ライフサイエンスコーパス: plethysmography

1 and calf vascular resistance (determined by plethysmography).

2 nce tomography and six respiratory inductive plethysmography).

3 Breathing was monitored with plethysmography.

4 by monitoring their sniffing with whole-body plethysmography.

5 r permeability was estimated by strain gauge plethysmography.

6 0% O2) with 5 min intervals, was measured by plethysmography.

7 nction was determined by peripheral arterial plethysmography.

8 composition was measured by air-displacement plethysmography.

9 choline challenge was measured by whole-body plethysmography.

10 russide were assessed using venous occlusion plethysmography.

11 ssessed by standard forearm venous occlusion plethysmography.

12 Respiration was examined by plethysmography.

13 sensitivity were assessed during LBNP using plethysmography.

14 ition was measured by using air-displacement plethysmography.

15 omposition was measured via air-displacement plethysmography.

16 week-old unanaesthetized rats via whole-body plethysmography.

17 re, and pulmonary function was determined by plethysmography.

18 oronasal airflow and respiratory inductance plethysmography.

19 ined by the forced oscillation technique and plethysmography.

20 elivery as measured by respiratory inductive plethysmography.

21 onchoconstriction was assessed by whole body plethysmography.

22 easured using venous-occlusion, strain-gauge plethysmography.

23 ted for every subject using venous occlusion plethysmography.

24 nitroprusside were measured by strain-gauge plethysmography.

25 CAD) using a robust, modified form of volume plethysmography.

26 re airway hyperreactivity through whole-body plethysmography.

27 ow responses were measured with strain-gauge plethysmography.

28 as measured using bilateral venous occlusion plethysmography.

29 position was measured using air displacement plethysmography.

30 (0.3 and 1 nmol/min) using venous occlusion plethysmography.

31 il) vasodilators were measured using forearm plethysmography.

32 coronary artery disease by venous occlusion plethysmography.

33 nitroprusside were measured by strain-gauge plethysmography.

34 and during reactive hyperemia by venous air plethysmography.

35 in 10 cirrhotic patients by venous occlusion plethysmography.

36 ults with concentrations of exhaled NO after plethysmography.

37 whites and 18 blacks by use of strain-gauge plethysmography.

38 ng, and body composition by air-displacement plethysmography.

39 lood flow was determined by venous occlusion plethysmography.

40 Forearm blood flow was determined by plethysmography.

41 g non-dominant forearm with venous occlusion plethysmography.

42 ntilation (VE) was measured using whole-body plethysmography.

43 ery and correlated well with measurements at plethysmography.

44 oxide production by forearm venous occlusion plethysmography.

45 d flow (FBF was measured by venous occlusion plethysmography.

46 ion reflex responses to ETS were examined by plethysmography.

47 d at postnatal day 21 using air-displacement plethysmography.

48 rimester was assessed using air displacement plethysmography.

49 diposity was assessed using air displacement plethysmography.

50 tolic BP (DBP) were measured using tail cuff plethysmography.

51 ) mice of C57BL6 background using whole body plethysmography.

52 e time as measuring breathing via whole body plethysmography.

53 sition was determined using air displacement plethysmography.

54 al pulse wave velocity, and venous occlusion plethysmography.

55 n at 2 wk was determined by air-displacement plethysmography.

56 y hyperresponsiveness was evaluated by using plethysmography.

57 ume measurements using respiratory inductive plethysmography.

58 nce in live anesthetized mice using invasive plethysmography.

59 ation, as measured by respiratory inductance plethysmography.

60 m vasodilator responses to isoprenaline with plethysmography.

61 pedance tomography and respiratory inductive plethysmography.

62 zoelectric sensor, and inductive respiratory plethysmography.

63 rements were taken by using air displacement plethysmography.

64 measured lung function using spirometry and plethysmography.

65 Ventilation was measured by whole-body plethysmography.

66 using invasive tracheostomy and unrestrained plethysmography.

67 k EMG recordings and breathing by whole-body plethysmography.

68 l arm blood flow as assessed by strain-gauge plethysmography.

69 h COPD underwent MR imaging, spirometry, and plethysmography.

70 tion was measured in nonanesthetized mice by plethysmography.

