ライフサイエンスコーパス: pleocytosis

1 d to patients with cerebrospinal fluid (CSF) pleocytosis.

2 viral and 87% (20/23) of bacterial cases had pleocytosis.

3 s diagnostic criteria due to HIV-related CSF pleocytosis.

4 ging and 43 patients had cerebrospinal fluid pleocytosis.

5 (91%) of 11 children had cerebrospinal fluid pleocytosis.

6 s with and without cerebrospinal fluid (CSF) pleocytosis.

7 diagnostic criteria, due to HIV-related CSF pleocytosis.

8 d lethargy, and lumbar puncture might reveal pleocytosis.

9 presenting to emergency departments with CSF pleocytosis.

10 sually shows modest elevation of protein and pleocytosis.

11 the diagnostic sensitivity or specificity of pleocytosis.

12 igns or symptoms despite cerebrospinal fluid pleocytosis.

13 repeated CSF culture, even in the absence of pleocytosis.

14 CSF often displayed lymphocytic pleocytosis.

15 mmation, including cerebrospinal fluid (CSF) pleocytosis.

16 NS NS with gadolinium enhancement and/or CSF pleocytosis.

17 ctable in all samples and no participant had pleocytosis.

18 ents with normal CSF cell counts, those with pleocytosis (1) more often reported radicular pain and m

19 6)/L), with predominant lymphocytic moderate pleocytosis (100 x10(6)/L; reference range, 0-5 x10(6)/L

20 Among 3295 patients with CSF pleocytosis, 121 (3.7%; 95% confidence interval [CI], 3.

21 tive patients had lower proportions with CSF pleocytosis (16% vs 26% with >=5 white cells/muL) and CS

22 atients presented CSF abnormalities, such as pleocytosis (18/25, 72.0%), oligoclonal bands (18/25, 72

23 rospinal fluid analysis revealed predominant pleocytosis (23 x 10(6)/L; normal range, [0-5] x 10(6)/L

24 al fluid (CSF) analysis revealed predominant pleocytosis (23 x 106/L; normal range, [0-5] x 106/L) (7

25 araclinical testing revealed CSF lymphocytic pleocytosis (all 4 tested), electrographic seizures (3 o

26 CSF pleocytosis and abnormal glucose/protein were poor predi

27 e were negative for SARS-CoV-2, positive for pleocytosis and hyperproteinorrachia, and showed increas

28 Consistent with increasing CSF pleocytosis and intrathecal immunoglobulin production, i

29 CSF pleocytosis and protein elevation were each seen in 87.5

30 Cerebrospinal fluid pleocytosis and protein elevation were each seen in 87.5

31 Cerebrospinal fluid pleocytosis and rash are absent in about a third of case

32 rolipram (0-0.01 microgram/kg/h), inhibited pleocytosis and reduced the lipopolysaccharide-induced i

33 Degree of CSF pleocytosis and skin-lesion severity were estimated by t

34 Our objective was to determine if pleocytosis and/or elevated protein levels were predicti

35 All patients had pleocytosis, and 3 had severe prodromal diarrhea of unkn

36 symptoms, neutrophilic cerebral spinal fluid pleocytosis, and complement consumption, especially if t

37 ty (agitation, myoclonus, tremor, seizures), pleocytosis, and frequent diarrhea at symptom onset.

38 tutional symptoms, cerebrospinal fluid (CSF) pleocytosis, and hilar adenopathy more frequently than N

39 gs suggested that disease stage, lymphocytic pleocytosis, and HIV-1 RNA levels in plasma may influenc

40 titers, time to maximum titer, degree of CSF pleocytosis, and severity of skin lesions differed signi

41 Children with cerebrospinal fluid (CSF) pleocytosis are routinely admitted to the hospital and t

42 rebrospinal fluid (CSF) for parasites and/or pleocytosis are sensitive, but recent evidence suggests

43 Despite pleocytosis being associated with some measures of clini

44 of evaluable cerebrospinal fluid samples had pleocytosis, but HPeV was detected in 95%.

45 was strongly correlated to plasma RNA and to pleocytosis, but in AIDS, CSF and plasma RNA were indepe

46 3.4% (59/93) of positive specimens exhibited pleocytosis, compared to 29.5% (233/789) of negative spe

47 Notably, the group without pleocytosis comprised similar proportions of infectious

48 included neutrophilic cerebral spinal fluid pleocytosis, constitutive complement activation, female

49 r time to disability milestones, whereas CSF pleocytosis could be protective.

50 January 1, 2001, and June 30, 2004, with CSF pleocytosis (CSF white blood cells > or =10 cells/microL

51 The use of a pleocytosis cutoff of >=10 cells/mm(3) would have missed

52 hotomous-stage diagnosis on the basis of CSF pleocytosis does not accurately reflect the biological c

53 CSF) analysis revealed lymphocytic/monocytic pleocytosis, elevated protein concentration, and intrath

54 rebrospinal fluid demonstrated a lymphocytic pleocytosis, elevated protein, and negative MPXV-specifi

55 CSF pleocytosis, elevated protein, or depressed glucose conc

56 The positive predictive value of pleocytosis for microbiologically proven borrelial infec

57 than 0.5, a neutrophilic cerebrospinal fluid pleocytosis (> 5 cells/muL), and fever.

