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1 r associated toxins to the liver through the portal circulation.
2 om its established signaling function in the portal circulation.
3 oxin within the gut lumen and its associated portal circulation.
4 ts constant exposure to foreign Ags from the portal circulation.
5 ial TLR ligands, which are delivered via the portal circulation.
6 he amino acid methionine or absorbed via the portal circulation.
7 ctor substances into the intestinal wall and portal circulation.
8  infused via intrasplenic injection into the portal circulation.
9 tes as direct vector administration into the portal circulation.
10  to export iron out of the cell and into the portal circulation.
11 bution in arterial, deep venous, and hepatic portal circulation.
12 s of >50% or the release of lactate into the portal circulation.
13 allogeneic hepatocytes transplanted into the portal circulation.
14 uctose uptake and transport into the hepatic portal circulation.
15 ation via the gut lymphatics rather than the portal circulation.
16 ried in the mesenteric lymph rather than the portal circulation.
17  as well as problems specifically related to portal circulation.
18 ated from the intestinal microbiota into the portal circulation.
19 ct delivery of the same vector dose into the portal circulation (1.3+/-0.3 microg/mL, or 26% of norma
20     When microspheres were injected into the portal circulation after nitroglycerine administration,
21                 Islets were infused into the portal circulation after transhepatic access.
22 ns, is present at high concentrations in the portal circulation and thus may play an important role i
23  the TMA produced is passively absorbed into portal circulation, and hepatic flavin-dependent monooxy
24 l permeability, release of microbes into the portal circulation, and increased serum levels and liver
25 uivocally that: (a) receptors in the coeliac-portal circulation are more sensitive in amplifying gast
26  of gut-derived antigens that arrive through portal circulation at homeostasis and protect these orga
27 tricular nucleus (PVN) and released into the portal circulation at the median eminence, is known to p
28    Repeated AVP/CRF release in the pituitary portal circulation, due to stress, sensitizes and potent
29                                          The portal circulation has a distinct immunological milieu c
30 s the ileocyte basolateral membrane into the portal circulation have not been fully identified.
31 divided into 4 groups based on the degree of portal circulation impairment: 63 patients with intrahep
32 p<.01) and the gut released lactate into the portal circulation in all animals.
33            Although transplantation into the portal circulation is clinically available, poor engraft
34 nsible for hepatic uptake of bile acids from portal circulation is Na+-taurocholate cotransporting po
35    The hepatic uptake of bile acids from the portal circulation is primarily dependent upon a sodium-
36 gh influx of TLR4 and TLR9 agonists into the portal circulation, leading to enhanced hepatic tumour-n
37 eptide (gene SLC10A1) efficiently clears the portal circulation of bile salts, and the apical bile sa
38 rved from 4 vectors after injection into the portal circulation of immunodeficient mice.
39 titrypsin (alpha-1AT) were injected into the portal circulation of immunodeficient mice.
40 des direct evidence for enhanced entrance to portal circulation of LPS enterally administered to the
41 f insulin secretion from beta-cells into the portal circulation of the islet during hypoglycemia is a
42 t of schistosomiasis, resides in the hepatic portal circulation of their human host up to 30 years wi
43 r level, microspheres were injected into the portal circulation of these animals.
44 le for basolateral bile acid export into the portal circulation remains to be determined.
45 suicide gene by RCR vectors infused into the portal circulation results in progressive transduction o
46 livered directly to the liver, via recipient portal circulation, stimulated long-term, high magnitude
47 creased levels of bacterial endotoxin in the portal circulation, suggesting a role for gut-derived to
48 y secreting inflammatory adipokines into the portal circulation that drains visceral fat, we determin
49  factor (CRF) into the hypothalamo-pituitary portal circulation, thereby stimulating adrenocorticotro
50 h the mucosal barrier and travel through the portal circulation to the liver, where they cause absces
51   Allogeneic hepatocytes introduced into the portal circulation via intrasplenic injection are immuno
52 f iron from the absorptive enterocyte to the portal circulation via the sole iron efflux transporter,
53 ologically active MAMPs translocate into the portal circulation, with flagellin (Toll-like receptor 5