF-AH] and
165     A recent report demonstrates that plasma platelet-activating factor acetylhydrolase activity incr
166 om allergy, such as suggesting that baseline platelet-activating factor acetylhydrolase activity leve
167                               Measurement of platelet-activating factor acetylhydrolase activity repr
168                                       Plasma platelet-activating factor acetylhydrolase becomes progr
169 lesterolemic human plasma with a recombinant platelet-activating factor acetylhydrolase completely ab
170 izygous mutations in the human gene encoding platelet-activating factor acetylhydrolase IB subunit al
171 imal study, transfection of tumor cells with platelet-activating factor acetylhydrolase inhibited tum
172 nt controversy over whether paraoxonase 1 or platelet-activating factor acetylhydrolase is responsibl
173 rpose of this study was to determine whether platelet-activating factor acetylhydrolase Sse and strep
174 creened by enzymatic assays for SpeB and the platelet-activating factor acetylhydrolase Sse, and a ne
175 coding complement component receptor 2/CD21, platelet-activating factor acetylhydrolase, and oxidized
176 tions in the relative levels of paraoxonase, platelet-activating factor acetylhydrolase, ceruloplasmi
177 lation and elongation protein zeta-1 (FEZ1), platelet-activating factor acetylhydrolase, isoform Ib,
178 e inconclusive in determining whether plasma platelet-activating factor acetylhydrolase, or lipoprote
179 nzymes, BChE and a new extracellular form of platelet-activating factor acetylhydrolase, PAFAH1b2.
180 ated phospholipase A2 (Lp-PLA2), also called platelet-activating factor acetylhydrolase, plays in ath
181                       Lp-PLA2, also known as platelet-activating factor acetylhydrolase, rapidly clea
182 hospholipase A(2) (Lp-PLA(2)), also known as platelet-activating factor acetylhydrolase.
183           In vivo and in vitro histamine and platelet-activating factor administration increases Fn14
184                             Finally, Tat and platelet-activating factor also increased neuronal vesic
185    In hippocampal slices exposed to a stable platelet-activating factor analogue, tetanic stimulation
186                                           As platelet-activating factor and HPC share structural semb
187 on anaphylaxis suggest an important role for platelet-activating factor and its relationship to the n
188 flammatory mediators (tumor necrosis factor, platelet-activating factor and prostaglandins), studies
189 locked by a combination of antihistamine and platelet-activating factor antagonist (but neither alone
190  Tsujimura et al. report that the release of platelet-activating factor by basophils stimulated with
191 se in cytosolic Ca2+ whereas the addition of platelet-activating factor did.
192 surfactant phospholipids and biosynthesis of platelet-activating factor in noninflammatory remodeling
193 -third, suggesting a role for toxin-produced platelet-activating factor in this process.
194           Using our model, we also show that platelet-activating factor is a crucial mediator of both
195 ceptor antagonists, or overexpression of the platelet-activating factor metabolizing enzyme acetylhyd
196  depletion by the G protein-coupled agonists platelet-activating factor or fMLP, but abolished agonis
197 tional state, and subsequent activation with platelet-activating factor or formyl-methionyl-leucyl-ph
198                                 Furthermore, platelet-activating factor or interleukin 8 acted in con
199 ormally sensitive to substance P, but not to platelet-activating factor or serotonin, suggesting that
200      Under the same experimental conditions, platelet-activating factor primed neutrophils for supero
201 factor, the ability of alpha-toxin to induce platelet-activating factor production was assessed, and
202 eir ability to generate oxidized lipids with platelet-activating factor receptor (PAF-R) agonist acti
203 by generating oxidized lipid agonists of the platelet-activating factor receptor (PAF-R).
204 er of melanoma metastasis, via activation of platelet-activating factor receptor (PAFR) and cAMP-resp
205 dditional recognition system mediated by the platelet-activating factor receptor (PAFr) and directed
206 e hypothesis that influenza up-regulates the platelet-activating factor receptor (PAFr) and thereby p
207                                Inhibitors of platelet-activating factor receptor (PAFR) blocked LTA-
208            We have previously shown that the Platelet-Activating Factor Receptor (PAFR) engagement in
209                                          The platelet-activating factor receptor (PAFr) has been sugg
210 hibitors identified a prominent role for the platelet-activating factor receptor (PAFr) in hypoxia-as
211                                              Platelet-activating factor receptor (PAFR) is a G protei
212                                              Platelet-activating factor receptor (PAFr) mediates pneu
213 f synaptic activity in a manner dependent on platelet-activating factor receptor (PAFR) signaling.