71 s, left/right limbs 14/9, using strain gauge plethysmography; 7/16 of these had thrombosis extending

72 sodilation, by both forearm venous occlusion plethysmography (93 +/- 67% vs. 145 +/- 106%, p = 0.006)

73 and FFM were assessed using air displacement plethysmography a median of 6 times between birth and 6

74 re, electrocardiogram, and oxygen saturation plethysmography activity were recorded and reviewed for

75 to determine the ability of air-displacement plethysmography (ADP) and formulas based on triceps skin

76 l age (GA), with the use of air-displacement plethysmography (ADP) and the lambda-mu-sigma method for

77 Air-displacement plethysmography (ADP) is a good candidate for monitoring

78 mposition measurement using air displacement plethysmography (ADP) that was used to calculate a refer

79 , 4.5, and 6 mo of age with air-displacement plethysmography (ADP) validated against a stable isotope

80 ave 4C measures taken: DXA, air-displacement plethysmography (ADP), and total body water (TBW).

81 ay absorptiometry (DXA) and air displacement plethysmography (ADP).

82 tokine profile from reactivated splenocytes, plethysmography after aerosol challenge to pollen, cell,

83 Analysis by plethysmography after challenge revealed an attenuation

84 ng forearm blood flow (FBF; venous occlusion plethysmography) after 5 minutes of arterial occlusion.

85 analysed in unanaesthetised animals by body plethysmography along with rate of O2 consumption (VO2))

86 rate respiratory defects based on quantified plethysmography analyses.

87 Lung volumes were estimated by plethysmography and a standard ECG obtained before and a

88 r ~5 y in >1200 children by air displacement plethysmography and anthropometrics.

89 udy, we established dwarf hamster whole-body plethysmography and assessed disease severity and propen

90 3 months corrected age with air displacement plethysmography and at 1-year-corrected age with skin-fo

91 factors were assessed with venous occlusion plethysmography and blood sampling during intra-arterial

92 , lung function was determined by whole-body plethysmography and bronchoalveolar lavage fluid was ana

93 ouble-blind, randomized case-control forearm plethysmography and crossover systemic interventional st

94 g were studied longitudinally with tail-cuff plethysmography and echocardiography.

95 espiratory circuits, we performed whole-body plethysmography and electrophysiological recordings from

96 ood pressure (SBP) was measured by tail-cuff plethysmography and fecal microbiota analyzed by16S rRNA

97 tions were determined noninvasively by photo-plethysmography and impedance cardiography.

98 Using barometric whole-body plethysmography and increases in enhanced pause (Penh) a

99 Forearm venous occlusion plethysmography and intra-arterial infusions of acetylch

100 n as 3-compartment model by air displacement plethysmography and isotope dilution in early (13-16 wee

101 Lung function was assessed with body plethysmography and lung clearance index (LCI).

102 Respiratory pattern was measured by plethysmography and quantified by an apnoea-hypopnoea in

103 tion was assessed through the use of forearm plethysmography and related to clinical disease activity

104 We measured lung function (plethysmography and spirometry) and airway hyper-reactiv

105 Respiratory effects were studied using plethysmography and the P-glycoprotein role at the blood

106 ed forearm blood flow (FBF; venous occlusion plethysmography) and calculated the vascular conductance

107 easured forearm blood flow (venous occlusion plethysmography) and calculated vascular conductance (FV

108 vascular conductance (FVC, venous occlusion plethysmography) and cutaneous vascular conductance (CVC

109 rm blood flow (measured via venous occlusion plethysmography) and intra-arterial blood pressure to qu

110 ulmonary function studies (spirometry and/or plethysmography), and a cardiology evaluation which incl

111 ath washout, compared with 14 (47%) of 30 by plethysmography, and 4 (13%) of 30 by spirometry.

112 observed through antinociception, whole-body plethysmography, and blood-brain biodistribution studies

113 Spirometry, plethysmography, and carbon monoxide transfer factor wer

114 lood flow was determined by venous occlusion plethysmography, and dose-response curves were generated

115 ional blood volume distribution by impedance plethysmography, and head-up tilt testing.

