ライフサイエンスコーパス: polymorphonuclear leukocyte

1 volving either platelet-monocyte or platelet-polymorphonuclear leukocyte.

2 the alpha-subunit after G-CSF expression on polymorphonuclear leukocytes.

3 P12-associating lectin-1 (MDL-1) on immature polymorphonuclear leukocytes.

4 strong activity for stimulating migration of polymorphonuclear leukocytes.

5 enuated for survival upon challenge by human polymorphonuclear leukocytes.

6 nuated in ability to resist killing by human polymorphonuclear leukocytes.

7 F1 decreases the antimicrobial activities of polymorphonuclear leukocytes.

8 ansfected HEK293T cells and peripheral blood polymorphonuclear leukocytes.

9 ct peribronchial infiltrate of predominantly polymorphonuclear leukocytes.

10 ive inflammation with granulation tissue and polymorphonuclear leukocytes.

11 tage to the bacteria in evading infiltrating polymorphonuclear leukocytes.

12 ls (PMVECs), alveolar macrophages (AMs), and polymorphonuclear leukocytes.

13 n, epithelial exfoliation, and the influx of polymorphonuclear leukocytes.

14 the assembly of the LT synthetic complex in polymorphonuclear leukocytes.

15 ed potent ADCC with mouse NK cells and mouse polymorphonuclear leukocytes.

16 MRSA exotoxins also caused neutrophil (polymorphonuclear leukocyte) activation, as measured by

17 olymerase chain reaction on peripheral blood polymorphonuclear leukocytes, acute rejection, and cardi

18 In contrast, polymorphonuclear leukocyte adhesion and chemotaxis were

19 Human polymorphonuclear leukocytes adhesion to endothelial cel

20 ption factor FOXO1, and increased numbers of polymorphonuclear leukocytes, all of which were signific

21 The observed dynamics in polymorphonuclear leukocyte alterations in peripheral bl

22 but reduced cytokines/chemokines as well as polymorphonuclear leukocyte and macrophage numbers.

23 H. ducreyi evades uptake by polymorphonuclear leukocyte and macrophage-like cell lin

24 r early source of chemokines associated with polymorphonuclear leukocyte and monocyte/macrophage infi

25 y increased and prolonged the formation of a polymorphonuclear leukocyte and mononuclear cell infiltr

26 associated with decreases in infiltration of polymorphonuclear leukocytes and activation of microglia

27 subsequent impairment of lung recruitment of polymorphonuclear leukocytes and clearance of Streptococ

28 ly 70% by 2C7-Ximab-E430G in the presence of polymorphonuclear leukocytes and complement.

29 re susceptible to oxidative killing by human polymorphonuclear leukocytes and displays decreased tiss

30 At 4 h, more polymorphonuclear leukocytes and fewer CD11c(+) cells we

31 Munro's microabscesses contain polymorphonuclear leukocytes and form specifically in th

32 he SBA assay, OMV ELISA, and OPA using human polymorphonuclear leukocytes and human complement but no

33 Primary polymorphonuclear leukocytes and keratinocytes were used

34 onstrated that the expression of FcRgamma on polymorphonuclear leukocytes and monocytes was not requi

35 ment and reduced intrahepatic recruitment of polymorphonuclear leukocytes and NKT cells after islet i

36 Measurement of sputum polymorphonuclear leukocytes and other analytes, cortiso

37 ROS generation by mononuclear cells and polymorphonuclear leukocytes and p47(phox) expression in

38 nsins are abundant antimicrobial peptides in polymorphonuclear leukocytes and play an important role

39 f CD11b adhesion molecules on the surface of polymorphonuclear leukocytes and status of iron-transfer

40 anied by a pronounced expression of CD11b in polymorphonuclear leukocytes and tissue sequestration of

41 converted to several novel products by human polymorphonuclear leukocytes and whole blood as well as

42 the gamma subunit on the surface of porcine polymorphonuclear leukocytes and with several other uniq

43 evaluated for total white blood cell (WBC), polymorphonuclear leukocyte, and monocyte counts.

44 ular phagocytosis with mouse NK cells, mouse polymorphonuclear leukocytes, and mouse macrophages.

