ライフサイエンスコーパス: popliteal

1 s were 199 superficial femoral arteries, 110 popliteal, 218 tibials, and 52 multilevel.

2 sustained iliac (2.3%), femoral (56.6%), and popliteal (41.1%) arterial injuries were included.

3 Accumulation of the nonspecific agent by the popliteal and axillary nodes at 6-hr postinjection was a

4 Popliteal and axillary nodes were then assayed for perce

5 , 25% and 8% (P < 0.05) in femoral Delta Q , popliteal and brachial artery FMD%, respectively, occurr

6 p < 0.01) were directly associated with both popliteal and carotid intimal-medial thicknesses.

7 e present analyses are based on the baseline popliteal and carotid ultrasonography examination in 10,

8 oxyglucose uptake for veins of interest (ie, popliteal and femoral).

9 ial (TRP) channel TRPC3 were present in both popliteal and first order intramuscular arterioles.

10 ode (LN) excision, consisting of ipsilateral popliteal and inguinal LN excision and to evaluate the i

11 ons in lymph drainage through tumor-draining popliteal and inguinal LNs versus contralateral uninvolv

12 The popliteal and inguinal lymph nodes were excised ipsilate

13 also related to early thickening of both the popliteal and the carotid artery walls.

14 imes to the common femoral artery (CFA), the popliteal and tibial arteries, and the corresponding vei

15 n and stiffness of the involved (femoral and popliteal) and uninvolved (brachial) arteries.

16 angioplasty, and atherectomy of the femoral, popliteal, and tibial vessels.

17 rd PTA for the Treatment of SFA and Proximal Popliteal Arterial Disease [INPACT SFA I], NCT01175850;

18 cond and first order arterioles vs. feed and popliteal arteries (58% and 16% vs. 5% and 3%; N = 10 im

19 surement of oxygen saturation in the femoral/popliteal arteries and veins during cuff-induced reactiv

20 based therapy in the superficial femoral and popliteal arteries in patients with peripheral artery di

21 or arterial remodelling, in the brachial and popliteal arteries of 13 healthy male subjects (21.6 +/-

22 ystem in which only the common iliac through popliteal arteries were considered.

23 flow (mean vessel sharpness: 44% vs 30% for popliteal arteries, 45% vs 28% for saphenous arteries; P

24 Popliteal arteries, subsequent gastrocnemius feed arteri

25 rtery disease of the superficial femoral and popliteal arteries.

26 e Superficial Femoral Artery and/or Proximal Popliteal Artery [MDT-2113 SFA], NCT01947478; The IN.PAC

27 uperficial Femoral Artery [SFA] and Proximal Popliteal Artery [PPA] [INPACT SFA II], NCT01566461; MDT

28 rficial Femoral Artery [SFA] and/or Proximal Popliteal Artery [PPA]) that enrolled 331 subjects with

29 ent placement for obstructive lesions of the popliteal artery achieves superior acute technical succe

30 U in the midabdominal aorta to 357 HU in the popliteal artery and 253 HU in the dorsalis pedis or pos

31 with additional 5 seconds +/- 2 to reach the popliteal artery and 7 seconds +/- 4 to reach the ankle

32 The relationship between PI values of the popliteal artery and the number of thrombosed calf veins

33 tion, the superficial femoral artery and the popliteal artery are subject to various forces e.g. thos

34 tentially associated with the development of popliteal artery atherosclerosis in a population-based s

35 h subsequently augmented insulin-stimulated popliteal artery blood flow and muscle perfusion.

36 with symptomatic superficial femoral and/or popliteal artery disease at 11 German centers between Se

37 ndovascular treatment of superficial femoral-popliteal artery disease in the Society for Vascular Sur

38 ality after treatment of superficial femoral-popliteal artery disease with paclitaxel and nonpaclitax

39 tment of symptomatic superficial femoral and popliteal artery disease.

40 stress reaction, periostitis, claudication, popliteal artery entrapment, and peripheral nerve entrap

41 andomization, included the ICD and ACD, ABI, popliteal artery flow with duplex and QOL* at baseline*,

42 udication distances, pressure indices [ABI], popliteal artery flow, and QOL with the short-form 36 He

43 Brachial and popliteal artery FMD and DC did not change in control su

44 Similarly, popliteal artery FMD increased from baseline (6.2 +/- 0.

45 Patients with popliteal artery injuries over the 10-year period ending

46 ve patients who sustained iliac, femoral, or popliteal artery injuries, and underwent surgery to atte

47 e limb salvage in association with repair of popliteal artery injuries.

