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1 I), a disorder characterized by polyuria and polydipsia.
2 rs in central DI, nephrogenic DI and primary polydipsia.
3 sed in FP KOs that exhibit mild polyuria and polydipsia.
4 ually involves symptoms such as polyuria and polydipsia.
5 trating ability of the kidney, polyuria, and polydipsia.
6 d 89 (56%) received the diagnosis of primary polydipsia.
7 HNF-1beta mutant mice exhibited polyuria and polydipsia.
8 stigate the BNST LFP in the schedule-induced polydipsia, an animal model of OCD.
9 ority trial, we assigned adult patients with polydipsia and hypotonic polyuria or a known diagnosis o
10  ratios (VBRs) than matched patients without polydipsia and intermittent hyponatremia and normal subj
11                                Patients with polydipsia and intermittent hyponatremia have greater ve
12 ished brain volume observed in patients with polydipsia and intermittent hyponatremia in previous stu
13 ions, eight male schizophrenic patients with polydipsia and intermittent hyponatremia were first assi
14  neurodegenerative disorder that presents as polydipsia and polyuria as a consequence of a loss of se
15 , a rare inherited disorder that presents as polydipsia and polyuria as a consequence of a loss of se
16 uld be a side effect of diabetes insipidus - polydipsia and polyuria seen in Hom rats due to loss of
17                 cph homozygotes demonstrated polydipsia and produced a copious amount of hypotonic ur
18  concentrate urine, which leads to polyuria, polydipsia and the risk of hypertonic dehydration.
19 esis combined with increased thirst, causing polydipsia and water intoxication.
20 vioural features, especially hyperphagia and polydipsia, and a normal body-mass index.
21 d increased urine volume, hypertonic plasma, polydipsia, and impaired urinary concentrating ability a
22  clinical diabetes markers such as polyuria, polydipsia, and polyphagia.
23 ere pollakiuria, thirst, fatigue, dry mouth, polydipsia, and polyuria.
24 ristic symptoms of XNDI, including polyuria, polydipsia, and resistance to the antidiuretic actions o
25 acute, with a few days to weeks of polyuria, polydipsia, and weight loss and lack of a precipitating
26 ually slow and symptoms such as polyuria and polydipsia are often subtle and may go unrecognized by t
27  AVP precursor (C67X) exhibited polyuria and polydipsia by 2 months of age and these features of DI p
28                            BackgroundPrimary polydipsia, characterized by excessive fluid intake, car
29                   Treatment of DI or primary polydipsia depends on the underlying aetiology and diffe
30 ldr1 in the mouse kidney causes polyuria and polydipsia due to renal concentrating defects.
31 tric patients and health enthusiasts but the polydipsia in a small subgroup of patients seems to be d
32 daily excretion of highly diluted urine) and polydipsia (increased water intake), both features of di
33 ine vasopressin (AVP) deficiency and primary polydipsia is challenging.
34                                      Primary polydipsia is most common in psychiatric patients and he
35 reduce fluid intake in patients with primary polydipsia.MethodsIn this randomized, double-blind, plac
36 ortex, and is active in the schedule-induced polydipsia model for obsessive compulsive disorders.
37 ze treatment for ingestive disorders such as polydipsia or obesity.
38      Nhe3-/- mice demonstrated a significant polydipsia (P < 0.03) and polyuria (P < 0.04), with a lo
39 uded reversible nausea, renal insufficiency, polydipsia, paresthesias, and myelosuppression.
40 uding a stilted gait, weight loss, anorexia, polydipsia, patterned motor behaviors, head and tail tre
41  of Na(+) and Cl(-), reduced blood pressure, polydipsia, polyuria, and poor urinary concentrating abi
42 f diabetes including blood glucose, insulin, polydipsia, polyuria, and weight loss were measured.
43 resented with back pain, general discomfort, polydipsia, polyuria, fatigue and recent weight loss of
44  histopathology; (6) attenuated diet-induced polydipsia/polyuria; and (7) reduced HbA1c.
45 y high drinking levels in a schedule-induced polydipsia procedure (SIP).
46 mpulsivity as measured in a schedule-induced polydipsia procedure.
47 ek crossover trial, 34 patients with primary polydipsia received weekly dulaglutide (1.5 mg, Trulicit
48 ve polydipsic drinking in a schedule-induced polydipsia (SIP) procedure before their fresh brains wer
49 hile most rats exposed to a schedule-induced polydipsia (SIP) procedure develop controlled, moderate,
50  Efficacy in a rat model of schedule-induced polydipsia supported the decision to select the compound
51           Among adult patients with polyuria polydipsia syndrome, AVP deficiency was more accurately
52  compulsive behavior in the schedule-induced polydipsia task.
53 schizophrenic patients with hyponatremia and polydipsia, thereby placing them at increased risk of li
54 ected individuals with profound polyuria and polydipsia were homozygous for an autosomal recessive mi
55 genic DI must be differentiated from primary polydipsia, which involves excessive intake of large amo
56 pmol per liter led to a diagnosis of primary polydipsia with a specificity of 91.4% (95% CI, 83.7 to
57 orted subjective improvement in polyuria and polydipsia with the use of dDAVP (1-desamino-8-D-arginin
58 final diagnosis of AVP deficiency or primary polydipsia with use of clinical information, treatment r