45 reus abscesses, including the involvement of polymorphonuclear leukocytes, and provide a brief overvi

46 ant, suggesting that macrophages (MP) and/or polymorphonuclear leukocytes are the key target cells no

47 ly, specific immunodepletion of neutrophils (polymorphonuclear leukocytes) blocked hCNS-SCns astrogli

48 it both apoptosis and the oxidative burst in polymorphonuclear leukocytes but were unable to identify

49 labeled bacteria was tested in primary mouse polymorphonuclear leukocytes by flow cytometry.

50 data further suggest that the expression of polymorphonuclear leukocyte CD11b and the response of ir

51 cytosis of G. bethesdensis by normal and CGD polymorphonuclear leukocytes (CGD PMN) required heat-lab

52 nd placenta components included and 2) using polymorphonuclear leukocyte characteristics to assign lo

53 Defective FPR1 expression and impaired polymorphonuclear leukocyte chemotaxis toward bacterial

54 Neutrophils, or polymorphonuclear leukocytes, comprise a crucial compone

55 Neutrophilic polymorphonuclear leukocytes contain glycosphingolipid-

56 blood counts were performed and examined for polymorphonuclear leukocyte content.

57 measures of cervical inflammation (elevated polymorphonuclear leukocyte count).

58 assessment of the dynamics of alteration in polymorphonuclear leukocyte counts and expression of CD1

59 ce expressing human alpha-defensins in their polymorphonuclear leukocytes (Def(+/+)).

60 n ligand uptake or C5aR endocytosis in human polymorphonuclear leukocytes, distinguishing its role fr

61 recruitment and the functional activities of polymorphonuclear leukocytes during infections caused by

62 lves (1) enhancement of SCD erythrocytes and polymorphonuclear leukocyte efferocytosis, (2) blunting

63 etics of enhanced green fluorescence protein-polymorphonuclear leukocyte (EGFP-PMN) influx within a w

64 (active matrix metalloproteinase-8 [aMMP-8], polymorphonuclear leukocyte elastase [PMN elastase], and

65 blood and during phagocytic interaction with polymorphonuclear leukocytes ex vivo We conclude that Fa

66 .)-deficient monocytes, dendritic cells, and polymorphonuclear leukocytes from gp91(PHOX)- or p47(PHO

67 f the bacteria compared with macrophages and polymorphonuclear leukocytes from wild-type mice in whic

68 eral strategy used by this pathogen to alter polymorphonuclear leukocyte function.

69 Hyperoxia also significantly increased polymorphonuclear leukocytes, growth-related oncogene-al

70 MP-inhibitor treatment significantly reduced polymorphonuclear leukocytes, growth-related oncogene/CI

71 The effect size was modest for polymorphonuclear leukocytes/high-powered field threshol

72 A definitions were constructed by 1) varying polymorphonuclear leukocytes/high-powered field threshol

73 novel pathogenic entity, the activated PMN (polymorphonuclear leukocyte, i.e., neutrophil)-derived e

74 , and decreases interstitial infiltration of polymorphonuclear leukocytes in a mouse model of TNF-alp

75 igration of naive murine bone marrow-derived polymorphonuclear leukocytes in an in vitro cell migrati

76 The transepithelial migration of polymorphonuclear leukocytes in response to chemokine ex

77 flammatory signaling to recruit and activate polymorphonuclear leukocytes in the airway.

78 Large numbers of polymorphonuclear leukocytes in the amnion and chorion d

79 since we observed increased accumulation of polymorphonuclear leukocytes in the colonic mucosa of ra

80 Early influx of polymorphonuclear leukocytes in the lungs of uninfected

81 uired to counter toxic effectors secreted by polymorphonuclear leukocytes in the tissues.

82 Depletion of polymorphonuclear leukocytes in vivo failed to prevent g

83 gion increase resistance to killing by human polymorphonuclear leukocytes, increase bacterial prolife

84 Protection was mediated by Gr-1(+) polymorphonuclear leukocytes, indicating a novel form of

85 normal epithelial architecture but increased polymorphonuclear leukocyte infiltration and large lymph

86 llin pretreatment resulted in suppression of polymorphonuclear leukocyte infiltration at a late stage

87 d equipotent (at nanogram dosages), limiting polymorphonuclear leukocyte infiltration in a dose-depen

88 The most polymorphonuclear leukocyte infiltration in periodontal

89 In zymosan-initiated peritonitis, neutrophil polymorphonuclear leukocyte infiltration in response to

90 ion, increased cell apoptosis, and decreased polymorphonuclear leukocyte infiltration in the healing

91 phagocytosis of Escherichia coli, decreased polymorphonuclear leukocyte infiltration, and counter-re

92 attenuated pulmonary membrane thickening and polymorphonuclear leukocyte infiltration, reduced NF-kap

93 ontrol of immune-mediated cardiac injury and polymorphonuclear leukocyte inflammation.

