1 arm (El Nino) and cold (La Nina) phases very
poorly.
2 By contrast,
poorly adapted avian-origin HAs contain predominately co
3 ly, vaccination with MF59 recruited NK cells
poorly and drove moderate monocyte phagocytic activity,
4 am signal transduction pathways involved are
poorly characterised.
5 These results advance the understanding of
poorly characterized ER stress-dependent RIP.
6 DZIP3/hRUL138 is a
poorly characterized RNA-binding RING E3-ubiquitin ligas
7 This injury is mitigated by a
poorly characterized, maladaptive repair response.
8 understanding of dog sleep patterns remains
poorly characterized.
9 such as MDR1, regulates other genes that are
poorly characterized.
10 imilation and fixation in paddy soils remain
poorly characterized.
11 eated people with HIV infection (PWH) remain
poorly characterized.
12 nd metabolic changes, these processes remain
poorly characterized.
13 opulation differences in immune responses is
poorly characterized.
14 trinsically disordered and therefore remains
poorly characterized.
15 heir long-term outcomes and resource use are
poorly characterized.
16 the N terminus of this large protein remains
poorly characterized.
17 e associated capsular polysaccharide remains
poorly characterized.
18 rcular RNAs (circRNAs) as biomarkers remains
poorly characterized.
19 lergy, and pain, their roles in COVID-19 are
poorly characterized.
20 Although the mechanisms remain
poorly clarified, drug-resistant strains are widely beli
21 particles in the presence of representative
poorly conductive oxide particles in our experimental co
22 as remained conceptual, experimental, and/or
poorly constrained.
23 serpentinization-driven H(2) fluxes, remain
poorly constrained.
24 These results confirm that when
poorly controlled, COVID-19 can infect a large proportio
25 Chronic cough and sputum production
poorly correlate with the presence of GCH or COPD.
26 Additionally, vaccine-induced CD8 T cells
poorly cross-recognized variant epitopes encoding HLA-I-
27 he telomere region, the integration locus is
poorly defined due to the low sequence complexity (TTAGG
28 clinically presenting patient groups remains
poorly defined for reasons including that somatic mutati
29 oint disease-associated variants that remain
poorly defined in their mechanism of action.
30 cytotoxicity (ADCC), during HCV infection is
poorly defined, while no study has explored the effects
31 The sinonasal microbiome remains
poorly defined, with our current knowledge based on a fe
32 utaneous coronary intervention (uLMS-PCI) is
poorly defined.
33 determinants of variability therefore remain
poorly defined.
34 y are impacted by enteric infections remains
poorly defined.
35 energy balance, and metabolic control remain
poorly defined.
36 odeling during cardiac pressure overload are
poorly defined.
37 that initiate type 2 immune responses remain
poorly defined.
38 lergy (FA) among European school children is
poorly defined.
39 y and affective integration, however, remain
poorly defined.
40 the priming of this T cell response is (are)
poorly defined.
41 relationship between these phenomena remains
poorly defined.
42 bution of ZO-1 to cardiac physiology remains
poorly defined.
43 successful mounting of T cell responses are
poorly defined.
44 ots of their inactive and active states, but
poorly describe the conformational dynamics of the allos
45 et, this specific subset of patients remains
poorly described.
46 Tumors from these mice were
poorly differentiated adenocarcinoma, characterized by e
47 a mesenchymal phenotype, similar to well and
poorly differentiated tumors, respectively.
48 nancies of the ear, tail, and foot comprised
poorly differentiated, round to spindle-shaped cells wit
49 lthy humans and, in contrast to WPI, zein is
poorly digestible.
50 e-body tumor volume for overall survival are
poorly elucidated to date.
51 poral nature of how cells respond to them is
poorly established.
52 e care and the impact on long-term health is
poorly evaluated.
53 ctors associated with these events have been
poorly explored.
54 ess sensitive to edge effects on spectra and
poorly focused regions of particles.
55 rupted the retinal vasculature, resulting in
poorly formed networks, especially in the venous drainag
56 Poorly functioning items were removed, and the model was
57 r have seen limited success in PDAC due to a
poorly immunogenic and exceedingly immunosuppressive tum
58 ozygous rutabaga/+;octbeta1r/+ flies perform
poorly in both aversive and appetitive conditioning, whi
59 -based genome-wide association study perform
poorly in Mexican Americans.
