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1 in the coding sequence of poliovirus (PV), a plus strand RNA virus.
2 tulated RNA domain within the 3' region of a plus-strand RNA virus.
3 brome mosaic bromovirus (BMV), a tripartite plus-strand RNA virus.
4 d intermediate, a hallmark of replication of plus-strand RNA viruses.
5 ion assay for tombusviruses, which are small plus-strand RNA viruses.
6 se properties appear to be similar for other plus-strand RNA viruses.
7 y be the most promising drug targets for all plus-strand RNA viruses.
8 bushy stunt virus (TBSV), which is a small, plus-stranded RNA virus.
9 organelles are important for replication of plus-stranded RNA viruses.
10 tion factors that inhibit the replication of plus-stranded RNA viruses.
12 tures in the 5' untranslated region (UTR) of plus-strand RNA viruses are known in many cases to funct
14 t-encoded proteins affect the replication of plus-stranded RNA viruses by acting as susceptibility fa
15 enal of proteins that inhibit replication of plus-stranded RNA viruses by functioning as cell-intrins
21 nal ribosomal entry sites (IRESs) of certain plus-strand RNA viruses direct cap-independent initiatio
22 umber necrosis tombusvirus (CNV), which is a plus-stranded RNA virus, from Saccharomyces cerevisiae.
24 tures in the 3' untranslated region (UTR) of plus-strand RNA viruses have been postulated to function
28 that forms a model for understanding single plus-stranded RNA virus infection, and reveals potential
29 tural life cycle of alphaviruses, a group of plus-strand RNA viruses, involves transmission to verteb
35 hich belongs to a different supergroup among plus-strand RNA viruses, is affected by vastly different
36 olated from tobacco plants infected with the plus-strand RNA virus, potato virus X (PVX), supported s
40 ore complexes; nuclear envelope reformation; plus-stranded RNA virus replication compartment formatio
43 es (DMVs) serve as replication organelles of plus-strand RNA viruses such as hepatitis C virus (HCV)
45 is a conserved protein fold found in several plus-strand RNA viruses that binds to the small molecule
47 potential as a reverse genetics platform for plus-stranded RNA viruses, the protocol has been success
49 viral pathogenesis factor, particularly for plus-strand RNA viruses, where immediate translation of
50 id and straightforward rescue of recombinant plus-stranded RNA viruses with high sequence fidelity us