94 Systemic treatment with PD1 reduced kidney polymorphonuclear leukocyte influx and, more importantly

95 ulted in improved oxygenation and diminished polymorphonuclear leukocyte influx into the isograft.

96 ice had increased efferocytosis of apoptotic polymorphonuclear leukocytes instilled into the peritone

97 However, the influx of polymorphonuclear leukocytes into the lung and the numbe

98 act of fruit reduced the edema, migration of polymorphonuclear leukocytes into the peritoneal cavity,

99 Peritoneal macrophages and polymorphonuclear leukocytes isolated from iNOS-/- mice

100 timulated COX-2 expression was suppressed in polymorphonuclear leukocytes isolated from MacKOs, but P

101 Upon stimulation of the human polymorphonuclear leukocyte, it was found that the abund

102 ls of erythrocyte lysis, resistance to human polymorphonuclear leukocyte killing, and pathogenesis in

103 laque rupture and consequent thrombosis, but polymorphonuclear leukocytes likely promote endothelial

104 aemophilus ducreyi is orchestrated by serum, polymorphonuclear leukocytes, macrophages, T cells, and

105 to invade epithelial cells and/or to induce polymorphonuclear leukocyte migration in a tissue cultur

106 culating blood elements including platelets, polymorphonuclear leukocytes, monocytes, and lymphocytes

107 (Hp) infection triggers a chronic influx of polymorphonuclear leukocyte neutrophils (PMNs) into the

108 We show that the specific immunodepletion of polymorphonuclear leukocyte neutrophils using anti-Ly6G

109 Polymorphonuclear leukocytes (neutrophils) are the prima

110 n of peri-implant inflammation: neutrophilic polymorphonuclear leukocytes (neutrophils) maximally acc

111 9 siRNA showed decreases in corneal opacity, polymorphonuclear leukocyte number, IL-12 and IFN-gamma

112 significantly elevated bacterial counts and polymorphonuclear leukocyte numbers at 1 and 3 days post

113 d latex bead-induced ROS production in human polymorphonuclear leukocytes occurred by the NADPH oxida

114 Neutrophils are polymorphonuclear leukocytes of the phagocytic system th

115 Cases had >=5 polymorphonuclear leukocytes on Gram stain plus symptoms

116 d for virulence and resistance to killing by polymorphonuclear leukocytes, one epa mutant (TX5179) wa

117 atment on the volume of nasal secretions, on polymorphonuclear leukocyte or interleukin-8 concentrati

118 ant for in vitro killing of P. gingivalis by polymorphonuclear leukocytes or AM and, moreover, the AM

119 ructure displayed cellular activity in human polymorphonuclear leukocytes, oral bioavailability, and

120 with NGU (visible urethral discharge or >/=5 polymorphonuclear leukocytes per high-power field [PMNs/

121 NGU was defined as >=5 polymorphonuclear leukocytes per high-power field on ure

122 nt score, and cervicitis was defined as >=30 polymorphonuclear leukocytes per high-power microscopic

123 ouse peritonitis and was more susceptible to polymorphonuclear leukocyte phagocytic killing.

124 pheral insulin resistance and alterations in polymorphonuclear leukocyte (PML) function.

125 ase of cytokines and chemokines that attract polymorphonuclear leukocytes (PML) and macrophages to th

126 trates the epidermis and reaches the dermis, polymorphonuclear leukocytes (PMLs) accumulate and an ab

127 inflammatory stimulus and results in tissue polymorphonuclear leukocyte (PMN) accumulation.

128 JAM-C mAb alone had no inhibitory effect on polymorphonuclear leukocyte (PMN) adhesion or transmigra

129 pothesis that these antibodies might augment polymorphonuclear leukocyte (PMN) adhesion to endotheliu

130 here that the saliva of ixodid ticks reduces polymorphonuclear leukocyte (PMN) adhesion via downregul

131 7 from Akt, prior to Akt activation, induced polymorphonuclear leukocyte (PMN) apoptosis.