116 methacholine was assessed by barometric body plethysmography, and numbers of lung eosinophils and lev

117 methacholine was assessed by barometric body plethysmography, and numbers of lung eosinophils and pro

118 hildren can perform multiple-breath washout, plethysmography, and spirometry at first attempt.

119 of the lung for carbon monoxide (TLco), body plethysmography, and spirometry were assessed post-bronc

120 performed multiple-breath inert gas washout, plethysmography, and spirometry.

121 graphy, graded treadmill exercise, tail cuff plethysmography, and telemetry (heart rate, activity, te

122 Blood flow was measured by venous occlusion plethysmography, and the percentage of HbO(2) perfusing

123 illary lactate (the primary endpoint), thumb plethysmography, and ulnar frame count to investigate th

124 ibrated and validated a new air-displacement plethysmography (AP) method for measuring body compositi

125 Recently, the use of air-displacement plethysmography (AP) was proposed as an accurate, comfor

126 , which was observed with the semi-automated plethysmography apparatus, but not a buried pellet test.

127 ycemia, we studied forearm blood flow (FBF) (plethysmography), arteriovenous glucose difference (AV-d

128 ss (FFM) were measured with air-displacement plethysmography at 2- and 6-mo corrected age.

129 ed as enhanced pause (Penh) with noninvasive plethysmography at Day 16 of age), and did not cause inf

130 ition was assessed by using air displacement plethysmography at day 2 and 2 months.

131 ition was assessed by using air-displacement plethysmography at discharge.

132 od flow, was measured noninvasively by using plethysmography at rest, after breathing supplemental ox

133 sophageal catheter or respiratory inductance plethysmography bands.

134 ial occlusion was determined by strain gauge plethysmography before and 4 h and six weeks after combi

135 ial acetylcholine) was assessed with forearm plethysmography before and after (1) 15-minute reperfusi

136 earm blood flow (FBF) using venous occlusion plethysmography before and after intra-arterial infusion

137 .2 microg/min) were assessed by strain-gauge plethysmography before and after nonselective blockade o

138 al voluntary contraction by venous occlusion plethysmography before and after regional inhibition of

139 rip and cold pressor test were determined by plethysmography before and during adrenergic receptor bl

140 latation were assessed with venous occlusion plethysmography before and during intra-arterial infusio

141 te-macrophage colony-stimulating factor) and plethysmography, before and after methacholine, to asses

142 blood flow was measured by venous occlusion plethysmography, before, and 8 min after, completing inf

143 y, nasal cannulas and respiratory inductance plethysmography belts connected to Nox-T 3 allows record

144 sistance or lung compliance measured by body plethysmography between infected and control mice.

145 re measured using bilateral venous occlusion plethysmography, bioimpedance cardiography, transthoraci

146 to 10 minutes of forearm arterial occlusion (plethysmography), blood pressure, and muscle sympathetic

147 ing) constant bias, and the air displacement plethysmography body density and SFT methods showed posi

148 -compartment, Db(VRJ), BIA, air displacement plethysmography body density, and SFT ranging from a mea

149 ood pressure and heart rate were measured by plethysmography; body composition was estimated by DEXA;

150 e-macrophage colony-stimulating factor), and plethysmography, both before and after methacholine trea

151 sed at birth and at 3 mo by air-displacement plethysmography by using the Pea Pod system (Cosmed) and

152 of 10 healthy volunteers enclosed in a body plethysmography chamber mimicking the entrapment environ

153 pliant protocol and underwent spirometry and plethysmography, completed the St George's Respiratory Q

154 Significantly, whole-body plethysmography confirmed that compound 53 reversed fent

155 ume (both at airway opening and by inductive plethysmography) could be identified on the deflation li

156 e (CV = 0.78-1.35%), except air displacement plethysmography (CV = 2.13%).

157 with the reduction in respiratory inductive plethysmography-derived lung volume, high continuous dis

158 tive pulmonary disease underwent spirometry, plethysmography, diffusing capacity of carbon monoxide,

159 We obtained spirometry, body plethysmography, diffusion capacity, respiratory muscle

160 cardiac output were made by venous occlusion plethysmography, Doppler flow wire and quantitative coro

161 pedance tomography and respiratory inductive plethysmography during a stepwise recruitment procedure.

