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1 othorax (lymphatic fluid accumulation in the pleural cavity).
2 d PAR2 (F2RL1) generated large tumors in the pleural cavity.
3 , was labeled by virus administered into the pleural cavity.
4 sive CD4+ T cells at the infection site, the pleural cavity.
5 ntained 78% of all CD4+ CTLA-4+ cells in the pleural cavity.
6 matically reduced neutrophil influx into the pleural cavity.
7 atory response around nurse cells and in the pleural cavity.
8 ion of radiolabeled transferrin in the mouse pleural cavity.
9 to PAF both in vitro and in vivo within the pleural cavity.
10 nsdiaphragmatically into the patient's right pleural cavity.
11 BALB/c mice, we analyze immune cells in the pleural cavity.
12 several human cancers to metastasize to the pleural cavity.
13 e of various human cancers metastatic to the pleural cavity.
14 hat predominantly inhabit the peritoneal and pleural cavities.
15 s in the mouse, including the peritoneal and pleural cavities.
16 ociated with an abnormal epithelium in their pleural cavities.
17 nd ascites accumulation in the peritoneal or pleural cavity, a major complication with a number of ma
18 re non-Hodgkin's lymphomas that arise in the pleural cavity after long-standing pleural inflammation
19 e increased proinflammatory cytokines in the pleural cavity and an influx of neutrophils within the l
20 uced recruitment of mononuclear cells to the pleural cavity and migration of macrophages at transwell
21 hysiological cues that activate FALCs in the pleural cavity and more generally the mechanisms control
22 y cell recruitment and bacterial load in the pleural cavity, and heightened levels of pleural and ser
24 gh found predominantly in the peritoneal and pleural cavities, B-1 cells are also present in other pe
25 deficiency virus (HIV)-negative patient with pleural cavity-based lymphoma that was passaged into tri
26 tes the pleuroperitoneal folds isolating the pleural cavities before the migration of the somitic myo
28 and inhibited tumour growth not only in the pleural cavity but also in the lung parenchyma, conferri
29 -derived cells accumulated in peritoneal and pleural cavities, but CD11c(+) CD226(+) macrophages were
30 od monocytes recruited to the peritoneal and pleural cavities constitutively, starting after birth, w
31 ytes present primarily in the peritoneal and pleural cavities is defined by the expression of CD5 and
32 eukocytes occupying the naive peritoneal and pleural cavities is higher in female than in male mice a
33 n of interleukin-10 (IL-10) by four distinct pleural cavity macrophage (M ) subpopulations characteri
34 number of macrophages and neutrophils in the pleural cavity, mainly by increasing the rate of neutrop
36 t drains the pericardial space into the left pleural cavity, might reduce the incidence of atrial fib
37 rease in the levels of IL-5 and IL-13 in the pleural cavities of sensitized NFAT1-/- mice after aller
40 r long CNTs or long asbestos fibers into the pleural cavity of mice induces mesothelioma that exhibit
42 isseminated pleural tumours, co-exist in the pleural cavity, presenting a major challenge for therape
43 lung inflammation, that inflammation of the pleural cavity rapidly activates mediastinal and pericar
44 crophage populations in mouse peritoneal and pleural cavities, the monocyte-derived, small peritoneal
45 instillation of long and short CNTs into the pleural cavity, the site of mesothelioma development, pr
47 t pleural macrophages (PMs) migrate from the pleural cavity to the lung after infection with IAV.
48 An injection of carrageenan (CAR) into the pleural cavity triggered an acute inflammatory response,
50 NK cells and DX5(+)/CD3(+) T cells into the pleural cavity, where the parasites were located, was ob
51 omised structural components surrounding the pleural cavity, which include rib protrusions, abnormal
52 n, and perhaps enzymatic pretreatment of the pleural cavity, will be necessary for efficient retrovir