コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 r frequently associated with the Merkel cell polyomavirus.
2 c acid, is the major attachment receptor for polyomavirus.
3 virus (MWPyV) is a recently identified human polyomavirus.
4 uce MCC in mice using oncoproteins from this polyomavirus.
5 Merkel cell carcinoma virus, which is also a polyomavirus.
6 ent of trichodysplasia spinulosa caused by a polyomavirus.
7 screen to examine the humoral response to BK polyomavirus.
8 tralize BK virus subtypes and the related JC polyomavirus.
9 skin cancer often caused by the Merkel cell polyomavirus.
10 the rearrangements typical for reactivating polyomaviruses.
11 g a proteostatic latency mechanism common to polyomaviruses.
12 ion of Fe/S coordination in the sTs of other polyomaviruses.
13 ural history of the more recently discovered polyomaviruses.
14 uses, including two papillomaviruses and two polyomaviruses.
15 ds sialylated glycan receptors in many other polyomaviruses.
16 mic and clinical features of these cutaneous polyomaviruses.
19 rus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPyV6], HPyV7, HPyV9, and Trichodysplasi
20 richodysplasia spinulosa polyomavirus, human polyomavirus 6 and 7 have been linked to specific skin d
21 richodysplasia spinulosa polyomavirus, human polyomavirus 6 and 7, and Malawi polyomavirus are shed f
25 el cell, HPyV7 and trichodysplasia spinulosa polyomaviruses after drug treatment were determined by i
27 teric viruses [human adenoviruses, JC and BK polyomaviruses, Aichi virus 1 (AiV-1), enteroviruses, an
28 In this Gem, we will discuss two viruses, polyomavirus and cytomegalovirus, in which cell culture
29 -is caused by the integration of Merkel cell polyomavirus and persistent expression of large T antige
32 Our findings also suggest that eukaryotic polyomaviruses and papillomaviruses with dsDNA genomes h
34 tical evidence in favor of an association of polyomaviruses and their hosts over millions of years.
36 V9, and Trichodysplasia spinulosa-associated polyomavirus) and examined factors associated with MCPyV
37 almonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine rotavirus A were present most f
38 irnaviruses, parvoviruses, papillomaviruses, polyomaviruses, and a gammaherpesvirus, were identified,
40 irus, human polyomavirus 6 and 7, and Malawi polyomavirus are shed from the skin, and Merkel cell pol
44 However, MCPyV appears different from other polyomaviruses, as it requires sulfated polysaccharides,
45 g drug regimens for transplant patients with polyomavirus-associated diseases may reduce symptomatic
48 nfiltrates in renal biopsy specimens with BK polyomavirus-associated nephropathy (BKPyVAN) and specim
50 With current immunosuppressive regimens, BK polyomavirus-associated nephropathy (BKPyVAN) is still a
53 omatic in healthy people; however, it causes polyomavirus-associated nephropathy in renal transplant
54 o a specific diagnosis), infections, such as polyomavirus-associated nephropathy, calcineurin inhibit
56 imental data on unperturbed adenoviruses and polyomaviruses, at the same time providing insight into
60 irus, and West Nile virus, as well the human polyomaviruses BK/JC/MCV, human adenoviruses, and human
64 ell epitopes that influence the course of BK polyomavirus (BKPyV) infection after kidney transplantat
65 t APOBEC3B is specifically upregulated by BK polyomavirus (BKPyV) infection in primary kidney cells a
69 ly target BKPyV-infected cells.IMPORTANCE BK polyomavirus (BKPyV) is an emerging pathogen that reacti
71 e-size primary transcripts.IMPORTANCE The BK polyomavirus (BKPyV) miRNA plays an important role in re
73 fter kidney transplantation, uncontrolled BK polyomavirus (BKPyV) replication causes kidney graft fai
74 Here, we demonstrate the detection of BK Polyomavirus (BKPyV), an emerging indicator of microbiol
76 d immunoglobulin G seroreactivity against 10 polyomaviruses (BKPyV, JCPyV, KIPyV, WUPyV, MCPyV, HPyV6
77 irus (CMV), Epstein-Barr virus (EBV), and BK polyomavirus (BKV) at transplant was a risk factor for p
78 uppression in kidney transplant patients, BK polyomavirus (BKV) has been shown to induce nephropathy
79 Recent case series describe detection of BK polyomavirus (BKV) in urinary tract cancers in kidney tr
80 we identified a peptide derived from the BK polyomavirus (BKV) minor structural proteins VP2/3 that
82 nses in a sophisticated manner.IMPORTANCE BK polyomavirus can cause serious problems in immune-suppre
85 affects the quantity and quality of the anti-polyomavirus CD8 T cell response and its differentiation
86 Merkel cell polyomavirus(+) and Merkel cell polyomavirus(-) cells express serine palmitoyl transfera
90 fectious entry.IMPORTANCE MCPyV is the first polyomavirus directly implicated in the development of a
91 based on CRISPR-Cas13 accurately detects BK polyomavirus DNA and cytomegalovirus DNA from patient-de
92 omaviruses, Merkel cell carcinoma-associated polyomavirus, Epstein-Barr virus, and Kaposi's sarcoma-a
93 cell polyomavirus (MCPyV) is the first human polyomavirus etiologically associated with Merkel cell c
95 y, and, ultimately, pathogenicity within the polyomavirus family and highlight the need for structure
96 hylogenetic relationships within the growing polyomavirus family and perhaps also for other viruses.