162 blood flow was measured by venous occlusion plethysmography during an intrabrachial infusion of brad

163 , freely behaving rat pups, using whole-body plethysmography during breathing of room air (RA), durin

164 blood flow was measured by venous occlusion plethysmography during intrabrachial infusions of apelin

165 of FM, and FFM measured by air-displacement plethysmography during the first 5 mo of life.Applying p

166 n 503 symptom-free children using whole-body plethysmography during tidal breathing.

167 asurements were performed using strain-gauge plethysmography during transfusion, followed by testing

168 sing deuterium dilution and air-displacement plethysmography), eating behavior (measured by using a 3

169 We used plethysmography, electrophysiology, functional magnetic

170 a selection from raw recordings derived from plethysmography experiments and the analysis of these da

171 ion was assessed by forearm venous occlusion plethysmography, flow-mediated dilation, and pulse wave

172 All three visits utilized ECG and finger plethysmography for haemodynamic measures, and the high

173 e (HR), forearm blood flow (venous occlusion plethysmography), FVR, and MSNA (obtained through direct

174 ne resorption in rat AIA were assessed using plethysmography, histopathologic analysis, and immunohis

175 r results show that in whole-body barometric plethysmography, hM3Dq-mediated, global Foxb1(+) neuron

176 y frequency (RF) was monitored by whole-body plethysmography immediately after the 4th challenge (ear

177 hma were characterized with spirometry, body plethysmography, impulse oscillometry, alveolar and bron

178 hysiological tests, such as spirometry, body plethysmography, impulse oscillometry, and multiple brea

179 essed all participants with spirometry, body plethysmography, impulse oscillometry, multiple breath n

180 BQ-123 were assessed using venous occlusion plethysmography in 10 patients with syndrome X and 10 ma

181 Lung volumes were measured by spirometry and plethysmography in 109 healthy subjects aged 7-21 years.

182 lood flow was measured with venous occlusion plethysmography in 12 cigarette smokers and 12 age- and

183 blood flow was measured by venous occlusion plethysmography in 16 volunteers during infusion of SFLL

184 enous tone using radionuclide forearm venous plethysmography in 24 healthy subjects with no cardiovas

185 ) receptor blocker (BQ-123) were analyzed by plethysmography in 37 normotensive patients and 27 hyper

186 Blood flow was measured by plethysmography in healthy men.

187 minute volume were measured using whole-body plethysmography in rats administered GHB.

188 blood flow was measured by venous occlusion plethysmography in response to serial intra-arterial inf

189 viously determined by respiratory inductance plethysmography in that subject (i.e., particles inhaled

190 responses were assessed by venous occlusion plethysmography in the brachial circulation before and a

191 lood flow was measured with venous occlusion plethysmography in the resting forearm.

192 lf blood flow was measured with strain gauge plethysmography in the two higher dose treatment groups

193 (brachial catheter) and forearm blood flow (plethysmography) in 19 healthy subjects at baseline and

194 l improved peak blood flow (venous occlusion plethysmography) in arms (+24%, P=0.027) and legs (+23%,

195 We used in vivo plethysmography, in vitro electrophysiology, neuropharma

196 ed by a combination of whole-body barometric plethysmography, invasive measurement of airway resistan

197 Whole-body air-displacement plethysmography is a new practical alternative to these

198 nhaled methacholine by barometric whole-body plethysmography is a valid indicator of airway hyperresp

199 ve surrogates for cystometry, such as penile plethysmography, lack sufficient evidence to allow recom

200 ition was measured by using air-displacement plethysmography </=3 d after birth.

201 In Study 1, MSNA, blood flow (plethysmography), mean arterial pressure (MAP) and heart

202 Unrestrained whole-body plethysmography measurement of enhanced pause revealed s

203 ficantly correlated to respiratory inductive plethysmography measurements in 12 patients (mean r = 0.

