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1 the diagnosis of demyelinating inflammatory polyradiculoneuropathy.
2 , such as chronic inflammatory demyelinating polyradiculoneuropathy.
3 ents with chronic inflammatory demyelinating polyradiculoneuropathy.
4 drome and chronic inflammatory demyelinating polyradiculoneuropathy.
5 ndrome or chronic inflammatory demyelinating polyradiculoneuropathy.
6 en in human acute inflammatory demyelinating polyradiculoneuropathy.
7 ses such as acute inflammatory demyelinating polyradiculoneuropathy.
8 rre syndrome (GBS) is a rare immune-mediated polyradiculoneuropathy.
9 portions of acute inflammatory demyelinating polyradiculoneuropathy (AIDP) (71.5% vs. 72%; p=1), axon
10 st cases of acute inflammatory demyelinating polyradiculoneuropathy (AIDP) from acute motor axonal ne
11 ance of the acute inflammatory demyelinating polyradiculoneuropathy (AIDP) subtype resembles experime
12 hose with chronic inflammatory demyelinating polyradiculoneuropathy and 56% of those with multifocal
13 part from chronic inflammatory demyelinating polyradiculoneuropathy and from systemic necrotizing vas
14 ents with chronic inflammatory demyelinating polyradiculoneuropathy and in none of the patients with
15 The diagnosis is based on having both the polyradiculoneuropathy and the monoclonal plasma cell di
16 treatments for immune-mediated demyelinating polyradiculoneuropathies are varied, with the authors ge
17 ents with chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) and antibodies directed ag
18 ortant in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) and Guillain-Barre syndrom
19 syndrome, chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) and multifocal motor neuro
20 caused by chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) but long-term effects have
28 ggressive chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) shows autoantibodies to co
29 f typical chronic inflammatory demyelinating polyradiculoneuropathy (CIDP), as well as have an aggres
33 points), chronic inflammatory demyelinating polyradiculoneuropathy (CIDP, n = 35, 70 time points), m
34 including chronic inflammatory demyelinating polyradiculoneuropathy (CIDP: N=20); amyloid polyneuropa
35 loplexus neuropathy (7/12 patients) or acute polyradiculoneuropathy (Guillain-Barre syndrome) (4/12).
36 enting as chronic inflammatory demyelinating polyradiculoneuropathy in infants of North-African Jewis
38 typical neuropathy types (radiculoplexus and polyradiculoneuropathies), monophasic course and tempora
39 cally presented as radiculoplexus neuropathy/polyradiculoneuropathy (more proximal involvement of sho
40 described chronic inflammatory demyelinating polyradiculoneuropathy, multifocal motor neuropathy with
41 gnoses of chronic inflammatory demyelinating polyradiculoneuropathy, myeloproliferative disorder, and
42 uropathies included axonal and demyelinating polyradiculoneuropathies (n = 2), length-dependent neuro
43 (n = 32), chronic inflammatory demyelinating polyradiculoneuropathy (n = 4) and healthy controls (n =
44 hort with chronic inflammatory demyelinating polyradiculoneuropathy (n=53) and Guillain-Barre syndrom
47 e defining features of the disease, that is, polyradiculoneuropathy, organomegaly, potentially includ
48 Guillain-Barre syndrome (GBS) is an acute polyradiculoneuropathy, thought to be an autoimmune proc