58 ts in all patients; however, 23.3% still had pleocytosis (>10 x 10(6) cells/L).

59 Cerebrospinal fluid (CSF) pleocytosis (>5 white blood cells/microL) was common (81

60 phalitis, mania, movement abnormalities, and pleocytosis (>=10 cells/mm(3) ) in cerebrospinal fluid a

61 On the other hand, the presence of CSF pleocytosis (>=5 cells/uL) increased time to moderate di

62 absence or presence of CSF white blood cell pleocytosis (>=5 cells/uL), to inform timely diagnosis a

63 d IL-8 levels independently from presence of pleocytosis/hyperproteinorracchia.

64 45%) and cerebrospinal fluid analysis showed pleocytosis in 17 of 22 patients (77%).

65 was present in 31 of 32 NS patients, and CSF pleocytosis in 27 of 32 (84%).

66 ts (65%) of patients and cerebrospinal fluid pleocytosis in 43 of 82 patients (52%).

67 -6, IL-8, chemokine ligand 2, and CXCL13 and pleocytosis in all infected animals, except dexamethason

68 However, an absence of CSF pleocytosis in encephalitis has been described, most not

69 All of the articles have shown CSF pleocytosis in MS.

70 tion should not be delayed in the absence of pleocytosis in patients with suspected encephalitis.

71 encephalitis, 47/247 (19%) had an absence of pleocytosis, including 18/76 (23.7%) with HSV-1 encephal

72 The presence of CSF pleocytosis increased ARR2 in RRMS (adjusted R(2)=0.036,

73 CSF pleocytosis is an important criterion for encephalitis d

74 CSF pleocytosis is associated with short-term inflammatory d

75 m 8 had CSF abnormalities (n = 3 lymphocytic pleocytosis, n = 5 elevated protein, n = 4 increased ope

76 patients (18%) and cerebrospinal fluid (CSF) pleocytosis occurred in 16 of 84 patients (19%).

77 CSF pleocytosis occurred in some participants receiving tofe

78 CSF pleocytosis occurred more commonly in infectious cases (

79 ients have a mononuclear cerebrospinal fluid pleocytosis, often with red blood cells.

80 ith HSV-1 encephalitis exhibit an absence of pleocytosis on initial LP.

81 CSF RNA levels were independent of CSF pleocytosis or antiretroviral exposure.

82 little specificity to the demonstration of a pleocytosis or increased overall or specific IgG product

83 Restricting FA-M/E orders based on pleocytosis or other abnormal parameters would have resu

84 ry parameter, including fever, leukocytosis, pleocytosis, or CSF protein and glucose, could reliably

85 1) and lower proportion of patients with CSF pleocytosis (P < .001).

86 meningismus (P = 0.04), cerebrospinal fluid pleocytosis (P = 0.04) or multifocal enhancing magnetic

87 at attack, p=0.007) and cerebrospinal fluid pleocytosis (p=0.005) were associated with a lower likel

88 ithout pleocytosis vs 71.1% in patients with pleocytosis; P < .001).

89 The CSF of all four patients contained a pleocytosis, predominantly mononuclear with elevated lev

90 All patients had abnormal CSF: six pleocytosis, six had increased protein concentration, an

91 ir administration (47.7% in patients without pleocytosis vs 71.1% in patients with pleocytosis; P < .

92 solated from EM skin lesions (odds ratio for pleocytosis was 31 times higher in patients with establi

93 The absence of pleocytosis was associated with a decreased rate of acyc

94 Cerebrospinal fluid pleocytosis was generally lacking.

95 p of patients aged >2 months, the absence of pleocytosis was highly predictive of a negative RT-PCR r

96 CSF pleocytosis was the most common PR006-related adverse ev

97 Severity of skin lesions and of CSF pleocytosis were inversely correlated (P=.005).

98 Of the 597 patients, 446 (74.7%) had CSF pleocytosis while 151 (25.3%) did not.

99 normalities with mildly elevated protein and pleocytosis with >90% lymphocytes, predominantly CD8, we

100 ebrospinal fluid analysis showed lymphocytic pleocytosis with elevated protein and normal glucose in

101 atients, cerebrospinal fluid analyses showed pleocytosis with elevated protein.

102 aboratory results (cerebrospinal fluid (CSF) pleocytosis with eosinophils and/or neutrophils, oligocl

103 evated protein concentration and lymphocytic pleocytosis with no malignant cells on cytological analy

104 Pleocytosis with white blood cell (WBC) levels of >=5 ce

105 Cerebrospinal fluid invariably shows a pleocytosis, with a predominance of neutrophils in up to

106 eadache, elevated intracranial pressure, and pleocytosis, with or without eosinophilia, particularly