214 polymeric immunoglobulin receptor (pIgR) and platelet-activating factor receptor (PAFr) to attach and
215 iciency in interleukin 1alpha (IL-1alpha) or platelet-activating factor receptor (PAFR), an important
216  of A. baumannii binds to A549 cells through platelet-activating factor receptor (PAFR), resulting in
217                      Moreover, expression of platelet-activating factor receptor (PAFR), which is kno
218 nvolved in immune responses include CD36 and platelet-activating factor receptor (PAFR).
219 r airway cells is mediated by host-expressed platelet-activating factor receptor (PAFR).
220 oupled receptors and excluded key candidates platelet-activating factor receptor and CCR5.
221 experiments revealed that L5 signals through platelet-activating factor receptor and lectin-like oxid
222 s demonstrate the differential expression of platelet-activating factor receptor and TLR-4 in uterine
223 nti-eotaxin and anti-RANTES mAbs, but not by platelet-activating factor receptor antagonists (CV6209,
224             Pretreatment of HaCaT cells with platelet-activating factor receptor antagonists, or over
225                                              Platelet-activating factor receptor blockade protected s
226 tion was assessed, and whether the epidermal platelet-activating factor receptor could augment toxin-
227 ctive effect resulted in part from decreased platelet-activating factor receptor expression on endoth
228 antibody or therapeutic agents that modulate platelet-activating factor receptor expression, may conf
229 stry studies demonstrate the presence of the platelet-activating factor receptor in both endometrial
230 nally, retroviral-mediated expression of the platelet-activating factor receptor into the platelet-ac
231                 Anaphylaxis was inhibited by platelet-activating factor receptor or histamine recepto
232 milar involvement for an unrelated GPCR (the platelet-activating factor receptor), indicating that it
233    Bacterial lipoteichoic acid activates the platelet-activating factor receptor, which is G protein-
234 rom the international phase III trial of the platelet-activating factor receptor-antagonist Lexipafan
235 platelet-activating factor receptor into the platelet-activating factor receptor-negative epithelial
236 ent studies used model systems consisting of platelet-activating factor receptor-positive and -negati
237 lin receptor, or cell wall phosphorylcholine/platelet-activating factor receptor.
238 signaling can be augmented via the epidermal platelet-activating factor receptor.
239 holine residues on pneumococcal cell wall to platelet-activating factor receptor.
240 hil, macrophage, and neutrophil secretion of platelet-activating factor subsequent to FcgammaR stimul
241  of PMN cationic entry after thapsigargin or platelet-activating factor suggested that SOCE occurs th
242             Lysophosphatidylcholine and lyso-platelet-activating factor were also associated directly
243 as prior exposure to a p38-activating agent (platelet-activating factor) diminished the TNFalpha-indu
244 some of these peptides (eg, endothelin-1 and platelet-activating factor) has decreased mortality and
245 nt chemicals (bradykinin, capsaicin, low pH, platelet-activating factor), whereas Adelta-fibres are r
246  that were 50-70% of those induced by 100 nM platelet-activating factor, a positive control.
247 toxic injury via activation of receptors for platelet-activating factor, a proinflammatory signaling
248 tic oxidatively truncated phospholipids, and platelet-activating factor, a remarkably potent and plei
249 ory cellular metabolite up-regulated by Tat, platelet-activating factor, also induced oxidative stres
250                               We report that platelet-activating factor, an inflammatory phospholipid
251 ory G protein-coupled chemoattractants fMLP, platelet-activating factor, and IL-8 elicit unique patte
252 ipids, including sphingosine, sphingomyelin, platelet-activating factor, and lysophosphatidylcholine.
