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1 ary embolism, 18 deep vein thrombosis, and 2 portal vein thrombosis).
2 ening islet survival and potentially causing portal vein thrombosis.
3 Three (2.9%) grafts were lost due to portal vein thrombosis.
4 source of infection and presenting with left portal vein thrombosis.
5 ing for age, sex, treatment, tumor size, and portal vein thrombosis.
6 a higher change of complications related to portal vein thrombosis.
7 ut was present in 62.5% of the patients with portal vein thrombosis.
8 There was no incidence of hepatic artery or portal vein thrombosis.
9 y independent risk factor for pre-transplant portal vein thrombosis.
10 es because of unexpected acute rejection and portal vein thrombosis.
11 patient required retransplantation, owing to portal vein thrombosis.
12 ies, 8 required surgical shunts for complete portal vein thrombosis.
13 e the relationship between folate status and portal vein thrombosis.
14 hic scan showed superior mesenteric vein and portal vein thrombosis.
15 t entry criteria, and 2 were excluded due to portal vein thrombosis.
16 y subjects were included, and 64 (15.7%) had portal vein thrombosis.
17 of 0.001, were significantly associated with portal vein thrombosis.
18 4), acute chest syndrome (5), pneumonia (2), portal vein thrombosis (1), priapism (1), hemolytic urem
19 his study found a notably high prevalence of portal vein thrombosis (15.7%) among hospitalized patien
20 were mainly rejected for comorbidity (19%), portal vein thrombosis (16%), previous surgery (9%), obe
21 ently in OPV than in cirrhosis: extrahepatic portal vein thrombosis (18 [43%] of 42 vs five [12%] of
22 erved post-COVID-19 vaccination, followed by portal vein thrombosis (52 cases), raised liver enzymes
25 ther evaluations revealed a mild ascites and portal vein thrombosis although the patient received pro
26 dy was conducted to assess the prevalence of portal vein thrombosis and associated factors among pati
27 .4%; 20-year survival, 12.5%). patients with portal vein thrombosis and biliary cirrhosis demonstrate
28 wo (16.3%) patients developed pre-transplant portal vein thrombosis and its presence had no impact in
29 tic thrombosis, and radiologic management of portal vein thrombosis and stenosis has decreased operat
31 C, 56% had more than 1 tumor nodule, 24% had portal vein thrombosis, and 29% did not receive any canc
32 ism, disseminated intravascular coagulation, portal vein thrombosis, and arterial thromboembolism.
34 inib secondary to cerebrovascular ischaemia, portal vein thrombosis, and coronary artery stenosis in
35 analysis Child-Pugh score, presence of HCC, portal vein thrombosis, and lack of secondary prophylaxi
36 e II diabetes mellitus, renal insufficiency, portal vein thrombosis, and poor performance status.
37 ced disease, increased inflammatory changes, portal vein thrombosis, and substantially lower survival
43 growing body of evidence has indicated that portal vein thrombosis, autoimmune hepatitis, raised liv
44 tis/cholecystitis, pancreatitis, hemorrhage, portal vein thrombosis, bowel wall perforation, or dehyd
45 f orthotopic liver transplantation, in which portal vein thrombosis developed in the immediate postop
47 s of the liver in children with extrahepatic portal vein thrombosis (EHPVT), with surgical outcome af
49 last decade a large number of patients with portal vein thrombosis have undergone successful liver t
50 igher mortality and complications, including portal vein thrombosis, hepatic encephalopathy, and gast
51 We aimed to characterize the pre-transplant portal vein thrombosis in a cohort of liver transplant r
52 ascular stent placement for the treatment of portal vein thrombosis in liver transplant recipients.
55 nt placement were successfully used to treat portal vein thrombosis in patients with recent liver tra
57 rahepatic portal vein occlusion (EHPVO) from portal vein thrombosis is a rare condition associated wi
58 In patients with cirrhosis, development of portal vein thrombosis is often insidious and remains un
60 patients with Budd-Chiari syndrome and with portal vein thrombosis, Kiladjian et al observed that JA
62 n associated with hepatic artery (n = 15) or portal vein thrombosis (n = 14).Mean surgical time was 1
63 ases for acute hepatic failure (n = 4, 50%), portal vein thrombosis (n = 25, 48.1%), splanchnic vein
64 liver disease (MELD) score <=25, absence of portal vein thrombosis, no mechanical ventilation at the
65 pared with non-BCS liver recipients), one of portal vein thrombosis (nonsignificant [NS]), and one of
67 cephalopathy (odds ratio, 4.4; P = .001) and portal vein thrombosis (odds ratio, 5.3; P = .001) were
68 d with the presence of SPSSs (any size) were portal vein thrombosis (odds ratio, 5.5; P = .008) and C
69 t survival for patients who have experienced portal vein thrombosis or stenosis is 61% and 67%, respe
71 ipients due to cardiovascular comorbidities, portal vein thrombosis, or to manage posttransplant comp
72 ardial infarct, perioperative hemorrhage, or portal vein thrombosis--or death occurred as a result of
73 001), MELD score greater than 10 (P = .001), portal vein thrombosis (P = .0001), and tumor diameter g
75 nt differences in hepatic artery thrombosis, portal vein thrombosis, primary nonfunction, and biliary
76 s possible even in the presence of recipient portal vein thrombosis, provided that hepatopetal portal
78 logy Group (ECOG), presence of cirrhosis and portal vein thrombosis (PVT) (none, branch, and main).
80 patients of liver cirrhosis associated with portal vein thrombosis (PVT) can be effectively treated
83 low-molecular-weight heparin, in preventing portal vein thrombosis (PVT) in patients with advanced c
93 ate and survival of hepatocellular carcinoma portal vein thrombosis (PVT) patients treated with (90)Y
96 Patients with Child-Pugh B disease who had portal vein thrombosis (PVT) survived 5.6 months (95% co
97 ed-stage hepatocellular carcinoma (HCC) with portal vein thrombosis (PVT) treated with (90)Y radioemb
98 onths with no significant difference between portal vein thrombosis (PVT) versus no PVT (7 versus 13
100 osal EV on contrast-enhanced CT, presence of portal vein thrombosis (PVT), and patent umbilical vein
101 tients with chronic noncirrhotic, nontumoral portal vein thrombosis (PVT), the usually recommended st
102 ocellular carcinoma (HCC), including 16 with portal vein thrombosis (PVT), were treated with (90)Y-lo
106 ion (Budd-Chiari Syndrome) and in those with portal vein thrombosis (second section); and we briefly
107 stribution hazard ratio, 2.9; P = .039), and portal vein thrombosis (subdistribution hazard ratio, 7.
109 atocellular carcinoma (HCC) with and without portal vein thrombosis underwent radioembolization with
110 ease, cytomegalovirus infection and disease, portal vein thrombosis, UNOS status, Childs-Pugh score,
117 60.1 years; range, 32-75 years) with HCC and portal vein thrombosis who were examined with both contr