132 ids participate in the suppression of murine polymorphonuclear leukocyte (PMN) bactericidal activitie

133 iodontitis (AgP) is associated with impaired polymorphonuclear leukocyte (PMN) chemotaxis toward bact

134 e, is susceptible to killing by a variety of polymorphonuclear leukocyte (PMN) components.

135 In vitro assays with human Polymorphonuclear leukocyte (PMN) demonstrated that CFTR

136 PD1 was a potent regulator of polymorphonuclear leukocyte (PMN) infiltration (approxim

137 logic sections showed more corneal edema and polymorphonuclear leukocyte (PMN) infiltration in contro

138 Histologic analysis suggested less polymorphonuclear leukocyte (PMN) infiltration into the

139 (2) formation was analyzed by lipidomics and polymorphonuclear leukocyte (PMN) infiltration quantifie

140 Polymorphonuclear leukocyte (PMN) infiltration was first

141 rsus the PBS-treated group were examined for polymorphonuclear leukocyte (PMN) infiltration, chemokin

142 Polymorphonuclear leukocyte (PMN) is the predominant inn

143 nslocation, biofilm formation, resistance to polymorphonuclear leukocyte (PMN) killing, and virulence

144 Extension of the polymorphonuclear leukocyte (PMN) leading edge toward a

145 Polymorphonuclear leukocyte (PMN) migration across the i

146 lts, larvae, and embryos), a transepithelial polymorphonuclear leukocyte (PMN) migration assay, and t

147 eltaybcL) failed to suppress transepithelial polymorphonuclear leukocyte (PMN) migration in vitro, a

148 ation but had no effect on bacterial load or polymorphonuclear leukocyte (PMN) numbers in the lung.

149 Using priming of the polymorphonuclear leukocyte (PMN) oxidase as a measure o

150 To model polymorphonuclear leukocyte (PMN) recognition of fungi u

151 a concentration where RvE1 potently reduced polymorphonuclear leukocyte (PMN) recruitment in zymosan

152 es (CORMs) suppress inflammation by reducing polymorphonuclear leukocyte (PMN) recruitment to the aff

153 ection, which coincided with the increase in polymorphonuclear leukocyte (PMN) recruitment to the lun

154 Polymorphonuclear leukocyte (PMN) recruitment to vascula

155 lular adhesion molecule 1 (ICAM-1)-dependent polymorphonuclear leukocyte (PMN) recruitment, functiona

156 ICAM-1) expression regulates the location of polymorphonuclear leukocyte (PMN) TEM.

157 tal staphylococcal infections by controlling polymorphonuclear leukocyte (PMN) trafficking.

158 nd counterregulatory actions that stop human polymorphonuclear leukocyte (PMN) transendothelial migra

159 sduction pathways that are engaged to induce polymorphonuclear leukocyte (PMN) transepithelial migrat

160 tactin phosphorylation in endothelium during polymorphonuclear leukocyte (PMN) transmigration through

161 n species (ROS) are critical for neutrophil (polymorphonuclear leukocyte (PMN)) microbicidal function

162 rate that epinephrine alters the neutrophil (polymorphonuclear leukocyte (PMN))-dependent inflammator

163 ant throughout the observation period, while polymorphonuclear leukocyte (PMN), S100A8, and interleuk

164 The polymorphonuclear leukocyte (PMN)-derived serine proteas

165 ction of cytoskeletal changes, inhibition of polymorphonuclear leukocyte (PMN)-endothelial cell inter

166 hydrogenase gene expression in macrophages, polymorphonuclear leukocyte (PMN)-like cells, and a mous

167 entify novel virulence factors in evasion of polymorphonuclear leukocyte (PMN)-mediated innate immuni

168 type 8 pneumococcus, but it does not promote polymorphonuclear leukocyte (PMN)-mediated pneumococcal

169 t defense against microbial infection is the polymorphonuclear leukocyte (PMN).

170 Neutrophil [polymorphonuclear leukocyte (PMN)] transepithelial migra

171 gue associated with extensive recruitment of polymorphonuclear leukocytes (PMN or neutrophils) to the

172 Polymorphonuclear leukocytes (PMN) achieve an intermedia

173 taphylococcus aureus include mobilization of polymorphonuclear leukocytes (PMN) and extracellular gro

174 (4) (LTB(4)) is a potent chemoattractant for polymorphonuclear leukocytes (PMN) and other cells.

