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1 he ER and were involved in PA production for pollen tube growth.
2 urrents of Ca2+, H+, and K+ are critical for pollen tube growth.
3 to previous models for Ca(2+) regulation of pollen tube growth.
4 ckening of the apical cell wall and inhibits pollen tube growth.
5 ck mechanism for calcium oscillations during pollen tube growth.
6 candidates for important roles in regulating pollen tube growth.
7 ith different ligands at different stages of pollen tube growth.
8 n in a signal transduction pathway mediating pollen tube growth.
9 LePRK2 might participate in signaling during pollen tube growth.
10 in a common pathway with NtRac1 to regulate pollen tube growth.
11 ve been shown to be important regulators for pollen tube growth.
12 ADF1, was used to dissect the role of ADF in pollen tube growth.
13 ins to their normal locations, and inhibited pollen tube growth.
14 lved in perceiving extracellular cues during pollen tube growth.
15 c events required for pollen germination and pollen tube growth.
16 of actin turnover by NtADF1 is critical for pollen tube growth.
17 to be essential during pollen hydration and pollen tube growth.
18 pathway that regulates pollen hydration and pollen tube growth.
19 d in the binding of a specific ligand during pollen tube growth.
20 acquire water from the female, thus enabling pollen tube growth.
21 s controlling the irreversible inhibition of pollen tube growth.
22 cium is essential for pollen germination and pollen tube growth.
23 e for SUZ1 and ZWI in pollen germination and pollen tube growth.
24 through the S phase of the cell cycle during pollen tube growth.
25 at the pea Rop GTPase Rop1Ps is critical for pollen tube growth.
26 len maturation which is essential for normal pollen tube growth.
27 ttracting protein needed for optimal in vivo pollen tube growth.
28 es of development and during germination and pollen tube growth.
29 ss effects on pollen hydration, adhesion and pollen tube growth.
30 expression of its putative target genes and pollen tube growth.
31 ze the mitochondria and contribute to arrest pollen tube growth.
32 ect (segregation 40%:51%:9%) due to aberrant pollen tube growth.
33 ough the male gametophyte due to a defect in pollen tube growth.
34 ng wound signaling, stomatal regulation, and pollen tube growth.
35 pollen, while NIP4;2 expression peaks during pollen tube growth.
36 luble inorganic pyrophosphatase required for pollen tube growth.
37 imilar pollen myosins had greater defects in pollen tube growth.
38 e cell walls that provide less resistance to pollen tube growth.
39 ect leading to more severe depolarization of pollen tube growth.
40 l-associated TGases are believed to regulate pollen tube growth.
41 reduced germination efficiency, and reduced pollen tube growth.
42 erspecific) pollen through the inhibition of pollen tube growth.
43 a dominant-active form of wild-type LTP5 in pollen tube growth.
44 rt for a redundant function of AGC1.5/1.7 in pollen tube growth.
45 en maturation, pollen grain germination, and pollen tube growth.
46 viability, delayed germination, and aberrant pollen tube growth.
47 the initiation and regulation of oscillatory pollen tube growth.
48 e pollen tubes are important for controlling pollen tube growth.
49 le transmission efficiency due to defects in pollen tube growth.
50 low detailed gene expression analyses during pollen tube growth.
51 egulate PRONE function, leading to polarized pollen tube growth.
52 ng tract and stigma development and impaired pollen tube growth.
53 ly increased medium ATP levels and inhibited pollen tube growth.
54 the effect of overexpressing this isoform on pollen tube growth.
55 ling network, resulting in the inhibition of pollen-tube growth.
56 ant and we used it to investigate aspects of pollen-tube growth.
57 Cooperation between ovule attraction and pollen tube growth acceleration favors conspecific ferti
59 importance of the functional connections in pollen tube growth and can help guide future research di
61 l- fluxes are not a significant component of pollen tube growth and Cl- itself is not required for gr
62 ses (PMEs) likely play a central role in the pollen tube growth and determination of pollen tube morp
70 s indicate that Rop controls actin-dependent pollen tube growth and H(2)O(2)-dependent defense respon
71 utocrine and paracrine signaling to maintain pollen tube growth and induce timely tube rupture at the
72 roduction, which fuels single nucleus-driven pollen tube growth and is essential for plant reproducti
73 -P2 act in a common pathway to control polar pollen tube growth and provide direct evidence for a fun
76 gk4-1/+ double heterozygote showed defective pollen tube growth and seed development because of nonvi
77 reeding system was determined by analysis of pollen tube growth and seed production from controlled p
78 the female organs of the flower that support pollen tube growth and sperm cell transfer along the tra
79 ndings provide insight into the mechanism of pollen tube growth and the oscillation of cellular signa
81 TPases (AHAs) have been proposed to energize pollen tube growth and underlie cell polarity, however,
83 1) exhibited ballooned pollen tubes, delayed pollen tube growth, and decreased numbers of fertilized
84 such as pollen tube incompatibility, slower pollen tube growth, and delayed generative cell mitosis.
86 e TT slowed and then arrested N. obtusifolia pollen tube growth, and was developmentally synchronized
87 sion and guidance for pollen germination and pollen tube growth are abundantly present in the extrace
89 morphological defects in the early stages of pollen tube growth, arising from frequent changes to pol
90 ered clearly from the metabolic state during pollen tube growth, as indicated by principal component
92 tion half-maximally at 50 nM, yet it blocked pollen tube growth at one-tenth of that concentration.