97 yV cell entry is unique among members of the polyomavirus family as it requires the engagement of two
99 These results suggest that, while intragraft polyomavirus gene expression may be useful as an ancilla
101 evidence that, consistent with this idea, BK polyomavirus genomes are depleted of APOBEC3B-preferred
108 titative PCR to test for cytomegalovirus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus,
109 avirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPyV6], HPyV7, HPyV9
110 cell polyomavirus, trichodysplasia spinulosa polyomavirus, human polyomavirus 6 and 7 have been linke
111 cell polyomavirus, trichodysplasia spinulosa polyomavirus, human polyomavirus 6 and 7, and Malawi pol
112 hybridization confirmed the presence of the polyomavirus in endothelial cells at sites of myositis a
114 associated crAssphage, Bacteroides spp., and polyomavirus in sewage samples from 49 wastewater facili
116 n of large tumor T antigens (TAg) encoded by polyomaviruses in mammalian cells results in increased t
117 vides insight into how DDRs are activated by polyomaviruses in primary cells with intact cell cycle c
119 ory of these viruses and the evolution of JC polyomavirus-induced progressive multifocal leukoencepha
122 res unravel a potent modality for inhibiting polyomavirus infection in kidney transplant recipients a
125 Tumour oncogenesis is linked to Merkel cell polyomavirus integration and ultraviolet-radiation-induc
128 en, as well as large T antigens from related polyomaviruses, is alone capable of upregulating APOBEC3
129 the basis by which MCPyV, among all 12 human polyomaviruses, is the only one that causes cancer in hu
130 the presence of this pore is conserved among polyomaviruses, its functional role in infection or asse
131 document a unique DNA recombination between polyomavirus JC (JC virus [JCV]) and Epstein-Barr virus
132 ting disease caused by the human neurotropic polyomavirus JC (JCV) and is found almost exclusively in
134 the white matter of the brain caused by the polyomavirus JC virus, which typically occurs only in im
136 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human po
139 a fatal disease caused by reactivation of JC polyomavirus (JCPyV) in immunosuppressed individuals and
140 nitoring, metagenomic sequencing detected JC polyomavirus (JCPyV) in the urine of 7 donors and their
144 nally, endocytosis-dependent infection by JC polyomavirus (JCPyV) was reduced in human cells with dec
145 rebral manifestations are associated with JC polyomavirus (JCPyV) which are diagnosed by detection of
147 the receptor-binding properties of human JC polyomavirus (JCPyV), a virus that resides in the kidney
148 ividuals are asymptomatically infected by JC polyomavirus (JCPyV), but if the host immune system is c
153 tients that treatment with interleukin 7, JC polyomavirus (JCV) capsid protein VP1, and a Toll-like r
154 of the central nervous system (CNS) with JC polyomavirus (JCV) usually occur as a result of immunoco
155 d persistence of 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyo
156 Combined, these results demonstrate that polyomaviruses lacking miRNAs can arise infrequently and
158 iated with clinical presentation: intrarenal polyomavirus load levels and Banff interstitial fibrosis
159 ic infection of oligodendrocytes by human JC polyomavirus may result in the development of progressiv
160 CD8 T cell responses to infections by human polyomaviruses may be influenced by VP1 mutations involv
161 rcinomas (MCCs) that contain the Merkel cell polyomavirus (MCPyV) and the clinical significance of tu
168 ng evidence indicates a role for Merkel cell polyomavirus (MCPyV) in the development of Merkel cell c
177 ns, copy number alterations, and Merkel cell polyomavirus (MCPyV) sequence were analyzed and compared
178 l integration of a polyomavirus, Merkel cell polyomavirus (MCPyV), and MCC tumor cells express putati
180 2008 to be caused by a PyV named Merkel cell polyomavirus (MCPyV), the first PyV linked to human canc
181 ectopic expression of different Merkel cell polyomavirus (MCPyV)-derived truncated large T antigens
185 incidence, prevalence, and persistence of 9 polyomaviruses (MCPyV, BK polyomavirus, KI polyomavirus,
190 hat harbor a clonally integrated Merkel cell polyomavirus (MCV) genome have low mutation burden and r
198 ntly associated with clonal integration of a polyomavirus, Merkel cell polyomavirus (MCPyV), and MCC
199 aneous tumor system with mice expressing the polyomavirus middle T (PyMT) oncogene under control of t
201 RT-deficient mice displayed marked delays in polyomavirus middle T oncogene-induced (PyMT-induced) ma
202 tors in the mouse mammary tumor virus (MMTV)-polyomavirus middle T oncoprotein (PyMT) mouse model of
203 k Institute set out to determine whether the polyomavirus middle T-transforming protein had a similar
212 alitative highly predictive urinary test for polyomavirus nephropathy (PVN) is the PV-Haufen test.
