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1 e cytological nuclear abnormalities (even in polyploid cells).
2 quitination of histone-H2A at lysine-119) in polyploid cell.
3 kinase activity was largely abolished in the polyploid cells.
4 ssors E2F7 and E2F8, leading to formation of polyploid cells.
5 rmed cardiomyocytes, are one such example of polyploid cells.
6 26 was required to activate the PIDDosome in polyploid cells.
7 ved in chronically damaged liver tissues, on polyploid cells.
8 n return to mitotic growth and go on to form polyploid cells.
9 agging chromosomes or micronuclei in mitotic polyploid cells.
10 ribed changes with respect to development in polyploid cells.
11  of chromosome structure in both diploid and polyploid cells.
12 aryocytes, as well as an increased number of polyploid cells.
13 from problems with chromosome segregation in polyploid cells.
14 g and functional analysis in post-mitotic or polyploid cells.
15 phology checkpoint to avoid the formation of polyploid cells.
16 ural chromosome instability of the resulting polyploid cells.
17 s in terminally differentiated larval midgut polyploid cells.
18 g cells replicate their DNA and propagate as polyploid cells.
19 ly in cells programmed to differentiate into polyploid cells.
20 bition by MK-0457, and preferentially killed polyploid cells.
21 le but skip late stages of mitosis to become polyploid cells.
22 s well as the endo cycle (S-G) that produces polyploid cells.
23  a reduced mitotic index and the presence of polyploid cells.
24 ns, failure of cytokinesis, and emergence of polyploid cells.
25  DNA synthesis resulted in the generation of polyploid cells.
26 6 were haploinsufficient with an increase in polyploid cells, a reduction in cell proliferation, elev
27                            Here we show that polyploid cells accumulate in the adult fly brain and th
28 in UV- and VP-16-treated cells and increases polyploid cells after VP-16 treatment.
29                                 Importantly, polyploid cells also exist in some normal tissues.
30 meiotic commitment can cause highly abnormal polyploid cells and can ultimately lead to germ cell tum
31 identifies inhibitors of bipolar division in polyploid cells and provides a rationale to understand c
32 ive stress promotes the appearance of highly polyploid cells, and antioxidant-treated NAFLD hepatocyt
33                                              Polyploid cells are a characteristic feature of certain
34                                  Binucleated polyploid cells are common in many animal tissues, where
35                                     However, polyploid cells are found in many tumors, and the enhanc
36             In many developmental processes, polyploid cells are generated by a variation of the norm
37                In these circumstances, large polyploid cells are produced in organisms that are prima
38 t precursor, the megakaryocyte, matures to a polyploid cell as a result of DNA replication in the abs
39                                           In polyploid cells, Bik1 is required before anaphase to mai
40 ogressing through the cell cycle faster than polyploid cells, both in vitro and during liver regenera
41 ntial recognition of autophagy-competent and polyploid cells by the innate and cellular immune system
42 p53-mediated suppression of proliferation of polyploid cells can be averted by increased levels of on
43                                      Somatic polyploid cells can be mononucleate or multinucleate, an
44               In mammals, the development of polyploid cells can contribute to tissue differentiation
45                                              Polyploid cells can originate from cell fusion, endorepl
46 end points are preceded by the appearance of polyploid cells caused by the suppression of Aurora kina
47                                              Polyploid cells contain more than two copies of each chr
48  The remaining 15 cells initiate a series of polyploid cell cycles to prepare for their role as nurse
49                                          How polyploid cells deal with increased centrosome numbers a
50 embly in polyploid cells, we found that most polyploid cells divide in a multipolar manner.
51 ly degraded by acetylated Skp2, resulting in polyploid cell division, genomic instability, and oncoge
52 o quantify aneuploidy, a possible outcome of polyploid cell divisions.
53                                    Normally, polyploid cells do not divide mitotically after initiati
54  meiosis-specific genes are expressed in the polyploid cells during depolyploidization.
55 pite its prevalence, major gaps exist in how polyploid cells emerge and affect tissue function.
56   While most cells maintain a diploid state, polyploid cells exist in many organisms and are particul
57         Our study shows that DENV uses these polyploid cells for its replication.
58                             Concomitant with polyploid cell formation, we observed the appearance of
59 ulture with thrombopoietin failed to develop polyploid cells greater than 8N.
60  begun to elucidate the signals required for polyploid cell growth as well as the advantages and disa
61        Tissue repair usually requires either polyploid cell growth or cell division, but the molecula
62 romotes the diploid-polyploid conversion and polyploid cell growth through the Akt-Skp2 axis.