48 accepted factors impacting amputation after popliteal artery injury include blunt trauma, prolonged

49 ed to evaluate those factors associated with popliteal artery injury that influence amputation, with

50 of Comprehensive Superficial Femoral and/or Popliteal Artery Lesions Using the IN.PACT Admiral Drug-

51 ated spectral doppler characteristics of the popliteal artery on the same side as the isolated calf v

52 To investigate the usefulness of popliteal artery spectral doppler findings as a complime

53 e Superficial Femoral Artery and/or Proximal Popliteal Artery Using the IN.PACT Admiral(TM) Drug-Elut

54 The WSS in the narrow popliteal artery was more sensitive to a reduction in ra

55 Lesions above the popliteal artery were a significant predictor of residua

56 ative superficial femoral artery or proximal popliteal artery with stenosis >=70%, vessel diameter of

57 tment of Superficial Femoral and/or Proximal Popliteal Artery) was designed to evaluate the patency o

58 n 5 cm in the superficial femoral artery and popliteal artery, and six patients had stenoses or occlu

59 y words were: "superficial femoral artery," "popliteal artery," "angioplasty," "drug-eluting balloon,

60 odium chloride (SALINE) was infused into the popliteal artery.

61 atients had stenoses or occlusions below the popliteal artery.

62 n, plasmid DNA was transferred to the distal popliteal artery.

63 t to the pulsatility index (PI) value of the popliteal artery.

64 class 2 to 5 who had a de novo lesion in the popliteal artery.

65 lar treatment for obstructive lesions of the popliteal artery.

66 luding superficial femoral, deep femoral and popliteal) artery models that were reconstructed from ma

67 Patients with symptomatic femoro-popliteal atherosclerotic disease undergoing percutaneou

68 re to induce alterations in the brachial and popliteal blood flow (BF).

69 Though popliteal blood flow appeared to be modulated by respira

70 ign also suggested a correlation between the popliteal-brachial gradient and aortic regurgitation sev

71 olume increased, in-hospital mortality after popliteal bypass decreased from 6.5% to 4.9% (P = 0.0045

72 thmia, aortic valve replacement, and femoral popliteal bypass graft) in isradipine (n=40; 9.05%) vs h

73 ased again by week 8 (6.5 +/- 0.6%), whereas popliteal DC progressively increased from baseline (8.9

74 Popliteal DCs transduced with an empty recombinant Adv u

75 During follow-up, popliteal deep-vein thrombosis was diagnosed in 1 patien

76 erford class 2-4) and superficial femoral or popliteal disease (>=70% stenosis) were randomized 1:1 t

77 rability for aortoiliac disease than femoral popliteal disease.

78 ude femorodistal bypass to ankle or foot and popliteal distal bypass using autogenous vein usually in

79 Iliofemoral DVT (n = 221 [71%]) and femoral-popliteal DVT (n = 79 [25%]) were treated with urokinase

80 tiveness ratio of $137 526/QALY; for femoral-popliteal DVT, standard therapy was an economically domi

81 y one patient required surgery for a delayed popliteal embolus.

82 h proximal deep-vein thrombosis of the legs (popliteal, femoral, or iliac vein thrombosis).

83 sions of the superficial femoral or proximal popliteal (femoropopliteal) artery.

84 5% CI, -0.2 to 2.1; P=0.116), peak hyperemic popliteal flow (0.0+/-0.4 mL/s; 95% CI, -0.8 to 0.8; P=0

85 Inter- and intragroup popliteal flow differences at 5/12 were small (P > 0.1).

86 time (PWT), collateral count, peak hyperemic popliteal flow, and capillary perfusion measured by magn

87 Skin grafts were placed distal to the popliteal fossa and mice were euthanized at day 10.

88 xcised collecting lymphatic vessels from the popliteal fossa of mice and removed their muscle cells t

89 nerve block was produced at the level of the popliteal fossa, and behavior was assessed using evoked

90 hy was performed from the iliac crest to the popliteal fossa.

91 ned contiguous images from the pelvis to the popliteal fossa.

92 om mouse collecting lymphatic vessels of the popliteal fossa.

