ライフサイエンスコーパス: pontine

1 iations between age and the mean cortical to pontine 18F-florbetapir standard uptake value ratios, pr

2 neurones, and increased c-fos expression in pontine A7 and other noradrenergic neurones.

3 rome is bilateral horizontal gaze palsy from pontine abducens nuclear defects, rather than abducens n

4 ate laterality, it was found that the dorsal pontine activation was ipsilateral in the right-sided an

5 espiratory drive; (b) even though changes in pontine activity are transient, they can persist after n

6 We hypothesized that DL pontine activity during delayed-I tests would be compara

7 (splenium and genu), two projection (cortico-pontine and anterior thalamic), and five bilateral assoc

8 While pontine and columnar neurons have been analyzed in detai

9 Central pontine and extrapontine myelinolysis (CPM/EPM) are seve

10 Central pontine and extrapontine myelinolysis patients experienc

11 orphic sets of small-field neurons including pontine and fb-eb neurons, and also isomorphic sets of l

12 cuitry governing REM sleep is located in the pontine and medullary brainstem and includes ascending a

13 and strong FLNa labeling was evident in the pontine and medullary raphe nuclei and in sensory and sp

14 ior colliculus, dorsal raphe, mesencephalic, pontine and medullary reticular formation, and the follo

15 The present study was undertaken to identify pontine and medullary structures that respond to noxious

16 itary tract (NTS), reticular formation (RF), pontine and midbrain vestibular nuclei, and medullary ra

17 cholinergic projections to MNTB arising from pontine and superior olivary nuclei.SIGNIFICANCE STATEME

18 Its receptor, FGFR2, is expressed by pontine and vestibular neurons when their axons (mossy f

19 those areas included limbic, basal ganglia, pontine, and cerebellar sites.

20 pare the power of template-based cerebellar, pontine, and cerebral white matter reference regions to

21 nges in an arousal network including limbic, pontine, and cortical areas underlying the decreased per

22 Parkinson's disease tremor is localized to pontine- and mesencephalic-cerebellar-thalamic circuits,

23 a cerebrovascular accident caused by a right pontine arteriovenous malformation and destruction of th

24 that in controls and three times the rate of pontine atrophy in PSP.

25 oradic PAPT, the combination of brainstem or pontine atrophy, parkinsonism, autonomic dysfunction or

26 including 1 proband from the first reported pontine autosomal dominant microangiopathy with leukoenc

27 ange dorsal guidance of inferior olivary and pontine axons after crossing the midline.

28 ed defects of the medial deep cerebellar and pontine axons observed in Unc5c(-/-) embryos, demonstrat

29 eus, reticular nuclei, nucleus raphe magnus, pontine blink premotor area, and superior salivatory nuc

30 d reciprocal inhibitory interactions between pontine brainstem monoaminergic and cholinergic neurons.

31 havioral arousal via descending input to the pontine brainstem.

32 The ability of pontine carbachol to elicit any cortical, hippocampal or

33 Both pontine cell groups express the transcription factor Fox

34 , the median raphe, caudal dorsal raphe, and pontine central gray are major recipients of LHb efferen

35 VTA, median raphe, caudal dorsal raphe, and pontine central gray reside in characteristic, but somet

36 s, and via caudal pontine reticular nucleus, pontine central gray, and MS, reached EC.

37 s in the LC; medial GABAergic neurons in the pontine central gray; ventromedial, small GABAergic neur

38 may be bridged by forebrain regions through pontine-cerebellar nuclear connections that can bypass c

39 ry cortex and detail several potential extra-pontine cerebrocerebellar pathways.