215 finitive PVN from the Banff Working Group on Polyomavirus Nephropathy, comprising nine transplant cen
218 ding for middle T antigen (MT) is the murine polyomavirus oncogene most responsible for tumor formati
220 MCPyV), and MCC tumor cells express putative polyomavirus oncoprotein small T antigen (sTAg) and trun
221 viruses, herpesviruses, papillomaviruses and polyomaviruses) over time were observed in individuals w
222 ttempted to reveal the composition of the JC polyomavirus population (the quasispecies, i.e., the who
243 study (n=40) to explore associations between polyomavirus reactivation and immune responses to the se
244 Furthermore, these results indicate that polyomavirus reactivation associates with immune respons
245 ore prevalent during year 1 in subjects with polyomavirus reactivation than in those without reactiva
246 It is unknown if they directly reactivate polyomavirus replication from latency beyond their gener
247 rs, at therapeutic levels, directly activate polyomavirus replication through a Skp2-dependent mechan
248 s-associated diseases may reduce symptomatic polyomavirus replication while maintaining allograft-spa
250 also found (gemycircularvirus [n = 5] and WU polyomavirus, Saffold virus, salivirus, cyclovirus-VN, a
259 xamined whether some of the functions of the polyomavirus small T antigens (ST) are shared by the E6
262 yomavirus, we show that brain-resident mouse polyomavirus-specific CD8 T cells, unlike memory cells i
266 for the transforming properties of nonhuman polyomaviruses, such as simian virus 40 (SV40), but is n
268 on (RTN) protects ER membrane integrity when polyomavirus SV40 escapes the ER to reach the cytosol en
272 tosol membrane transport of the nonenveloped polyomavirus SV40, a decisive infection step, a cytosoli
275 me E2F site is required for A3B induction by polyomavirus T antigen indicating a shared molecular mec
277 ng CNS infection caused by JC virus (JCV), a polyomavirus that commonly establishes persistent, asymp
278 Jersey polyomavirus (NJPyV-2013) is a novel polyomavirus that may have tropism for vascular endothel
279 f membrane behavior for all AB(5) toxins and polyomaviruses that bind glycosphingolipids to invade ho
280 question by focusing on a single lineage of polyomaviruses that infect both humans and their closest
281 cell carcinoma (MCC), making MCPyV the first polyomavirus to be clearly associated with human cancer.
283 en (MT), the principal oncoprotein of murine polyomavirus, transforms by association with cellular pr
285 irus are shed from the skin, and Merkel cell polyomavirus, trichodysplasia spinulosa polyomavirus, hu
286 we show that manifestations of the causative polyomavirus (TSPyV) occur during primary infection of t
287 witnessed the discovery of eleven new human polyomaviruses, two of which cause unusual and rare canc
288 ogous peptide exhibits similar binding to JC polyomavirus VP1 and inhibits infection with similar pot
289 arison with other structurally characterized polyomavirus VP1 proteins enhances our understanding of
300 We review what is currently known about JC polyomavirus, what is suspected, and what remains to be
301 (MCPyV, BK polyomavirus, KI polyomavirus, JC polyomavirus, WU polyomavirus, Human polyomavirus 6 [HPy