204 On the basis of test lung data and plethysmography measurements, we also conclude that heli

205 Parameters studied included strain-gauge plethysmography measures of peripheral circulatory funct

206 electroencephalography, electrocardiography, plethysmography, mechanical ventilation or pharmacologic

207 lood pressure data by the indirect tail-cuff plethysmography method consistently shows increased pres

208 lood flow were evaluated by venous occlusion plethysmography, MSNA by microneurography, and blood pre

209 control subjects underwent spirometry, body plethysmography, multiple-breath inert gas washout (with

210 otocols mimicking those imposed by tail-cuff plethysmography (novel environment, heat, restraint, inf

211 abdominal wall motion using opto-electronic plethysmography (OEP), (ii) intra-thoracic and intra-abd

212 In vivo double-chamber plethysmography of Tau-P301L mice showed significantly r

213 nction in children with CF more readily than plethysmography or spirometry.

214 We used several techniques (plethysmography, organ baths, confocal microscopy, RT-PC

215 linical Severity Scoring (VCSS) systems, air-plethysmography (outflow fraction [OF], venous filling i

216 es (deltaV(L)[t]) via respiratory inductance plethysmography over a range of P(aw) settings in five p

217 Lung volume (respiratory inductive plethysmography), oxygen saturation, transcutaneous carb

218 ung volume measured by respiratory inductive plethysmography, oxygen saturation, perfusion index, reg

219 ted well with volumes obtained by total body plethysmography (p < 0.0001).

220 A-602 or vehicle and conscious, unrestrained plethysmography performed on days 0, 3, and 5 (n = 7 to

221 Subjects then underwent plethysmography (PL) followed by PET measurements of blo

222 l pressure (MAP), electrocardiography (ECG), plethysmography (Pleth), and the test systems utilized e

223 Keywords: whole body plethysmography; polymorphonuclear leukocytes; minute ve

224 monitoring was limited to blood pressure and plethysmography preoperatively and intraoperatively.

225 ) data available from respiratory inductance plethysmography provided important insight to changes in

226 t a constant level to acquire a pulse volume plethysmography (PVP) waveform and calibrate it to brach

227 h airway resistance measured with whole-body plethysmography (Raw-p) in 10 of the 16 infants.

228 l artery to measure forearm blood flow (FBF, plethysmography) responses to administration of isoprena

229 n, forearm blood flow (FBF; venous occlusion plethysmography) responses to brachial artery administra

230 Forearm blood flow (FBF, by strain-gauge plethysmography) responses to local intra-arterial infus

231 Calibrated inductive plethysmography revealed that 12 of 12 patients with Che

232 Respiratory parameters from plethysmography revealed that Dex-Asthma mice compensate

233 contribute to eventual neonatal death, since plethysmography revealed that E18.5 Spi1(-/-) embryos ar

234 Whole-body plethysmography revealed that KO neonates had aberrant r

235 Respiratory inductance plethysmography (RIP) and electrical impedance tomograph

236 utility of calibrated respiratory inductance plethysmography (RIP) and esophageal manometry to identi

237 nd, to address whether respiratory inductive plethysmography (RIP) could be used to monitor changes i

238 Respiratory inductance plethysmography (RIP) provides a noninvasive method of P

239 ed home monitor using respiratory inductance plethysmography (RIP) with simultaneously recorded polys

240 2) differentiated sum respiratory inductance plethysmography (RIP), and (3) nasal pressure.

241 ments were measured by respiratory inductive plethysmography (RIP).

242 ermination of cardiac function (by impedance plethysmography), serial pulmonary function tests (spiro

243 Using whole-body plethysmography, SH attenuated the acute (5 min) hypoxic

244 Supine venous occlusion plethysmography showed no differences between blood flow

245 lsatile and the nonpulsatile portions of the plethysmography signal and is, in part, determined by st

246 ultiple breath inert gas washout (MBW), body plethysmography, single-breath determination of carbon m

247 Tests included spirometry, plethysmography, sputum cell count, exhaled nitric oxide

248 s measured by spirometry (FEV1; n = 521) and plethysmography (sRaw; n = 567), and AHR by methacholine

249 pump function (CPF) measurements from venous plethysmography studies from 1998 to 2015.