40 omparison with other mammals, the numbers of pontine cholinergic (126,776) and noradrenergic (122,878

41 itative analysis reveals that the numbers of pontine cholinergic (259,578) and noradrenergic (127,752

42 tative analysis revealed that the numbers of pontine cholinergic (274,242) and noradrenergic (203,686

43 enlarged posterior commissure, supernumerary pontine cholinergic and noradrenergic cells, and an enla

44 arged posterior commissure and supernumerary pontine cholinergic and noradrenergic neurons indicate t

45 sions, and may result from disruption of the pontine component of associative corticopontocerebellar

46 isite recordings were made within a putative pontine-cortical micturition circuit from the pontine mi

47 ot dystrophin is required for normal basilar pontine development.

48 Pontine efferent axons terminate mainly in V and rostral

49 Cortico-pontine fibers arose from the PcG, the caudal/medial SFG

50 Cortico-pontine fibers travel through the cerebral peduncles and

51 the generation of active sleep is gated by a pontine GABAergic system that exerts its effects postsyn

52 aracteristics in pediatric diffuse intrinsic pontine glioma (DIPG) and to correlate these metrics wit

53 ch tumors in children with diffuse intrinsic pontine glioma (DIPG) by measuring the tumor uptake of (

54 s significantly reduced in diffuse intrinsic pontine glioma (DIPG) cells that carry a lysine-to-methi

55 Diffuse intrinsic pontine glioma (DIPG) comprise a subset of HGG that aris

56 Diffuse intrinsic pontine glioma (DIPG) is a childhood brainstem tumor wit

57 Diffuse intrinsic pontine glioma (DIPG) is a fatal brain cancer that arise

58 Diffuse intrinsic pontine glioma (DIPG) is a fatal childhood cancer.

59 Diffuse intrinsic pontine glioma (DIPG) is a fatal malignancy of the child

60 Diffuse intrinsic pontine glioma (DIPG) is a fatal pediatric cancer with l

61 Diffuse intrinsic pontine glioma (DIPG) is a highly aggressive pediatric b

62 Diffuse intrinsic pontine glioma (DIPG) is a lethal pediatric brain cancer

63 Diffuse intrinsic pontine glioma (DIPG) is a lethal pediatric tumor with n

64 Diffuse intrinsic pontine glioma (DIPG) is a surgically unresectable and d

65 Diffuse intrinsic pontine glioma (DIPG) is an incurable pediatric brain tu

66 survival for children with diffuse intrinsic pontine glioma (DIPG) is less than 10%, and new therapeu

67 survival for children with diffuse intrinsic pontine glioma (DIPG) is less than one year.

68 and response assessment of diffuse intrinsic pontine glioma (DIPG) is poorly defined.

69 R1 have been identified in diffuse intrinsic pontine glioma (DIPG) tumors.

70 roved for the treatment of diffuse intrinsic pontine glioma (DIPG), an aggressive pediatric cancer re

71 gliomas (DMGs), including diffuse intrinsic pontine glioma (DIPG), have dismal outcomes.

72 tic mutations in pediatric diffuse intrinsic pontine glioma (DIPG), we performed whole-genome sequenc

73 tal pediatric brain tumor, diffuse intrinsic pontine glioma (DIPG).

74 tic oligodendroglioma, and diffuse intrinsic pontine glioma (DIPG).

75 are frequently observed in diffuse intrinsic pontine glioma (DIPGs), an incurable pediatric cancer.

76 tric high-grade glioma and diffuse intrinsic pontine glioma and 20 publicly available datasets in an

77 dren and young adults with diffuse intrinsic pontine glioma and to develop a consensus on recommendat

78 uct of clinical trials for diffuse intrinsic pontine glioma involves use of consistent, objective dis

79 Diffuse intrinsic pontine glioma is an aggressive pediatric cancer for whi

80 d vascular morphology in a diffuse intrinsic pontine glioma patient was performed.

81 childhood brainstem tumor diffuse intrinsic pontine glioma to harbor somatic mutations in ACVR1.

82 w- and high-grade gliomas, diffuse intrinsic pontine glioma, and ependymoma) and some selected rare t

83 ressive and chemoresistant diffuse intrinsic pontine glioma.

84 in a spontaneous model of diffuse intrinsic pontine glioma.

