ライフサイエンスコーパス: poor control

1 duals diagnosed with the condition remain in poor control.

2 ardiovascular events than those with fair or poor control.

3 ategies at exacerbation or in the context of poor control.

4 ulting in slow polymerization and relatively poor control.

5 recognized as a risk for asthma severity and poor control.

6 , whereas those with the worst score (4) had poor control (29.4%) and high hazard of hemorrhage (haza

7 L) within the past year; only 9% experienced poor control (a HbA1c level > 12% or mean blood glucose

8 rom asthma, comorbidities that contribute to poor control and lifestyle/environmental factors that re

9 Inefficient induction of ISGs contributes to poor control and persistence of hepatitis C virus infect

10 .e., control and clearance versus partial or poor control and persistent infection).

11 ints that are magnified in wildlife, such as poor control and substantial trait variation within and

12 Patients with severe illness and poor control appear to bear the most burden in terms of

13 health risks and management challenges, with poor control attributed to survival of treatment-resista

14 itoring especially in those with evidence of poor control before pregnancy.

15 The disease in patients with no poor control criteria was defined as well controlled, an

16 Severe asthmatics often exhibit poor control despite high doses of inhaled corticosteroi

17 In patients who have persistently poor control despite optimal medication compliance, newl

18 ded to identify the barriers contributing to poor control even in populations with access to care.

19 GHb <7) soon after induction of diabetes, or poor control for 6 months with 6 months' good control.

20 ozotocin-induced diabetes were in a state of poor control (GHb >11%) for 12 months, good control (GHb

21 4 years of follow-up, subjects who were in "poor" control (glycosylated hemoglobin (GHb) > or = 11%)

22 their HbA(1c) was in the lowest quartile and poor control if it was in the highest quartile.

23 roid therapy (ICS) is a major contributor to poor control in difficult asthma, yet it is challenging

24 ivity observed in particular cases is due to poor control in ylide conformation or due to partial rev

25 ndings suggest that in diabetic adolescents, poor control is associated with a significant depletion

26 Poor control of a local activation process of this syste

27 res knowledge of the factors associated with poor control of asthma symptoms.

28 d CXCL1/keratinocyte-derived chemokine (KC), poor control of bacterial replication, and exacerbated i

29 ertension, lack of appropriate diagnosis and poor control of blood pressure among those with a diagno

30 a well-recognized factor contributing to the poor control of blood pressure in hypertension.

31 Poor control of cancer pain is a major public health pro

32 These findings indicate poor control of cancer pain.

33 association between social risk factors and poor control of cardiovascular disease (CVD) risk factor

34 f core outcome measures used by studies, and poor control of coexisting psychopathology.

35 ily driven by fishing vessel flags linked to poor control of corruption by the flag state, high owner

36 Poor control of diabetes mellitus (DM) increases active

37 factor for several autoimmune diseases, and poor control of EBV viral load and enhanced anti-EBV res

38 Poor control of electrode properties can lead to subopti

39 alian cells by high background fluorescence, poor control of expression, and low GFP maturation effic

40 Poor control of eye movement may lead to unstable binocu

41 Poor control of gastric pH was not associated with feedi

42 Poor control of health risk factors and worsening enviro

43 Poor control of HIV replication and detectable EBV repli

44 tion, CD8(+) T-cell alveolitis, smoking, and poor control of human immunodeficiency virus (HIV) are f

45 ack race, were independently associated with poor control of hypertension among those who were aware

46 Poor control of inflammation results in higher rates of

47 lammatory responses that are associated with poor control of M. tuberculosis infection.

48 These data reveal the persistently poor control of pneumococcal colonization in HIV-infecte

49 has recently been challenged with reports of poor control of reflux and the inability to prevent prog

50 functions, and lack of either element led to poor control of responder cell proliferation and cytokin

51 s suffer from a number of limitations (e.g., poor control of solid tumors), and while combining ACT w

52 In case of poor control of symptoms despite guideline-directed phar

53 ector size and volume of injected fluid, and poor control of the amount of injected material.

54 cessing annealing step at 140 degrees C with poor control of the bonding geometry.

55 eneral nonpermissiveness of primate cells or poor control of the IFN response.

56 Poor control of the laser beam leads to potentially mult

57 s often a trial-and-error process limited by poor control of the local catalyst environment and few s

58 with diabetes are at risk for DKD because of poor control of their blood glucose, as well as nonmodif

59 , the expression of Tim-3 is associated with poor control of viral burden and impaired antiviral immu

60 dysregulated immunity, which contributes to poor control of viral infection.

61 Poor control of viral replication as well as TNF-alpha a

62 ected individuals or humanized mice reported poor control of virus replication and the rapid emergenc

63 Negative immune regulation can result in the poor control of virus replication in chronic infections

64 database who began insulin (2000-2007) after poor control on oral glucose-lowering agents (OGLD) were

65 h often suffer from preservation issues, and poor control on the timing of oxidation leaves geochemic

66 We report that, relative to GTs, STs have poor control over attentional performance, due in part t

67 entrations during the polymerization provide poor control over film thickness.

68 information density, few functional groups, poor control over folding, and difficulties in forming c

69 otein matrices that suffer from variability, poor control over matrix biochemistry, and inability to

70 n such systems is often difficult because of poor control over molecular morphology.

71 e reintubated within the same day because of poor control over secretions, airway spasm, or hypoventi

72 ibitory control deficits possibly underlying poor control over stereotyped and repetitive and compuls

73 o- and heterosolvated dimers but demonstrate poor control over structure.

74 rs comprised participants who showed similar poor control over symptoms with the distribution of the

75 he lack of truly tumor-specific antigens and poor control over T cell activity.

76 olloidal assemblies, but also because of the poor control over the assembly of nanocrystals within a

77 n material modulation(5), which suffers from poor control over the energy and position of trapping po

78 ifficult task for nonswimming organisms with poor control over their orientation.

79 +/- 1.0 kg/ m(2), HbA(1c) 5.9 +/- 0.2%) and poor control (PC) (n = 10, age 50.0 +/- 2.5 years, BMI 2

80 ld male patient with diabetes mellitus under poor control presented to our emergency room with fever,

81 In rats with poor control, retinal GAPDH activity and expressions wer

82 marked decrease in PEPCK content, suggesting poor control strength for this enzyme in gluconeogenic r

83 otein, supporting the concept that PEPCK has poor control strength over the gluconeogenic pathway in

84 ere dampened in co-housed mice, resulting in poor control upon challenge.

85 Poor control was defined as polyp recurrence (polyp grow

86 /toluene mixture, while no polymerization or poor control was observed with Ru(bpy)3.

87 Subjects who were in poor control were somewhat more likely to develop overt

88 In contrast, patient level predictors of poor control were: short acting bronchodilator overuse [