250 vity was measured by non-invasive whole-body plethysmography, Th2 response and airway inflammation by

251 y pattern of the rat pups using flow-through plethysmography, then formed in situ preparations from t

252 kg (airway opening tidal volume), and 13.7% (plethysmography tidal volume) compared with total lung c

253 llary density (GCD); and 3) venous occlusion plethysmography to assess endothelium-dependent (% Hyper

254 and K(+)) analyses; and 2) venous occlusion plethysmography to assess peripheral microvascular filtr

255 to use the technique of abdominal inductance plethysmography to compare diurnal variation in girth in

256 ratory monitors using respiratory inductance plethysmography to detect apnea and obstructed breathing

257 nt, and ventilatory parameters by whole body plethysmography to determine the responses elicited by b

258 Subjects were studied using strain gauge plethysmography to measure blood flow, P(v), and the ven

259 Using barometric plethysmography to measure respiratory function, we foun

260 Finally, using whole-body plethysmography to measure volume changes in the lungs,

261 aired in the mutant mice, we used whole-body plethysmography to quantitatively measure odor-evoked sn

262 arm blood flow (FBF) responses (strain-gauge plethysmography) to intra-arterial infusion of a selecti

263 arm blood flow (FBF) responses (strain gauge plethysmography) to intraarterial infusion of a selectiv

264 We measured forearm blood flow responses (plethysmography) to isocapnic hypoxia (arterial saturati

265 Forearm blood flow responses (plethysmography) to mental stress were compared in 12 no

266 The following measurements were taken: FM by plethysmography, total and specific immunoglobulin E (Ig

267 , we introduce the concept of terahertz-wave-plethysmography (TPG), which detects blood volume change

268 ecorded in newborn rat pups using whole-body plethysmography under normoxic and hypoxic conditions.

269 healthy control subjects, with strain-gauge plethysmography used to measure venous pressure (Pv), fo

270 sessed by bilateral forearm venous occlusion plethysmography using acetylcholine and N-monomethyl-L-a

271 d methacholine were determined by whole body plethysmography using changes in enhanced pause (Penh) a

272 including compression ultrasound, impedance plethysmography, ventilation-perfusion scanning, compute

273 c wall motion were obtained using inductance plethysmography via adaptable belts.

274 ism of death in cases of viral encephalitis, plethysmography was evaluated in mice infected with 3 fl

275 Strain gauge plethysmography was performed on twenty-one young subjec

276 Venous occlusion plethysmography was used to assess forearm blood flow in

277 Venous occlusion plethysmography was used to assess resistance vessel res

278 Air-displacement plethysmography was used to measure body composition.

279 sponse), and in 6 subjects, venous occlusion plethysmography was used to measure forearm blood flow d

280 To address this question, venous occlusion plethysmography was used to measure forearm blood flow r

281 Venous occlusion plethysmography was used to measure peak blood flow and

282 Forearm blood flow (venous occlusion plethysmography) was measured during brachial intraarter

283 Ventilation, assessed via plethysmography, was attenuated during quiet breathing a

284 Pulmonary airflow resistance, as measured by plethysmography, was detected 1 day postinoculation and

285 pathology is the need to combine whole-body plethysmography (WBP) to measure respiration with electr

286 ) or noninvasive techniques, like whole body plethysmography (WBP), assesses the severity of pulmonar

287 Using impedance plethysmography, we assessed blood redistribution during

288 Using plethysmography, we investigated the effects of menthol

289 Using plethysmography, we studied the change in forearm blood

290 h the dorsal hand vein technique and forearm plethysmography, we studied the effects of PAR-2 activat

291 phageal manometry and respiratory inductance plethysmography were made preextubation during airway oc

292 spiration and expiration), and spirometry or plethysmography were performed during a 2-hour visit to

293 py, ultrasonic vocalizations, and whole-body plethysmography were used to assess VF motion, swallow f

294 h recorded CPF (28% air and 72% strain gauge plethysmography) were included; 591 (38.5%) had normal C

295 ial artery catheter) and forearm blood flow (plethysmography) were measured and vascular conductance

296 ram) and beat-to-beat blood pressure (finger plethysmography) were recorded from all participants, wh

297 Subjects (n=157) underwent venous occlusion plethysmography with acetylcholine, bradykinin, glyceryl

298 ssessed by standard forearm venous occlusion plethysmography with acetylcholine, nitroprusside, and v

299 ultaneously in both arms by venous occlusion plethysmography with mercury-in-Silastic strain gauges w

300 taneous breathing, assessed using whole-body plethysmography without anesthesia.