93 eral erythematous halo was noted in the left popliteal fossa; the ulcer had begun as an asymptomatic

94 CT venograms from the iliac crests to the popliteal fossae were reviewed for presence and location

95 in sites relevant to AD: the antecubital and popliteal fossae, nasal tip, and cheek.

96 adjustment for covariates, both carotid and popliteal intimal-medial thicknesses were strongly assoc

97 of Obstructive Superficial Femoral Artery or Popliteal Lesions With A Novel Paclitaxel-Coated Percuta

98 and biomechanical properties of the oblique popliteal ligament (OPL).

99 ells were observed in maximal numbers in the popliteal LN at day 1 and in marginal zones and T-depend

100 MicroCT imaging of murine popliteal LN showed that capillaries were responsible fo

101 tal three-photon microscopy to visualize the popliteal LN through its entire depth (600-900 mum).

102 and knee joint synovial volumes and draining popliteal LN volumes before and after 8 weeks of treatme

103 icated that most of the blood flow in rabbit popliteal LN was at velocities lower than 5 mm/sec.

104 erations in contrast agent drainage into the popliteal LN, while lower molecular weight or albumin-bi

105 IVM of popliteal LNs after intradermal (i.d.) injection of bact

106 ds In vivo studies were carried out to image popliteal LNs of two healthy male New Zealand white rabb

107 pliteal LNs, lymphatic drainage from paws to popliteal LNs, and the number of VEGF-C-expressing CD11b

108 he number of lymphatic vessels in joints and popliteal LNs, lymphatic drainage from paws to popliteal

109 reatment significantly decreased the size of popliteal LNs, the number of lymphatic vessels in joints

110 und the cortex and medulla of tumor-draining popliteal LNs, while they were restricted to the cortex

111 l formation and the morphology of joints and popliteal LNs.

112 of VEGF-C-expressing CD11b+ myeloid cells in popliteal LNs.

113 ine and chemokine production in the draining popliteal LNs.

114 y in lymphatic vessels afferent to collapsed popliteal LNs.

115 ion on lymph transport from paws to draining popliteal LNs.

116 model of inflammatory-erosive arthritis, the popliteal lymph node (PLN) enlarges during the pre-arthr

117 its the sentinel macrophages in the draining popliteal lymph node (pLN) to infect highly permissive i

118 was confirmed by longitudinal assessment of popliteal lymph node (PLN) volume via ultrasound, PLV co

119 ural and functional changes of the adjoining popliteal lymph node (PLN), detectable by contrast-enhan

120 high CD25+) CD8+ T cells within the draining popliteal lymph node (PLN).

121 d by highly expanded B cells in the draining popliteal lymph node (PLN).

122 Here, we show that the popliteal lymph node (popLN) receives direct neural inpu

123 otyping, and gene expression profiles in the popliteal lymph node and inflamed joints, two pathogenic

124 the inflammatory infiltrate in the draining popliteal lymph node and the site of the infection using

125 The popliteal lymph node assay was used to study the role of

126 We analyzed clonally related VDJ genes from popliteal lymph node B cells responding to primary, seco

127 Ab blockade of ICOS ligand, expressed by popliteal lymph node B cells, but not dendritic cells, a

128 Flow cytometric analysis of popliteal lymph node cells demonstrated similar profiles

129 Spleen and popliteal lymph node cells from OVA-primed mice 3 or 7 d

130 vitro proliferative response of OVA-specific popliteal lymph node cells was assessed.

131 d vessel growth; however, the tumor-draining popliteal lymph node featured greatly increased lymphati

132 , amides show inhibitory activity in the rat popliteal lymph node hyperplasia assay.

133 CCR2(+) monocytic dendritic cells within the popliteal lymph node in comparison with B6.WT mice.

134 dary challenge (the increase in the draining popliteal lymph node mass, cell number, and lymphocyte t

135 C cell lines (P < 0.01) in vitro, as well as popliteal lymph node metastases of ESCC cells in nude mi

136 IL-6 expression was dramatically enhanced in popliteal lymph node tissue after fracture.

137 ype littermates to quantify the synovial and popliteal lymph node volumes and the patella and talus b

138 t respond to the host antigens measured by a popliteal lymph node weight gain assay.

139 of myeloid and lymphoid DCs in the draining popliteal lymph node, but not in other lymphoid organs.

140 l as from Ag-induced germinal centers of the popliteal lymph node.

141 -positive cells were readily detected in the popliteal lymph nodes (pLN) of VLP-inoculated mice.