85 explain lack of effect in diffuse intrinsic pontine glioma.

86 Diffuse intrinsic pontine gliomas (DIPG) are incurable brain tumors with a

87 pediatric HGGs, including diffuse intrinsic pontine gliomas (DIPGs) and non-brainstem HGGs (NBS-HGGs

88 gh-grade glioma (pHGG) and diffuse intrinsic pontine gliomas (DIPGs) are aggressive pediatric brain t

89 Diffuse intrinsic pontine gliomas (DIPGs) are aggressive pediatric brain t

90 Diffuse Intrinsic Pontine Gliomas (DIPGs) are deadly paediatric brain tumo

91 H3K27M diffuse intrinsic pontine gliomas (DIPGs) are fatal and lack treatments.

92 Diffuse intrinsic pontine gliomas (DIPGs) are highly aggressive tumors of

93 Diffuse intrinsic pontine gliomas (DIPGs) are highly infiltrative malignan

94 Diffuse intrinsic pontine gliomas (DIPGs) are incurable childhood brainste

95 We report that human diffuse intrinsic pontine gliomas (DIPGs) containing the K27M mutation dis

96 n childhood HGG, including diffuse intrinsic pontine gliomas, and gene expression analyses supported

97 target for pediatric HGG, including diffuse pontine gliomas.

98 ainstem gliomas, including Diffuse Intrinsic Pontine Gliomas.

99 easure the progress in management of diffuse pontine gliomas.

100 r and superior colliculi, periaqueductal and pontine gray matter, and the red nucleus.

101 of postnatal Sox2(+)Olig2(+) progenitors in pontine growth and oligodendrogenesis.

102 ediatric gliomas; however, the mechanisms of pontine growth remain poorly understood.

103 As pontine hypoplasia has been reported in some clinical ca

104 alities, in particular callosal agenesis and pontine hypoplasia, delayed myelination and, less freque

105 re characterized by the presence of multiple pontine infarcts in all symptomatic mutation carriers.

106 Medial rectus motoneurons receive two main pontine inputs: abducens internuclear neurons, whose axo

107 It has been recently shown that the pontine intertrigeminal region (ITR) plays an important

108 nization at inferior levels and the midbrain-pontine isthmus suggests a vulnerable region of passage

109 on of fiber passage occurred at the midbrain-pontine isthmus where all of the fiber bundles overlappe

110 e rostrally in the pons, and in the midbrain-pontine junction part of the nucleus, orexin-A-immunopos

111 superior cerebellar peduncle at the midbrain pontine junction.

112 Since the pontine Kolliker-Fuse area (KF) drives post-inspiration,

113 lary pre-Botzinger complex (preBotC) and the pontine Kolliker-Fuse nucleus (KF) are considered critic

114 The pontine Kolliker-Fuse nucleus (KF) controls upper airway

115 Neurons in the pontine Kolliker-Fuse nucleus (KF) were inhibited with b

116 t-inspiration, achieved by inhibition of the pontine Kolliker-Fuse nucleus, removed post-inspiratory

117 teral nucleus of the solitary tract, and the pontine Kolliker-Fuse, intertrigeminal region, and later

118 cts in the basilar pons to determine whether pontine lacunar syndromes conform to discrete clinical e

119 r nucleus of the hypothalamus (PVN), and the pontine lateral parabrachial nucleus (PBL; an important

120 ical laughter result from rostral and medial pontine lesions, and may result from disruption of the p

121 SX, transection of the brain stem at the mid-pontine level abolished PD in response to unilateral ESV

122 and transection of the brain stem at the mid-pontine level blocks access of vagus-induced activity th

123 control posit a key modulatory role for the pontine locus coeruleus-norepinephrine (LC-NE) system.