94 rain and a functional phase representing the pollen tube growth, beginning with the landing of the po
97 vers of reproductive thermoresilience during pollen tube growth by comparing a set of thermotolerant
98 s thaliana Rho family GTPase, ROP1, controls pollen tube growth by regulating apical F-actin dynamics
101 ignaling network, resulting in inhibition of pollen tube growth, cytoskeletal alterations, and progra
102 haliana delays pollen germination and causes pollen tube growth defects, leading to drastically reduc
103 Ca(2+) availability partially suppresses the pollen tube growth defects, suggesting that LRX proteins
104 tween SI and UI in the tomato clade, in that pollen tube growth differs between these two rejection s
106 ces in our understanding of the mechanism of pollen tube growth, focusing on such basic cellular proc
107 e by examining seed set, pollen fitness, and pollen tube growth for knockout mutants of five of the s
108 role in the regulation of calcium-dependent pollen tube growth, H(2)O(2)-mediated cell death, and ma
109 OP1-overexpression-induced depolarization of pollen-tube growth identified REN1 (ROP1 enhancer 1) in
112 el that describes vesicle trafficking during pollen tube growth in Arabidopsis (Arabidopsis thaliana)
113 rol UCI phenotype by independently affecting pollen tube growth in both antagonistic and synergistic
114 abacum resulted in the loss of inhibition of pollen tube growth in Nicotiana obtusifolia (synonym Nic
115 s characterized by evaluating N. obtusifolia pollen tube growth in normal and TT-ablated N. tabacum s
118 genes required for pollen grain development, pollen tube growth in the stigma and style, or pollen tu
119 nscript levels during stamen development and pollen tube growth in the transgenic trees of a stamen-s
121 a tabacum (tobacco), TTS protein, stimulates pollen tube growth in vivo and in vitro and attracts pol
125 nteraction of cognate PrsS and PrpS triggers pollen tube growth inhibition and programmed cell death
126 s vital and in vitro germination normal, but pollen tube growth inside stylar tissues appeared less d
133 dient and tip-localized Rho-family GTPase in pollen tube growth is established, the existence and fun
134 The claim of a central role for Cl- in lily pollen tube growth is further undermined by the fact tha
138 ycin, antimycin A, and cyanide, we find that pollen tube growth is much less sensitive to respiratory
140 w both in vitro and in vivo that asf1 mutant pollen tube growth is stunted, limiting fertilisation to
141 he response is biphasic; rapid inhibition of pollen-tube growth is followed by PCD, which is involved
142 ing genes involved in pollen germination and pollen tube growth might account for the fertility break
146 peated pattern of faster and longer-distance pollen tube growth often within solid pathways in phylog
148 posure to high temperature solely during the pollen tube growth phase limits fruit biomass and seed s
149 o determine how high temperature affects the pollen tube growth phase, taking advantage of cultivars
150 s pollen tube elongation but does not affect pollen tube growth polarity and shows Rop1-independent l
151 of catalytically modified Pi CDPK1 disrupted pollen tube growth polarity, whereas expression of Pi CD
152 later defects affecting pollen germination, pollen tube growth, polarity or guidance, or pollen tube
154 nsmitting tissue-specific (TTS) protein is a pollen tube growth-promoting and attracting glycoprotein
155 Comparative analyses point to accelerated pollen tube growth rate as a critical innovation that pr
156 llose-walled growth pattern with accelerated pollen tube growth rate underlies a striking repeated pa
160 tigmas, pollen development, pollination, and pollen tube growth require spatial and temporal regulati
161 Inhibition of N. obtusifolia and N. repanda pollen tube growth required accumulation of PELPIII in t
164 netic evidence presented here indicates that pollen tube growth requires cyclic nucleotide-gated chan
167 use severe defects in pollen germination and pollen tube growth, resulting in a reduced seed set.
168 e of sterility was identified as a defect in pollen tube growth, resulting in tubes that were kinky,
169 h predictions made by a mechanical model for pollen tube growth revealed the importance of pectin dee
170 n tubes and reduced its inhibitory effect on pollen tube growth significantly, suggesting that phosph
172 nteractions and the precision of directional pollen tube growth suggest that signals are continually
173 lay a crucial role in pollen germination and pollen tube growth, the proteins that mediate their acti
174 its known role in transporting sperm during pollen tube growth, the vegetative cell also contributes
175 ubstantiating a mechanistic role for AHAs in pollen tube growth through plasma membrane hyperpolariza
176 Pollen Receptor-Like Kinases (PRKs) control pollen tube growth through the pistil in response to ext
178 a indicate that the increased sensitivity of pollen tube growth to LATB was not due to general destab
182 ing or the actin cytoskeleton, then examined pollen tube growth using fluorescent protein markers tha
184 ning of pollinated pistils demonstrated that pollen tube growth was affected only when both parents b
186 pool was necessary to half-maximally inhibit pollen tube growth, whereas a approximately 100% increas
187 rane at the tip and caused depolarization of pollen tube growth, which was less severe than that indu
188 ecreased pollen germination rate and reduced pollen tube growth, which were all closely related to lo
189 screen and select among male gametes during pollen tube growth within the female tissues of the stig