142 arance at the site of infection and draining popliteal lymph nodes (PLNs), and impaired functions of

143 ed B cell accumulation within tumor-draining popliteal lymph nodes (TDLN).

144 Tissues (draining popliteal lymph nodes [LN], spleens, and thymi) were rem

145 Furthermore, cLTalpha(-/-) popliteal lymph nodes contained a higher proportion of n

146 e, control of RVFVmiR-142 replication in the popliteal lymph nodes correlated with an increased type

147 estimulation, and secondary responses in the popliteal lymph nodes following in vivo challenge and in

148 erived myeloid dendritic cells trafficked to popliteal lymph nodes from paw pads, the expression of C

149 ces potent IL-4 expression by T cells in the popliteal lymph nodes of mice following footpad immuniza

150 rminal center responses were detected in the popliteal lymph nodes of wild-type, but not in IL-6(-/-)

151 B lymphocytes from popliteal lymph nodes or Peyer's patch activated in vitr

152 ompared with the paraaortic lymph nodes, the popliteal lymph nodes retain greater than 95% of the rad

153 nt and subsequent MRI of rabbit axillary and popliteal lymph nodes revealed significant contrast enha

154 e for a T cell residing 24 hours in a murine popliteal lymph nodes to interact with a DC was 8%, 58%,

155 In selected mice, popliteal lymph nodes underwent Cerenkov luminescence im

156 Fluorescence within all popliteal lymph nodes was easily detected by the FireFly

157 Impression smears from skin lesions and popliteal lymph nodes were prepared from all cases, wher

158 sis of Ly6C(+) macrophages in the ankles and popliteal lymph nodes, decreased migration of monocytes

159 One and 36 h after injection, popliteal lymph nodes, representing the SLNs, were disse

160 antly to early induced IL-4 responses in the popliteal lymph nodes.

161 local (mesenteric) and distant (inguinal and popliteal) lymph nodes of mice with induced polymicrobia

162 after a single fraction of 20 Gy radiation, popliteal lymphadenectomy, and lymphatic vessel ablation

163 IL-6(-/-) fracture mice displayed reduced popliteal lymphadenopathy after fracture.

164 Loss of popliteal lymphatic vessel (PLV) contractions, which is

165 e inguinal and popliteal nodes with draining popliteal lymphatic vessel significantly decreased the p

166 As ultrastructural studies of joint-draining popliteal lymphatic vessels (PLVs) in TNF-Tg mice reveal

167 Spontaneous contractions of popliteal lymphatics from wild-type (WT) (C57BL/6J) mice

168 dtype and four core genotypes (FCG) mice and popliteal lymphnode cellularity and gene expression.

169 IFN-gamma secretion by cultured popliteal lymphocytes decreased in TG animals by 83% aft

170 minal centers in the spleen, plasma cells in popliteal lymphoid nodes, bone marrow cells and granuloc

171 gament in 48%, and the fibular origin of the popliteal muscle in 53% of the patients, whereas standar

172 nd-paw incision model by peri-incisional and popliteal nerve block administration combined with mecha

173 Upon stimulation of popliteal node cells, in vitro induction of regulatory c

174 Necropsy at 24 h revealed that the popliteal node on the experimental leg receiving the avi

175 nd enhanced NK cell activity in the draining popliteal node.

176 At both doses (1.7 and 8.4 nmol), the popliteal nodes had higher (P < 0.050) optical fluoresce

177 Excision of the inguinal and popliteal nodes with draining popliteal lymphatic vessel

178 des) and partial systemic (inguinal, but not popliteal nodes) loss of DCs from lymph nodes in septic

179 ial occlusions and 24 (83%) of 29 SFA and/or popliteal occlusions were longer than 10 cm.

180 tients with tibial occlusions and SFA and/or popliteal occlusions, respectively, as scored with modif

181 ilar if not lower after treatment of femoral-popliteal occlusive disease with paclitaxel versus nonpa

182 ce of symptomatic or asymptomatic DVT in the popliteal or femoral veins.

183 mies, iliac or femoral arteries in 25 (18%), popliteal or tibial arteries in 25 (18%), carotid arteri

184 d with LTbetaR-Ig had no axillary, inguinal, popliteal, or peripancreatic lymph nodes.

185 as absent during mild calf contraction where popliteal outflow was phasic with the concentric phase o

186 onspecific agents were observed for both the popliteal (p < 0.006) and axillary (p < 0.012) nodes.

