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1 DNA double-strand breaks (>/=5% gamma-H2A.X-positive cells).
2 c markers (p53, cleaved caspase-7, and TUNEL-positive cells).
3 repression of rat macrophages by human CD47-positive cells.
4 nitor cells, and decreased the count of Sox1 positive cells.
5 xpress FLPo recombinase specifically in GFAP-positive cells.
6 g ADC could selectively target and kill HER2-positive cells.
7 an increased number of glucose transporter-1-positive cells.
8 groups exhibited less than 5% of basal layer positive cells.
9 g this multisubunit repressor complex in EBV-positive cells.
10 as positively correlated with TCTP in HpslyD-positive cells.
11 aminotransferase levels and numbers of TUNEL-positive cells.
12 rkedly reduced cyclobutane pyrimidine dimers-positive cells.
13 cient differentiation of CD4(+)CD8(+) double positive cells.
14 et-like structures containing MAFA/C-peptide-positive cells.
15 with camptothecin while sparing isogenic p53-positive cells.
16 ained a large number of CD11c, CD68, and CD3 positive cells.
17 minimal changes were seen in IL-4- and IL-5-positive cells.
18 ts accompanied by an expansion of Krt5/Krt14-positive cells.
19 N knockdown promotes PTK6 activation in PTEN positive cells.
20 groups according to the percentage of PD-L1-positive cells.
21 creased number of BrdU- and beta-III tubulin-positive cells.
22 , which colocalized with megalin, in podocin-positive cells.
23 origenic than the freshly isolated Aldefluor-positive cells.
24 genome amplification in differentiating HPV-positive cells.
25 tive (HBsA-positive) and HBV DNA- and cccDNA-positive cells.
26 stain intensity and the percentage of immune-positive cells.
27 a-SMA positive cells but showed very few NG2 positive cells.
28 and identifies a related population of NREP-positive cells.
29 ssing red fluorescent protein in retinal Cre-positive cells.
30 r alpha-SMA while they completely lacked NG2 positive cells.
31 ation of tartrate-resistant acid phosphatase-positive cells.
32 er with high expression of alpha-SMA and NG2 positive cells.
33 atory infiltrate has a larger amount of CD20 positive cells.
34 ximity to nuclear Cajal bodies in telomerase-positive cells.
35 ells and premitotic arrest of CD4/CD8 double-positive cells.
36 s with multiple islets with numerous insulin-positive cells.
37 significant specific phototoxicity in GLP-1R-positive cells (2.3% +/- 0.8% and 2.7% +/- 0.3% remainin
38 latelet-derived growth factor receptor-alpha-positive cells, 4) alpha-smooth muscle actin-positive bl
39 the A gate produced significantly more Vasa-positive cells (45.0% +/- 15.2%) than the "B" gate (0.0%
40 alists showed that all had scattered insulin-positive cells; 59 additionally had few insulin-positive
41 ed growth factor receptor alpha (PDGFRalpha) positive ( + ) cells accumulate in CTS SSCT and that the
43 ucleated tartrate-resistant acid phosphatase-positive cells along the alveolar bone surface was signi
45 4 deficiency also significantly reduced Ki67-positive cells and beta-catenin expression compared with
46 ha smooth muscle actin (alphaSMA) and desmin-positive cells and collagen deposition, independent of i
47 governing entry of measles virus into SLAMF1-positive cells and identified endocytic uptake of viral
48 eta-cell mass, decreased the number of TUNEL positive cells and improved glucose tolerance after gluc
49 MG before differentiation reduced hemoglobin-positive cells and increased stem/progenitor cell surfac
50 ilar distributions in genomic regions of EBV-positive cells and is associated with oncogenic super-en
51 antly, the levels of p73 are elevated in HPV-positive cells and its knockdown impairs HPV genome ampl
52 matrix metalloproteinase (MMP) 9-, and MMP13-positive cells and more proliferating cell nuclear antig
53 idine triphosphate nick end-labeling (TUNEL)-positive cells and mortality compared with vehicle-injec
54 This uptake pathway is specific to SLAMF1-positive cells and occurs within 60 min of viral attachm
56 ed by the reduced number of propidium iodide-positive cells and the cleavage of caspase-3 and poly (A