60 tastatic or recurrent disease continue to do
poorly,
indicating a need for new treatments.
61 ey lack methodological transparency; or they
poorly integrate qualitative and quantitative sources of
62 is a highly relevant functional measurement
poorly investigated due to postmortem degradation of syn
63 The extent of this
poorly ionizable pool of carbon is unknown, is presumabl
64 diated by physiology and behavior, which are
poorly known in high-elevation taxa.
65 rized microtubule ends, to which KIF5B binds
poorly,
likely because its cofactors, MAP7-family protei
66 n lung and their contribution to disease are
poorly mapped out.
67 anthracene-substituted derivatives fluoresce
poorly (
Phi < 0.01).
68 ) LSCs were capable of self-renewal and were
poorly proliferative.
69 on in mosquitoes, is a well-acknowledged but
poorly quantified phenomenon that forms the basis for th
70 ns from oil and gas activities are large and
poorly quantified, with onshore studies showing systemat
71 Their mother had
poorly reactive pupils with asymptomatic cerebral arteri
72 -sensitive, prone to photodecomposition, and
poorly reactive towards sterically bulky alkene substrat
73 bA1c (glycated hemoglobin A1c) levels, which
poorly reflects direct glucose variation.
74 nsformed during ozone pretreatment that were
poorly removed in the open-water wetland.
75 -alkylated thymidine lesions are known to be
poorly repaired and persist in mammalian tissues.
76 blood transcriptome whose cellular origin is
poorly resolved.
77 efit in highly metastatic murine TNBC models
poorly responsive to PD-1 blockade.
78 CD4+ T cells were
poorly restored specifically in the lung interstitium, d
79 tain the challenges of being time-consuming,
poorly sensitive and expensive.
80 cific chemotactile receptors (CRs) to detect
poorly soluble natural products, thereby defining a form
81 lain by a variable thickness duricrust, with
poorly sorted, unconsolidated sand with rocks beneath.
82 ptical highlighting indicate that papilin, a
poorly studied glycoprotein, is the most abundant compon
83 nd the accumulation of adrenic acid (AdA), a
poorly studied n-6 PUFA.
84 The resulting surgeon workload is
poorly studied with little knowledge of the contributing
85 Native joint septic arthritis (NJSA) is
poorly studied.
86 es suggest and speculate mechanisms that are
poorly supported by the design and reporting of data in
87 uciferase-luciferin pairs emit light that is
poorly tissue penetrant, hindering efforts to visualize
88 rotic lung lesions of TB patients responding
poorly to antibiotic therapy, supporting the role of NET
89 vation; some scrub typhus patients responded
poorly to doxycycline, which investigators attributed to
90 The few simulation tools that exist scale
poorly to large modern phylogenies, which can comprise t
91 n immunosuppressive microenvironment respond
poorly to therapy.
92 ed potent antitumor activity in vivo but was
poorly tolerated, which was hypothesized to be the resul
93 sorders are some of the most devastating and
poorly treated conditions in developed nations, yet effe
94 n disorders, is highly prevalent but remains
poorly treated.
95 (AOS) interprets social chemosignals, but we
poorly understand AOS information processing.
96 biological heterogeneity in schizophrenia is
poorly understood and confounds current analyses.
97 dividual residues to specificity are usually
poorly understood and often obscured by mutational robus
98 c spectrum disorders, their etiology remains
poorly understood and their phenomenology incompletely c
99 ent; the underlying molecular mechanisms are
poorly understood and warrant investigation given the in
100 cular rules driving TCR cross-reactivity are
poorly understood and, consequently, it is unclear the e
101 status (SES) in transplantation outcomes is
poorly understood because of limitations of conventional
102 lipedes and centipedes, is a fascinating but
poorly understood branch of life, including species with
103 in complex 4 (AP-4) lead to prototypical yet
poorly understood forms of childhood-onset and complex h
104 repertoire of divergent CDKs and cyclins of
poorly understood function and interdependency.
105 variation in molar proportions, what remains
poorly understood is if molar shape, or specifically com
106 onto a periplasmic protein bridge through a
poorly understood mechanism.