253                Lysophosphatidylcholine, lyso-platelet-activating factor, and several phospholipid fat
254 production induced by TNF-alpha, GM-CSF, and platelet-activating factor, and this loss of pyk2 activi
255 e mediated by the autocrine activity, not of platelet-activating factor, but rather of endogenous CCR
256 arturition signals, surfactant protein A and platelet-activating factor, by the developing mouse feta
257 irway eosinophils to chemoattractants (FMLP, platelet-activating factor, CCL11, CCL5, CXCL8) with res
258 cellular calcium concentration stimulated by platelet-activating factor, fMLP, and SP-G.
259 duction of prostaglandins, leukotrienes, and platelet-activating factor, is present in EAE lesions.
260 merous chemoattractants, such as chemokines, platelet-activating factor, or FMLP, through a process k
261 ls with G protein-coupled receptor agonists, platelet-activating factor, or FMLP.
262  mediators, including TNF-alpha, GM-CSF, and platelet-activating factor, overcomes the adhesion-media
263 -alkyl-2-acetyl-sn-glycero-3-phosphocholine (platelet-activating factor, PAF) and the generation of 2
264 phils were treated with fMLP with or without platelet-activating factor, PMA alone, or tumor necrosis
265 ity, we found that histamine, serotonin, and platelet-activating factor, small molecule vascular perm
266 lus for the synthesis for the lipid mediator platelet-activating factor, the ability of alpha-toxin t
267       Depletion of basophils and blockade of platelet-activating factor, the key components of the Ig
268 ructurally diverse neutrophil-priming agents platelet-activating factor, TNF-alpha, and the substance
269 5-hydroxytryptamine), platelet factor 4, and platelet-activating factor, which specifically stimulate
270 lpha-toxin resulted in significant levels of platelet-activating factor, which were approximately 50%
271 l, lipid mediators lysophosphatidic acid and platelet-activating factor, whose signals are implicated
272 via CD14-, NF-kappaB-, and p44/42-dependent, platelet-activating factor-independent activation of the
273 otected primary astrocytes from H(2)O(2)- or platelet-activating factor-induced apoptosis.
274                        In ADSA lymphoblasts, platelet-activating factor-induced Ca(2+) mobilization w
275 ependent manner, LXs significantly inhibited platelet-activating factor-induced increases in leukocyt
276                                    Following platelet-activating factor-induced shock, MK886 increase
277  TWEAK-blocking antibody prevented histamine/platelet-activating factor-induced vascular subcutaneous
278 gosine blocks thapsigargin-, ionomycin-, and platelet-activating factor-mediated SOCE despite normal
279 hown that the protein acts primarily as lyso-platelet-activating factor-phospholipase C.
280 ins in whole blood and dampened responses to platelet-activating factor.
281 ed priming of this response by TNF-alpha and platelet-activating factor.
282 is induced by administration of histamine or platelet-activating factor.
283 rmation was also reversed by the addition of platelet-activating factor.
284 trates for the production of eicosanoids and platelet-activating factor.
285 -containing lipids, such as lyso-PC, PC, and platelet-activating factor.
286 2-AG) are unaffected by LPS but increased by platelet-activating factor.
287  human epithelial cells via the receptor for platelet-activating factor.
288 ector cells, and the mediators histamine and platelet-activating factor.
289 uding vascular endothelial growth factor and platelet-activating factor; by contrast, endothelial cel
290 owing to elevated levels of pro-inflammatory platelet-activating factors.
291            Immunoassays fail to discriminate platelet-activating from nonpathogenic antibodies.
292 action of unusual pathogenesis that features platelet-activating immunoglobulin G antibodies.
293 signaling cascade and inhibits virtually all platelet-activating key mechanisms.
294 f antibodies with strong heparin-independent platelet-activating properties are formed.
295 isposed to forming recurrent antibodies with platelet-activating properties.
296 bodies exhibiting strong heparin-independent platelet-activating properties.
297 tin-like receptor 2 (CLEC-2) is an essential platelet-activating receptor in hemostasis and thrombosi
298  C-type lectin-like receptor 2 are essential platelet activating receptors in hemostasis and thrombo-
299 omponents that specifically target different platelet-activating receptors such as the collagen-bindi
300 rades adenosine 5'-diphosphate (ADP), a main platelet activating/recruiting agent.

 
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