59 1, P-selectin, cluster of differentiation 45-positive cells); and oxidative stress (increased malondi
60 latelet-derived growth factor receptor-alpha-positive cells, and alpha-smooth muscle actin-positive b
61 amma antibody led to a decrease in IFN-gamma-positive cells, and an increase in IL-5-positive cells,
62 cultured with supernatants from Tr1 or FOXP3-positive cells, and analyzed by immunofluorescence and f
63 were associated with increased HA, vimentin-positive cells, and fibrosis in Hyal2(-/-) compared with
64 brain atrophy, neuron loss, loss of poly(PR)-positive cells, and gliosis, culminating in motor and me
65 ion of interleukin-1beta by CD11b- and Iba-1-positive cells, and loss of olfactory sensory neurons (O
67 local muscle cell compartments, such as PAX7 positive cells, and recruited macrophages during skeleta
68 rted liver morphology, accumulation of TUNEL-positive cells, and transcriptional upregulation of apop
69 ncubated with supernatants from Tr1 or FOXP3-positive cells, and transepithelial electrical resistanc
76 cantly higher number of TRAP and Osteocalcin positive cells at 4 weeks in the cell-seeded group compa
78 Immunohistochemistry showed increased KRT80-positive cells at relapse and, using several clinical en
79 vor somatostatin- and pancreatic polypeptide-positive cells at the expense of insulin- and glucagon-p
81 mpared with primary tumors (17% nanoparticle-positive cells) because they reside close to blood vesse
82 When cocultured on exosomes from Nischarin-positive cells, breast cancer cells exhibited reduced su
83 ycle arrest and eliminated Ki-67 mRNA in RB1-positive cells but had no effect in RB1-negative cells,
85 amma-positive cells, and an increase in IL-5-positive cells, but did not impact clearance of Pneumocy
86 RT promoter in both telomerase-negative and -positive cells, but it activated the repressed hTERT pro
88 ctivated for infection of CD4-negative, CCR5-positive cells, but the infection of CD4-positive, CCR5-
89 on reduced the percentage of nucleoprotein 1-positive cells by 85, 56, and 66%, respectively (P < 0.0
92 r- and progesterone receptor-negative (< 10% positive cells by immunohistochemistry) early breast can
93 active Rho1 and a decrease in phosphomyosin-positive cells by immunostaining, while expression of co
94 ies of reports document induction of the CK5-positive cells by progestins, it is unknown if other 3-k
95 activated the infection of CD4-negative CCR5-positive cells by several HIV-1 strains resistant to the
97 In this study, we demonstrate that insulin-positive cells can be generated in vitro from human indu
98 ased pAKT signal suggesting that the variant-positive cells can induce lesion formation in a non-cell
100 heres containing HBMSCs and HUVECs, and CD31-positive cell clusters were prominent within HUVEC-impla
104 ment reduced (P < 0.001) the number of TUNEL-positive cells compared to vehicle at 24 h and day 7 pos
105 f cells in micrometastases (50% nanoparticle-positive cells) compared with primary tumors (17% nanopa
106 diminished the procoagulant activity of EMT-positive cells, confirming a functional role for TF in t
107 elanocytes, immunotargeting of the chemokine-positive cells, continuous loss of the pigment-producing
109 specificity to HER2 and integrin alphavbeta6 positive, cell culture-derived, breast and pancreatic ca
110 a filamentous pattern along regions of inner positive-cell curvature by its coiled-coil motifs, and i
111 Cdkn1a, Nr4a1, Gata2, Junb and Btg2, and the positive cell cycle regulators were correspondingly down
112 in cell-death-deficient mutants lacking the positive cell death regulator ORESARA1/ANAC092 (ORE1).
116 opes were defined, with peptide-MHC pentamer-positive cells displaying the central and effector memor
118 d; (b) Layer A1 exhibits increases in SMI-32-positive cells earlier than layer A; (c) The general tra
121 elomeric lagging strands, whereas telomerase positive cells exhibited similar elongation between lead
125 an average efficiency of 55% into C-peptide-positive cells, expressing markers of mature beta-cells,
127 numbers of antigen-specific CD8(+) IFN-gamma-positive cells following injection into BALB/c mice.