107 ate across the outer bacterial membrane by a
poorly understood mechanism.
108 by genetic and environmental modifiers, via
poorly understood mechanisms.
109 Exosome secretion by cells is a complex,
poorly understood process.
110 bacteriocins to translocate into cells by a
poorly understood process.
111 ties of dense hydrogen, including a rich and
poorly understood solid polymorphism(1,3-5), an anomalou
112 ospheric particles and a highly variable and
poorly understood source of INPs.
113 t colitis response, including members of the
poorly understood TM7 phylum.
114 It is currently
poorly understood whether localized S. aureus skin infec
115 time, but the contributing factors are still
poorly understood(1).
116 mental aspects of protein-DNA binding remain
poorly understood(1,2).
117 by which SCN8A variants lead to epilepsy are
poorly understood, although heterologous expression syst
118 achinery and its biological relevance remain
poorly understood, although NTA has gained recognition a
119 driving sarcomatoid dedifferentiation remain
poorly understood, and information and treatment options
120 the etiology of the cognitive impairment is
poorly understood, and no satisfactory cognitive treatme
121 but the underlying molecular features remain
poorly understood, and therapeutic options are limited.
122 Its pathogenesis is
poorly understood, and there are no effective treatments
123 Polycomb system selects its target genes is
poorly understood, and whether its histone-modifying act
124 The etiology of aortic aneurysms is
poorly understood, but it is associated with atheroscler
125 n of drug-resistant tuberculosis (TB) remain
poorly understood, despite over half a million incident
126 s and their transfer mechanisms still remain
poorly understood, especially for the cases of liquid-so
127 nges of the bacterial wine microbiota remain
poorly understood, especially in the context of red and
128 m cells (HSCs) within the leukemic niche are
poorly understood, especially in the human context.
129 n regulating multiple ecosystem functions is
poorly understood, limiting our ability to predict how s
130 the ser/thr protein phosphatase 2A (PP2A) is
poorly understood, limiting our understanding of PP2A-re
131 irment (MCI) and Alzheimer's disease (AD) is
poorly understood, particularly at early stages precedin
132 How this resistance is mediated is
poorly understood, particularly during long-term exposur
133 limited array of G proteins and effectors is
poorly understood, particularly in native cell systems.
134 edictors of BE progression to HGD or EAC are
poorly understood, with multiple contradictory studies.
135 key disease features, and pathophysiology is
poorly understood.
136 y elements, yet how this is achieved remains
poorly understood.
137 he R loop-associated genomic stress is still
poorly understood.
138 d into proliferation-quiescence decisions is
poorly understood.
139 tivalent WW domain-PPXY assemblies are still
poorly understood.
140 inding, selectivity, and signaling are still
poorly understood.
141 operate with known somatic driver events are
poorly understood.
142 interactions with cellular chaperones remain
poorly understood.
143 he mechanisms underlying this disruption are
poorly understood.
144 rities and institutional constraints remains
poorly understood.
145 the mechanisms that govern this process are
poorly understood.
146 ative regulator of mTOR/protein kinase B, is
poorly understood.
147 nctional properties of cortical circuits are
poorly understood.
148 to many populations that live today, remains
poorly understood.
149 ient signals control IME1 expression remains
poorly understood.
150 which TBC1D23 regulates cargo transport are
poorly understood.
151 acteria, but their interrelationships remain
poorly understood.
152 events that drive metastatic recurrence are
poorly understood.
153 nic obstructive pulmonary disease (COPD) are
poorly understood.
154 neurons are produced from progenitors remain
poorly understood.
155 (LOC) and affects sensory processing remains
poorly understood.
156 l translocation, and inflammation in PHIV is
poorly understood.
157 virus-driven germinal center response remain
poorly understood.
158 characterized, the underlying mechanisms are
poorly understood.
159 ostoperative prescribing among providers are
poorly understood.
160 icated; however, the genetic architecture is
poorly understood.
161 ms that link these pathways, however, remain
poorly understood.
162 r, its mechanism of transport into plants is
poorly understood.
163 ionary origins and ancestral function remain
poorly understood.
164 it recapitulates embryonic mechanisms remain
poorly understood.