124 We used anaesthetized rats in which pontine microinjections of a cholinergic agonist, carbac

125 corticotropin-releasing hormone (Crh) in the pontine micturition center (PMC) is electrophysiological

126 ontine-cortical micturition circuit from the pontine micturition center (PMC), locus coeruleus (LC) a

127 her densities of fibers originating from the pontine micturition center in the lumbosacral gray matte

128 erruption of the descending pathway from the pontine micturition center to the sacral spinal cord in

129 trigger voiding and thereby function as the "pontine micturition center." Lacking detailed informatio

130 been disconnected by spinal injury from the pontine micturition centre, vanilloid-sensitive fibres a

131 lar afferent systems in DS, including fronto-pontine (middle cerebellar peduncle) and olivo-cerebella

132 dritic spine formation, axon elongation, and pontine midline crossing in a FEZF2-dependent manner.

133 s specifically arrest the extension of their pontine mossy fiber afferents, leading us to propose tha

134 In the caudal pontine MTN, only scattered orexin-A-immunoreactive fibe

135 t (ICU) admission, and occurrence of central pontine myelinolysis (CPM).

136 uronal population within ventral, dorsal and pontine network compartments.

137 (no-inflation (no-I or delayed-I) tests), DL pontine neuronal activity increased the strength and con

138 We conclude that (a) DL pontine neurones receive both vagal-dependent excitatory

139 f the NTS) and the second-order dorsolateral pontine neurons (pre-LC and PB-el inner neurons).

140 the maturation of mossy fiber afferents from pontine neurons and the expression of the alpha6 GABA(A)

141 Instead, ectopic clusters of pontine neurons are found lateral to their normal site,

142 ontrol animals, and that significantly fewer pontine neurons are produced.

143 enic expression of Unc5c in deep neurons and pontine neurons by the Atoh1 promoter rescued defects of

144 However, abnormal migration of facial and pontine neurons found in NMHC II-B mutant and ablated mi

145 naline-containing fibers that originate from pontine neurons in the A5, locus coeruleus (LC), and A7

146 Netrin-1 fails to attract pontine neurons lacking Robo3, and attraction can be res

147 the basilar pontine nuclei is delayed, with pontine neurons migrating 1-2 days later than in control

148 We also identified a novel set of pontine neurons that connect contralateral segments in t

149 by YO at 5.0 mg/kg BW included medullary and pontine neurons that project to the central nucleus of t

150 Pontine neurons, along with other precerebellar neurons

151 chain in migrating, compared with stationary pontine neurons, supports an active role for myosin II i

152 e CX are formed by columnar, tangential, and pontine neurons.

153 5c(-/-) mice, as were anterior migrations of pontine neurons.

154 myocytes and in migration of the facial and pontine neurons.

155 recerebellar migratory stream, including the pontine neurons.

156 specific class of bi-columnar elements, the pontine neurons.

157 Thus, the pontine noradrenergic cell groups project in a roughly t

158 The pontine noradrenergic cell groups, A5, A6 (locus coerule

159 They also innervate pontine noradrenergic cell groups, including the locus c

160 t is, at least in part, due to activation of pontine noradrenergic neurones, but is not mediated by O

161 e re-examined the effect of C1 activation on pontine noradrenergic neurons (LC and A5) using a more s

162 requency stimulation of C1 neurons activates pontine noradrenergic neurons and sympathetic nerve disc

163 ess tau-pathology in motor cortex, striatum, pontine nuclei and cerebellum in PSP-PNLA.

164 formance and prevented neuronal cell loss in pontine nuclei and substantia nigra regions.

165 presses high levels of Nfi proteins, and the pontine nuclei are greatly reduced in mice lacking a fun

166 VGLUT1 and -2 were strongly expressed in the pontine nuclei but could not be detected in LC or seroto

167 Coupling discrete stimulation of pontine nuclei controlling vigilance state with analytic

168 Ultimately, we wanted to determine if the pontine nuclei could be a component of the descending au

169 ne inferior colliculus of 10 animals and the pontine nuclei examined under a light microscope to dete

170 eled fibers were observed in the ipsilateral pontine nuclei in 70% of the animals.