63          The newly discovered versatility of polyploid cells has the potential to provide alternative
64                                              Polyploid cells have genomes that contain multiples of t
65  p53 activity leads to a greater fraction of polyploid cells, higher mean and maximum ploidy, acceler
66 w cytometry confirmed a greater frequency of polyploid cells in basal zones of leaf blades, consisten
67  and the enhanced chromosomal instability of polyploid cells in culture suggests that such cells cont
68 0% in humans, the specialized role played by polyploid cells in liver homeostasis and disease remains
69 f p53 in 4pX-1 cells increases by 5-fold the polyploid cells in response to pX expression, indicating
70 allele system to genetically label and trace polyploid cells in situ.
71 leads to an earlier onset of polyploidy, and polyploid cells in the adult brain are more resistant to
72 o apoptosis and to allow for accumulation of polyploid cells in vitro.
73        The mitotic arrest occurring in these polyploid cells involves novel alterations in the cdk1/c
74 igenesis, primarily because cell division in polyploid cells is error-prone and produces aneuploid ce
75      Underreplication of specific regions in polyploid cells is proposed to be due to a slower S phas
76                           The elimination of polyploid cells largely depends on the pro-apoptotic BAX
77 takes 2-3 months and can be applied to other polyploid cell lines or high-copy-number genes.
78                    Thus, disrupting genes in polyploid cell lines or when using poorly performing sgR
79                                          The polyploid cells lost the capacity for proliferation and
80  where haploid 5-mum cells convert to highly polyploid cells of >10 mum with distinct but poorly unde
81  minichromosome Dp(1;f)1187 are shortened in polyploid cells of both the ovary and salivary gland but
82 ithin sequences that are underrepresented in polyploid cells of Drosophila melanogaster.
83 ocycles, as we find only the M-phase-capable polyploid cells of the papillae and female germline can
84  known about the fates or functions of these polyploid cells or how they affect development of liver
85 e mechanisms that lead to the development of polyploid cells, our current state of understanding of h
86 thout significant genetic instability (i.e., polyploid cell populations were not observed).
87     Whole-genome duplication (WGD) generates polyploid cells possessing more than two copies of the g
88 ra-nuclear protein translocation of FoxM1 in polyploid cells, respectively.
89             The accumulation of aneuploid or polyploid cells resulting from a disrupted mitotic spind
90  the formation of tetraploid or higher-order polyploid cells resulting from the culture of human colo
91 rch, and how they relate to studies of other polyploid cells, such as cancer cells.
92 ergo cytokinesis, producing aploid cells and polyploid cells that contain multiple SPBs.
93 tion of Aurora-A in adult tissues results in polyploid cells that display a DNA-damage-like response
94 ary infection, cryptococcal cells form large polyploid cells that exhibit increased resistance to hos
95                    Megakaryocytes are large, polyploid cells that produce platelets.
96 ollectively as endoreplication, resulting in polyploid cells that support specific aspects of develop
97 o the sustained increase in the frequency of polyploid cells, the level of polyploidization was downr
98 In this study we employed sorted, pX-induced polyploid cells to investigate their growth and oncogeni
99  in diploid cells resulted in acquisition of polyploid cell traits.
100 ker gene expression, the appearance of large polyploid cells (trophoblast giant cells), and the expre
101 riptional drivers of all but the most highly polyploid cell types of the placenta.
102 inantly enhance the dap phenotype in several polyploid cell types.
103  live cell-sorting, nearly 40% of pX-induced polyploid cells undergo apoptosis, whereas the surviving
104                     Moreover, the changes in polyploid cell wall composition promoted saccharificatio
105 r cells to study mitotic spindle assembly in polyploid cells, we found that most polyploid cells divi
106 ic checkpoint abnormality with production of polyploid cells when exposed to microtubule-targeting dr
107 e heterozygotes with such mutations generate polyploid cells when exposed to spindle depolymerizing a
108 mosome structure is a characteristic of some polyploid cells where several to thousands of chromatids
109                                              Polyploid cells, which contain more than two genome copi
110 intain meiosis can result in highly aberrant polyploid cells, which could lead to oncogenesis in the
111 cation remains unfinished in many Drosophila polyploid cells, which harbor disproportionately fewer c
112 d Kinesin-14 HSET increased the frequency of polyploid cells, which resulted from a failure in cytoki
113 NA content with each cell cycle resulting in polyploid cells with an intricate demarcation membrane s
114 because Aurora-A-deficient tumors accumulate polyploid cells with limited proliferative potential.
115         Given that mitochondrial genomes are polyploid, cells with advantageous levels of mtDNA mutat
116  are terminally differentiated, uncultivable polyploid cells, with remarkably elongated and even bran
117 ientation of individual chromosomes in large polyploid cells would not hamper reproductive success as
118  were specifically exposed on the surface of polyploid cells, yet lost upon passage of such cells thr

 
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