175 presentation of phagocytes in these lesions: polymorphonuclear leukocytes (PMN) are more prevalent in

176 Polymorphonuclear leukocytes (PMN) are the first respond

177 Polymorphonuclear leukocytes (PMN) from patients with ch

178 Depletion of circulating polymorphonuclear leukocytes (PMN) had a similar therape

179 tussis is able to avoid bacterial killing by polymorphonuclear leukocytes (PMN) if specific opsonic a

180 expression in human uroepithelial cells and polymorphonuclear leukocytes (PMN) in vitro and in bladd

181 duced chronic injury in the cornea triggered polymorphonuclear leukocytes (PMN) infiltration, patholo

182 flammation is traditionally characterized by polymorphonuclear leukocytes (PMN) influx followed by ph

183 Transendothelial migration (TEM) of polymorphonuclear leukocytes (PMN) involves a carefully

184 ng upon this model, we investigated the role polymorphonuclear leukocytes (PMN) play in the control o

185 Subcellular fractionation studies in polymorphonuclear leukocytes (PMN) revealed similar patt

186 sly localized the anion transporter ClC-3 to polymorphonuclear leukocytes (PMN) secretory vesicles an

187 xperiments, conditioned media collected from polymorphonuclear leukocytes (PMN) selectively increased

188 LFA-1 bonds help anchor polymorphonuclear leukocytes (PMN) to inflamed endotheli

189 We have shown that polymorphonuclear leukocytes (PMN) treated with the sali

190 trate by flow cytometric analysis that mouse polymorphonuclear leukocytes (PMN) up-regulate surface e

191 nfected individuals contain large numbers of polymorphonuclear leukocytes (PMN) with ingested gonococ

192 ays, we found that RAGE mediates neutrophil (polymorphonuclear leukocytes (PMN)) adhesion to, and sub

193 reported previously that human neutrophils (polymorphonuclear leukocytes (PMN)) express multiple SIR

194 . bethesdensis resists killing by serum, CGD polymorphonuclear leukocytes (PMN), and antimicrobial pe

195 J774 cells, murine polymorphonuclear leukocytes (PMN), human monocytes, and

196 depends on a prompt response by circulating polymorphonuclear leukocytes (PMN).

197 atterns of JNK/SAPK in wild-type and JNK2-/- polymorphonuclear leukocytes (PMN).

198 dal polypeptides secreted by macrophages and polymorphonuclear leukocytes (PMN).

199 spergillus are killed by normal, but not CGD polymorphonuclear leukocytes (PMN); however, the few stu

200 , sustained recruitment to the lymph node of polymorphonuclear leukocytes (PMN, or neutrophils), the

201 st immune responses, including the influx of polymorphonuclear leukocytes (PMN; neutrophils).

202 y with IL-1beta to promote early neutrophil (polymorphonuclear leukocyte [PMN]) recruitment.

203 Neutrophil (polymorphonuclear leukocytes [PMN]) infiltration plays a

204 Neutrophil (polymorphonuclear leukocytes [PMN]) transepithelial migr

205 ed to increased infiltration of neutrophils (polymorphonuclear leukocytes [PMN]), macrophages (MPhi),

206 Neutrophil (polymorphonuclear leukocyte; PMN) inflammatory functions

207 by the provider, who recorded the number of polymorphonuclear leukocytes (PMNLs) per epithelial cell

208 HEK293) cell line expressing 5-LO, and human polymorphonuclear leukocytes (PMNLs) we investigated the

209 Polymorphonuclear leukocytes (PMNs or neutrophils) are e

210 Transmigration of polymorphonuclear leukocytes (PMNs) across CRECs monolay

211 The migration of polymorphonuclear leukocytes (PMNs) across the intestina

212 We hypothesized that polymorphonuclear leukocytes (PMNs) and less mature myel

213 nstrate its ability in separating/recovering polymorphonuclear leukocytes (PMNs) and mononuclear leuk

214 s a host foreign body response, during which polymorphonuclear leukocytes (PMNs) and then monocytes (

215 Human neutrophilic polymorphonuclear leukocytes (PMNs) are central to innat

216 Polymorphonuclear leukocytes (PMNs) are innate immune ce

217 e hypersensitivity and autoimmunity in which polymorphonuclear leukocytes (PMNs) are involved.