187 ciated with thickened carotid (p < 0.01) and popliteal (p < 0.05) intimal-medial thicknesses, hormone

188 /CT-derived changes in femoral (P=0.008) and popliteal (P=0.002) vein inflammation were significantly

189 matory response in the femoral (P=0.012) and popliteal (P=0.013) veins of patients who experienced a

190 nd with plasma triglycerides (women only for popliteal) (p < 0.01).

191 ements were carried out on brachial (BA) and popliteal (PA) arteries using a 2-D B-mode ultrasound.

192 Popliteal percent injected dose at 24 hr was 3.00 +/- 0.

193 t 6 months, and reinterventions after femoro-popliteal percutaneous transluminal angioplasty up to 1

194 oated balloons in patients undergoing femoro-popliteal percutaneous transluminal angioplasty.

195 pain attributable to superficial femoral and popliteal peripheral artery disease were randomly assign

196 nts were randomly assigned (1:1:1) to femoro-popliteal plain balloon angioplasty with or without bare

197 th CEAP clinical class (P < .01) in femoral, popliteal, posterior tibial, peroneal, gastrocnemial, an

198 Van der Woude syndrome (VWS) and popliteal pterygium syndrome (PPS) are autosomal dominan

199 Popliteal pterygium syndrome (PPS; OMIM 119500) is a dis

200 sufficiency of IRF6 causes Van der Woude and popliteal pterygium syndrome, 2 syndromic forms of cleft

201 ries of devastating birth defects, including popliteal pterygium syndrome, cocoon syndrome, and Barts

202 genital disorders Van der Woude syndrome and popliteal pterygium syndrome, have a hyperproliferative

203 ermal disorders Bartsocas-Papas syndrome and popliteal pterygium syndrome, respectively.

204 6 (IRF6) underlie Van der Woude syndrome and popliteal pterygium syndrome.

205 otypes in individuals with Van der Woude and popliteal pterygium syndromes, suggesting that the TGFbe

206 fore and after light pulses presented to the popliteal region (behind the knee).

207 It occurs around the popliteal region between the plantaris muscle, the poste

208 ular femoro-popliteal, with or without infra-popliteal, revascularisation.

209 d 10 patients who received ultrasound-guided popliteal sciatic nerve block for the relief of severe r

210 Ultrasound-guided popliteal sciatic nerve block provides effective pain co

211 e been shown to reduce CD-TLR in the femoral-popliteal segment in de- novo and restenotic lesions.

212 linical benefit over PBA+/-BMS in the femoro-popliteal segment in patients with chronic limb threaten

213 .3%) was characterized by two tendons in the popliteal sulcus.

214 R imaging sequence performed parallel to the popliteal tendon proximally was added to our routine stu

215 ral collateral ligament of the knee, and the popliteal tendon.

216 eks from training cessation, in the femoral, popliteal (treated with stretching), and brachial arteri

217 femoral vein (CFV), mid-SFV, distal SFV, and popliteal vein (PV).

218 ases (99%), either the common femoral or the popliteal vein was involved.

219 analyses were 0.76 (femoral vein) and 0.77 (popliteal vein) based on incidence of VTE occurrence.

220 T) isolated to the calf veins (distal to the popliteal vein) is frequently detected with duplex ultra

221 ed US examinations of the common femoral and popliteal veins only, followed by traditional US of the

222 The common femoral and popliteal veins were assessed for compressibility.

223 ws thrombosis of the superficial femoral and popliteal veins.Onchest computed tomography (CT) angiogr

224 thrombus in the left superficial femoral and popliteal veins; follow-up chest CT angiogram shows no e

225 ive blood oxygenation time-course of femoral/popliteal vessels in: 1) young healthy subjects (YH) (n

226 ial and superior femoral artery [SFA] and/or popliteal vessels, n = 22).

227 osclerotic disease and who required an infra-popliteal, with or without an additional more proximal i

228 -threatening ischaemia who required an infra-popliteal, with or without an additional more proximal i

229 threatening ischaemia who required an infra-popliteal, with or without an additional more proximal i

230 ing ischaemia undergoing endovascular femoro-popliteal, with or without infra-popliteal, revascularis

231 otein cholesterol and carotid (p < 0.01) and popliteal (women only) (p < 0.05) intimal-medial thickne