165 covered, but its mechanism of action remains
poorly understood.
166 how this may contribute to tumorigenesis is
poorly understood.
167 er, the patterns for grants and grantees are
poorly understood.
168 -ALL, the mechanisms of GC resistance remain
poorly understood.
169 logical processes modified by nesting remain
poorly understood.
170 riants on protein function and physiology is
poorly understood.
171 of TRIM21 during bacterial infection remains
poorly understood.
172 t and dyke propagation through the crust are
poorly understood.
173 tion is limited and functional importance is
poorly understood.
174 ions to recent regulatory adaptations remain
poorly understood.
175 ation of leptin in polygenic obesity remains
poorly understood.
176 -mediated immunity (AMI) to Y. pestis remain
poorly understood.
177 for its entry, replication, and assembly are
poorly understood.
178 complex signaling pathways, which are still
poorly understood.
179 late fascin dynamics within filopodia remain
poorly understood.
180 However, ASD pathogenic mechanisms are
poorly understood.
181 nals to produce a given behavioral output is
poorly understood.
182 y cells in neurodegenerative diseases remain
poorly understood.
183 iatric surgery in this patient population is
poorly understood.
184 lowing performance, the mechanics of TBR are
poorly understood.
185 the underlying causes of this variation are
poorly understood.
186 hanical properties of the mineral phases are
poorly understood.
187 chanisms controlling digit morphology remain
poorly understood.
188 in dynamics and the function of a protein is
poorly understood.
189 homeostatic mechanisms during pregnancy are
poorly understood.
190 behavior and subsequent reinstatement remain
poorly understood.
191 evels, but its neural implementation remains
poorly understood.
192 cular mechanisms underlying ECM layering are
poorly understood.
193 o dictate the fates of cellular RNAs remains
poorly understood.
194 g increased susceptibility to infections are
poorly understood.
195 ongenita mutations remain uncharacterized or
poorly understood.
196 on and differentiation of cDCs are currently
poorly understood.
197 water-stress are shaped by host genotype are
poorly understood.
198 n the brain as this learning takes place are
poorly understood.
199 air NHE6 activity and endosomal function are
poorly understood.
200 d the influence of exercise intensity remain
poorly understood.
201 atomical basis of this heterogeneity remains
poorly understood.
202 and behavioral consequences, if any, remain
poorly understood.
203 orces driving their emergence and spread are
poorly understood.
204 ve information about reward outcomes remains
poorly understood.
205 The etiology of autism spectrum disorder is
poorly understood.
206 een specific dietary fats and cell fates are
poorly understood.
207 hrough the blood-brain barrier (BBB) remains
poorly understood.
208 tion mechanisms in estuarine wetlands remain
poorly understood.
209 complex is assembled upon binding to IFN is
poorly understood.
210 nicotine withdrawal on cognitive control are
poorly understood.
211 nelles is determined and maintained is still
poorly understood.
212 adapt to a particular internal state remain
poorly understood.
213 e of scaffold proteins in these processes is
poorly understood.
214 n mediating autophagy to suppress viruses is
poorly understood.
215 e onset but the underlying mechanisms remain
poorly understood.
216 ry, gene expression, assembly, and egress is
poorly understood.
217 for severe inflammatory side effects remain
poorly understood.
218 tions in sympathetic neuron survival is very
poorly understood.
219 of epithelial cells to the host response are
poorly understood.
220 he persistent accumulation of Mo/MPhi remain
poorly understood.
221 ying this so-called foraging response remain
poorly understood.
222 pathophysiology of Lassa fever in humans is
poorly understood.
223 habitable for terrestrial organisms remains
poorly understood.
224 nce of variable environmental forces remains
poorly understood.
225 fates are triggered by environmental cues is
poorly understood.
226 health risks associated with ENDS use remain
poorly understood.
227 echanisms underlying this resistant is still
poorly understood.
228 echanisms that initiate this activity remain
poorly understood.
229 pecificity of their function is achieved are
poorly understood.
230 d yet how the brain represents shape remains
poorly understood.
231 derived exosomes in metabolic homeostasis is
poorly understood.
232 the role of the thalamus in this process is
poorly understood.
233 e activation and IL-1beta secretion, remains
poorly understood.