218 Polymorphonuclear leukocytes (PMNs) are key in innate im

219 e, we demonstrate that RvD1 actions on human polymorphonuclear leukocytes (PMNs) are pertussis toxin

220 Y. pestis and that CD11b(+) cells other than polymorphonuclear leukocytes (PMNs) are selectively lost

221 oeae recruits and interacts extensively with polymorphonuclear leukocytes (PMNs) during infection.

222 of CA-MRSA, we evaluated the lysis of human polymorphonuclear leukocytes (PMNs) during phagocytic in

223 A flow-cytometric analysis of polymorphonuclear leukocytes (PMNs) exposed to supernata

224 We tested cytotoxicity of LukGH toward polymorphonuclear leukocytes (PMNs) from mice, rabbits,

225 Polymorphonuclear leukocytes (PMNs) from subjects with l

226 into host cells, and both pathogens recruit polymorphonuclear leukocytes (PMNs) from the submucosa t

227 nd cell migration are essential functions of polymorphonuclear leukocytes (PMNs) in host defense.

228 orresponding decrease in the accumulation of polymorphonuclear leukocytes (PMNs) in lungs.

229 progenitor cells (HSPCs) differentiate into polymorphonuclear leukocytes (PMNs) in the bone marrow.

230 ntratracheal administration of LPS increased polymorphonuclear leukocytes (PMNs) in the bronchoalveol

231 n the percentage of dendritic cells (DCs) or polymorphonuclear leukocytes (PMNs) in the early stage o

232 del of LPS-induced lung injury, migration of polymorphonuclear leukocytes (PMNs) into the different c

233 lografts is initiated by the infiltration of polymorphonuclear leukocytes (PMNs) into the graft.

234 ine receptor 2 (CXCR2) mediates migration of polymorphonuclear leukocytes (PMNs) into the lung.

235 f pathogenesis of pneumonia is the influx of polymorphonuclear leukocytes (PMNs) into the lungs.

236 elial cells (HPMEC) prior to the addition of polymorphonuclear leukocytes (PMNs) or dendritic cells (

237 Bacteria and polymorphonuclear leukocytes (PMNs) per cornea were quan

238 Massive infiltration of polymorphonuclear leukocytes (PMNs) to the corneal endot

239 oeae (Gc), is characterized by the influx of polymorphonuclear leukocytes (PMNs) to the site of infec

240 nococcus), is characterized by the influx of polymorphonuclear leukocytes (PMNs) to the site of infec

241 opsonophagocytosis of P. gingivalis by human polymorphonuclear leukocytes (PMNs) using a fluorescent

242 A few polymorphonuclear leukocytes (PMNs) were already present

243 On necropsy, a large number of necrotic polymorphonuclear leukocytes (PMNs) were observed in the

244 ized by a mucopurulent exudate that contains polymorphonuclear leukocytes (PMNs) with intracellular g

245 flammatory response that is characterized by polymorphonuclear leukocytes (PMNs) with intracellular g

246 ite the fundamental function of neutrophils (polymorphonuclear leukocytes (PMNs)) in innate immunity,

247 Neutrophils (or polymorphonuclear leukocytes (PMNs)) readily clear blast

248 ing in septic plasma required C5a receptors, polymorphonuclear leukocytes (PMNs), and the Nacht-, LRR

249 e HUVEC monolayer permeability, and activate polymorphonuclear leukocytes (PMNs), as reflected in CD1

250 aggregated with human platelets and bound to polymorphonuclear leukocytes (PMNs), as shown by the sti

251 Neutrophil granulocytes, also called polymorphonuclear leukocytes (PMNs), extrude molecular l

252 by an inflammatory infiltrate that includes polymorphonuclear leukocytes (PMNs), monocytes, lymphocy

253 -derived alveolar macrophages (AMs), but not polymorphonuclear leukocytes (PMNs), to phagocytose bact

254 Our method reproducibly recovers 94.0% of polymorphonuclear leukocytes (PMNs), with up to 10(5) PM

255 din (PVL), a pore-forming toxin that targets polymorphonuclear leukocytes (PMNs).

256 ession in epithelial cells, fibroblasts, and polymorphonuclear leukocytes (PMNs).

