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1 olecules retained the ability to bind to the polymeric Ig receptor.
2 mologous to two other IgM-binding receptors (polymeric Ig receptor and Fcalpha/muR) but exhibits an e
3 aralleling the gradient in expression of the polymeric Ig receptor and the transport of its ligand, o
4 to form dimeric (d) IgA that is bound by the polymeric Ig-receptor ectodomain, called secretory compo
8 by heterosubtypic immunity, indicating that polymeric Ig receptor-mediated transport is not required
10 dney (MDCK) cells transfected with the human polymeric Ig receptor (pIgR) and the cells studied by fl
15 rming growth factor-B (TGF-B) down-regulates polymeric Ig receptor (pIgR) on mucosal epithelium, resu
16 form dimeric (d) IgA, which is bound by the polymeric Ig receptor (pIgR) on the basolateral surface
17 nding of dimeric immunoglobulin (Ig)A to the polymeric Ig receptor (pIgR) stimulates transcytosis of
22 epithelial cells transfected to express the polymeric Ig receptor (pIgR), were transfected with HIV
23 and IgM) across epithelia is mediated by the polymeric Ig receptor (pIgR), which is expressed on the
24 om the basolateral to the apical surface via polymeric Ig receptor (pIgR)-mediated binding and the in
26 s study aimed to address the hypothesis that polymeric Ig receptor (pIgR)-mediated secretory IgA immu