234 during development by mechanisms that remain
poorly understood.
235 factors to variation in immune responses are
poorly understood.
236 chanisms underlying diverse morphologies are
poorly understood.
237 The mechanisms mediating AMC remain
poorly understood.
238 -specific effects on receptor activation are
poorly understood.
239 lex mechanisms driving immune evasion remain
poorly understood.
240 ulation is regulated or impacts longevity is
poorly understood.
241 that regulates Parkin protein level remains
poorly understood.
242 ted, regulation of membrane dynamics remains
poorly understood.
243 ecosystems, yet their formation mechanism is
poorly understood.
244 rctic Ice Sheet (EAIS), to global warming is
poorly understood.
245 determinants of recognition specificity are
poorly understood.
246 hat underlie their ionic conductivity remain
poorly understood.
247 ular force regulates these processes remains
poorly understood.
248 sses underpinning flowering in nature remain
poorly understood.
249 igated, the genetic etiology of NTDs remains
poorly understood.
250 helial-to-hematopoietic transition (EHT) are
poorly understood.
251 owever, the adaptive mechanisms involved are
poorly understood.
252 their effect on the soil microbiome remains
poorly understood.
253 ve rise to correlated activity are, however,
poorly understood.
254 xtend and/or progress into adulthood remains
poorly understood.
255 h rates of relative sea level rise (RSLR) is
poorly understood.
256 of myosin 1s distinctive lipid targeting is
poorly understood.
257 y risk factors for adulthood fatty liver are
poorly understood.
258 blasts to persist in fibrotic tissues remain
poorly understood.
259 l basis of Wnt signaling through Fzd remains
poorly understood.
260 cisely how gamma-TuRC nucleates a MT remains
poorly understood.
261 perature signaling pathways in plants remain
poorly understood.
262 t predicts the response to immunotherapy are
poorly understood.
263 n of GPR139 and its signaling mechanisms are
poorly understood.
264 they affect substrate conformations remains
poorly understood.
265 inst hemibiotrophic fungal infection remains
poorly understood.
266 under pathologically relevant conditions is
poorly understood.
267 ion of this function to tumor suppression is
poorly understood.
268 hts, are overlooked and their impacts remain
poorly understood.
269 are event which significance for immunity is
poorly understood.
270 ics of monsoonal precipitation and runoff is
poorly understood.
271 ut how these are implemented in the brain is
poorly understood.
272 eural mechanisms underlying this process are
poorly understood.
273 to reach taste receptors, processes that are
poorly understood.
274 cardiac neovascularization after injury are
poorly understood.
275 large-scale hydroclimate variability remains
poorly understood.
276 higher up in the cortical hierarchy remains
poorly understood.
277 e played in the evolution of bat immunity is
poorly understood.
278 utational selection in normal tissues remain
poorly understood.
279 ylceramide, and the origin of psychosine are
poorly understood.
280 n the membrane biophysical properties remain
poorly understood.
281 eatment failure for Mycoplasma genitalium is
poorly understood.
282 chanisms linking these two phenomena, remain
poorly understood.
283 timuli while ignoring distractor stimuli are
poorly understood.
284 ities and site-selectivities in chromatin is
poorly understood.
285 Tim-3, but the molecular determinants remain
poorly understood.
286 ctional relevance of these modifications are
poorly understood.
287 lation, particularly in human cells, remains
poorly understood.
288 hip between cell number and body size remain
poorly understood.
289 sparities in cancer incidence and outcome is
poorly understood.
290 thway in promoting lipid accumulation remain
poorly understood.
291 hanisms that control myotube guidance remain
poorly understood.
292 motor neurons to OPCs with distinct fates is
poorly understood.
293 rotoxicity, the underlying mechanisms remain
poorly understood.
294 e cis-isomers from trans-violaxanthin remain
poorly understood.
295 and neurogenetics of animal migration remain
poorly understood.
296 g factors and strategies to address this are
poorly understood.
297 ctor sensing triggers NLR activation remains
poorly understood.
298 e canine population and yet, its etiology is
poorly understood.
299 t the cues promoting Trm cell generation are
poorly understood.
300 The dosage formulation of
poorly water-soluble drugs often faces several challenge