257 pact interactions between N. gonorrhoeae and polymorphonuclear leukocytes (PMNs).

258 ion conferring protection against killing by polymorphonuclear leukocytes (PMNs).

259 eported, decrease membrane fluidity on mouse polymorphonuclear leukocytes (PMNs).

260 ositive strains is an elevation in levels of polymorphonuclear leukocytes (PMNs).

261 revisiae, release chemoattractants for human polymorphonuclear leukocytes (PMNs).

262 , as they prevented an influx of Siglec-F(+) polymorphonuclear leukocytes (PMNs).

263 infections depends on bacterial clearance by polymorphonuclear leukocytes (PMNs); however, excessive

264 hemorrhage, and accumulation of neutrophils [polymorphonuclear leukocytes (PMNs)] in the alveolar com

265 Human polymorphonuclear leukocytes (PMNs, or neutrophils) are

266 Human polymorphonuclear leukocytes (PMNs, or neutrophils) are

267 Polymorphonuclear leukocytes (PMNs, or neutrophils) are

268 The transmigration of polymorphonuclear leukocytes (PMNs; neutrophils) into th

269 Neutrophils (polymorphonuclear leukocytes [PMNs]) are critical to the

270 Human neutrophils (polymorphonuclear leukocytes [PMNs]) generate inflammato

271 ytes, the interactions of human neutrophils (polymorphonuclear leukocytes [PMNs]) with both TSP-4 var

272 n to avoid destruction by human neutrophils (polymorphonuclear leukocytes [PMNs]), which are crucial

273 n by human epithelial cells and neutrophils (polymorphonuclear leukocytes [PMNs]).

274 ous system (CNS) include infiltrating cells (polymorphonuclear leukocytes [PMNs], macrophages, and na

275 Neutrophils (polymorphonuclear leukocytes, PMNs) are vital to innate

276 O+/+) mice exhibited comparable increases in polymorphonuclear leukocytes, predominately neutrophils,

277 The total number of bronchoalveolar lavage polymorphonuclear leukocytes recovered from the mice exp

278 on chromosome 2, and a region affecting both polymorphonuclear leukocyte recruitment and TNF-alpha le

279 the omega-3 group correlated with decreased polymorphonuclear leukocyte recruitment, chemokine and c

280 ng inflammation, characterized by increasing polymorphonuclear leukocyte recruitment, is a major caus

281 Finally, depletion of polymorphonuclear leukocytes reversed the enhanced susce

282 thal disease, in association with widespread polymorphonuclear leukocyte-rich microabscesses but with

283 sion of NF-kappaB activity and restored lung polymorphonuclear leukocyte sequestration in response to

284 In polymorphonuclear leukocytes, SipA or other Salmonella p

285 s, but PG formation was not even detected in polymorphonuclear leukocyte supernatants from control mi

286 ated expression of MMP-26 in macrophages and polymorphonuclear leukocytes supports the functional rol

287 distribution in leukocytes (110 copies/10(5) polymorphonuclear leukocytes) than the R-/D+ subgroup.

288 mine (GPEtn) species after exposure of human polymorphonuclear leukocytes to A23187 and granulocyte m

289 e attributed to a defect in the migration of polymorphonuclear leukocytes to infected sites during ea

290 Bacterial invasion of host tissues triggers polymorphonuclear leukocytes to release DNA [neutrophil

291 iae-infected Mmp2/9(-/-) mice recruited more polymorphonuclear leukocytes to the lung but had higher

292 RvD1 regulates human polymorphonuclear leukocyte transendothelial migration a

293 ntercellular adhesion molecule-1 expression, polymorphonuclear leukocyte transendothelial migration,

294 AC753S mutant had reduced Salmonella-induced polymorphonuclear leukocytes transepithelial migration.

295 ies of AnxA1 become operative to finely tune polymorphonuclear leukocytes transmigration to the site

296 Polymorphonuclear leukocytes undergo directed movement t

297 multisubunit complex from Brij 96 lysates of polymorphonuclear leukocytes using the G7 mAb, which bin

298 sing the RosetteSep antibody cocktail, while polymorphonuclear leukocytes were separated with density

299 By 6 h after infection, polymorphonuclear leukocytes with intracellular spores w

300 y detection of viral DNA in peripheral blood polymorphonuclear leukocytes within the first few months