128 , and presence of binucleate hepatocytes and positive cells for AlkP in periodontitis versus control
132 ly GD2-expressing osteosarcomas (32%-69% GD2-positive cells) from high GD2 expressors (>99%, P < 0.05
134 d insulin independence, all examined insulin-positive cells had lost expression of the end-differenti
137 ion for why this microRNA is targeted in HPV-positive cells.IMPORTANCE We describe here for the first
138 F1, a TopBP1-binding partner, and p73 in HPV-positive cells in contrast to its effects in other cell
139 a decrease in infectious virus and fewer RSV-positive cells in cultures after neutrophil exposure tha
141 ammation, BOT-64 reduced the number of SETD1-positive cells in inflamed periodontal tissues, restored
143 The percentage of CXCR3 expression in CD3-positive cells in PBMCs was inversely correlated with se
145 developmental emergence of parvalbumin (PV)-positive cells in the BLA and increased anatomical conne
148 further detected a significant loss of OLIG2-positive cells in the corpus callosum of Tmem106b-/- mic
149 ric metaplasia to include Gkn3 mRNA and GKN3-positive cells in the corpus, allowing a more accurate a
150 t the large majority (80%) of the calretinin positive cells in the ganglion cell layer are ganglion c
151 sion and stimulates accumulation of alphaSMA-positive cells in the gel, while knockdown of endothelia
153 cleotidyl transferase dUTP nick-end labeling positive cells in the hippocampus, and improved spatial
154 iments yielded no glycinergic or cholinergic positive cells in the IC, and descending projections to
155 PAE cells stimulate accumulation of alphaSMA-positive cells in the implanted gel, while Twist1 knockd
156 n addition, a small proportion of calretinin-positive cells in the inner nuclear layer and in the gan
158 e ionized calcium-binding adapter molecule 1-positive cells in the ischemic cortex of GPR37 KO mice,
159 the DDC-stimulated number of cytokeratin-19-positive cells in the liver of wild-type animals was sli
161 on, there were fewer 5-bromo-2'-deoxyuridine-positive cells in the LK population in Chd7(f/f)Mx1-CreC
162 e in proliferative alpha-smooth muscle actin-positive cells in the lungs of ITSN-deficient mice, tran
163 timal hyperplasia (alpha-smooth muscle actin positive cells in the neointima) and endothelial activat
165 wever, there were significantly more IL-17RA-positive cells in the submucosa and epithelium in childr
166 cells (P < .01) and with the presence of CD8-positive cells in the surrounding stroma (P = .04).
167 onfirmed the presence of alphaFAP- and FSP-1-positive cells in the tumor stroma, and their presence c
168 We found fewer beta-catenin and more pSMAD5 positive cells in the uninjured AC of MRL/MpJ mice than
169 as performed to identify and quantify CHI3L1-positive cells in tissue sections from ALS, DCs and non-
171 erial and assessed the frequency of BCR-ABL1-positive cells in various hematopoietic subpopulations;
172 oxin-based fusion protein that depletes CD25-positive cells including regulatory T cells and has been
173 nct subtypes of somatostatin+/Reelin+ double-positive cells, including Hpse+ layer IV cells targeting
174 d to a more potent nanomedicine against HER2-positive cells; incorporation of the chemotherapeutic pa
175 CD163-positive, CD206-positive, and arginase-positive cells, indicating a M2 macrophage phenotype.
177 s tartrate-resistant acid phosphatase (TRAP)-positive cell induction than M0 or M2 macrophage transfe
180 our samples revealed that the number of ODZ1-positive cells inversely correlated with overall and pro
185 ormed with C4-2 cells, a subline of the PSMA-positive cell line LNCaP (human lymph node carcinoma of
186 ted HLA-B*51:08 subtype expressed in a Hap10-positive cell line was isolated, characterized by mass s
187 gen receptor (ER)/progesterone receptor (PR)-positive cell line) with negligible cytotoxicity against
188 ibiting cell proliferation in T47D, a triple positive cell line, it had limited activity in TNBC.
191 SR1 (Y537S) mutations in MCF7 and SUM44 ESR1-positive cell lines after acquisition of resistance to l
194 telomerase RNA) gene knockouts in telomerase positive cell lines that resulted in long-term surviving
196 ell cycle arrest and apoptosis of human EVI1-positive cell lines, and prolonged survival in both orth
197 ter analyzing human lymphoma tissues and EBV-positive cell lines, we now document a strong correlatio
199 During this process, agonist-selected double-positive cells lose CD4/8 coreceptor expression and masq
200 significant proportion of hepatocyte marker-positive cells maintaining a less well-differentiated ph
202 ther groups were confirmed by a greater TRAP-positive cell number and reabsorption surface on both th
203 BMP-2-induced 5-bromo-2'-deoxyuridine (BrdU)-positive cell numbers at the injected site on day 7 and
206 f15 is normally expressed within aquaporin 1-positive cells of the S3 segment of the proximal tubule,
210 h was suppressed with S3I-201 (percentage of positive cells per field: 31.7% +/- 3.4 vs 3.8% +/- 1.7;
212 hage and decreased alpha-smooth muscle actin-positive cell population, fibrous cap thinning, and decr
215 in immunoreactivity and tyrosine hydroxylase-positive cell populations in specific larval brain regio
216 confirmed the proliferative advantage of MB-positive cell populations relative to MB-negative and G2
218 tonin immunoreactivity, tyrosine hydroxylase-positive cell populations, and rescued venlafaxine-media
219 f FITC-labeled Ki-67 antibody TuBB-9 in EGFR-positive cells pre-loaded with the photoactive dye BPD.
220 o-infected with rAAV-CFP-Neo, 27% of the CFP-positive cells present rHSV48Y replication compartments.