171 ojection from the inferior colliculus to the pontine nuclei in guinea pig.

172 ojection from the inferior colliculus to the pontine nuclei in guinea pig.

173 , such as the spinal cord, basal ganglia and pontine nuclei in the brainstem, can be involved.

174 ndicates that the development of the basilar pontine nuclei is delayed, with pontine neurons migratin

175 lt intake, c-Fos activation increased within pontine nuclei that relay gustatory (caudal medial PB) a

176 Tracer overlap in the pontine nuclei was significantly higher than in the othe

177 ere observed in the dorsolateral area of the pontine nuclei, adjacent to the lateral lemniscus.

178 input to the thalamus, thalamic input to the pontine nuclei, and cerebellar feedback to the thalamus.

179 such population, the neurons of the basilar pontine nuclei, expresses high levels of Nfi proteins, a

180 ene including tooth buds, thymic epithelium, pontine nuclei, fastigial cerebellar nuclei, and cerebra

181 ent (striatum, substantia nigra, olivary and pontine nuclei, hippocampus, forebrain and thalamus, ant

182 nucleus, paralemniscal nucleus, red nucleus, pontine nuclei, inferior colliculus and the parvocellula

183 us, in the thalamus, hypothalamus, amygdala, pontine nuclei, spinal cord, and dorsal root ganglion.

184 s hypoglossi, the subtrigeminal nucleus, the pontine nuclei, the dorsal tegmental nucleus, the locus

185 eus, dorsal raphe nucleus, medial lemniscus, pontine nuclei, vagus nerve, inferior olive, abducens nu

186 put from the medial auditory thalamus to the pontine nuclei, which in turn affects input to the cereb

187 ranule cell layer and a reduction in size of pontine nuclei.

188 tercalatus, reticularis tegmenti pontis, and pontine nuclei.

189 cerebellum by way of a synaptic link in the pontine nuclei.

190 ed axonal growth in the deafferented basilar pontine nuclei.

191 t of neuropil in the superior colliculus and pontine nuclei.

192 xternal granule layer and development of the pontine nuclei.

193 cephalic and pontine reticular formation and pontine nuclei.

194 biotinylated dextran amine into the lateral pontine nucleus (PL), and led to extensive retrograde la

195 nucleus (IPN) of the cerebellum and basilar pontine nucleus (PN) during different phases of training

196 nce in the frequency of NCIs in the putamen, pontine nucleus and inferior olivary nucleus between the

197 e tracing experiments identified the basilar pontine nucleus at the confluence of outputs from CeA th

198 -/-) mice and found that they have disrupted pontine nucleus development.

199 In mammals, the pontine nucleus locus ceruleus (LC) is the sole source o

200 oliation defects, motor impairments, partial pontine nucleus migration defects, cochlear hair cell de

201 c nuclei, superior colliculus, zona incerta, pontine nucleus, multiple other brainstem areas, and the

202 olfactory bulb, red nucleus, facial nucleus, pontine nucleus, oculomotor nucleus, substantia nigra, d

203 tegmentum and can be traced into the medial pontine nucleus.

204 a (LHb), ventral tegmental area, and lateral pontine nucleus.

205 egion are most medial and include the median pontine nucleus.

206 om the medial central nucleus to the basilar pontine nucleus.

207 oxb2 deletion resulted in a reduction of the pontine oligodendrogenic domain.

208 cortex, hippocampus, hypothalamus, midbrain, pontine olivary nuclei, trigeminal nuclei, cerebellum, a

209 system by removing the potent inhibition of pontine omnipause neurons (OPNs).

210 eruleus, median raphe, parabrachial complex, pontine oralis, pedunculopontine and ventral tegmental a

211 electrical stimulation of afferents from the pontine parabrachial area (part of the spino-parabrachio

212 NTS) and their efferent target nuclei in the pontine parabrachial complex (PB) in rats during sodium

213 The pontine parabrachial nucleus (PBN) and medullary reticul

214 oral roles for cannabinoid mechanisms of the pontine parabrachial nucleus (PBN) in modulating intake

215 e CGRP innervation of BNST originates in the pontine parabrachial nucleus and targets its anterolater

216 provide evidence that the convergent basilar pontine pathways carry corollary discharges from upper b

217 sory (upper body proprioceptive) and basilar pontine pathways onto individual granule cells and mappe

218 rts on chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids

219 Chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids

220 Chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids

221 named 'chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids'

222 Nfia and Nfix null mice exhibit no apparent pontine phenotype, implying specificity in the action of

223 mplitude of the anticipatory decrease in the pontine portion of DBS was associated with greater activ

224 also appears to be active in normal ventral pontine precursor-like cells of the mouse, and unregulat

225 ith a clinically and radiologically distinct pontine-predominant encephalomyelitis we have named 'chr

226 ulus input to the cerebellum via the thalamo-pontine projection.

227 absent in Fezl(-/-) mice, corticotectal and pontine projections were severely reduced, and Fezl-expr

228 ls retrogradely, BDA3k was injected into the pontine pyramidal tract.

229 the RTN is innervated by both medullary and pontine raphe and receives inputs from thyrotropin-relea

230 ar stem, periaqueductal gray, area postrema, pontine raphe nucleus, gracile nucleus, spinal trigemina

231 cholinergic and noradrenergic nuclei of the pontine region revealed that in comparison with other ma

232 ths, and activity in limbic, paralimbic, and pontine regions decreased.

233 activity in the right prefrontal cortex and pontine regions in the brainstem; no brain regions showe

234 ze of the cerebral cortical, cerebellar, and pontine regions.

235 rainstem respiratory centers via a brainstem pontine relay located in the parabrachial/Kolliker-Fuse

236 the stria terminalis) that in turn regulates pontine REM generator regions.

237 rahypoglossal and dorsal parafacial regions, pontine respiratory group, and ventromedial medulla.

238 (DLSC), periaqueductal grey (PAG), or caudal pontine reticular formation (cPRF), which are implicated

239 duce a rise in activity in the mesencephalic-pontine reticular formation (MPRF), an area of the DPMS

240 KA subunits were also measured in the medial pontine reticular formation (mPRF), medial prefrontal co

241 The oral pontine reticular formation (PnO) of rat is one region i

242 or of adenylyl cyclase into the caudal, oral pontine reticular formation (PnOc) of the rat induces a

243 superior colliculus (SC) and the paramedian pontine reticular formation (PPRF).

244 ic and glycinergic fibers ascending from the pontine reticular formation (PRF) of the brainstem evoke

245 Aergic transmission suggest that GABA in the pontine reticular formation (PRF) promotes wakefulness a

246 ons with characteristic locations within the pontine reticular formation (PRF).

247 nucleus of Darkschewitsch, mesencephalic and pontine reticular formation and pontine nuclei.

248 ity of the PCC to the ventral tegmental area/pontine reticular formation and thalamus, in addition to

249 sis studies revealed that ACh release in the pontine reticular formation is significantly altered by

250 The caudal pontine reticular formation nucleus (cPRF) is implicated

251 , zona incerta (ZI), anterior pretectum, and pontine reticular formation) provides temporally precise

252 ojections to the facial nucleus, surrounding pontine reticular formation, and spinal cord.

253 n the brainstem, including the mesencephalic pontine reticular formation, and the anterior thalami re

254 ort to discrete regions of the medullary and pontine reticular formation, cerebellum, parabrachial nu

255 rinic receptor agonists into the caudal oral pontine reticular formation.

256 nt magnitudes of G protein activation in the pontine reticular formation.

257 the hypothesis that GABA(A) receptors in the pontine reticular nucleus, oral part (PnO) of mouse modu

258 ed from the cochlear nucleus, and via caudal pontine reticular nucleus, pontine central gray, and MS,

259 crest, facial mesenchyme, mesencephalon, and pontine reticular nucleus.