222 uated MPTP-induced CSF1R activation and Iba1-positive cell proliferation, without a reduction of the
223 repair, CHK1 and Wee1, are suppressed in HPV-positive cells, providing an explanation for why this mi
224 Analysis of 16S rRNA sequences from BONCAT-positive cells recovered by fluorescence-activated cell
225 tic drop in the number of Pax7- and myogenin-positive cells relative to WT muscles, suggesting that d
229 th conditional activation of KRAS in the PGR positive cells reveal an increase of SIRT1 expression in
233 NA sequencing comparison of VEC-null and VEC-positive cells suggested a more general role of VEC in a
234 o facilitate EBV latency, though whether HPV-positive cells support a latent EBV infection has not be
235 ed proliferation did not affect basal, Krt14-positive cells, supporting the fact that Foxa1-regulated
237 genome levels in their TG but many fewer LAT-positive cells than wild-type mice from day 7 to day 30
238 destly GD2-expressing osteosarcomas (32% GD2-positive cells) than in a GD2-negative tumor (9.8% vs. 1
239 ut also to maintain higher cell uptake (>90% positive cells) than the most densely PEGylated particle
242 oplastic growth of smooth muscle-alpha-actin-positive cells that destroy lung parenchyma and by the f
243 t cardiac fibroblasts into Periostin (Postn)-positive cells that express high levels of extracellular
244 entify a population of glutamatergic Adcyap1-positive cells, the activity of which accurately determi
245 The mean numbers of CD31-, KI67-, and MYOD1-positive cells, the optical density of TGF-beta, and the
246 filtration of Ly-6G-, CD11b-, Iba-1- and CD3-positive cells, the production of interleukin-1beta by C
247 crine progenitors into insulin- and glucagon-positive cells through non-cell-autonomous regulation of
248 in mortality and in brain cell death (TUNEL positive cells) throughout the whole brain, and in the i
249 eurons, the parvalbumin and the somatostatin positive cells, tightly control both up-to-down and down
250 s9 to delete PML and APB components from ALT-positive cells to cleanly define the function of APBs in
251 lass of inhibitory interneurons-somatostatin-positive cells-to the generation of slow waves during NR
252 mouse, we recently discovered that an Hnf1b positive cell type in the lung is sensitive to adenoma f
253 e to loss of Atoh1 function than other Atoh1-positive cell types and that heterozygous mice actually
254 get for molecular imaging of different CXCR4-positive cell types in cardiovascular diseases such as a
255 lammatory cytokine secretion from other ROCK-positive cell types, corroborating the selective in vivo
261 bears striking resemblance to a related Gram-positive cell-wall remodeling strategy that also promote
268 cytosed myelin within Iba1-positive or P2Y12-positive cells was significantly lower after DHA treatme
269 ing these ALT cells with parental telomerase positive cells, we observed that ALT cells possess exces
270 by using a model of in vivo ablation of PAX7 positive cells, we show that this radiosensitive skeleta
271 tral fovea and found evidence that rod opsin positive cells were absent and violet-sensitive cone and
272 Following IUCT, the antihuman mitochondria-positive cells were detected in the liver of recipient r
276 oliferating cell nuclear antigen- and pSMAD5-positive cells were found in the DMM-injured AC in MRL/M
277 ctive within glial fibrillary acidic protein-positive cells were generated and this allowed for selec
279 ged with angiotensin II, PDGF receptor alpha-positive cells were increased in the skin and heart.
280 MS lesions, and changes in the percentage of positive cells were more relevant in MS than in other wh
281 atic increase in alpha-SMA(+), EP4(+) double-positive cells were observed in EP4cKO(S100a4) suggestin
284 ng hypertrophic-like scars, whereas few CD26-positive cells were present in the regenerated gingival
287 ition, alpha-SMA (alpha-smooth muscle actin)-positive cells were reduced in the infarcted myocardium
290 egative, stem cell antigen-1-positive, c-Kit-positive) cells were quantified and proliferation assess
291 or killing epithelial cell adhesion molecule-positive cells when used as a targeting domain on chimer
292 MSPCs are observed as leptin receptor (LepR)-positive cells, whereas osteoblasts can be classified as
293 panded epithelia are Sox9-, Pitx2-, and Tbx1-positive cells, which are markers of the dental epitheli
294 nding protein 1 foci, after incubating SSTR2-positive cells with (177)Lu-diethylene triamine pentaace
296 itive cells; 59 additionally had few insulin-positive cells within a few islets; and 14 additionally
297 eriostin and transforming growth factor beta-positive cells within Asm bundles, in addition to lower
299 on therapy (ADT), destroy the bulk of the AR-positive cells within the tumor, eradicating this popula