260 We recently characterized physiologically a pontine reticulospinal (pRS) projection in the neonatal

261 nergic neurons in the NTS, and from the same pontine sites that receive major inputs from the HSD2 ne

262 Cortical-to-pontine standard-uptake value ratios were used to charac

263 blink-conditioned responses established with pontine stimulation in 12-day-old rats were reversibly a

264 show associative eyeblink conditioning when pontine stimulation is used in place of an external (e.g

265 isolated CNS histoplasmosis presenting with pontine stroke and meningitis.

266 , and that ipsilateral dysmetria after large pontine stroke represents a disconnection syndrome.

267 s anatomical arrangement suggests that small pontine strokes spare sufficient decussating pontocerebe

268 agnetic resonance imaging revealed bilateral pontine strokes, and the working diagnosis was ischemic

269 greement with the general modulatory role of pontine structures in autonomic activities including res

270 are at risk for alcohol-related reduction of pontine structures that are not necessarily affected by

271 cate that medullary circuits, independent of pontine structures, are sufficient to produce 5-HT induc

272 tion of the state required activation of the pontine subceruleus (SubC) by cholinergic inputs.

273 Cerebral-to-white matter, cerebellar, and pontine SUVRs were characterized in terms of their longi

274 pure motor hemiplegia) to incomplete basilar pontine syndrome and restricted deficits after small foc

275 (100%), with cerebral atrophy or dentate and pontine T2 hyperintensities observed in 28%.

276 ve neurons in the laterodorsal and pedunculo-pontine tegmental nuclei (LDT and PPT), compared with ad

277 dorsal, pedunculopontine, and subpeduncular pontine tegmental nuclei.

278 riaqueductal grey matter (vlPAG) and lateral pontine tegmentum (LPT)) and vice versa.

279 Lesions in the midbrain peduncle and pontine tegmentum alongside the caudate nucleus were imp

280 the neuroaxis and is located in the rostral pontine tegmentum extending from the level of the inferi

281 ithin the nucleus pontis oralis (NPO) of the pontine tegmentum is critically involved in the generati

282 on of nerve growth factor (NGF) into the cat pontine tegmentum rapidly induces rapid eye movement (RE

283 distribution of noradrenergic neurons in the pontine tegmentum that project to the cochlear nucleus w

284 ; amygdala; hippocampus; and dorsal midbrain/pontine tegmentum, including the periaqueductal gray/nuc

285 e ventrolateral periaqueductal gray, lateral pontine tegmentum, locus ceruleus, and dorsal raphe.

286 iculus, the ventral central gray matter, the pontine tegmentum, the amygdala, the reticular formation

287 um lesion overlap was centred in the core of pontine tegmentum.

288 ensive production of oligodendrocytes in the pontine territory (r4d) and delayed and reduced oligoden

289 ary DIPG samples and 5 rare normal pediatric pontine tissue samples, revealing clinically relevant fu

290 on of forskolin-stimulated cAMP in dissected pontine tissue slices containing the PnOc incubated with

291 Compared with non-malignant pediatric pontine tissue, we identify and functionally validate bo

292 hypercapnia (7-10% CO(2)) was abolished with pontine transections or chemical suppression of retrotra

293 ts, Losartan, followed by pre-collicular and pontine transections, failed to reduce tSNA, whereas tra

294 of hippocampal sleep dynamics by cholinergic pontine transients may promote systems and synaptic memo

295 lined the ventral and rostral aspects of the pontine trigeminal sensory nucleus.

296 In the primary pontine tumor, all samples showed similar vascular morph

297 hese animals exhibited an increase in phasic pontine-wave (P-wave) activity during post-training REM

298 nt rapid eye movement (REM) sleep and phasic pontine-wave (P-wave) activity, followed by improvement

299 es hippocampal network events through phasic pontine waves known as pontogeniculooccipital waves (PGO

300 ociation cortex and fractional anisotropy of pontine white matter pathways within the CTCC.